ライフサイエンスコーパス: gonococcal infection

1 aneous bacterial peritonitis, and outpatient gonococcal infection.

2 immune responses in a mouse model of genital gonococcal infection.

3 sion of IgD(+)CD27(+) B cells in response to gonococcal infection.

4 oluble 17beta-estradiol to promote long-term gonococcal infection.

5 rations in host innate responses may enhance gonococcal infection.

6 ffects of H(2)O(2)-producing lactobacilli on gonococcal infection.

7 lain species-specific restriction of natural gonococcal infection.

8 mens from female subjects with uncomplicated gonococcal infection.

9 a key arm of innate immune defenses against gonococcal infection.

10 ge of nucleic acid amplification testing for gonococcal infection.

11 the human host, the only known reservoir for gonococcal infection.

12 BV), often without concomitant chlamydial or gonococcal infection.

13 erapeutic and prophylactic compounds against gonococcal infection.

14 play a role in tubal scarring in response to gonococcal infection.

15 implications for understanding asymptomatic gonococcal infection.

16 gnificantly to the reduction in the rates of gonococcal infection.

17 samples from individuals with no history of gonococcal infection.

18 ls of IL-6 and IL-8 cytokines in response to gonococcal infection.

19 e by LCR following therapy for uncomplicated gonococcal infection.

20 ated in the pathogenesis of various forms of gonococcal infection.

21 roteins was observed to occur in response to gonococcal infection.

22 ent, a physiologically relevant state during gonococcal infection.

23 (14.8% of the 608) had a total of 112 repeat gonococcal infections.

24 gical memory is not induced by uncomplicated gonococcal infections.

25 96.9 to 100% using LCA for the detection of gonococcal infections.

26 et for beta-lactam antibiotics used to treat gonococcal infections.

27 G, which for over 40 years was used to treat gonococcal infections.

28 rkers, a continuing problem for treatment of gonococcal infections.

29 arm of the innate immune system that combats gonococcal infections.

30 nd a collection of strains from disseminated gonococcal infections.

31 ent in the prevalence of both chlamydial and gonococcal infections.

32 ases ranging from urethritis to disseminated gonococcal infections.

33 Positive test result for chlamydial or gonococcal infections.

34 s to present a challenge to the treatment of gonococcal infections.

35 o = 0.50; 95% CI, 0.28 to 0.88; P = .02) and gonococcal infections (48 vs 54 participants, respective

36 For identification of gonococcal infections, all assays performed similarly (P

37 proven to be an efficient means of studying gonococcal infection and focusing vaccine development.

38 gulated tbpA and tbpB genes are expressed in gonococcal infection and that male subjects with mucosal

39 mportant tool for epidemiological studies of gonococcal infection and transmission.

40 a FSWs, we evaluated factors associated with gonococcal infection and with gonococcal antimicrobial r

41 and public health strategy for management of gonococcal infections and antimicrobial resistance.

42 her insights into the species specificity of gonococcal infections and proof-of-concept of a novel th

43 ere calculated separately for chlamydial and gonococcal infections and were stratified by assay and p

44 ulation aged 18 to 35 years has an untreated gonococcal infection, and 3.0% (SE, 0.8%) is estimated t

45 ialoglycoprotein receptor increase following gonococcal infection, and, like the primary cells, the i

46 Chlamydial and gonococcal infections are important causes of pelvic inf

47 ensitive for the detection of chlamydial and gonococcal infection at the rectal site than is culture.

48 ay have resulted in decreased chlamydial and gonococcal infections at the population level.

49 ral barriers to screening for chlamydial and gonococcal infections, but most test samples are obtaine

50 Gonococcal infections cause significant morbidity, parti

51 Symptomatic gonococcal infection, caused by the pathogen Neisseria g

52 Symptomatic gonococcal infection, caused exclusively by the human-sp

53 The gel group had 116 diagnosed gonococcal infections, chlamydial infections, or both fo

54 ined from female subjects with uncomplicated gonococcal infection corroborated our in vitro findings

55 ent of a C1q-dependent experimental model of gonococcal infection created an opportunity to evaluate

56 for simultaneous detection of chlamydial and gonococcal infections demonstrated superior sensitivity

57 specific vaginal bacteria and chlamydial or gonococcal infection detected by strand displacement ass

58 Disseminated gonococcal infection (DGI) and pelvic inflammatory disea

59 the isolates from patients with disseminated gonococcal infection (DGI).

60 uce the serious complication of disseminated gonococcal infection (DGI).

61 Also, previous history of gonococcal infection did not increase antibody levels in

62 Gonococcal infection did not induce evident immunity to

63 Gonococcal infections do not elicit protective immunity,

64 n with the same serovar can occur, and prior gonococcal infection does not alter the Ig response upon

65 of participants with a laboratory-confirmed gonococcal infection during the 36-month follow-up.

66 nfection and that male subjects with mucosal gonococcal infections exhibit antibodies to these protei

67 tal model that may mimic the transmission of gonococcal infection from mother to the fetus during pre

68 ariable pathophysiology of meningococcal and gonococcal infections given that after an initial exposu

69 Screening for chlamydial and gonococcal infection has been strongly recommended for a

70 Here, we demonstrate that women with gonococcal infections have levels of sialidases present

71 the contributions of MisR and MisS (CpxA) to gonococcal infection in a murine model of cervicovaginal

72 d genes are expressed in vivo during mucosal gonococcal infection in men, which suggests that this or

73 are invaded by Neisseria gonorrhoeae during gonococcal infection in men.

74 not constitutive activation is required for gonococcal infection in mice.

75 erefore the potential for spread, (b) use of gonococcal infection in the animal model system to study

76 The RR for gonococcal infection in the gel group vs the condom grou

77 and Prevention STD treatment guidelines for gonococcal infections in adolescents and adults.

78 the evidence on screening for chlamydial and gonococcal infections in asymptomatic patients from stud

79 he prevalence of asymptomatic chlamydial and gonococcal infections in male and female military popula

80 specificity of polymerase chain reaction to gonococcal infections in men was 90.4%.

81 rnative treatment regimen, and management of gonococcal infections in persons with severe cephalospor

82 e regarding the prevalence of chlamydial and gonococcal infections in the general young adult populat

83 The estimated number of untreated gonococcal infections in the population (9241; SE, 2441)

84 ions in men; and 55.6%, 91.3%, and 84.9% for gonococcal infections in women.

85 n vivo would require the presence of a mixed gonococcal infection, in which an individual is concurre

86 Gonococcal infection induced a significant increase in s

87 Initiation of a gonococcal infection involves attachment of Neisseria go

88 The role of autolysis during gonococcal infection is not known, but possible advantag

89 During and after gonococcal infection, local and systemic antigonococcal

90 Although gonococcal infection of B cells produced small amounts o

91 mphenicol demonstrated that the Opa-mediated gonococcal infection of Chang cells required bacterial p

92 gested a role for the cervical epithelium in gonococcal infection of females; however, the nature of

93 that an intact MisRS system is required for gonococcal infection of mice.

94 ansferrin, nonhemoglobin iron sources during gonococcal infection of the female genital tract.

95 Gonococcal infection of the male urogenital tract has be

96 infection of females; however, the nature of gonococcal infection of the normal uterine cervix remain

97 irectly influences B cells, we observed that gonococcal infection prolonged viability of primary huma

98 Most gonococcal infections remain localized to the genital tr

99 Neisseria gonorrhoeae to cause disseminated gonococcal infection requires that such strains resist t

100 were phenotypically similar and responded to gonococcal infection similarly to primary cells.

101 Gonococcal infection studies and electron microscopy sho

102 For gonococcal infection, the overall prevalence per 100 pop

103 In a laboratory model of mixed gonococcal infections, the por type of one strain could

104 tudy, we examined the immune response during gonococcal infection to the individual transferrin bindi

105 In this work, a cell culture model of gonococcal infection was adapted to examine the effects

106 By multivariate analysis, gonococcal infection was associated with sex with a new

107 Specificity for gonococcal infections was >/= 99.8%.

108 ence of coinfection with both chlamydial and gonococcal infections was 0.030% (95% CI, 0.18%-0.49%).

109 regulated genes in vivo during uncomplicated gonococcal infection, we examined gene expression profil

110 A microarrays and a tissue culture model for gonococcal infection, we examined global changes in gene

111 To better understand the role of Opa in gonococcal infections, we created and characterized a de

112 inal pH, positive whiff test, and concurrent gonococcal infection were positively associated with TV

113 No gonococcal infections were detected.

114 es were evaluated, and 281 chlamydial and 69 gonococcal infections were identified.

115 However, among men with urethral gonococcal infections who attended one sexually transmit

116 The unsialylated gonococcal infections, with a median incubation time of

117 genic diversity and allow the persistence of gonococcal infection within the human population.