ライフサイエンスコーパス: goiter

1 's effects on the body (eg, polycythemia and goiter).

2 ctice in nontoxic uninodular or multinodular goiter.

3 esis of Cowden Disease and sporadic nontoxic goiter.

4 drome, which is associated with deafness and goiter.

5 and with a maternal history of hepatitis or goiter.

6 ssive disorder characterized by deafness and goiter.

7 ed, especially in patients with pre-existing goiter.

8 diffuse goiter and 41 patients had a nodular goiter.

9 38%) but not in 18 follicular adenomas and 6 goiters.

10 weed was used as a source of iodine to treat goiters.

11 m in newborns that, if untreated, results in goiter along with serious cognitive and growth defects.

12 atients, of which 233 patients had a diffuse goiter and 41 patients had a nodular goiter.

13 mice had hypothyroidism, dwarfism, alopecia, goiter and cardiac abnormalities including hypertrophy,

14 ome is a major cause of congenital deafness, goiter and defective iodide organification.

15 hyroid abnormalities, including multinodular goiter and follicular adenomas, and are at increased ris

16 animal model of Graves disease to show that goiter and hyperthyroidism occur to a much greater exten

17 oid-specific expression of BRAFV600E induces goiter and invasive PTC, which transitions to poorly dif

18 Graves' disease is hyperthyroidism, diffuse goiter, and exophthalmos.

19 der characterized by sensorineural deafness, goiter, and impaired iodide organification.

20 rrhythmia or ischemic heart disease, size of goiter, and severity of thyrotoxicosis.

21 at exposure and at examination, presence of goiter, and urban/rural residency.

22 d multinodular (OR = 0.69, p = 3.9 x 10(-5)) goiters, and thyrotoxicosis (OR = 0.76, p = 1.5 x 10(-3)

23 a murine model prevented the development of goiter as well as the induction of inflammatory and fibr

24 o were scheduled for total thyroidectomy for goiter, benign nodular disease, suspected thyroid cancer

25 found that Pten mutant mice develop diffuse goiter characterized by extremely enlarged follicles, in

26 Goiter development due to incomplete thyrotropin suppres

27 causes neonatal hypothyroidism and prevents goiter development.

28 tic thyrocyte cell death, preventing thyroid goiter formation in rdw/rdw rats.

29 an important cause of congenital hypothyroid goiter; further, homozygous mice expressing two cog/cog

30 Patients with toxic nodular goiter had an SMR of 1.16 (95% confidence interval [CI],

31 90 papillary carcinomas, and 0 of 10 nodular goiters had 3p25 rearrangements by interphase fluorescen

32 ich a lack of secondary thyroid enlargement (goiter) implicates death of thyrocytes as part of diseas

33 icient to eliminate or postpone the onset of goiter in individuals with DFNB4.

34 enocytes from NODCCR7(ko/ko) animals induced goiter in NOD.SCID recipients, demonstrating that autore

35 g transcytosis in vivo, using a rat model of goiter induced by aminotriazole, in which increased rele

36 al hypothyroidism, and in the cog congenital goiter mouse and rdw rat dwarf models, thyroid hormone s

37 All 16 thyroid masses were due to goiter; none of these were changed at follow-up CT 1 yea

38 Of 59 patients referred for a diffuse goiter or a multinodular gland, ultrasonography detected

39 4.3% [70 of 288], P < .001) and multinodular goiter or thyroid nodule (40.7% [50 of 123] vs 29.2% [84

40 (OR, 7.63; 95% CI, 3.49-16.69), multinodular goiter or thyroid nodule (OR, 1.82; 95% CI, 1.01-3.28),

41 oidism and hyperthyroidism in adults without goiter or thyroid nodules.

42 erparathyroidism, and in patients with large goiters or moderate to severe thyroid eye disease who ca

43 e to suppress TSH because of thyroid cancer, goiters, or nodules seem to have an adverse effect on bo

44 otid artery and thyroid vein of 10 euthyroid goiter patients and one patient with a toxic solitary ad

45 to segregate with retinitis pigmentosa (RP), goiter, primary ovarian insufficiency, and mild intellec

46 lly are associated with the development of a goiter, provided that the bioactivity and action of thyr

47 n in rural and urban areas included maternal goiter (rural odds ratio (OR) = 5.14, 95% confidence int

48 hibit polycythemia, pericardial effusion, or goiter should be evaluated for cobalt exposure.

49 , especially co-existing with an adenomatous goiter, should prompt a work-up for thyroid metastasis.

50 d a marked enlargement of the thyroid gland (goiter) that was associated with circulating autoantibod

51 h as Graves' disease and functioning nodular goiters, there are more than 20 less common causes of el

52 A growth or mass (including hernias and goiters) was the most commonly reported potentially surg

53 nimals suffering from congenital hypothyroid goiter with defective thyroglobulin, GRP94 and thyroglob