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1 sting or reducing anginal episodes or use of glyceryl trinitrate.
2 ere was no significant change in response to glyceryl trinitrate.
3 onduit artery selectivity similar to that of glyceryl trinitrate (0.013-4.4 nmol/min) over resistance
4 achidonic acid: 0.2, 2, and 20 nmol/min; and glyceryl trinitrate 1, 2, and 4 pmol/min) were construct
6 The metabolism of nitrovasodilators such as glyceryl trinitrate and nitroprusside provides the activ
7 P=0.045) after adjusting for the response to glyceryl trinitrate and was independent of conventional
8 , D-S-nitrosocysteine, S-nitrosoglutathione, glyceryl trinitrate, and sodium nitroprusside produced m
10 ependent dilation, EDD) and after sublingual glyceryl trinitrate (causing endothelium-independent dil
12 .01% (P=0.036) with no change in response to glyceryl trinitrate (endothelial-independent dilatation)
13 ers of each sex, we assessed FMD response to glyceryl trinitrate (GTN) at baseline and at 8 hours and
18 Therefore, we investigated the effect of glyceryl trinitrate (GTN) on the efficacy of cell engraf
25 ent) and response to 50 microg of sublingual glyceryl trinitrate (GTN, endothelium independent) were
26 abolism of the cornerstone nitrovasodilator, glyceryl trinitrate (GTN; 0.1-100 mg/kg), in the rat in
28 mediated dilatation [FMD]) and -independent (glyceryl trinitrate [GTN]) changes in brachial artery ar
29 yl dinitrate and nitrite from nitroglycerin (glyceryl trinitrate [GTN]) within mitochondria, leading
30 ular mechanisms through which nitroglycerin (glyceryl trinitrate, GTN) elicits nitric oxide (NO)-base
31 atalyzes the bioactivation of nitroglycerin (glyceryl trinitrate, GTN) in blood vessels, resulting in
33 ry flow-mediated (endothelium-dependent) and glyceryl trinitrate-induced (endothelium-independent) di
35 15) and lower FMD percentage (P = 0.026) and glyceryl trinitrate-induced dilation (P = 0.012) than he
36 pattern was identical in migraineurs during glyceryl trinitrate-induced migraine attacks as well as
37 ix control subjects at rest and during acute glyceryl trinitrate-induced migraine attacks using posit
40 thysmography with acetylcholine, bradykinin, glyceryl trinitrate, norepinephrine, and l-NG-monomethyl
41 Sublingual administration of the NO donor glyceryl trinitrate normalized platelet VASP phosphoryla
42 harged on the same day with diclofenac, 0.2% glyceryl-trinitrate ointment, lactulose, a telephone num
43 rently available nitric oxide donors such as glyceryl trinitrate or isosorbide mononitrate could be u
45 been short-term; a new topical, easy-to-use glyceryl trinitrate preparation has been shown to improv
46 inistration of the nitric oxide (NO) mimetic glyceryl trinitrate prevented altered uteroplacental per
49 ts, intravenous infusion of three NO donors, glyceryl trinitrate, sodium nitroprusside, or 3'-morphol
50 pothalamus, seen in the premonitory phase of glyceryl trinitrate-triggered migraine attacks can expla
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