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1 inhibitors [AGIs], and insulin adjusted for glycosylated hemoglobin).
2 001) between serum protein argpyrimidine and glycosylated hemoglobin.
3 values, as well as significant decreases in glycosylated hemoglobin.
4 tis is associated with a slight elevation in glycosylated hemoglobin.
5 cerides, SBP, DBP, BMI, waist/hip ratio, and glycosylated hemoglobin.
6 esterol, total to HDL cholesterol ratio, and glycosylated hemoglobin].
7 lic blood pressure (-9.37 mm Hg; p < 0.001), glycosylated hemoglobin (-0.92%; p < 0.001), and smoking
8 table plaque and healed infarct had elevated glycosylated hemoglobin (10.2 +/- 5.0% versus 6.4 +/- 0.
10 f fasting serum glucose, insulin, C-peptide, glycosylated hemoglobin A (HbA1c), and Homeostasis Model
12 with fluorinated compounds to detect tHb and glycosylated hemoglobin A1c (GHbA1c) in human whole bloo
13 ation of less than 7.8 mmol/L (140 mg/dL) or glycosylated hemoglobin A1c (HbA1c) below 7% is unknown.
16 plasma glucose by 0.69 mmol/L [1.32; 0.07], glycosylated hemoglobin A1C by 0.37% [0.54; 0.20], body
18 tegerin, osteocalcin, osteopontin, and serum glycosylated hemoglobin A1c, insulin, and glucose were a
19 adjusting for duration of diabetes mellitus, glycosylated hemoglobin A1c, statin use, and end-stage r
21 herapy during the DCCT, smoking, and greater glycosylated hemoglobin and AER at DCCT closeout but not
23 yperglycemic at 26 weeks of age had elevated glycosylated hemoglobin and sciatic nerve sorbitol level
24 six subjects with normal fasting glucose and glycosylated hemoglobin and seven overtly diabetic subje
25 and duration of diabetes, level of control (glycosylated hemoglobin), and demographic data were reco
30 s in a 24-site US AMI registry (2005-08) had glycosylated hemoglobin assessed at a core laboratory, w
31 terol, high-density lipoprotein cholesterol, glycosylated hemoglobin, cigarette smoking) in addition
32 Total cholesterol (TC), HDL cholesterol, glycosylated hemoglobin, cigarette smoking, and hyperten
35 ithout affecting body weight, blood glucose, glycosylated hemoglobin, creatinine, or creatinine clear
37 adjusting for age, gender, body mass index, glycosylated hemoglobin, diabetes duration, systolic blo
38 r decline was higher after age 35 yr or when glycosylated hemoglobin exceeded 9% but did not vary wit
39 low-up, subjects who were in "poor" control (glycosylated hemoglobin (GHb) > or = 11%) at baseline we
40 han values in the WT mice; blood glucose and glycosylated hemoglobin (GHb) levels did not differ in t
41 nsive description of glycemic control (total glycosylated hemoglobin (GHb)) up to 4.5 years duration
42 cholesterol, multivessel CAD, diabetes with glycosylated hemoglobin >7%, and persistent angina were
45 strates that hydroxychloroquine (HCQ) lowers glycosylated hemoglobin (HbA(1c) ) in diabetes patients
46 primary outcome (simultaneous control with a glycosylated hemoglobin (HbA(1c)) <7.0%, blood pressure
47 y disease; however, the relationship between glycosylated hemoglobin (HbA(1c)) as a marker of chronic
48 aptoglobin (Hp) polymorphism rs#72294371 and glycosylated hemoglobin (HbA(1c)) on risk of coronary he
49 al glucose tolerance test); 2) with baseline glycosylated hemoglobin (HbA1) less than 9%; and 3) who
50 8 (16.8) U/d of insulin, and had a mean (SD) glycosylated hemoglobin (HbA1) level of 10.0% (1.9%).
53 ncluded low-density lipoprotein </= 70mg/dl, glycosylated hemoglobin (HbA1c) </= 6.5%, and systolic b
54 4, OR10-14 years = 2.07, OR15+years = 3.99), glycosylated hemoglobin (HbA1c) (OR6.5-6.9% = 1.33, OR7-
55 designed to examine the relationship between glycosylated hemoglobin (HbA1C) and adverse outcomes in
56 abetes control was assessed by percentage of glycosylated hemoglobin (HbA1c) and divided into three c
57 the effects of ranolazine versus placebo on glycosylated hemoglobin (HbA1c) at 6- and 12-month follo
62 Mental State Examination of 28.6 +/- 1.5, a glycosylated hemoglobin (HbA1C) of 5.88 +/- 0.74%, and a
65 the levels of the fasting plasma glucose and glycosylated hemoglobin (HbA1c) were compared in both gr
67 nder-the-curve glucose after glucose load or glycosylated hemoglobin (HbA1c), and measures of insulin
68 index (BMI), blood pressure, serum glucose, glycosylated hemoglobin (HbA1c), blood urea nitrogen, se
70 rement of random blood glucose levels, serum glycosylated hemoglobin (HbA1c), serum porcine C peptide
73 h type 2 diabetes and poor glycemic control (glycosylated hemoglobin [HbA1c] levels > or =8.0%) and p
74 DM (mean duration, 7.7 [SD, 7.2] years; mean glycosylated hemoglobin [HbA1c] value, 7.4% [SD, 1.0%]),
75 hypertension (>130/80 mm Hg) and type 2 DM (glycosylated hemoglobin [HbA1c], 6.5%-8.5%) and were rec
76 factor changes (fasting blood glucose [FBG], glycosylated hemoglobin [HbA1c], systolic or diastolic b
77 s to these bacteria, and blood sugar levels (glycosylated hemoglobin, HbAlc) were studied before and
78 estimates for glycosylated hemoglobin (total glycosylated hemoglobin, hemoglobin A1, or hemoglobin A1
79 r increased extent of macular edema), higher glycosylated hemoglobin, history of diabetic neuropathy,
80 sting glucose is 91.6+/-13.8 mg/dl, and mean glycosylated hemoglobin is 5.1+/-0.7% (normal range: 4.3
82 he purpose of this study was to determine if glycosylated hemoglobin is elevated in patients with per
83 luded age, gender, BP, body mass index, GFR, glycosylated hemoglobin, LDL cholesterol, HDL cholestero
84 inuria of short duration, salutary levels of glycosylated hemoglobin (less than 8 percent), low systo
85 represented a 1-percentage point increase in glycosylated hemoglobin level (95% CI, 1.10 to 1.26) in
86 ssure (diabetic and nondiabetic patients) or glycosylated hemoglobin level (diabetic patients only).
87 hospitalization were longitudinally measured glycosylated hemoglobin level (rate ratio = 1.5 per 2% i
89 of metformin-rosiglitazone, 28.1% achieved a glycosylated hemoglobin level of 7% or less [corrected].
90 ose level of 12.0 mmol/L (216 mg/dL), a mean glycosylated hemoglobin level of 8.8%, and a mean body m
91 during the open treatment phase (mean +/- SD glycosylated hemoglobin level reduction, -0.4% +/- 1.4%;
92 diabetes, chronic hyperglycemia (assessed by glycosylated hemoglobin level) is related to the develop
94 italization in the preceding 6 months, lower glycosylated hemoglobin level, and questionnaire respons
95 controlling for age, sex, cigarette smoking, glycosylated hemoglobin level, insulin use, plasma C-pep
99 style, there was a significant difference in glycosylated hemoglobin levels between those who rated t
102 reatment are associated with improvements in glycosylated hemoglobin levels for at least 1 year.
106 our postdextrose glucose concentrations, and glycosylated hemoglobin levels were available from 11 27
107 than for white girls in every age group, and glycosylated hemoglobin levels were highest for black an
108 reports from 1966 through June 1994 in which glycosylated hemoglobin levels were measured concurrentl
110 ferent in the two groups, but follow-up mean glycosylated hemoglobin levels were significantly better
112 ts, emergency department visits, death rate, glycosylated hemoglobin levels, medications, and past me
118 ssure, high-density lipoprotein cholesterol, glycosylated hemoglobin, lipoprotein(a) and fibrinogen l
119 e <120/80 mm Hg, fasting glucose <100 mg/dl, glycosylated hemoglobin <5.7%, and total cholesterol <20
123 of diabetic retinopathy was associated with glycosylated hemoglobin (odds ratio [OR], 1.27; 95% CI,
125 nor documentation of DM in the chart (median glycosylated hemoglobin of unrecognized patients, 6.7%;
126 to the previously established role for total glycosylated hemoglobin, other factors including height,
127 plasma glucose concentration, percentage of glycosylated hemoglobin, plasma insulin concentration, a
128 rs of progression to definite DSP were total glycosylated hemoglobin (relative risk (RR) for increase
130 and endorsed as an acceptable screen for DM, glycosylated hemoglobin testing should be considered for
133 rovascular disease; however, the relation of glycosylated hemoglobin to macrovascular disease is less
134 MI); lipid profile; and levels of glucose or glycosylated hemoglobin, to identify those with metaboli
136 e incidence of neuropathy was related to the glycosylated hemoglobin value and the duration of diabet
137 liter], P<0.001), respectively, and the mean glycosylated hemoglobin value decreased 1.2 percentage p
138 ent glycosylated hemoglobin value, change in glycosylated hemoglobin value during the follow-up perio
139 ary albumin excretion rate and with the mean glycosylated hemoglobin value during the mean duration (
141 we found that duration of diabetes, current glycosylated hemoglobin value, change in glycosylated he
146 ations during fasting and postprandially and glycosylated hemoglobin values were measured periodicall
147 nsulin-dependent (type 2) diabetes mellitus (glycosylated hemoglobin values, 8 to 12 percent; normal,
149 c model assessment of insulin resistance and glycosylated hemoglobin were measured from a fasting blo
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