ライフサイエンスコーパス: glybenclamide

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1 apamin, 1 mm 4-aminopyridine, and 10 microm glybenclamide.

2 c acid, diphenylamine-2-carboxylic acid, and glybenclamide.

3 icroM; inhibits KCa and Kv1.2 channels), and glybenclamide (0.1-100 microM; inhibits KATP channels) h

4 cts of pinacidil were completely reversed by glybenclamide (10 micromol/L, n=4) and partially reverse

5 ener diazoxide, 3) the K(ATP) channel closer glybenclamide, 4) diazoxide plus the GABA(A) receptor ag

6 Inhibition of IK(ATP) by glybenclamide, a selective K(ATP) channel inhibitor, dep

7 The KATP channel blockers glybenclamide and 5-hydroxydecanoate prevented these eff

8 mbrane conductance regulator (CFTR) blockers glybenclamide and diphenylamine-2-carboxylate did not af

9 ormational change in SUR to increase the SUR/glybenclamide binding affinity.

10 Strikingly, it increased glybenclamide binding by decreasing the K(d).

11 a2+ in isolated bile duct cells suggest that glybenclamide directly activates Na+-K+-2Cl- cotransport

12 Denatonium did not displace glybenclamide from its binding sites on the sulfonylurea

13 4'-diisothiocyanostilbene-2,2'-disulfonate > glybenclamide >> diphenlyamine-2-carboxylate, tamoxifen,

14 twofold greater decrease in GABA levels, and glybenclamide increased VMH GABA levels by 57%.

15 verapamil and K(ATP) channel inhibitors like glybenclamide on pharmacologic stress using adenosine or

16 hannel blocker barium chloride (but not TEA, glybenclamide or tertiapin-Q) significantly occluded the

17 his efflux was not inhibited by brefeldin A, glybenclamide, or intracellular ATP depletion but was in

18 the GABA(A) receptor agonist muscimol, or 5) glybenclamide plus the GABA(A) receptor antagonist bicuc

19 Glybenclamide potently blocked all sK(ATP) channels, but

20 Here we report that the sulfonylurea glybenclamide potently stimulates bile flow and bicarbon

21 glucagon and epinephrine responses, whereas glybenclamide raised glucose infusion rates in conjuncti

22 n- or estrogen-induced cholestasis show that glybenclamide retains its stimulatory effects on bile fl

23 ctivity, in airway epithelial cells, that is glybenclamide sensitive.

24 ATP release from the RBCs was inhibited with glybenclamide, the platelet fluorescence intensity decre

25 Pretreatment with glybenclamide to assess the role of ATP-sensitive K+ cha

26 Inhibition of lipid efflux by glybenclamide treatment or by mutation of the ATP-bindin

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