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1 which is specifically esterified to the sn-2 glyceryl carbon of phosphatidylglycerol.
2 doglycan and the lipopeptide tri-palmitoyl-S-glyceryl-Cys-Ser-(Lys)(4) that occurred earlier and was
3 conjugated to the TLR2 agonist dipalmitoyl-S-glyceryl cysteine stimulated CD4 T lymphocyte IFN-gamma
4 embrane signal transducer for tripalmitoyl-S-glyceryl-cysteine (Pam(3)Cys)-modified lipoproteins, inc
5  recombinant OspA or OspB, or tripalmitoyl-S-glyceryl-cysteine had little or no effect on expression
6 e stability of DNA duplexes bearing isolated glyceryl cytosine residues has also been investigated.
7 ppended with templates incorporating atactic glyceryl cytosine residues.
8                                      Atactic glyceryl cytosine units are found to impede, but not to
9  that the C20BAS derivative has a short sn-1 glyceryl diether C20H40 transmembrane alkyl chain and tw
10  specifically catalyzes the formation of 1,2-glyceryl dinitrate and nitrite from GTN, leading to prod
11 se 2 (mtALDH) catalyzes the formation of 1,2-glyceryl dinitrate and nitrite from nitroglycerin (glyce
12  thionitrate intermediate and release of 1,2-glyceryl dinitrate.
13 2(S)-Hydroxyeicosa-5,8,10,14-tetraenoic acid glyceryl ester (12-HETE-G) was produced following additi
14 r release of 15-hydroxyeicosatetraenoic acid glyceryl ester (15-HETE-G), establishing that lipoxygena
15 , to quantitatively analyze changes in those glyceryl ester molecular species containing identical fa
16 poxygenase (12-LOX) oxygenated 2-AG to the 2-glyceryl ester of 12(S)-hydroperoxyeicosa-5,8,10,14-tetr
17                           We report that the glyceryl ester of PGE(2), PGE(2)-G, triggers rapid, conc
18                                    Thus, the glyceryl ester of prostaglandin E2, PGE2-G, mobilizes Ca
19 iding 15(S)-hydroperoxyeicosatetraenoic acid glyceryl ester.
20 thod for the quantification of prostaglandin glyceryl esters (PG-Gs), a newly discovered class of eic
21 ), to prostaglandins (PGs) and prostaglandin glyceryl esters (PG-Gs), respectively.
22 caused release of 2-AG and production of the glyceryl esters of PGE2 and PGI2 over 60 min.
23       Its turnover leads to the formation of glyceryl esters of prostaglandins (PG-Gs), a subset of w
24 thanolamide to prostaglandins, prostaglandin glyceryl esters, and prostaglandin ethanolamides, respec
25  stereoselective formation of 1'-(Z)-alkenyl glyceryl ethers.
26 g, UDP from a hydrolyzed donor, and an alpha-glyceryl-GlcNAc product formed in situ.
27  caryophilli PG2982, was observed to utilize glyceryl glyphosate as a sole phosphorus source.
28 that hydrolyzes phosphonate esters including glyceryl glyphosate to glyphosate and glycerol was clone
29 hytotoxicity resulted from the hydrolysis of glyceryl glyphosate to glyphosate and subsequent inhibit
30                            The hydrolysis of glyceryl glyphosate to glyphosate by a phosphonate ester
31 hereas its phosphonate monoester derivative, glyceryl glyphosate, is approximately 50-fold less activ
32  the pehA gene were killed when treated with glyceryl glyphosate.
33 promoter and treating the mature plants with glyceryl glyphosate.
34       The use of solid support bearing diDMT-glyceryl-linker permitted convenient synthesis of immuno
35 cenoic acid in the sn-2 position of its core glyceryl moiety.
36 rger (approximately 10-fold) than in neutral glyceryl monooleate (GMO) membranes.
37 ediated dilatation/Deltadilation response to glyceryl nitrate (95% CI 0.13-2.11).
38 pain, nonpungent analogs such as olvanil and glyceryl nonivamide (GLNVA) were synthesized to enhance
39          Evidence suggests that both D and L glyceryl nucleoside configurations at a given template p
40 ned sunflower oils, which 1% w/w addition of glyceryl oleate was determined as the optimal.
41 ne or more alkyl- (C2-C12), branched alkyl- (glyceryl or aminobutyryl-propanediol), or ethyleneglycol
42  (GGGP synthase) catalyzes alkylation of (S)-glyceryl phosphate [(S)-GP] by geranylgeranyl diphosphat
43 glyceryl prostaglandins, some of which (e.g. glyceryl prostaglandin E(2) and glyceryl prostaglandin I
44  which (e.g. glyceryl prostaglandin E(2) and glyceryl prostaglandin I(2)) exhibit interesting biologi
45                      The sterically hindered glyceryl prostaglandin was the poorest substrate examine
46                                              Glyceryl prostaglandins (PG-Gs) are generated by the oxy
47 gical activities of 2-arachidonylglycerol or glyceryl prostaglandins in humans.
48 of COX-2 oxygenation of endocannabinoids are glyceryl prostaglandins, some of which (e.g. glyceryl pr
49 monstrated by analysis of prostaglandins and glyceryl prostaglandins.
50 erred to acyl groups plus fatty acids, or to glyceryl structures.
51                                              Glyceryl tri-oleate but not OAEE resulted in 70% mortali
52                The triglyceride of OA (i.e., glyceryl tri-oleate) or OAEE was injected into the pancr
53  disperse nanoparticles of PGT (propoxylated glyceryl triacylate) that contain a glaucoma drug timolo
54                                              Glyceryl tributyrate was used for the in situ selective
55 by intraductal injection of the triglyceride glyceryl trilinoleate alone or with the chemically disti
56                                  Intraductal glyceryl trilinoleate with or without the lipase inhibit
57 ydroxytoluene (BHT) on the biodegradation of glyceryl trilinoleate, a model vegetable oil highly susc
58           Both lipase inhibitors reduced the glyceryl trilinoleate-induced increase in serum lipase,
59 onduit artery selectivity similar to that of glyceryl trinitrate (0.013-4.4 nmol/min) over resistance
60 f albuterol (5 microg/min, DeltaRI(ALB)) and glyceryl trinitrate (5 microg/min, DeltaRI(GTN)).
61 ependent dilation, EDD) and after sublingual glyceryl trinitrate (causing endothelium-independent dil
62 .01% (P=0.036) with no change in response to glyceryl trinitrate (endothelial-independent dilatation)
63 ers of each sex, we assessed FMD response to glyceryl trinitrate (GTN) at baseline and at 8 hours and
64 donor drugs isosorbide mononitrate (ISMN) or glyceryl trinitrate (GTN) for stable angina.
65                  Nitric oxide donors such as glyceryl trinitrate (GTN) have been shown to cause a rev
66 n each group, a migraine was induced using a glyceryl trinitrate (GTN) infusion.
67                                  Infusion of glyceryl trinitrate (GTN) into patients with migraine pr
68     Therefore, we investigated the effect of glyceryl trinitrate (GTN) on the efficacy of cell engraf
69                                 The NO donor glyceryl trinitrate (GTN) provokes delayed migraine atta
70                   Albuterol (salbutamol) and glyceryl trinitrate (GTN) were administered locally by b
71                               The effects of glyceryl trinitrate (GTN), amlodipine, and atenolol were
72                                        Using glyceryl trinitrate (GTN), an agonist of nitric oxide (N
73 ing pacing and after intragraft injection of glyceryl trinitrate (GTN).
74 h after infusing the NO donor nitroglycerin [glyceryl trinitrate (GTN)] to migraineurs.
75 ent) and response to 50 microg of sublingual glyceryl trinitrate (GTN, endothelium independent) were
76 abolism of the cornerstone nitrovasodilator, glyceryl trinitrate (GTN; 0.1-100 mg/kg), in the rat in
77                                      We used glyceryl trinitrate (nitroglycerin) to trigger premonito
78 educed responses to bradykinin (P=0.005) and glyceryl trinitrate (P=0.006).
79 achidonic acid: 0.2, 2, and 20 nmol/min; and glyceryl trinitrate 1, 2, and 4 pmol/min) were construct
80 mediated dilatation [FMD]) and -independent (glyceryl trinitrate [GTN]) changes in brachial artery ar
81 yl dinitrate and nitrite from nitroglycerin (glyceryl trinitrate [GTN]) within mitochondria, leading
82  The metabolism of nitrovasodilators such as glyceryl trinitrate and nitroprusside provides the activ
83 P=0.045) after adjusting for the response to glyceryl trinitrate and was independent of conventional
84     In addition, exogenous acetylcholine and glyceryl trinitrate both increase arterial distensibilit
85    There was no significant change in FMD or glyceryl trinitrate during the placebo phase.
86    Sublingual administration of the NO donor glyceryl trinitrate normalized platelet VASP phosphoryla
87 rently available nitric oxide donors such as glyceryl trinitrate or isosorbide mononitrate could be u
88  been short-term; a new topical, easy-to-use glyceryl trinitrate preparation has been shown to improv
89 inistration of the nitric oxide (NO) mimetic glyceryl trinitrate prevented altered uteroplacental per
90                Infusion of acetylcholine and glyceryl trinitrate reduced PWV significantly, by 6+/-4%
91                      Responses to sublingual glyceryl trinitrate were unchanged.
92 , D-S-nitrosocysteine, S-nitrosoglutathione, glyceryl trinitrate, and sodium nitroprusside produced m
93 ular mechanisms through which nitroglycerin (glyceryl trinitrate, GTN) elicits nitric oxide (NO)-base
94 atalyzes the bioactivation of nitroglycerin (glyceryl trinitrate, GTN) in blood vessels, resulting in
95                               Nitroglycerin (glyceryl trinitrate, GTN), originally manufactured by Al
96 thysmography with acetylcholine, bradykinin, glyceryl trinitrate, norepinephrine, and l-NG-monomethyl
97                         In in vitro studies, glyceryl trinitrate, sodium nitroprusside, and 3'-morpho
98 ts, intravenous infusion of three NO donors, glyceryl trinitrate, sodium nitroprusside, or 3'-morphol
99 ry flow-mediated (endothelium-dependent) and glyceryl trinitrate-induced (endothelium-independent) di
100                                 In contrast, glyceryl trinitrate-induced dilatation was similar in th
101 15) and lower FMD percentage (P = 0.026) and glyceryl trinitrate-induced dilation (P = 0.012) than he
102  pattern was identical in migraineurs during glyceryl trinitrate-induced migraine attacks as well as
103 ix control subjects at rest and during acute glyceryl trinitrate-induced migraine attacks using posit
104 ins tight for dihydroergotamine during acute glyceryl trinitrate-induced migraine attacks.
105 pothalamus, seen in the premonitory phase of glyceryl trinitrate-triggered migraine attacks can expla
106 ere was no significant change in response to glyceryl trinitrate.
107 sting or reducing anginal episodes or use of glyceryl trinitrate.
108 (BK) and the endothelium-independent dilator glyceryl-trinitrate (GTN).
109 harged on the same day with diclofenac, 0.2% glyceryl-trinitrate ointment, lactulose, a telephone num
110 eeding the fly mutant one of the components, glyceryl trioleate oil, blocked the accumulation of exce
111 rmed from the model compounds palmitic acid, glyceryl tripalmitate and 1,3-dipalmitoyl-2-oleoylglycer

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