ライフサイエンスコーパス: gluten-free diet

1 way for enzyme therapy as an adjunct to the gluten free diet.

2 al diagnosis may need more time to adjust to gluten free diet.

3 en and in treated CD adults (T-CD, n = 6) on gluten free diet.

4 estigational therapeutic alternatives to the gluten-free diet.

5 d therapeutic interventions alternative to a gluten-free diet.

6 aseline levels following implementation of a gluten-free diet.

7 rsist or recur despite strict adherence to a gluten-free diet.

8 containing, gluten-free diet vs. a naturally gluten-free diet.

9 n in persons with celiac disease following a gluten-free diet.

10 pation, both of which were responsive to the gluten-free diet.

11 nsive to oral pancreatic enzyme therapy or a gluten-free diet.

12 ded by early diagnosis and compliance with a gluten-free diet.

13 hea in these patients after treatment with a gluten-free diet.

14 isorders is based on a lifelong, and strict, gluten-free diet.

15 years who had coeliac disease and were on a gluten-free diet.

16 treatment is strict adherence to a life-long gluten-free diet.

17 l and histological features and adherence to gluten-free diet.

18 ls from patients who had been on a long-term gluten-free diet.

19 t diagnosis and after at least one year on a gluten-free diet.

20 gliadinases may provide an alternative to a gluten-free diet.

21 tients with CD and monitoring adherence to a gluten-free diet.

22 gnancy each can be reduced by adherence to a gluten-free diet.

23 ass) circulating in CD patients' sera before gluten-free diet.

24 th celiac disease who improved with a strict gluten-free diet.

25 nding celiac disease that was managed with a gluten-free diet.

26 40 celiac disease patients after 6 months of gluten-free diet.

27 h care provider diagnosis and adherence to a gluten-free diet.

28 lth-related quality of life despite a strict gluten-free diet.

29 ective treatment for this disease is a total gluten-free diet.

30 treated coeliac disease and the effects of a gluten-free diet.

31 The current treatment is a strict gluten-free diet.

32 s or their presenting clinical features on a gluten-free diet.

33 stionnaire while undergoing treatment with a gluten-free diet.

34 merging in celiac patients unresponsive to a gluten-free diet.

35 accine in patients with coeliac disease on a gluten-free diet.

36 pain improved after diagnosis and initiated gluten-free diet.

37 liac disease usually resolve with a lifelong gluten-free diet.

38 l children develop celiac disease or require gluten-free diets.

39 eir symptoms improve when they are placed on gluten-free diets.

40 rexate restored the patients' sensitivity to gluten-free diets.

41 th 30 of 52 patients who did not adhere to a gluten-free diet (58%) (P < .0001).

42 according to their subsequent responses to a gluten-free diet, 7 of the 17 who were responsive to glu

43 45 of 148 patients who adhered strictly to a gluten-free diet (98%) had reduced symptoms, compared wi

44 nt may facilitate standardized evaluation of gluten-free diet adherence.

45 th celiac disease who no longer responded to gluten-free diets after they developed T-cell LGL, with

46 indings, one year following institution of a gluten free diet, also suggest that ulcerative jejunitis

47 tivity reflecting a strict compliance to the gluten-free diet and a good clinical response to gluten

48 cantly correlated with the low compliance to gluten-free diet and a mild clinical response.

49 odenal histology, comorbidities, response to gluten-free diet and complications were evaluated.

50 While typically managed by gluten-free diet and dapsone, treatment of DH refractory

51 Clinico-pathological features heal on a gluten-free diet and relapse when gluten is reintroduced

52 clearly are needed to assess the efficacy of gluten-free diet and to address the underlying mechanism

53 ash resolves when the patient is placed on a gluten-free diet and/or dapsone.

54 ional strategies (elimination of cow's milk, gluten-free diets, and 4-food elimination diet) were als

55 ogenesis, novel therapies alternative to the gluten-free diet are currently in advanced phase of deve

56 Gluten-free diets are now readily achievable with approp

57 ive serologic findings found initiation of a gluten-free diet associated with small improvement in ga

58 Patients' symptoms improved on a gluten-free diet at 12- to 18-month follow-up.

59 ectable in biopsy samples from patients on a gluten-free diet but expanded rapidly and specifically a

60 oeliac patients that initially improved on a gluten-free diet but then relapsed despite a strict diet

61 fore, AN-PEP should not be used to replace a gluten free diet, but rather to support digestion of occ

62 After treatment of celiac sprue with a gluten-free diet, chronic diarrhea persists in a substan

63 Gluten-free diet, claimed to be helpful in treating the

64 celiac disease in the context of an everyday gluten-free diet containing daily up to 2 g gluten.

65 Many patients following a gluten-free diet continue to have symptoms and have smal

66 coeliac patients who normally responded to a gluten-free diet (controls) were collected among 11 Ital

67 A gluten-free diet currently remains the only treatment op

68 n gastrointestinal symptoms compared with no gluten-free diet (difference less than 1 point on a scal

69 Although both conditions are treated with a gluten-free diet, distinguishing between celiac disease

70 However, compliance with the gluten-free diet, especially in the United States, is ex

71 In celiac patients on a gluten-free diet, exposure to gluten induces the appeara

72 bers of individuals are empirically trying a gluten-free diet for a variety of signs and symptoms.

73 Participants reported following a gluten-free diet for at least 1 year before the study be

74 c sprue (59 women and 19 men) treated with a gluten-free diet for at least 12 months were surveyed ab

75 years old; 17 male), who had been on strict gluten-free diets for at least 3 months, were given a 3-

76 lenge of 59 individuals on a self-instituted gluten-free diet, for whom celiac disease had been exclu

77 evices for an efficient, simple and accurate gluten free diet (GFD) monitoring as well as therapy fol

78 opsy-proven CD who were being treated with a gluten-free diet (GFD) for > or =2 y.

79 A strict gluten-free diet (GFD) for life is the only treatment fo

80 actively sought as alternative therapy to a gluten-free diet (GFD) for patients with coeliac disease

81 illous atrophy despite strict adherence to a gluten-free diet (GFD) have non-responsive celiac diseas

82 To assess the effectiveness of a Gluten-Free Diet (GFD) in seven adult female screening-d

83 objective evidence regarding the impact of a Gluten-Free Diet (GFD) in the large proportion of asympt

84 A gluten-free diet (GFD) is the only effective treatment f

85 growing number of individuals adhering to a gluten-free diet (GFD) without exclusion of celiac disea

86 tients with celiac disease should maintain a gluten-free diet (GFD), excluding wheat, rye, and barley

87 Six DH patients were studied while on a gluten-free diet (GFD), whereas four continued on a norm

88 symptoms are common despite treatment with a gluten-free diet (GFD), yet no approved or proven nondie

89 an impressive increase in popularity of the gluten-free diet (GFD)-now the most trendy alimentary ha

90 er active celiac disease (ACD) or those on a gluten-free diet (GFD).

91 tment for celiac disease (CeD) is a lifelong gluten-free diet (GFD).

92 y are commonly used to monitor patients on a gluten-free diet (GFD).

93 h endomysial antibodies (EmA) benefit from a gluten-free diet (GFD).

94 bowel syndrome (IBS-D) could benefit from a gluten-free diet (GFD).

95 coming a fraction of individuals following a gluten-free diet (GFD).

96 ease in individuals who have self-prescribed gluten-free diets (GFD) or have atypical presentations.

97 Difficulties adhering to a gluten-free diet have led to the development of non-diet

98 Poor adherence to a gluten-free diet, HLA-DQ2 homozygosity, and late diagnos

99 mins, that has been recommended as part of a gluten-free diet; however, few experimental data support

100 he main therapeutic intervention for CD is a gluten-free diet; however, novel nondietary agents are u

101 Initiated treatment with a gluten-free diet improves the situation for celiac disea

102 Studies of gluten-free diet in patients with gluten sensitivity and

103 ore, it allowed monitoring of adherence to a gluten-free diet in patients.

104 mptoms before and after the institution of a gluten-free diet in the patients, all of whom had been g

105 ciated with prevalence of celiac disease and gluten-free diet in the United States.

106 in antibodies of IgG class after 6 months of gluten-free diet; in contrast, 16/40 (40%) of celiac pat

107 tion of clinical manifestations follows if a gluten-free diet is adopted.

108 Although a gluten-free diet is an effective treatment in most indiv

109 A gluten-free diet is an effective treatment, but complian

110 In addition, a gluten-free diet is highly burdensome and can profoundly

111 ening for celiac disease must occur before a gluten-free diet is implemented, since once a patient in

112 nal histology before and after introducing a gluten-free diet is not yet established.

113 A gluten-free diet is the mainstay of safe and effective t

114 A gluten-free diet is the only available treatment for cel

115 A gluten-free diet is the only means to manage coeliac dis

116 up children with CD after prescription of a gluten-free diet is unclear.

117 Despite adherence to a gluten-free diet, many patients remain symptomatic and s

118 th poor prognosis, but strict adherence to a gluten-free diet may prevent its occurrence.

119 ure, omega-3 fatty acid supplementation, and gluten-free diet, may have additional benefits, as do po

120 New approaches to making the gluten-free diet more palatable are being studied.

121 After diagnosis, a strict gluten-free diet must be followed, which in most cases w

122 A gluten-free diet normalizes liver enzymes and histologic

123 zes a review of scientific studies using the gluten-free diet, nutritional risk factors, controversia

124 s designed to characterize the effect of the gluten-free diet on anti-gliadin antibodies of IgG class

125 Our aim was to investigate the effect of a gluten-free diet on celiac disease related symptoms, hea

126 We examined the effect of a gluten-free diet on gastrointestinal symptoms in a cohor

127 With a gluten-free diet, patients have substantial and rapid im

128 Loss of response to a gluten-free diet (refractory sprue) and ulcerative jejun

129 Treatment options other than the restrictive gluten-free diet remain limited.

130 Adherence to a gluten-free diet remains paramount as the recognition of

131 of coeliac disease, early introduction of a gluten-free diet remains the cornerstone of treatment.

132 in our understanding of celiac disease, the gluten-free diet remains the only current viable therapy

133 Strict adherence to a gluten-free diet remains the only way to prevent EATL.

134 scopy, 1 year and 4 years following a strict gluten free diet showed endoscopic and histological norm

135 mplemented, since once a patient initiates a gluten-free diet, testing for celiac disease is no longe

136 Because the symptoms of CS respond to a gluten-free diet, testing for CS in IBS may prevent year

137 Therapy is a gluten-free diet that requires avoidance of wheat, rye,

138 Despite his compliance to the gluten-free diet the symptoms persisted and we eventuall

139 red for screening for gluten sensitivity and gluten-free diet trials.

140 disease that do not improve despite a strict gluten-free diet (type A cases) and previously diagnosed

141 On a gluten-free diet, visits to primary care decreased signi

142 s choosing a strict wheat starch-containing, gluten-free diet vs. a naturally gluten-free diet.

143 ring phenomenon with the administration of a gluten-free diet with or without dapsone were observed.

144 : A man in his 80s with DH not controlled by gluten-free diet (with poor adherence), dapsone, and con

145 celiac disease was defined as adherence to a gluten-free diet without a diagnosis of celiac disease.