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1 xyprogesterone in response to stimulation by gonadotrophin.
3 n of differentiation markers human chorionic gonadotrophin and placental alkaline phosphatase was una
4 em gene expression depended on androgens and gonadotrophins, as demonstrated by a lack of expression
5 (i.e., her offspring's) homologous chorionic gonadotrophin beta5 (CGB5) and CSH1 genes and her post-p
8 tuitarism in humans that is characterized by gonadotrophin deficiency known as hypogonadotrophic hypo
10 ack of expression in hypophysectomized mice, gonadotrophin-deficient hypogonadal (hpg) mutant mice, a
11 chytene spermatocyte that is under increased gonadotrophin drive because of testicular atrophy induci
12 ne (alpha-fetoprotein and/or human chorionic gonadotrophin) during the first two cycles of chemothera
14 s, with up-regulation and down-regulation of gonadotrophin gene expression during the breeding and no
15 Using primers specific for human chorionic gonadotrophin gene, the presence of human DNA was confir
16 h-affinity binding of LH and human chorionic gonadotrophin (hCG) causes secondary hormone or receptor
17 alpha-fetoprotein (AFP) and human chorionic gonadotrophin (HCG) during the first two cycles of chemo
19 ractions between an antigen, human chorionic gonadotrophin (hCG), and antibodies, anti-alpha-hCG and
20 e pretreatment level of HCG; human chorionic gonadotrophin (HCG; < or = 100 times the upper limit of
21 ded by alpha-fetoprotein and human chorionic gonadotrophin in the management of germ cell tumor (GCT)
25 The importance of accurate human chorionic gonadotrophin monitoring and the types of human chorioni
28 ell line-derived recombinant human chorionic gonadotrophin (r-alpha hCG), a protein that is glycosyla
30 a sustained increase in pulsatile release of gonadotrophin releasing hormone (GnRH) from the hypothal
32 as following the downregulation of pituitary gonadotrophin releasing hormone (GnRH) receptors and the
33 ergic transmission regulates the activity of gonadotrophin-releasing hormone (GnRH) neurons in the pr
34 ously shown that central CGRP suppresses the gonadotrophin-releasing hormone (GnRH) pulse generator,
35 dealing with various stressors that regulate gonadotrophin-releasing hormone (GnRH) secretion in a va
36 for vasoactive intestinal peptide (VIP) and gonadotrophin-releasing hormone (GnRH) was carried out o
37 e axis, specifically inhibition of pulsatile gonadotrophin-releasing hormone (GnRH)/luteinising hormo
38 7 alpha-hydroxyprogesterone to leuprolide, a gonadotrophin-releasing hormone agonist, and performed o
39 logue (CEP-701), as the trk inhibitor, and a gonadotrophin-releasing hormone agonist, Leuprolide, to
40 and progesterone stimulate their growth, and gonadotrophin-releasing hormone agonists shrink them.
41 el SERMs, aromatase inhibitors/inactivators, gonadotrophin-releasing hormone agonists, retinoids, sta
42 nous reproductive hormone production using a gonadotrophin-releasing hormone antagonist (GnRHant) for
43 although reproduction relies on hypothalamic gonadotrophin-releasing hormone output, and most cells p
44 in-deficient states, leptin therapy restores gonadotrophin secretion, as well as luteinizing hormone
47 o promote desensitization of human chorionic gonadotrophin-stimulated AC activity, in the presence of
48 ied beta-arrestin-1 mimicked human chorionic gonadotrophin to promote desensitization of human chorio
49 ariety of sequences, such as human chorionic gonadotrophin, ubiquitin, TFIIA, guanine nucleotide-bind
50 arkers alpha-fetoprotein and human chorionic gonadotrophin were correlated with treatment outcome as
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