ライフサイエンスコーパス: gonadotropin

1 irculating concentrations of human chorionic gonadotropin.

2 regulation on androgen production induced by gonadotropin.

3 a) and the hormone-specific beta-subunits of gonadotropin.

4 progesterone metabolites and human chorionic gonadotropin.

5 rd, along with a greater ovarian response to gonadotropins.

6 ehaviors and the control of the secretion of gonadotropins.

7 its expression is regulated directly by the gonadotropins.

8 of gonadotropin-releasing hormone (GnRH) and gonadotropins.

9 ey responded efficiently to a single dose of gonadotropins.

10 rtility defects were secondary to suppressed gonadotropins.

11 ovarian stimulation (up to four cycles) with gonadotropin (301 women), clomiphene (300), or letrozole

12 Subsequent treatment with gonadotropins allowed the generation of mature oocytes c

13 9%; P=0.44) but was lower than the rate with gonadotropin alone (34 of 107, 32%; P=0.006).

14 py (gonadotropin or clomiphene) (P=0.003) or gonadotropin alone (P<0.001) but not with clomiphene alo

15 tropin hormone alpha-subunit gene, chorionic gonadotropin alpha (Cga), is responsible for Cga cell-sp

16 ts in hallmarks of POI including stereotyped gonadotropin alterations indicative of early menopause,

17 ased blood level of the beta human chorionic gonadotropin and a histopathological examination, the di

18 ys 1, 8, and 15]), patients' human chorionic gonadotropin and alfa-fetoprotein concentrations were me

19 with a favourable decline in human chorionic gonadotropin and alfa-fetoprotein continued BEP (Fav-BEP

20 ly published data sets for annual changes in gonadotropins and estradiol in rainbow trout.

21 at the end of drug administration, and serum gonadotropins and oestradiol measured.

22 ary during the estrous cycle and the role of gonadotropins and ovarian steroid hormones in ESR36 expr

23 gical activity was validated by secretion of gonadotropins and sex steroids.

24 mulation triggered a significant increase in gonadotropins and testosterone levels in Gnaq(d/d) mice.

25 od sampling for thyroid stimulating hormone, gonadotropin, and prolactin deficiencies, whereas for AC

26 rimary site and the level of human chorionic gonadotropin as independent factors.

27 ndent microRNAs, predicted in silico to bind gonadotropin beta subunit mRNAs, were suppressed in puri

28 t if microRNAs regulate the hormone-specific gonadotropin beta subunits in vivo, we deleted Dicer in

29 a that was confirmed by beta human chorionic gonadotropin (beta-HCG) levels and histopathology.

30 dotropes resulted in profound suppression of gonadotropin-beta subunit proteins and, consequently, th

31 phoblast cells that produce equine chorionic gonadotropin between days 40 and 120 of normal pregnancy

32 hat have not been previously associated with gonadotropin biosynthesis and/or secretion.

33 er frequency of live birth, as compared with gonadotropin but not as compared with clomiphene.

34 rmatids in the SLL, showing that circulating gonadotropin can reach the intratubular compartment.

35 After treatment with gonadotropin, clomiphene, or letrozole, clinical pregnan

36 otein substrates by agalacto human chorionic gonadotropin, comprising 29 nM for beta4GalNAc-T3 and 1.

37 arboring SOX2 mutations are at high risk for gonadotropin deficiency, which has important implication

38 ulature relationship maps indicated age- and gonadotropin-dependent increases in vasculature and bran

39 of the maternal gametic genome to exogenous gonadotropins during the endocrine stimulation of follic

40 g induction of ovulation by equine chorionic gonadotropin (eCG)/human CG (hCG) treatment and mating,

41 Pituitary gonadotropins follicle-stimulating hormone and luteinizi

42 gonadotropes to direct the synthesis of the gonadotropins, follicle-stimulating hormone (FSH), and l

43 ed a single injection of pregnant mare serum gonadotropin followed by progesterone or vehicle.

44 ible for generating the pulsatile release of gonadotropins from the pituitary gland are unknown.

45 on of Homer1 splicing by GnRH contributes to gonadotropin gene control.

46 tone 3 (H3S10p) as part of its regulation of gonadotropin gene expression, possibly involving cross-t

47 sults uncover differential regulation of the gonadotropin genes by Galpha(q/11) and by Galpha(s) and

48 atory loops in the differential induction of gonadotropin genes.

49 e time of GCT diagnosis, and human chorionic gonadotropin >/= 1,000 mIU/mL at initiation of HDCT.

50 fetoprotein (AFP) 2.0 ng/mL, human chorionic gonadotropin (hCG) 151,111 IU/L, and lactate dehydrogena

51 or two model cancer markers, human chorionic gonadotropin (hCG) and prostate specific antigen (PSA),

52 uteinizing hormone (hLH) and human chorionic gonadotropin (hCG) are human glycoprotein hormones each

53 Serum human chorionic gonadotropin (hCG) concentration was 200 mIU/ml; she was

54 astic disease include raised human chorionic gonadotropin (hCG) concentrations 6 months after uterine

55 d that the pregnancy hormone human chorionic gonadotropin (hCG) efficiently attracts human Tregs to t

56 Human chorionic gonadotropin (hCG) induces de novo synthesis of STAR, a

57 ompromised mice that secrete human chorionic gonadotropin (hCG) into the host mouse and include small

58 Human chorionic gonadotropin (hCG) is an important biomarker for the dia

59 Human chorionic gonadotropin (hCG) is necessary for the maintenance of e

60 not establish a single serum human chorionic gonadotropin (hCG) level that is diagnostic of ectopic p

61 affinity and specificity for human chorionic gonadotropin (hCG) protein.

62 The human chorionic gonadotropin (hCG) proteins constitute a diverse group o

63 Treatment of mice with human chorionic gonadotropin (hCG) resulted in increased circulating tes

64 In the present work human chorionic gonadotropin (hCG) was used as a model protein in a proo

65 ms of the human pregnancy hormone, chorionic gonadotropin (hCG), in the liver.

66 using the mediator molecule human chorionic gonadotropin (hCG), we interface the intracellular infor

67 before and after exposure to human chorionic gonadotropin (hCG).

68 for the efficient binding of Human Chorionic Gonadotropin (hCG).

69 ntracellular beta subunit of human chorionic gonadotropin (hCGbeta) and peroxisome proliferator activ

70 uction of aromatase (hCYP19A1) and chorionic gonadotropin (hCGbeta) expression.

71 DICER-dependent microRNAs are essential for gonadotropin homeostasis and fertility in mice.

72 The distal enhancer of the gonadotropin hormone alpha-subunit gene, chorionic gonad

73 immunorecognition assays of human chorionic gonadotropin hormone are well below the visual inspectio

74 circuit that evokes the pulsatile release of gonadotropin hormones (luteinizing hormone and follicle-

75 imulate synthesis and secretion of pituitary gonadotropin hormones and thereby mediate control of rep

76 Pituitary gonadotropin hormones are regulated by gonadotropin-rele

77 reproduction by regulating expression of the gonadotropin hormones, which are responsible for follicl

78 Release of gonadotropins in adult rodents is sex specific and depen

79 inbow trout and new data on the synthesis of gonadotropins in the pituitary.

80 ed pups, whereas administration of exogenous gonadotropins induced normal ovulation in these mice.

81 hat low doses of exogenous androgens enhance gonadotropin-induced ovulation in mice, further demonstr

82 Gonadotropin-inhibitory hormone (GnIH) acts as a negativ

83 Recently, gonadotropin-inhibitory hormone (GnIH) has emerged as an

84 -related peptides (RFRPs) are orthologous to gonadotropin-inhibitory hormone (GnIH) inhibiting gonado

85 Gonadotropin-inhibitory hormone (GnIH) is a neuropeptide

86 with testosterone or in vitro treatment with gonadotropin-inhibitory hormone (GnIH) reduced GnRH rele

87 s, gonadotropin-releasing hormone (GnRH) and gonadotropin-inhibitory hormone (GnIH), respectively, re

88 ted peptide-3 [RFRP-3; mammalian ortholog to gonadotropin-inhibitory hormone (GnIH)] in neuroendocrin

89 Inhibitory action of gonadotropin-inhibitory hormone on the signaling pathway

90 ind increased follicle numbers and decreased gonadotropin levels in aging FOXO3-transgenic mice compa

91 serum alpha-fetoprotein and human chorionic gonadotropin levels were normal.

92 ls and a compensatory elevation in levels of gonadotropin luteinizing hormone (LH).

93 NAP fusion reporter protein (human chorionic gonadotropin-O(6) -alkylguanine-DNA alkyltransferase) le

94 f 100 ng/mL or greater or of human chorionic gonadotropin of 5,000 U/L or greater (group B) were addi

95 ce and highly elevated serum human chorionic gonadotropin or alfa-fetoprotein concentrations that mat

96 not differ significantly from the rate with gonadotropin or clomiphene (42 of 192, 22%; P=0.15) or c

97 py for women with unexplained infertility is gonadotropin or clomiphene citrate.

98 lower than the rates with standard therapy (gonadotropin or clomiphene) (P=0.003) or gonadotropin al

99 oocyte maturation, whereas the daily dose of gonadotropin or the total number of metaphase II oocytes

100 Although basal gonadotropin production in these mice is relatively norm

101 produce extensive amounts of human chorionic gonadotropin, progesterone, placental growth factor, and

102 The luteinizing hormone chorionic gonadotropin receptor (LHCGR) is a G(s)-coupled GPCR tha

103 reated OSPW the abundances of transcripts of gonadotropin receptors and several enzymes of sex hormon

104 s unique to the ovary because in the testes, gonadotropin receptors are expressed in separate compart

105 W there was less abundance of transcripts of gonadotropin receptors in gonads, as well as less abunda

106 ypes and maintained responsiveness to KP and gonadotropins reflect Galphaq/11-independent GnRH secret

107 A helicase (GRTH/DDX25) is a testis-specific gonadotropin-regulated RNA helicase that is present in L

108 Gonadotropin-regulated testicular RNA helicase (GRTH/DDX

109 diol replacement after ovariectomy inhibited gonadotropin release to a similar extent in both groups.

110 tion, insufficient energy reserves attenuate gonadotropin release, leading to infertility.

111 otropin-inhibitory hormone (GnIH) inhibiting gonadotropin release.

112 direct hypophysiotropic effect on pituitary gonadotropin release.

113 In gonadotropin-release experiments, dendrimer 3 was shown

114 17beta-estradiol (E2) regulating release of gonadotropin releasing hormone (GnRH) and luteinizing ho

115 Release of gonadotropin releasing hormone (GnRH) from the medial ba

116 stone deacetylases (HDACs) in the control of gonadotropin releasing hormone (GnRH) neuronal developme

117 odulate the central driver of fertility: the gonadotropin releasing hormone (GnRH) neuronal system.

118 onadotropic hypogonadism to study effects of gonadotropin releasing hormone (GnRH) neurons on neurona

119 e associated with regulation by activins and gonadotropin releasing hormone (GnRH).

120 Gonadotropin releasing hormone antagonism successfully r

121 o the antral stage in both immature mice and gonadotropin releasing hormone antagonist-treated adult

122 differences in T through administration of a gonadotropin releasing hormone antagonist.

123 ive control, including direct innervation of gonadotropin releasing hormone neurons.

124 Pharmacoperones of the gonadotropin releasing hormone receptor (GnRHR) have eff

125 in-conjugated gold nanorods (gGNRs) promotes gonadotropin releasing hormone receptor-mediated interna

126 onsiveness to the endogenous natural ligand, gonadotropin releasing hormone, and an agonist that is s

127 Finasteride and gonadotropin-releasing analogues are associated with inc

128 acetate, a synthetic nonapeptide analogue of gonadotropin-releasing hormone (GnRH or LHRH), is the ac

129 Gonadotropin-releasing hormone (GnRH) acts at gonadotrop

130 Gonadotropin-releasing hormone (GnRH) agonists (e.g., tr

131 bjected to 26 ART treatment cycles receiving gonadotropin-releasing hormone (GnRH) agonists and recom

132 Studies of the use of gonadotropin-releasing hormone (GnRH) agonists to protec

133 n were on antiandrogens (AA), 26,959 were on gonadotropin-releasing hormone (GnRH) agonists, and 3,74

134 ective randomized trial evaluated the use of gonadotropin-releasing hormone (GnRH) analog triptorelin

135 Women treated with gonadotropin-releasing hormone (GnRH) analogs may develo

136 estrogens, finasteride, spironolactone, and gonadotropin-releasing hormone (GnRH) analogs.

137 Both gonadotropin-releasing hormone (GnRH) and activins, memb

138 ted with oocyte meiosis, TGF-beta signaling, gonadotropin-releasing hormone (GnRH) and epidermal grow

139 In vertebrates, gonadotropin-releasing hormone (GnRH) and gonadotropin-i

140 and controls the synthesis and/or release of gonadotropin-releasing hormone (GnRH) and gonadotropins.

141 Elagolix, an oral, nonpeptide, gonadotropin-releasing hormone (GnRH) antagonist, produc

142 Congenital gonadotropin-releasing hormone (GnRH) deficiency manifes

143 Individuals with an inherited deficiency in gonadotropin-releasing hormone (GnRH) have impaired sexu

144 that FOXO1 repressed basal transcription and gonadotropin-releasing hormone (GnRH) induction of both

145 amic median eminence (ME) where they release gonadotropin-releasing hormone (GnRH) into a specialized

146 Gonadotropin-releasing hormone (GnRH) is a key regulator

147 Gonadotropin-releasing hormone (GnRH) is a trophic pepti

148 Gonadotropin-releasing hormone (GnRH) is a trophic pepti

149 propriate tissue-specific gene expression of gonadotropin-releasing hormone (GnRH) is critical for pu

150 Gonadotropin-releasing hormone (GnRH) is secreted in bri

151 Gonadotropin-releasing hormone (GnRH) is secreted in bri

152 Gonadotropin-releasing hormone (GnRH) is the central reg

153 The gonadotropin-releasing hormone (GnRH) is the master regu

154 The gonadotropin-releasing hormone (GnRH) is the master regu

155 We have previously shown that gonadotropin-releasing hormone (GnRH) ligand-independent

156 The gonadotropin-releasing hormone (GnRH) neuron is the pivo

157 An attractive model is the gonadotropin-releasing hormone (GnRH) neuron system, mas

158 (RFRP-3) neurons have been shown to inhibit gonadotropin-releasing hormone (GnRH) neuronal activity

159 a profound influence on the activity of the gonadotropin-releasing hormone (GnRH) neuronal network c

160 Estradiol feedback regulates gonadotropin-releasing hormone (GnRH) neurons and subseq

161 Gonadotropin-releasing hormone (GnRH) neurons are born i

162 Gonadotropin-releasing hormone (GnRH) neurons are neuroe

163 Gonadotropin-releasing hormone (GnRH) neurons are the fi

164 Gonadotropin-releasing hormone (GnRH) neurons are the fi

165 The gonadotropin-releasing hormone (GnRH) neurons are the ke

166 sspeptin neurons act cooperatively to excite gonadotropin-releasing hormone (GnRH) neurons during pos

167 oductive functioning in mammals depends upon gonadotropin-releasing hormone (GnRH) neurons generating

168 in mice for controlling the activity of the gonadotropin-releasing hormone (GnRH) neurons in vivo to

169 Hypothalamic gonadotropin-releasing hormone (GnRH) neurons integrate

170 Gonadotropin-releasing hormone (GnRH) neurons originate

171 Gonadotropin-releasing hormone (GnRH) neurons produce th

172 Gonadotropin-releasing hormone (GnRH) neurons represent

173 onstructions and electrophysiology, that the gonadotropin-releasing hormone (GnRH) neurons that contr

174 rculating estradiol on proestrus to activate gonadotropin-releasing hormone (GnRH) neurons that, in t

175 nisms through which estradiol (E2) regulates gonadotropin-releasing hormone (GnRH) neurons to control

176 s how nerve terminal Ca(2+) is controlled in gonadotropin-releasing hormone (GnRH) neurons via action

177 peptin regulates reproduction by stimulating gonadotropin-releasing hormone (GnRH) neurons via the ki

178 icating abnormal steroid hormone feedback to gonadotropin-releasing hormone (GnRH) neurons.

179 , is enriched in cilia projecting from mouse gonadotropin-releasing hormone (GnRH) neurons.

180 o adulthood and depends upon the activity of gonadotropin-releasing hormone (GnRH) neurons.

181 ARH)), which drive the pulsatile activity of gonadotropin-releasing hormone (GnRH) neurons.

182 TEMENT The brain regulates fertility through gonadotropin-releasing hormone (GnRH) neurons.

183 f reproduction by acting on gonadotropes and gonadotropin-releasing hormone (GnRH) neurons.

184 Hypothalamic gonadotropin-releasing hormone (GnRH) plays a critical r

185 The gonadotropin-releasing hormone (GnRH) receptor is a drug

186 ion as a measure of information transfer via gonadotropin-releasing hormone (GnRH) receptors (GnRHR)

187 Pulsatile gonadotropin-releasing hormone (GnRH) release is critica

188 important in modulating the tonic pulsatile gonadotropin-releasing hormone (GnRH) release.

189 of reproduction by brain-secreted pulses of gonadotropin-releasing hormone (GnRH) represents a longs

190 Acquisition of a mature pattern of gonadotropin-releasing hormone (GnRH) secretion from the

191 Gonadotropin-releasing hormone (GnRH) secretion is regul

192 wn to be key components in the regulation of gonadotropin-releasing hormone (GnRH) secretion.

193 ic hypogonadism had a measurable response to gonadotropin-releasing hormone (GnRH) stimulation, sugge

194 lmann syndrome is an inherited deficiency of gonadotropin-releasing hormone (GnRH) that is characteri

195 revealed that IKK-beta and NF-kappaB inhibit gonadotropin-releasing hormone (GnRH) to mediate ageing-

196 itary gonadotropin hormones are regulated by gonadotropin-releasing hormone (GnRH) via MAPK signaling

197 duce ciliated neurons that express genes for gonadotropin-releasing hormone (GnRH), a G-protein-coupl

198 ) designed to express an antibody that binds gonadotropin-releasing hormone (GnRH), a master regulato

199 (MCH), thyrotropin-releasing hormone (TRH), gonadotropin-releasing hormone (GnRH), and kisspeptin.

200 sprouting in hypothalamic neurons secreting gonadotropin-releasing hormone (GnRH), the neuropeptide

201 Gonadotropin-releasing hormone (GnRH)-expressing neurons

202 lamic-pituitary-gonadal axis is dependent on gonadotropin-releasing hormone (GNRH)-stimulated synthes

203 ted as an increase in pulsatile secretion of gonadotropin-releasing hormone (GnRH).

204 ontrols reproduction in mammals by secreting gonadotropin-releasing hormone (GnRH).

205 neural network that drives the secretion of gonadotropin-releasing hormone (GnRH).

206 n mammals by secreting the 'master molecule' gonadotropin-releasing hormone (GnRH).

207 e reproduction requires pulsatile release of gonadotropin-releasing hormone (GnRH1) from the hypothal

208 In mammals, the receptor of the neuropeptide gonadotropin-releasing hormone (GnRHR) is unique among t

209 Gene expression of salmon gonadotropin-releasing hormone (sGnRH) and NR1 increased

210 to provide an episodic, excitatory drive to gonadotropin-releasing hormone 1 (GnRH) neurons, the syn

211 nit, which is transcriptionally regulated by gonadotropin-releasing hormone 1 (GNRH).

212 asic ovarian sex hormone fluctuation using a gonadotropin-releasing hormone agonist (GnRHa) and evalu

213 rted previously that after 1-year follow up, gonadotropin-releasing hormone agonist (GnRHa) did not p

214 by surgical castration and those who receive gonadotropin-releasing hormone agonist (GnRHa) therapy.

215 nditions: ovarian suppression induced by the gonadotropin-releasing hormone agonist leuprolide acetat

216 umulation increased after treatment with the gonadotropin-releasing hormone agonist leuprolide, which

217 Gonadotropin-releasing hormone agonist therapy is associ

218 over study, 12 healthy, young males received gonadotropin-releasing hormone agonist treatment 1 month

219 nditions: ovarian suppression induced by the gonadotropin-releasing hormone agonist, leuprolide aceta

220 al women, or premenopausal women receiving a gonadotropin-releasing hormone agonist, with estrogen re

221 The authors examined the effects of gonadotropin-releasing hormone agonist-induced ovarian s

222 Gonadotropin-releasing hormone agonists (GnRHa) have bee

223 herapies (OR, 4.04 [95% CI, 1.88-8.69]), and gonadotropin-releasing hormone agonists (OR, 1.93 [95% C

224 androgen deprivation therapy in the form of gonadotropin-releasing hormone agonists and newer antago

225 Gonadotropin-releasing hormone agonists given with chemo

226 y a combined androgen blockade consisting of gonadotropin-releasing hormone agonists with oral antian

227 er treatments such as surgical oophorectomy, gonadotropin-releasing hormone agonists, chemotherapy-in

228 rized into 1 of 6 mutually exclusive groups: gonadotropin-releasing hormone agonists, oral antiandrog

229 Gonadotropin-releasing hormone analogue triptorelin, bil

230 rel-releasing intrauterine system (LNG-IUD), gonadotropin-releasing hormone analogues (GnRHa; nafarel

231 Gonadotropin-releasing hormone analogues and laparoscopi

232 n suppression (castration via orchiectomy or gonadotropin-releasing hormone analogues) suppresses cir

233 Gonadotropin-releasing hormone analogues, danazol, and d

234 ikely to be mediated by reduced secretion of gonadotropin-releasing hormone and our results support t

235 le-stimulating hormone and administration of gonadotropin-releasing hormone antagonist to prevent pre

236 levels are increased somewhat by exposure to gonadotropin-releasing hormone but are not necessarily l

237 st gonadotropin responses, suggesting normal gonadotropin-releasing hormone neuronal and gonadotrope

238 lamic nucleus and send axonal projections to gonadotropin-releasing hormone neurons and regulate repr

239 In immortalized immature gonadotropin-releasing hormone neurons endogenously expr

240 Notably, TRPC1 suppressed the migration of gonadotropin-releasing hormone neurons without affecting

241 ive Ca(2+) imaging in the nerve terminals of gonadotropin-releasing hormone neurons.

242 targeting of tumor cells overexpressing the gonadotropin-releasing hormone receptor (GnRH-R).

243 icipate in partitioning mutant conformers of gonadotropin-releasing hormone receptor (GnRHR), a G pro

244 oter activity, highlighting a role of SET in gonadotropin-releasing hormone regulation of gene expres

245 from the Kiss1 promoter disrupted pulsatile gonadotropin-releasing hormone release, delayed puberty

246 ns, suggesting sensitization of the NKB-NK3R-gonadotropin-releasing hormone signaling pathway under m

247 androgen receptors, and by the hypothalamic gonadotropin-releasing hormone through activation of PKA

248 olic, and other factors control secretion of gonadotropin-releasing hormone to determine initiation o

249 uires the pulsatile, coordinated delivery of gonadotropin-releasing hormone to the pituitary and the

250 a neuropeptide homologous to the vertebrate gonadotropin-releasing hormone, is downregulated as work

251 nsgenic mice, and is released in response to gonadotropin-releasing hormone, similar to LH.

252 uctive function requires timely secretion of gonadotropin-releasing hormone, which is controlled by a

253 production was achieved with the use of the gonadotropin-releasing-hormone agonist triptorelin, ooph

254 In particular, gonadotropin replacing hormone (GnRH) is released in rhy

255 re;Lepr(lox/lox) female mice elicited robust gonadotropin responses, suggesting normal gonadotropin-r

256 ovarian follicle development and antagonizes gonadotropin responsiveness in granulosa cells by suppre

257 a key regulator of follicle development and gonadotropin responsiveness.

258 a key regulator of follicle development and gonadotropin responsiveness.-Abedini, A., Zamberlam, G.,

259 GnRH regulates the pituitary gonadotropin's follicle-stimulating hormone (FSH) and lu

260 othesis that a NKB antagonist would decrease gonadotropin secretion and inhibit folliculogenesis in h

261 Kisspeptin administration restored gonadotropin secretion and ovarian cyclicity, suggesting

262 low oxygen leads to reduced human chorionic gonadotropin secretion and STB-associated gene expressio

263 owever, only GATAD1 abundance increases when gonadotropin secretion is suppressed during late infancy

264 n mice induced anovulation, reduced GnRH and gonadotropin secretion, and diminished kisspeptin expres

265 ssociation with the pubertal reactivation of gonadotropin secretion.

266 egions critical for kisspeptin regulation of gonadotropin secretion.

267 in birds and mammals by inhibiting GnRH and gonadotropin secretion.

268 H) neurons generating a pulsatile pattern of gonadotropin secretion.

269 Serum gonadotropins, sex steroids, glucose, insulin, ghrelin,

270 NT5a functions as a physiologic inhibitor of gonadotropin signaling.

271 the carboxyterminal heptapeptide in LH is a gonadotropin-sorting determinant in vivo that directs pu

272 ular basis for this evolutionarily conserved gonadotropin-specific secretion pattern is not understoo

273 ation from ICSI by subjecting female mice to gonadotropin stimulation and then allowing them to produ

274 er IVF cycle included the usual cocktail for gonadotropin stimulation and was uncomplicated, except f

275 spermatogenic cells, providing evidence that gonadotropin stimulation contributes to the induction of

276 Our results suggest that gonadotropin stimulation protocols used in conjunction w

277 by both in vivo and in vitro human chorionic gonadotropin stimulation.

278 ed in GCs and its expression is modulated by gonadotropin stimuli.

279 nadotrope decodes GnRH frequency to regulate gonadotropin subunit genes differentially have been prop

280 Although transcriptional regulation of gonadotropin subunits has been well studied, the post-tr

281 died, the post-transcriptional regulation of gonadotropin subunits is not well understood.

282 and extragonadal androgen actions (including gonadotropin suppression and sexual behavior) were asses

283 Inhibition of the gonadotropin surge by phenobarbital treatment on D4:1100

284 signaling in the regulation of the ovulatory gonadotropin surge, a result at variance with experiment

285 e post-gonadectomy-induced rise in pituitary gonadotropin synthesis and secretion were both abolished

286 ate microRNAs in gonadal feedback control of gonadotropin synthesis and secretion.

287 ncy of GnRH pulses determines the pattern of gonadotropin synthesis.

288 ned as a negative serum beta-human chorionic gonadotropin test (beta-hCG < 6 IU/L) 17 days after egg

289 ute levels, effects the release of pituitary gonadotropins that drive steroid production in the gonad

290 ed commercial strip test for human chorionic gonadotropin, the hormone used to detect pregnancy, and

291 women with low responses to the traditional gonadotropin therapy.

292 negative feedback of exogenous T to suppress gonadotropins, thereby blocking the testicular T product

293 pendently or additively with human chorionic gonadotropin to enhance androstenedione secretion.

294 ene and letrozole groups were twins, whereas gonadotropin treatment resulted in 24 twin and 10 triple

295 collected from women between days 3 and 9 of gonadotropin treatment.

296 GRTH is transcriptionally upregulated by gonadotropin via cyclic AMP/androgen through androgen re

297 subtype and increase of beta-human chorionic gonadotropin were not significantly correlated with wors

298 nscripts of genes important for synthesis of gonadotropins were greater in brains from both male and

299 g hormone (GnRH) is the central regulator of gonadotropins, which stimulate gonadal function.

300 would maintain sexual function and suppress gonadotropins without simultaneously activating spermato