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1 ace of Sec was commonly observed in GPx1 and glutathione peroxidase 4.
2 s the lipid hydroperoxide-detoxifying enzyme glutathione peroxidase 4.
3 antly down-regulating the antioxidant enzyme glutathione peroxidase-4.
4 elevated levels of the mitochondrial form of glutathione peroxidase 4 and was fully dependent on the
5 strated by decreased expression of catalase, glutathione peroxidase 4, and glutathione synthetase gen
7 educed levels of superoxide dismutase (Sod), glutathione peroxidase 4 (Gpx4) and peroxiredoxin 3 (Prd
13 te-specific knockout mice lacking either the glutathione peroxidase 4 (GPx4) or thioredoxin reductase
16 orm of cell death triggered by inhibition of glutathione peroxidase 4 (GPX4), which catalyzes the red
22 mine, an anticancer drug, as an inhibitor of glutathione peroxidase 4 lipid repair activity, which wa
24 including thioredoxin reductases 1 and 3 and glutathione peroxidase 4, were expressed in normal or re
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