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1 s derive from a common precursor pool during gonadogenesis.
2 ng that lines represses hub cell fate during gonadogenesis.
3 nd identified twenty-four genes required for gonadogenesis.
4 t for testis differentiation at the onset of gonadogenesis.
5 that is required for Sox9 expression during gonadogenesis.
6 lineages and have distinct roles in somatic gonadogenesis.
7 rphic and initiates sex-specific programs of gonadogenesis.
8 howed that C. elegans gon-14 is required for gonadogenesis.
9 plays several distinct roles during somatic gonadogenesis.
10 b on a Numbl mutant background just prior to gonadogenesis.
11 primary focus has been the role of hnd-1 in gonadogenesis.
12 ate morphogenic processes are at work during gonadogenesis.
13 well as their migration and guidance during gonadogenesis.
14 ut does not appear to have any role in later gonadogenesis.
15 ng functions with hnd-1 in embryogenesis and gonadogenesis.
16 partially penetrant defects in viability and gonadogenesis.
17 with FOG co-factors exhibit abnormalities in gonadogenesis.
18 on-4 in an attempt to learn how it regulates gonadogenesis.
19 he regulation of both hermaphrodite and male gonadogenesis.
20 1 to early L2 stage hermaphrodites to permit gonadogenesis.
21 ee posterior parasegments at early stages in gonadogenesis.
23 e ubiquitously expressed HDA-1 in C. elegans gonadogenesis and place hda-1 in the Notch signaling pat
24 r findings shed light on the complexities of gonadogenesis and the genetic regulation required for pr
25 e that the lag-2 gene, which plays a role in gonadogenesis and vulval development and encodes a Notch
27 ay in which germ cell pressure may influence gonadogenesis, and also predicts that adult germ cells m
29 the C. elegans gonad is defined during early gonadogenesis by two somatic gonadal precursor cells, Z1
30 we describe a novel regulator of Drosophila gonadogenesis, clift, mutations in which abolish gonad f
31 (q645) allele is fully penetrant for the Sys gonadogenesis defect in hermaphrodites, but affects male
32 ch has also been called a gon-10 mutant (for gonadogenesis defective 10) and show that loss of HDA-1
33 We find that sys-1 is haplo-insufficient for gonadogenesis defects and that sys-1 and pop-1 mutants d
38 nimals, GSCs are established during preadult gonadogenesis following the proliferation and migration
40 is of distal tip cell (DTC) migration during gonadogenesis in Caenorhabditis elegans has revealed the
41 blish a link between ribosome biogenesis and gonadogenesis in Caenorhabditis elegans that affects ger
45 Neuronal vpr-1 expression is sufficient for gonadogenesis induction during a limited time period sho
48 of Pitx2 on the right side before and during gonadogenesis is sufficient to transform the right gonad
49 or 1 (Sf1), a nuclear receptor essential for gonadogenesis, is reduced to minimal levels in the Lhx9-
50 ing other genes that mediate early stages of gonadogenesis, Lhx9 mutants do not exhibit additional ma
53 to the seminiferous tubules of the testis as gonadogenesis proceeds, and both Sertoli cells and germ
55 of these male-biased genes were involved in gonadogenesis, spermatogenesis, testicular determination
56 determination system, but genes involved in gonadogenesis, spermatogenesis, testicular determination
57 antagonistic effect on fkh-6 expression and gonadogenesis, which is relieved by cyclin D activity.
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