ライフサイエンスコーパス: gonadotropin-releasing hormone

1 l basis for a putative role of sbGnRH as the gonadotropin-releasing hormone.

2 gonadotropes, and this effect is enhanced by gonadotropin-releasing hormone.

3 to provide an episodic, excitatory drive to gonadotropin-releasing hormone 1 (GnRH) neurons, the syn

4 nit, which is transcriptionally regulated by gonadotropin-releasing hormone 1 (GNRH).

5 Gonadotropin-releasing hormone 1 (GnRH1) from the brain

6 thalamic neurons that synthesize and release gonadotropin-releasing hormone 1 (GnRH1), but the precis

7 Pulsatile secretion of gonadotropin-releasing hormone-1 (GnRH-1) is essential f

8 cystokinin (CCK) in the developing olfactory-gonadotropin-releasing hormone-1 (GnRH-1) neuroendocrine

9 Gonadotropin-releasing hormone-1 (GnRH-1) neurons migrat

10 asic ovarian sex hormone fluctuation using a gonadotropin-releasing hormone agonist (GnRHa) and evalu

11 rted previously that after 1-year follow up, gonadotropin-releasing hormone agonist (GnRHa) did not p

12 by surgical castration and those who receive gonadotropin-releasing hormone agonist (GnRHa) therapy.

13 nditions: ovarian suppression induced by the gonadotropin-releasing hormone agonist leuprolide acetat

14 We administered the gonadotropin-releasing hormone agonist leuprolide acetat

15 umulation increased after treatment with the gonadotropin-releasing hormone agonist leuprolide, which

16 Gonadotropin-releasing hormone agonist therapy is associ

17 over study, 12 healthy, young males received gonadotropin-releasing hormone agonist treatment 1 month

18 ceptive use or progesterone treatment, prior gonadotropin-releasing hormone agonist treatment, and pr

19 nditions: ovarian suppression induced by the gonadotropin-releasing hormone agonist, leuprolide aceta

20 al women, or premenopausal women receiving a gonadotropin-releasing hormone agonist, with estrogen re

21 nmetastatic disease, intervention group with gonadotropin-releasing hormone agonist-based ADT, contro

22 The authors examined the effects of gonadotropin-releasing hormone agonist-induced ovarian s

23 othersome side effects than treatment with a gonadotropin-releasing hormone agonist.

24 efore and after treatment with a long-acting gonadotropin-releasing hormone agonist.

25 production was achieved with the use of the gonadotropin-releasing-hormone agonist triptorelin, ooph

26 Gonadotropin-releasing hormone agonists (GnRHa) have bee

27 herapies (OR, 4.04 [95% CI, 1.88-8.69]), and gonadotropin-releasing hormone agonists (OR, 1.93 [95% C

28 androgen deprivation therapy in the form of gonadotropin-releasing hormone agonists and newer antago

29 Gonadotropin-releasing hormone agonists decrease bone mi

30 Gonadotropin-releasing hormone agonists given with chemo

31 y a combined androgen blockade consisting of gonadotropin-releasing hormone agonists with oral antian

32 er treatments such as surgical oophorectomy, gonadotropin-releasing hormone agonists, chemotherapy-in

33 rized into 1 of 6 mutually exclusive groups: gonadotropin-releasing hormone agonists, oral antiandrog

34 ess of depot leuprolide acetate, a synthetic gonadotropin-releasing hormone analog (GnRH-a), for prot

35 of glucocorticoid replacement, 19.2%; and of gonadotropin-releasing hormone analog therapy, 34.2%.

36 Ten women (23 +/- 1 years) received a gonadotropin releasing hormone analogue (GnRHa), leuprol

37 Gonadotropin-releasing hormone analogue triptorelin, bil

38 rel-releasing intrauterine system (LNG-IUD), gonadotropin-releasing hormone analogues (GnRHa; nafarel

39 Gonadotropin-releasing hormone analogues and laparoscopi

40 n suppression (castration via orchiectomy or gonadotropin-releasing hormone analogues) suppresses cir

41 Gonadotropin-releasing hormone analogues, danazol, and d

42 399R) had attenuated secretion of endogenous gonadotropin-releasing hormone and a left-shifted dose-r

43 siveness to both exogenous gonadotropins and gonadotropin-releasing hormone and had normal levels of

44 ikely to be mediated by reduced secretion of gonadotropin-releasing hormone and our results support t

45 Moreover, ERbeta2 is coexpressed in gonadotropin-releasing hormone and oxytocin neurons.

46 onsiveness to the endogenous natural ligand, gonadotropin releasing hormone, and an agonist that is s

47 Gonadotropin releasing hormone antagonism successfully r

48 o the antral stage in both immature mice and gonadotropin releasing hormone antagonist-treated adult

49 differences in T through administration of a gonadotropin releasing hormone antagonist.

50 (ii) a single subcutaneous injection of the gonadotropin-releasing hormone antagonist (GnRH-A), acyl

51 le-stimulating hormone and administration of gonadotropin-releasing hormone antagonist to prevent pre

52 of short-term castration (1 month) using the gonadotropin-releasing hormone antagonist, Acyline, vers

53 been found for other classes of drugs (e.g., gonadotropin-releasing hormone antagonists, neurokinin r

54 tostatin agonists, urotensin II antagonists, gonadotropin-releasing hormone antagonists, neurotensin

55 e and a left-shifted dose-response curve for gonadotropin-releasing hormone as compared with six pati

56 h, and adrenal maturation, is initiated when gonadotropin-releasing hormone begins to be secreted by

57 levels are increased somewhat by exposure to gonadotropin-releasing hormone but are not necessarily l

58 e likely to be factors in the suppression of gonadotropin releasing hormone (GnRH) activity.

59 17beta-estradiol (E2) regulating release of gonadotropin releasing hormone (GnRH) and luteinizing ho

60 tress index, CSI), women self administered a gonadotropin releasing hormone (GnRH) antagonist for 16

61 The complete Serine 8-type gonadotropin releasing hormone (GnRH) coding sequence wi

62 ntrolled in part by the pulsatile release of gonadotropin releasing hormone (GnRH) from the hypothala

63 Release of gonadotropin releasing hormone (GnRH) from the medial ba

64 stone deacetylases (HDACs) in the control of gonadotropin releasing hormone (GnRH) neuronal developme

65 odulate the central driver of fertility: the gonadotropin releasing hormone (GnRH) neuronal system.

66 gy balance and season, time the awakening of gonadotropin releasing hormone (GnRH) neurons at the ons

67 onadotropic hypogonadism to study effects of gonadotropin releasing hormone (GnRH) neurons on neurona

68 regions contain cell bodies and/or fibers of gonadotropin releasing hormone (GnRH) neurons, suggestin

69 tory receptor (ORNs), vomeronasal (VRNs) and gonadotropin releasing hormone (GnRH) neurons.

70 Gonadotropin releasing hormone (GnRH) plays an important

71 e associated with regulation by activins and gonadotropin releasing hormone (GnRH).

72 acetate, a synthetic nonapeptide analogue of gonadotropin-releasing hormone (GnRH or LHRH), is the ac

73 Gonadotropin-releasing hormone (GnRH) acts at gonadotrop

74 a potent and very long acting antagonist of gonadotropin-releasing hormone (GnRH) after subcutaneous

75 mone but apparently normal responsiveness to gonadotropin-releasing hormone (GnRH) agonist and antago

76 Androgen deprivation therapy with a gonadotropin-releasing hormone (GnRH) agonist is associa

77 bserved by MIS, which enhances the effect of gonadotropin-releasing hormone (GnRH) agonist on the FSH

78 Gonadotropin-releasing hormone (GnRH) agonists (e.g., tr

79 bjected to 26 ART treatment cycles receiving gonadotropin-releasing hormone (GnRH) agonists and recom

80 Gonadotropin-releasing hormone (GnRH) agonists are assoc

81 Gonadotropin-releasing hormone (GnRH) agonists decrease

82 Gonadotropin-releasing hormone (GnRH) agonists decrease

83 Studies of the use of gonadotropin-releasing hormone (GnRH) agonists to protec

84 n were on antiandrogens (AA), 26,959 were on gonadotropin-releasing hormone (GnRH) agonists, and 3,74

85 ective randomized trial evaluated the use of gonadotropin-releasing hormone (GnRH) analog triptorelin

86 Because gonadotropin-releasing hormone (GnRH) analogs constitute

87 Women treated with gonadotropin-releasing hormone (GnRH) analogs may develo

88 estrogens, finasteride, spironolactone, and gonadotropin-releasing hormone (GnRH) analogs.

89 Both gonadotropin-releasing hormone (GnRH) and activins, memb

90 ted with oocyte meiosis, TGF-beta signaling, gonadotropin-releasing hormone (GnRH) and epidermal grow

91 In vertebrates, gonadotropin-releasing hormone (GnRH) and gonadotropin-i

92 and controls the synthesis and/or release of gonadotropin-releasing hormone (GnRH) and gonadotropins.

93 ons are thought to regulate the secretion of gonadotropin-releasing hormone (GnRH) and thus coordinat

94 Elagolix, an oral, nonpeptide, gonadotropin-releasing hormone (GnRH) antagonist, produc

95 Based on the SAR from bicyclic gonadotropin-releasing hormone (GnRH) antagonists such a

96 Neurons that produce gonadotropin-releasing hormone (GnRH) are the final comm

97 amining the distribution of cells containing gonadotropin-releasing hormone (GnRH) as well as estroge

98 male rat preoptic area (POA), containing the gonadotropin-releasing hormone (GnRH) cell bodies, treat

99 ypothalamic-pituitary-ovarian axis, in which gonadotropin-releasing hormone (GnRH) controls the relea

100 Isolated gonadotropin-releasing hormone (GnRH) deficiency caused

101 othalamic amenorrhea is a reversible form of gonadotropin-releasing hormone (GnRH) deficiency commonl

102 Gonadotropin-releasing hormone (GnRH) deficiency in the

103 Congenital gonadotropin-releasing hormone (GnRH) deficiency manifes

104 Neurons that produce gonadotropin-releasing hormone (GnRH) drive the reproduc

105 In mice, gonadotropin-releasing hormone (GnRH) expression is limi

106 The pulsatile secretion of gonadotropin-releasing hormone (GnRH) from normal and im

107 been established that episodic secretion of gonadotropin-releasing hormone (GnRH) from the hypothala

108 Individuals with an inherited deficiency in gonadotropin-releasing hormone (GnRH) have impaired sexu

109 action on puberty by stimulating release of gonadotropin-releasing hormone (GnRH) in the hypothalamu

110 levated levels of transcripts for the salmon gonadotropin-releasing hormone (GnRH) in the male telenc

111 Administration of gonadotropin-releasing hormone (GnRH) induces a surge of

112 that FOXO1 repressed basal transcription and gonadotropin-releasing hormone (GnRH) induction of both

113 amic median eminence (ME) where they release gonadotropin-releasing hormone (GnRH) into a specialized

114 Hypothalamic gonadotropin-releasing hormone (GnRH) is a decapeptide t

115 Gonadotropin-releasing hormone (GnRH) is a key regulator

116 Gonadotropin-releasing hormone (GnRH) is a neuropeptide

117 Gonadotropin-releasing hormone (GnRH) is a trophic pepti

118 Gonadotropin-releasing hormone (GnRH) is a trophic pepti

119 propriate tissue-specific gene expression of gonadotropin-releasing hormone (GnRH) is critical for pu

120 In vertebrates, gonadotropin-releasing hormone (GnRH) is essential to th

121 Gonadotropin-releasing hormone (GnRH) is exclusively exp

122 Gonadotropin-releasing hormone (GnRH) is found in a wide

123 n of ERK1/2 by the hypothalamic neuropeptide gonadotropin-releasing hormone (GnRH) is relatively sust

124 Gonadotropin-releasing hormone (GnRH) is released in a p

125 Pulsatile release of gonadotropin-releasing hormone (GnRH) is required for fe

126 The hypothalamic neuropeptide gonadotropin-releasing hormone (GnRH) is secreted in a p

127 Gonadotropin-releasing hormone (GnRH) is secreted in bri

128 Gonadotropin-releasing hormone (GnRH) is secreted in bri

129 Gonadotropin-releasing hormone (GnRH) is the central reg

130 Gonadotropin-releasing hormone (GnRH) is the central reg

131 Gonadotropin-releasing hormone (GnRH) is the central reg

132 The gonadotropin-releasing hormone (GnRH) is the master regu

133 The gonadotropin-releasing hormone (GnRH) is the master regu

134 We have previously shown that gonadotropin-releasing hormone (GnRH) ligand-independent

135 The gonadotropin-releasing hormone (GnRH) neuron is the pivo

136 An attractive model is the gonadotropin-releasing hormone (GnRH) neuron system, mas

137 (RFRP-3) neurons have been shown to inhibit gonadotropin-releasing hormone (GnRH) neuronal activity

138 a profound influence on the activity of the gonadotropin-releasing hormone (GnRH) neuronal network c

139 Estradiol feedback regulates gonadotropin-releasing hormone (GnRH) neurons and subseq

140 Gonadotropin-releasing hormone (GnRH) neurons are born i

141 Gonadotropin-releasing hormone (GnRH) neurons are critic

142 Gonadotropin-releasing hormone (GnRH) neurons are neuroe

143 Gonadotropin-releasing hormone (GnRH) neurons are neuroe

144 Gonadotropin-releasing hormone (GnRH) neurons are the ce

145 Gonadotropin-releasing hormone (GnRH) neurons are the ce

146 Gonadotropin-releasing hormone (GnRH) neurons are the fi

147 Gonadotropin-releasing hormone (GnRH) neurons are the fi

148 The gonadotropin-releasing hormone (GnRH) neurons are the ke

149 Gonadotropin-releasing hormone (GnRH) neurons are the pr

150 sspeptin neurons act cooperatively to excite gonadotropin-releasing hormone (GnRH) neurons during pos

151 Gonadotropin-releasing hormone (GnRH) neurons exhibit bu

152 Gonadotropin-releasing hormone (GnRH) neurons form the f

153 al axis is dependent on correct migration of gonadotropin-releasing hormone (GnRH) neurons from the n

154 oductive functioning in mammals depends upon gonadotropin-releasing hormone (GnRH) neurons generating

155 in mice for controlling the activity of the gonadotropin-releasing hormone (GnRH) neurons in vivo to

156 Hypothalamic gonadotropin-releasing hormone (GnRH) neurons integrate

157 ut from the suprachiasmatic nucleus (SCN) to gonadotropin-releasing hormone (GnRH) neurons is critica

158 Secretion from gonadotropin-releasing hormone (GnRH) neurons is necessa

159 Gonadotropin-releasing hormone (GnRH) neurons migrate fr

160 Gonadotropin-releasing hormone (GnRH) neurons originate

161 Gonadotropin-releasing hormone (GnRH) neurons produce th

162 Gonadotropin-releasing hormone (GnRH) neurons represent

163 n, rostral projections from the AVPV contact gonadotropin-releasing hormone (GnRH) neurons surroundin

164 onstructions and electrophysiology, that the gonadotropin-releasing hormone (GnRH) neurons that contr

165 rculating estradiol on proestrus to activate gonadotropin-releasing hormone (GnRH) neurons that, in t

166 estradiol (E2) regulates the activity of the gonadotropin-releasing hormone (GnRH) neurons through bo

167 nisms through which estradiol (E2) regulates gonadotropin-releasing hormone (GnRH) neurons to control

168 s how nerve terminal Ca(2+) is controlled in gonadotropin-releasing hormone (GnRH) neurons via action

169 peptin regulates reproduction by stimulating gonadotropin-releasing hormone (GnRH) neurons via the ki

170 Gonadotropin-releasing hormone (GnRH) neurons, a small n

171 mice lacking necdin have reduced numbers of gonadotropin-releasing hormone (GnRH) neurons, but the m

172 eactive fibres also abut the preoptic-septal gonadotropin-releasing hormone (GnRH) neurons, suggestin

173 siological processes including activation of gonadotropin-releasing hormone (GnRH) neurons, the neura

174 ne hormone secretion, especially that of the gonadotropin-releasing hormone (GnRH) neurons, via neura

175 arcuate nucleus (Arc) provide tonic drive to gonadotropin-releasing hormone (GnRH) neurons, which in

176 o a developmental defect in the migration of gonadotropin-releasing hormone (GnRH) neurons.

177 l pathways contain cell bodies and fibers of gonadotropin-releasing hormone (GnRH) neurons.

178 ARH)), which drive the pulsatile activity of gonadotropin-releasing hormone (GnRH) neurons.

179 TEMENT The brain regulates fertility through gonadotropin-releasing hormone (GnRH) neurons.

180 f reproduction by acting on gonadotropes and gonadotropin-releasing hormone (GnRH) neurons.

181 icating abnormal steroid hormone feedback to gonadotropin-releasing hormone (GnRH) neurons.

182 , is enriched in cilia projecting from mouse gonadotropin-releasing hormone (GnRH) neurons.

183 o adulthood and depends upon the activity of gonadotropin-releasing hormone (GnRH) neurons.

184 yclicity, including long-acting analogues of gonadotropin-releasing hormone (GnRH) or oestradiol (adm

185 Most cells are immunoreactive for either gonadotropin-releasing hormone (GnRH) or RF-amide-like p

186 Gonadotropin-releasing hormone (GnRH) plays a central ro

187 Hypothalamic gonadotropin-releasing hormone (GnRH) plays a critical r

188 ty of kisspeptin-10 to elicit the release of gonadotropin-releasing hormone (GnRH) precociously, and

189 genes are expressed under the control of the gonadotropin-releasing hormone (GnRH) promoter has made

190 Convergent evolution of the gonadotropin-releasing hormone (GnRH) receptor (GnRHR) p

191 Efforts to develop orally available gonadotropin-releasing hormone (GnRH) receptor antagonis

192 The gonadotropin-releasing hormone (GnRH) receptor is a drug

193 The human gonadotropin-releasing hormone (GnRH) receptor is evolut

194 Gonadotropin-releasing hormone (GnRH) receptor mutants f

195 o and caused cell death in cells bearing the gonadotropin-releasing hormone (GnRH) receptor, we could

196 ion as a measure of information transfer via gonadotropin-releasing hormone (GnRH) receptors (GnRHR)

197 Activation of gonadotropin-releasing hormone (GnRH) receptors inhibits

198 have specialized adaptations associated with gonadotropin-releasing hormone (GnRH) regulation to opti

199 NMDA and kisspeptins can stimulate gonadotropin-releasing hormone (GnRH) release after peri

200 Pulsatile gonadotropin-releasing hormone (GnRH) release is critica

201 A robust surge of gonadotropin-releasing hormone (GnRH) release triggers t

202 ing hormone, the latter reflecting increased gonadotropin-releasing hormone (GnRH) release.

203 important in modulating the tonic pulsatile gonadotropin-releasing hormone (GnRH) release.

204 of reproduction by brain-secreted pulses of gonadotropin-releasing hormone (GnRH) represents a longs

205 Neurons that produce gonadotropin-releasing hormone (GnRH) reside in the basa

206 otropic hypogonadism (IHH) due to defects of gonadotropin-releasing hormone (GnRH) secretion and/or a

207 Ovulation is initiated by a surge of gonadotropin-releasing hormone (GnRH) secretion by the b

208 Acquisition of a mature pattern of gonadotropin-releasing hormone (GnRH) secretion from the

209 Gonadotropin-releasing hormone (GnRH) secretion is regul

210 in the regulation of pubertal and adulthood gonadotropin-releasing hormone (GnRH) secretion, and mut

211 wn to be key components in the regulation of gonadotropin-releasing hormone (GnRH) secretion.

212 try regulating estrogen negative feedback on gonadotropin-releasing hormone (GnRH) secretion.

213 uctive development and for the regulation of gonadotropin-releasing hormone (GnRH) secretion.

214 plicated in the neuroendocrine regulation of gonadotropin-releasing hormone (GnRH) secretion.

215 ic hypogonadism had a measurable response to gonadotropin-releasing hormone (GnRH) stimulation, sugge

216 A robust gonadotropin-releasing hormone (GnRH) surge is a prerequ

217 lmann syndrome is an inherited deficiency of gonadotropin-releasing hormone (GnRH) that is characteri

218 sulting in a massive release of hypothalamic gonadotropin-releasing hormone (GnRH) that peaks within

219 rmal puberty promotes the central release of gonadotropin-releasing hormone (GnRH) that, in turn, lea

220 revealed that IKK-beta and NF-kappaB inhibit gonadotropin-releasing hormone (GnRH) to mediate ageing-

221 The binding of hypothalamic gonadotropin-releasing hormone (GnRH) to the pituitary G

222 itary gonadotropin hormones are regulated by gonadotropin-releasing hormone (GnRH) via MAPK signaling

223 duce ciliated neurons that express genes for gonadotropin-releasing hormone (GnRH), a G-protein-coupl

224 ) designed to express an antibody that binds gonadotropin-releasing hormone (GnRH), a master regulato

225 and negative modulators drives secretion of gonadotropin-releasing hormone (GnRH), a neuropeptide th

226 (MCH), thyrotropin-releasing hormone (TRH), gonadotropin-releasing hormone (GnRH), and kisspeptin.

227 brain controls fertility through release of gonadotropin-releasing hormone (GnRH), but the mechanism

228 he FSHbeta subunit is primarily regulated by gonadotropin-releasing hormone (GnRH), gonadal steroids,

229 under the control of pulsatile hypothalamic gonadotropin-releasing hormone (GnRH), is essential for

230 Pubertal onset, initiated by pulsatile gonadotropin-releasing hormone (GnRH), only occurs in a

231 pite of the fact that hypothalamic levels of gonadotropin-releasing hormone (GnRH), pituitary release

232 sprouting in hypothalamic neurons secreting gonadotropin-releasing hormone (GnRH), the neuropeptide

233 blished that hypothalamic neurons, including gonadotropin-releasing hormone (GnRH), VP, OT, beta-endo

234 The decapeptide gonadotropin-releasing hormone (GnRH), which regulates r

235 ndent upon the appropriate neurosecretion of gonadotropin-releasing hormone (GnRH), yet the endogenou

236 Gonadotropin-releasing hormone (GnRH)-expressing neurons

237 Mammalian reproduction requires gonadotropin-releasing hormone (GnRH)-mediated signaling

238 lamic-pituitary-gonadal axis is dependent on gonadotropin-releasing hormone (GNRH)-stimulated synthes

239 ontrols reproduction in mammals by secreting gonadotropin-releasing hormone (GnRH).

240 neural network that drives the secretion of gonadotropin-releasing hormone (GnRH).

241 or action of the master reproductive hormone gonadotropin-releasing hormone (GnRH).

242 etic disorders associated with deficiency of gonadotropin-releasing hormone (GnRH).

243 genital defect in the secretion or action of gonadotropin-releasing hormone (GnRH).

244 in the pituitary gonadotrope by hypothalamic gonadotropin-releasing hormone (GnRH).

245 n mammals by secreting the 'master molecule' gonadotropin-releasing hormone (GnRH).

246 e a class of neuroendocrine cells containing gonadotropin-releasing hormone (GnRH).

247 ted as an increase in pulsatile secretion of gonadotropin-releasing hormone (GnRH).

248 They secrete gonadotropin-releasing hormone (GnRH-1), communicate wit

249 rmone-releasing hormone (LHRH, also known as gonadotropin-releasing hormone [GnRH]), a key neurohormo

250 e reproduction requires pulsatile release of gonadotropin-releasing hormone (GnRH1) from the hypothal

251 In mammals, the receptor of the neuropeptide gonadotropin-releasing hormone (GnRHR) is unique among t

252 basis of selectivity of naturally occurring gonadotropin-releasing hormones (GnRHs) from different s

253 Gonadotropin-releasing hormone-I (GnRH-I) cells are loca

254 r fibers were observed in close proximity to gonadotropin-releasing hormone-I, dopamine, beta-endorph

255 ing hormone-I, dopamine, beta-endorphin, and gonadotropin-releasing hormone-II neurons in the preopti

256 n-releasing hormone and had normal levels of gonadotropin-releasing hormone in the hypothalamus.

257 a neuropeptide homologous to the vertebrate gonadotropin-releasing hormone, is downregulated as work

258 ty and infertility as a result of defects in gonadotropin-releasing hormone neuron development or fun

259 st gonadotropin responses, suggesting normal gonadotropin-releasing hormone neuronal and gonadotrope

260 ive control, including direct innervation of gonadotropin releasing hormone neurons.

261 se neurons also precede the emergence of the gonadotropin-releasing hormone neurons and ensheathing g

262 lamic nucleus and send axonal projections to gonadotropin-releasing hormone neurons and regulate repr

263 In immortalized immature gonadotropin-releasing hormone neurons endogenously expr

264 Notably, TRPC1 suppressed the migration of gonadotropin-releasing hormone neurons without affecting

265 Z formed appositions with orexin neurons and gonadotropin-releasing hormone neurons, two cell populat

266 ive Ca(2+) imaging in the nerve terminals of gonadotropin-releasing hormone neurons.

267 ed hypothalamic cells and immortalized GnRH (gonadotropin-releasing hormone) neurons (GT1-7 cells) tr

268 g hormone, growth hormone-releasing hormone, gonadotropin-releasing hormone, oxytocin, somatostatin,

269 g that this receptor is essential for normal gonadotropin-releasing hormone physiology and for pubert

270 cant relation between the number of doses of gonadotropin-releasing hormone received during the 12 mo

271 Pharmacoperones of the gonadotropin releasing hormone receptor (GnRHR) have eff

272 ere designed and synthesized as potent human gonadotropin releasing hormone receptor antagonists.

273 The gonadotropin releasing hormone receptor, because of its

274 in-conjugated gold nanorods (gGNRs) promotes gonadotropin releasing hormone receptor-mediated interna

275 eptide agonists and antagonists of the human gonadotropin-releasing hormone receptor (GnRH-R) are wid

276 eurons (GT1-7 cells), agonist binding to the gonadotropin-releasing hormone receptor (GnRH-R) causes

277 of a series thienopyrimidinediones with the gonadotropin-releasing hormone receptor (GnRH-R).

278 targeting of tumor cells overexpressing the gonadotropin-releasing hormone receptor (GnRH-R).

279 r reproductive traits, growth hormone (GHR), gonadotropin-releasing hormone receptor (GNRHR) and neur

280 Long-term suppression of the gonadotropin-releasing hormone receptor (GnRHR) is an im

281 The gonadotropin-releasing hormone receptor (GnRHR) is expre

282 on in pedigree 1 and a compound heterozygous gonadotropin-releasing hormone receptor (GNRHR) mutation

283 icipate in partitioning mutant conformers of gonadotropin-releasing hormone receptor (GnRHR), a G pro

284 mines bearing a butyric acid as potent human gonadotropin-releasing hormone receptor (hGnRH-R) antago

285 he C-terminal domains of either GluR1 or the gonadotropin-releasing hormone receptor permits efficien

286 y of several potent antagonists of the human gonadotropin-releasing hormone receptor.

287 regulation (reduced pituitary sensitivity to gonadotropin-releasing hormone), reduced conception rate

288 oter activity, highlighting a role of SET in gonadotropin-releasing hormone regulation of gene expres

289 from the Kiss1 promoter disrupted pulsatile gonadotropin-releasing hormone release, delayed puberty

290 Three prepro-gonadotropin-releasing hormones, seabream GnRH (sbGnRH),

291 Gene expression of salmon gonadotropin-releasing hormone (sGnRH) and NR1 increased

292 ns, suggesting sensitization of the NKB-NK3R-gonadotropin-releasing hormone signaling pathway under m

293 nsgenic mice, and is released in response to gonadotropin-releasing hormone, similar to LH.

294 lular: oxytocin, vasopressin; parvicellular: gonadotropin-releasing hormone, somatostatin, thyrotropi

295 these two factors is essential in mediating gonadotropin-releasing hormone stimulation of LHbeta pro

296 androgen receptors, and by the hypothalamic gonadotropin-releasing hormone through activation of PKA

297 olic, and other factors control secretion of gonadotropin-releasing hormone to determine initiation o

298 uires the pulsatile, coordinated delivery of gonadotropin-releasing hormone to the pituitary and the

299 Responsiveness to exogenous gonadotropin-releasing hormone was assessed in both the

300 uctive function requires timely secretion of gonadotropin-releasing hormone, which is controlled by a