ライフサイエンスコーパス: glutathione peroxidase

1 plexes I, II, III; superoxide dismutase; and glutathione peroxidase).

2 hione and the phase II detoxification enzyme glutathione peroxidase.

3 of superoxide dismutase (SOD), catalase and glutathione peroxidase.

4 s immediately upstream of ospR and encodes a glutathione peroxidase.

5 bition against glutathione S-transferase and glutathione peroxidase.

6 activating factor acetylhydrolase (PAF), and glutathione peroxidase.

7 drogenase (G6PD), glutathione reductase, and glutathione peroxidase.

8 affected in both catalase and TR but not in glutathione peroxidase.

9 include catalase, superoxide dismutase, and glutathione peroxidase.

10 n down-regulation of thioredoxin, MnSOD, and glutathione peroxidase.

11 tiporter, gamma-glutamylcysteine ligase, and glutathione peroxidase.

12 ncluding superoxide dismutase, catalase, and glutathione peroxidase.

13 le in antioxidant defenses as a component of glutathione peroxidase.

14 ession of superoxide dismutase isoform 2 and glutathione peroxidase.

15 ar to that found in mammalian selenocysteine glutathione peroxidases.

16 peroxidases; however, they lack catalase and glutathione peroxidases.

17 f manganese superoxide dismutase (MnSOD) and glutathione peroxidase 1 (GPx).

18 ine-induced suppression of the selenoprotein glutathione peroxidase 1 (GPx-1) and endothelial dysfunc

19 Glutathione peroxidase 1 (GPx-1) has been implicated in

20 n the gene for the antioxidant selenoprotein glutathione peroxidase 1 (GPx-1) in the risk or etiology

21 In animal models, glutathione peroxidase 1 (GPx1) activity is reduced afte

22 Hepatic glutathione peroxidase 1 (GPx1) and selenoprotein S (Sel

23 with (75)Se revealed a loss of the abundant glutathione peroxidase 1 (GPx1) band, whereas other sele

24 Among genes in the GSH family, glutathione peroxidase 1 (GPX1) had the most significant

25 , a mitochondrial complex III inhibitor, and glutathione peroxidase 1 (GPX1), a scavenger of hydrogen

26 don 200 (Pro200Leu) of the gene encoding for glutathione peroxidase 1 (GPx1), a selenium-dependent en

27 investigate the impact of overexpression of glutathione peroxidase 1 (GPX1), an intracellular seleno

28 zymes, including heme oxygenase-1 (HO-1) and glutathione peroxidase 1 (Gpx1).

29 in mice lacking the antioxidant selenoenzyme glutathione peroxidase 1 (GPx1).

30 rtion into thioredoxin reductase 1 (TR1) and glutathione peroxidase 1 (GPx1).

31 in reductase 1 concentrations and lymphocyte glutathione peroxidase 1 and 4 activities and concentrat

32 ice but showed higher levels and activity of glutathione peroxidase 1 and methionine-R-sulfoxide redu

33 Human glutathione peroxidase 1 could thereby also be produced,

34 Sec tRNA([Ser]Sec), and consequently reduced glutathione peroxidase 1 expression.

35 ne of the major antioxidant defense enzymes, glutathione peroxidase 1 knockout mice are protected dur

36 C57Bl6 and glutathione peroxidase 1 knockout mice.

37 of hydrogen peroxide in bacterially infected glutathione peroxidase 1 macrophages and that restoring

38 Infected glutathione peroxidase 1 mice showed an early and signif

39 protective effect was suppressed by treating glutathione peroxidase 1 mice with an interleukin-1 rece

40 nit (GCLC), glutathione S-transferasemu, and glutathione peroxidase 1 were down-regulated in the live

41 liver but not stress-related proteins (e.g. glutathione peroxidase 1).

42 2.5-fold) (REV3L, XPMC2H, HNRPUL1, TOR1AIP1, glutathione peroxidase 1, and SCFD2), with CGIs of inter

43 4 did not cause alterations in activities of glutathione peroxidase 1, catalase, Cu/Zn superoxide dis

44 detoxifying enzymes (superoxide dismutase 2, glutathione peroxidase 1, thioredoxin, and peroxiredoxin

45 Rdx12 was found to interact with glutathione peroxidase 1, whereas 14-3-3 protein was ide

46 a reactive oxygen species scavenging enzyme glutathione peroxidase 1.

47 Several selenoproteins, including glutathione peroxidases 1 and 3, SelR, and SelT, were no

48 We have found that suppression of glutathione peroxidase-1 (GPx-1) in human microvascular

49 Glutathione peroxidase-1 (GPx-1) is a crucial antioxidan

50 Glutathione peroxidase-1 (GPx-1) is a selenocysteine-con

51 d mice overexpressing the antioxidant enzyme glutathione peroxidase-1 (Gpx1) are protected from exper

52 tical redox axis with the antioxidant enzyme glutathione peroxidase-1 (GPX1) at its hub and identify

53 e identified a role for the network hub gene glutathione peroxidase-1 (GPX1) in pathological remodeli

54 Glutathione peroxidase-1 (GPX1) represents the first ide

55 of the most abundant hepatic selenoprotein, glutathione peroxidase-1 (Gpx1), to 15% of the selenium-

56 nt genes than chow-fed young mice, including glutathione peroxidase-1 and -4, catalase, superoxide di

57 Levels of glutathione peroxidase-1 and catalase, however, were not

58 n the medium Se group the expression of lung glutathione peroxidase-1 and liver selenoprotein P were

59 Transgenic expression of the glutathione peroxidase-1 antioxidant enzyme (GPX1) in db

60 l of hexokinase-2, hormone sensitive lipase, glutathione peroxidase-1, and myosin heavy chain IIa in

61 of antioxidants, superoxide dismutase 1 and glutathione peroxidase-1, were significantly upregulated

62 ere, we studied how the H2O2-reducing enzyme glutathione peroxidase 2 (GPx2) regulates H2O2 stress an

63 ntioxidant and prooxidant enzymes, including glutathione peroxidase 2 and 3 (GPx2 and GPx3), peroxire

64 ntioxidant mechanism that uses mitochondrial glutathione peroxidase 2.

65 human and mouse Grx2 was lower than that of glutathione peroxidases (2.5 and 0.8x10(4) s(-1) M(-1),

66 oxidase-1 (GPx-1), gastrointestinal specific glutathione peroxidase-2 (GPx-2), thioredoxin reductase-

67 We report a glutathione peroxidase 3 (AaGPx3) involved in the comple

68 Expression of glutathione peroxidase 3 (GPx3) is down-regulated in a v

69 Plasma Se, SePP, and glutathione peroxidase 3 (GPx3) levels increased with su

70 the reactive oxygen species (ROS) scavenger glutathione peroxidase 3 (GPx3) positively correlates wi

71 this report, we found that the expression of glutathione peroxidase 3 (GPx3) was widely inactivated i

72 ein 1 (HIVEP1), von Willebrand factor (VWF), glutathione peroxidase 3 (GPX3), and platelet-derived gr

73 Glutathione peroxidase 3 is a selenium-dependent enzyme

74 reas antioxidant proteins, paraoxonase 2 and glutathione peroxidase 3, were down-regulated in the eye

75 Glutathione peroxidase-3 (GPx-3) is a selenocysteine-con

76 The other extracellular selenoprotein, glutathione peroxidase-3 (Gpx3), has not been shown to t

77 Glutathione peroxidase 4 (GPX4) and arachidonic acid 15-

78 educed levels of superoxide dismutase (Sod), glutathione peroxidase 4 (Gpx4) and peroxiredoxin 3 (Prd

79 The antioxidant enzyme glutathione peroxidase 4 (Gpx4) is a key regulator of ox

80 The selenoenzyme glutathione peroxidase 4 (Gpx4) is a major scavenger of

81 Glutathione peroxidase 4 (Gpx4) is an essential antioxid

82 The selenoenzyme glutathione peroxidase 4 (Gpx4) is an intracellular anti

83 Glutathione peroxidase 4 (Gpx4) is uniquely involved in

84 te-specific knockout mice lacking either the glutathione peroxidase 4 (GPx4) or thioredoxin reductase

85 Small molecules that inhibit glutathione peroxidase 4 (GPX4), a phospholipid peroxida

86 Glutathione peroxidase 4 (GPX4), an antioxidant defense

87 orm of cell death triggered by inhibition of glutathione peroxidase 4 (GPX4), which catalyzes the red

88 This inhibition was mediated by glutathione peroxidase 4 (GPx4), which preferentially de

89 ted necrosis that is critically dependent on glutathione peroxidase 4 (GPX4).

90 inhibiting the phospholipid hydroperoxidase glutathione peroxidase 4 (GPX4).

91 elevated levels of the mitochondrial form of glutathione peroxidase 4 and was fully dependent on the

92 naturally occurring selenoprotein homolog of glutathione peroxidase 4 in fowlpox virus.

93 mine, an anticancer drug, as an inhibitor of glutathione peroxidase 4 lipid repair activity, which wa

94 In addition, glutathione peroxidase 4 was found to be a Rdx12 redox t

95 strated by decreased expression of catalase, glutathione peroxidase 4, and glutathione synthetase gen

96 including thioredoxin reductases 1 and 3 and glutathione peroxidase 4, were expressed in normal or re

97 ace of Sec was commonly observed in GPx1 and glutathione peroxidase 4.

98 s the lipid hydroperoxide-detoxifying enzyme glutathione peroxidase 4.

99 Glutathione peroxidase-4 (GPx4), which specifically meta

100 antly down-regulating the antioxidant enzyme glutathione peroxidase-4.

101 05; copper/zinc superoxide dismutaseP< 0.05; glutathione peroxidase 4P< 0.01] and increased lipoxygen

102 ysteine insertion sequences (SECIS) of human glutathione peroxidase (58 nt) and thioredoxin reductase

103 ton's disease, we identify the age-regulated glutathione peroxidase 6 (Gpx6) gene as a modulator of m

104 The glutathione peroxidases, a family of selenocysteine-cont

105 F2-isoprostanes, and a moderate reduction in glutathione peroxidase activities and porin levels were

106 improved superoxide dismutase, catalase and glutathione peroxidase activities in H2O2 treated CCD an

107 nted a mercuric chloride-induced decrease in glutathione peroxidase activity and completely prevented

108 Changes in platelet glutathione peroxidase activity and in plasma selenium a

109 d decreases in glutathione concentration and glutathione peroxidase activity and increased glutathion

110 oxidative parameters in the brain, enhanced glutathione peroxidase activity as well as reduced nitri

111 Platelet glutathione peroxidase activity did not change significa

112 ium cyanide but had no effect on catalase or glutathione peroxidase activity in the presence of kaini

113 worsening, CuZnSOD overexpression increased glutathione peroxidase activity in the presence of sodiu

114 proximately 22 kDa and has little detectable glutathione peroxidase activity in vitro.

115 way modulation; however, treatment to reduce glutathione peroxidase activity increased 5-LO metabolit

116 However, glutathione peroxidase activity was an order of magnitud

117 elenium-deficient human subjects showed that glutathione peroxidase activity was optimized before sel

118 Sepp1 and selenium concentrations as well as glutathione peroxidase activity were determined in the t

119 ted genes in the small intestine, intestinal glutathione peroxidase activity, secreted Relm-beta prot

120 Indeed, a recombinant SelH shows significant glutathione peroxidase activity.

121 Oxidative stress was assayed by measuring glutathione peroxidase activity.

122 idase and a 2-fold increase in mitochondrial glutathione peroxidase activity.

123 ntioxidants in fa/fa rats and also decreased glutathione peroxidase activity.

124 chondrial manganese superoxide dismutase and glutathione peroxidase and a 2-fold increase in mitochon

125 nction as mimetics of the antioxidant enzyme glutathione peroxidase and catalyze the reduction of hyd

126 PB also down-regulated glutathione peroxidase and glutathione reductase, which

127 ncreased the activities of hepatic catalase, glutathione peroxidase and glutathione S transferase com

128 iet improved superoxide dismutase, catalase, glutathione peroxidase and GR activities and eNOS, iNOS

129 ene expression of a cadre of cytosolic (e.g. glutathione peroxidase and heat shock proteins) and mito

130 Cu,Zn superoxide dismutase or intracellular glutathione peroxidase and non-transgenic mice were expo

131 nthase, and the antioxidant enzymes cellular glutathione peroxidase and paraoxonase).

132 edoxin 6 (Prdx6), a bifunctional enzyme with glutathione peroxidase and phospholipase A2 (PLA(2)) act

133 Glutathione peroxidase and Sepp1Delta240-361 accounted f

134 ectopic expression of ROS-scavenging enzymes glutathione peroxidase and superoxide dismutase effectiv

135 The activities of catalase, glutathione peroxidase and superoxide dismutase were sig

136 met assay), activity of detoxifying enzymes (glutathione peroxidase and superoxide dismutase) in eryt

137 intake is needed to maximize the activity of glutathione peroxidases and other selenoproteins.

138 re affected by this H2O2 including catalase, glutathione peroxidase, and 4 alpha-carbinolamine dehydr

139 id-reactive substances (TBARS), glutathione, glutathione peroxidase, and 6-hydroxy-2,5,7,8-tetramethy

140 in and mRNA expression, as well as catalase, glutathione peroxidase, and glutathione reductase tissue

141 constitutively higher glutathione reductase, glutathione peroxidase, and higher ratios of reduced to

142 modest (approximately 2-fold) inductions of glutathione peroxidase, and novel inductions of glutathi

143 de [MDA]) and antioxidant enzymes (catalase, glutathione peroxidase, and superoxide dismutase).

144 ditionally, the activities of peroxidase and glutathione peroxidase, and the peroxiredoxin abundance

145 in breast cancer cells, such as catalase and glutathione peroxidase, and to inactivate tumor suppress

146 es such as superoxide dismutases, catalases, glutathione-peroxidases, and peroxiredoxins.

147 The fact that genes encoding putative glutathione peroxidases are found in the genomes of many

148 ralizing enzyme of many organisms, and their glutathione peroxidases are in the phospholipid class wi

149 However, glutathione peroxidases are not well studied among proka

150 Glutathione peroxidases are widespread among eukaryotic

151 and lipid peroxidation assays, we identified glutathione peroxidase as a candidate for reducing PPD.

152 al, and GPXs, APX, and MSRA2 genes (encoding glutathione peroxidase, ascorbate peroxidase, and methio

153 nd between H and L mice in liver (except for Glutathione Peroxidase), brain or mammary glands.

154 d immunodetectable endogenous or recombinant glutathione peroxidase but reduced the specific activity

155 as Cu/Zn-superoxide dismutase, catalase, and glutathione peroxidase, but also significantly decreased

156 ieved in part by antioxidant enzymes such as glutathione peroxidase, catalase, and superoxide dismuta

157 A expression levels of other peroxiredoxins, glutathione peroxidases, catalase, superoxide dismutases

158 and restored superoxide dismutase, catalase, glutathione peroxidase, coenzyme Q(10) and ORAC levels i

159 active metalloporphyrin or overexpression of glutathione peroxidase, decreased DOX-induced apoptosis

160 utathione, total antioxidant capacities, and glutathione peroxidase enzymatic activity than did vecto

161 In search for better mimics of the glutathione peroxidase enzymes, pyridoxine-like diseleni

162 ridoxines could also mimic the action of the glutathione peroxidase enzymes.

163 nificantly elevated levels of mRNAs encoding Glutathione Peroxidase enzymes.

164 ects of G418, an aminoglycoside, on cellular glutathione peroxidase expression and function in mammal

165 mechanisms (increased levels of catalase and glutathione peroxidase expression), observed with both P

166 Selenite-induced up-regulation of GPx (glutathione peroxidase) expression-enhanced roGFP2 respo

167 enzyme (superoxide dismutase, catalase, and glutathione peroxidase) expression.

168 nd catalase activities (FA200), erythrocytes glutathione peroxidase (FB400) and thiobarbituric acid-r

169 enzyme activities related to the GSH usage (glutathione peroxidase, gamma-glutamyl transpeptidase, a

170 Some genes including glucanase, glutathione peroxidase, glutaredoxin, and a profilin wer

171 of malondialdehyde (MDA), and activities of glutathione peroxidase, glutathione reductase and supero

172 nzymatic antioxidants (superoxide dismutase, glutathione peroxidase, glutathione) in human HepG2 cell

173 e this, serum SOD activity and proteins, the glutathione peroxidase/glutathione antioxidant system, a

174 ular oxidants and counteracts suppression of glutathione peroxidase/glutathione reductase (GSH-Px/GSS

175 rds xenobiotics including herbicides and for glutathione peroxidase (GPOX) activity.

176 rowth factor (VEGF), glycolytic enzymes, and glutathione peroxidase (GPX) (P<0.05), and a higher expr

177 dehyde 3-phosphate dehydrogenase (GAPDH) and glutathione peroxidase (GPx) activities and suppressed p

178 pacity (TEAC) levels, and catalase (CAT) and glutathione peroxidase (GPx) activities.

179 Here we consider glutathione peroxidase (GPx) activity as a determinant o

180 ntrol, p<0.1); most likely through enhancing glutathione peroxidase (GPx) activity in liver (4.3-fold

181 variants modify the response of whole-blood glutathione peroxidase (GPx) activity to selenium supple

182 which increased superoxide levels, decreased glutathione peroxidase (GPx) activity, decreased glutath

183 Plasma SEPP1 concentration and glutathione peroxidase (GPX) activity, the latter due la

184 d their ability to induce selenium-dependent glutathione peroxidase (GPx) activity, which indicates t

185 dium and used its selenium to increase their glutathione peroxidase (Gpx) activity.

186 for humans as it plays an important role in glutathione peroxidase (GPx) activity.

187 The antioxidant glutathione peroxidase (GPx) and inflammation biomarkers

188 alase (CAT), superoxide dismutase (SOD), and glutathione peroxidase (GPx) and the levels of malondial

189 (GR) by UV spectrophotometry and determined glutathione peroxidase (GPx) expression therein by weste

190 ismutase (SOD) (activity and expression) and glutathione peroxidase (GPx) expression.

191 pendent superoxide dismutase (Cu,Zn-SOD) and glutathione peroxidase (GPX) following nigral dopaminerg

192 ynitrite reductase activity of selenoprotein glutathione peroxidase (GPx) has been investigated using

193 10-fold the protein levels of SOD, CAT, and glutathione peroxidase (GPX) in rat primary cortical cul

194 alase (CAT), superoxide dismutase (SOD), and glutathione peroxidase (GPX) in vivo.

195 Two glutathione peroxidase (GPX) isozymes, GPX-1 and GPX-2 (

196 nitric oxide (NO), superoxide dismutase, and glutathione peroxidase (GPX) levels in serum were measur

197 atalase (CAT), reduced glutathione (GSH) and glutathione peroxidase (GPx) levels.

198 Glibenclamide decreased GSH levels and glutathione peroxidase (GPx) of PMNs after exposed to li

199 t during oligodendrocyte maturation, whereas glutathione peroxidase (GPx) was upregulated with a twof

200 line erythrocyte superoxide dismutase (SOD), glutathione peroxidase (GPx), and catalase (CAT) activit

201 oward MDR cells is achieved by inhibition of glutathione peroxidase (GPx), and the mode of inhibition

202 activity of superoxide dismutases (SOD) and glutathione peroxidase (GPx), glutathione (GSH), and cyt

203 superoxide dismutase (SOD), catalase (CAT), glutathione peroxidase (GPx), glutathione reductase (GR)

204 se superoxide dismutase (Mn-SOD), aconitase, glutathione peroxidase (GPx), heat shock protein 70, iso

205 superoxide dismutase (Mn-SOD), catalase, and glutathione peroxidase (GPX), increased accumulation of

206 ing mimetics of the antioxidant selenoenzyme glutathione peroxidase (GPx), via oxidation to the corre

207 yme activity, superoxide dismutase (SOD) and glutathione peroxidase (GPx), vitamin E, lipid peroxidat

208 er that beta-cell-specific overexpression of glutathione peroxidase (GPx)-1 significantly ameliorated

209 s azo-bis-ebselens 7 were poor mimics of the glutathione peroxidase (GPx)-enzymes, nitroebselens 3, 6

210 peroxidase activities of PGHS-2, PGHS-1, and glutathione peroxidase (GPx).

211 evels of intracellular glutathione (GSH) and glutathione peroxidase (GPx).

212 as well as cellular protection genes such as glutathione peroxidase (GPx).

213 ow antioxidant protection by glutathione and glutathione peroxidase (GPX).

214 and H2O2 is converted to H2O by catalase or glutathione peroxidase (GPX).

215 water and oxygen by either catalase (CAT) or glutathione peroxidase (GPX).

216 , non-enzymatic (total thiol) and enzymatic [glutathione peroxidase (GPx)] antioxidant contents and i

217 xide dismutase (CuZn SOD, Mn SOD), catalase, glutathione peroxidase (GPX)], nitric oxide synthases (N

218 Cellular glutathione peroxidase (GPx-1), a selenocysteine-contain

219 Plasma glutathione peroxidase (GPx-3) is a selenocysteine-conta

220 Mice deficient in two glutathione peroxidases (GPX), Gpx1 and Gpx2, [Gpx1/2-do

221 m is an essential component of antioxidative glutathione peroxidases (GPx).

222 implicated the selenium-containing cytosolic glutathione peroxidase, GPx-1, as a determinant of cance

223 response to G418, whereas expression of the glutathione peroxidases GPX1 and GPX2 was marginally aff

224 rasting manner, with levels of Sep15 and the glutathione peroxidases GPx1 and GPx4 being substantiall

225 -mediated iron uptake), or transfection with glutathione peroxidase (GPx1) enzyme inhibits intracellu

226 binding sites for TFAP2C, we identified that glutathione peroxidase (GPX1) is regulated by TFAP2C thr

227 ts in a decrease in the activity of cellular glutathione peroxidase (GPx1), an intracellular antioxid

228 Gene delivery of glutathione peroxidase (GPx1), which detoxifies H(2)O(2)

229 Using glutathione peroxidase, Gpx1 and Gpx2, double knockout (

230 calization requires the participation of the glutathione peroxidase Gpx3 and the Yap1-binding protein

231 The glutathione peroxidase GPX3 is the only extracellular me

232 tructural components of the sperm, including glutathione peroxidase GPx4/PHGPx.

233 y on pathways converging on the phospholipid glutathione peroxidase (GPX4), a selenocysteine-containi

234 Glutathione peroxidases (GPXs) are a group of enzymes th

235 As complementary to RNA (cDNAs) encoding two glutathione peroxidases (GpXs) from a plant parasitic ne

236 letion of glutathione causes inactivation of glutathione peroxidases (GPXs) in response to one class

237 Two of these suppressors encode glutathione peroxidases (GPxs), which are conserved anti

238 creased (p<0.05) the activities of catalase, glutathione peroxidase (GSH-Px) and glutathione reductas

239 -hydroxynonenal (HNE) and enzyme activity of glutathione peroxidase (GSH-Px) were analyzed with enzym

240 nhibited by antioxidant enzymes catalase and glutathione peroxidase (H(2)O(2) scavenger), but not by

241 response and detoxification genes, like the glutathione peroxidase homologous gene GPXH/GPX5 and the

242 ivity of catalase, superoxide dismutase, and glutathione peroxidase in atrial tissue of the supplemen

243 rovide the first demonstration of a role for glutathione peroxidase in bacterial virulence.

244 essed a mammalian phospholipid hydroperoxide glutathione peroxidase in Escherichia coli from a constr

245 ismutase (MnSOD), copper/zinc, catalase, and glutathione peroxidase in normal human pancreas and in t

246 s more resistant to suppression by cytosolic glutathione peroxidase in PGHS-2 than in PGHS-1.

247 hat GpoA glutathione peroxidase is the major glutathione peroxidase in S. pyogenes and is essential f

248 ransgenic cassava overexpressing a cytosolic glutathione peroxidase in storage roots showed delayed P

249 thione reductase inhibition and no effect by glutathione peroxidase inhibition.

250 Cellular glutathione peroxidase is a key intracellular antioxidan

251 d tissue, it was of interest to determine if glutathione peroxidase is important for virulence.

252 In contrast, glutathione peroxidase is not essential for virulence in

253 In this study, we report that GpoA glutathione peroxidase is the major glutathione peroxida

254 Here, we found that GPX2, which encodes a glutathione peroxidase, is up-regulated by p63 but not p

255 iminished enzyme activity of the antioxidant glutathione peroxidase, leading to an increase in intrac

256 ) gene encoding a phospholipid hydroperoxide glutathione peroxidase (LePHGPx).

257 The glutathione peroxidase-like activity of 4 was measured i

258 ogen peroxide in the presence of thiophenol (glutathione peroxidase-like activity).

259 xide formation, and it is distinguished from glutathione peroxidase-like activity.

260 Cys36-Cys82 disulfide bond at pH 6.0 in the glutathione peroxidase-like enzyme, oxidant receptor pro

261 tive stress (superoxide dismutase, catalase, glutathione peroxidase, lipidic and protein peroxidation

262 score, had decreased expression of catalase, glutathione peroxidase, manganese superoxide dismutase,

263 y pathogenic bacterial species suggests that glutathione peroxidase may have a general role in bacter

264 Quantitative data for two phospholipid glutathione peroxidases (MdesPHGPX-1 and MdesPHGPX-2), t

265 In contrast, ebselen (a glutathione peroxidase mimetic and inhibitor of fatty ac

266 nzyme inhibitor diphenylene iodonium, or the glutathione peroxidase mimetic ebselen significantly att

267 onal cells and determined whether ebselen, a glutathione peroxidase-mimetic, protected against MPTP-i

268 Adolescent treatment with the glutathione peroxidase mimic ebselen also reversed behav

269 N-acetyl-L-cysteine (NAC), catalase, and the glutathione peroxidase mimic ebselen.

270 or amino acid hydroperoxides with ebselen, a glutathione peroxidase mimic, were also determined, and

271 equence in the 3'-untranslated region of the glutathione peroxidase mRNA, and other translational cof

272 e protein disulfide isomerase, glutaredoxin, glutathione peroxidase, NK-lysin/granulysin, HIV Tat pro

273 Adenoviral overexpression of glutathione peroxidase or cytosolic or mitochondrial cat

274 , such as superoxide dismutase, catalase, or glutathione peroxidase, our results suggest that the TP0

275 roxide dismutase (SOD) mimetic (EUK-134) and glutathione peroxidase overexpression prevented the hypo

276 ession of a novel phospholipid hydroperoxide glutathione peroxidase (PHGPx), which incorporates cyste

277 along with other phospholipid hydroperoxide glutathione peroxidases (PHGPx) from several organisms.

278 dismutase; erythrocyte, platelet, and plasma glutathione peroxidase; platelet and leukocyte cytochrom

279 he preservation of total glutathione levels, glutathione peroxidase protein abundance, and a decrease

280 capsule, SMCP and phospholipid hydroperoxide glutathione peroxidase, provide outstanding examples of

281 Combined treatments increased liver glutathione peroxidase, serum catalase, and colon myelop

282 ytosolic superoxide dismutase (SmCT-SOD) and glutathione peroxidase (SmGPX), and a partial coding seq

283 ivity of the three main antioxidant enzymes: glutathione peroxidase, superoxide dismutase and catalas

284 arameters and antioxidant enzymes, including glutathione peroxidase, superoxide dismutase, and catala

285 ), glutathione S-transferase (GST) and total glutathione peroxidase (t-GPx) were decreased compared w

286 thione S-transferase peroxidase kappa 1, and glutathione peroxidase) than the BN rat, suggesting that

287 eins, one of which is a selenocysteine-based glutathione peroxidase, the first found in insects.

288 In the absence of catalase and glutathione peroxidase, the parasites rely primarily on

289 idants, catalase, phospholipid hydroperoxide glutathione peroxidase, thioredoxin, and glutathione wer

290 these data suggest that S. pyogenes requires glutathione peroxidase to adapt to oxidative stress that

291 cted in either Cu,Zn superoxide dismutase or glutathione peroxidase transgenic mice.

292 rved in either Cu,Zn superoxide dismutase or glutathione peroxidase transgenic mice.

293 oxidant stress by expressing enzymes such as glutathione peroxidase type 1 (GPx-1).

294 ductase, tryparedoxin (Tpx), and nonselenium glutathione peroxidase-type enzymes.

295 nuclear (HvGPx42) phospholipid hydroperoxide glutathione peroxidase were isolated from Hydra vulgaris

296 ant strains deficient for peroxiredoxins and glutathione peroxidases were equally sensitive to fatty

297 positive modifications, like an increase in glutathione peroxidase, were detected in muscle (29%) an

298 VC%, respectively), positive associations of glutathione peroxidase with FEV1% and FVC%, and an inver

299 The associations of TBARS and glutathione peroxidase with FVC% in men remained statist

300 isticated fine control of peroxiredoxins and glutathione peroxidases with their backup systems as wel