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1 ransfer bound phosphates between cytosol and glycosome.
2 essential bound-phosphate moiety within the glycosome.
3 y and localized the endogenous enzyme to the glycosome.
4 s and that it is sequestered in the parasite glycosome.
5 que metabolic organelle in the parasite: the glycosome.
6 s in a unique single-membrane organelle, the glycosome.
7 strong driving force for maintenance of the glycosome.
8 athway of these organisms resides within the glycosome.
9 nals (PTS2s) to direct their import into the glycosome.
10 novani HGPRT is localized exclusively to the glycosome.
11 of HGPRT, all of which was localized to the glycosome.
12 suggesting a heretofore unknown role of the glycosome.
13 roxide dismutases (SODs) in mitochondria and glycosomes.
14 intermediary metabolism and hence are called glycosomes.
15 ile TbPAGM localized to both the cytosol and glycosomes.
16 mes within peroxisome-like organelles called glycosomes.
17 steps inside specialized peroxisomes, named glycosomes.
18 ytic direction with production of ATP in the glycosomes.
19 e topogenic signal targeting proteins to the glycosome, a fuel-metabolizing microbody unique to these
22 g signal (PTS-1) that steers proteins to the glycosome, an organelle unique to Leishmania and related
23 PPP is localized to both the cytosol and the glycosome and adding it to the glycolytic model without
25 is compartmentalized exclusively within the glycosome and that the COOH-terminal tripeptide of the p
29 ng proteins to these organelles suggest that glycosomes and peroxisomes may have evolved from a commo
40 man host, and the vital importance of proper glycosome biogenesis to the parasite, render these perox
43 arasite; and (iv) that ARG is present in the glycosome, but this subcellular milieu is not essential
45 ycolytic steps need to be regenerated in the glycosomes by kinases, such as phosphoenolpyruvate carbo
46 n of protein import into this organelle, the glycosome, can be accomplished through RNA interference
48 for the exchange of metabolites between the glycosomes, cytosol, mitochondrion and the host medium.
50 ve shown that in the absence of glucose, the glycosome exhibits mild acidification from pH 7.4 +/- 0.
51 isolate the first Leishmania mutant (gim1-1 [glycosome import] mutant) with a defect in the import of
52 C)}GGAKL) for investigating pH regulation of glycosomes in live procyclic form Trypanosoma brucei Whe
54 inant proteins localized endogenous TbPMM to glycosomes in the bloodstream form of the parasite, whil
55 l diseases, contain unique organelles called glycosomes in which the first seven glycolytic enzymes a
57 disease, compartmentalize glycolysis within glycosomes, metabolic organelles related to peroxisomes.
59 etabolically specialized peroxisomes include glycosomes of trypanosomes, which have come to compartme
60 luding additional enzymatic reactions in the glycosome, or (ii) adding a mechanism to transfer bound
61 ays in unique subcellular microbodies called glycosomes, organelles related to the peroxisomes of mam
64 L. major; (ii) sequestration of GPI-PLCp to glycosomes protects free protein-GPIs from cleavage by t
66 Taken together, these data indicate that the glycosome provides significant, but not complete, protec
69 3-phosphate, which is produced in vivo by a glycosome-resident glycerol kinase, mitigated acid inact
70 These observations support a model in which glycosome sequestration of a catabolic GPI-PLCp preserve
71 of gim1-1 and distinctive role of Leishmania glycosomes suggest that future studies of this system wi
72 HK1, particularly given the acidification of glycosomes that can be induced under a variety of parasi
74 ll as gain insights into the function of the glycosome, we used a positive genetic selection procedur
77 bsequently demonstrated to be present in the glycosome, whereas the polyamine biosynthetic enzymes, i
78 icroscopy also revealed increased numbers of glycosomes, while immunofluorescence microscopy showed i
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