ライフサイエンスコーパス: gonococcus

1 may also play a role in the survival of the gonococcus.

2 etence, and epithelial cell adherence of the gonococcus.

3 actually influence DNA transformation of the gonococcus.

4 porA in some meningococci was absent in the gonococcus.

5 i functioned for the counterselection in the gonococcus, albeit with low efficiency.

6 the recombination-dependent processes of the gonococcus and further define the pathways of homologous

7 orA was acquired by a common ancestor of the gonococcus and meningococcus but inactivated in the gono

8 e fusion with pedestal formation between the gonococcus and the epithelial cell, gonococci within vac

9 irst major discriminating factor between the gonococcus and the other Neisseria and carries genes for

10 biotic resistance, or when such pathogens as gonococcus are being considered.

11 We show that actin footprints are induced in gonococcus-challenged primary urethral epithelium.

12 ercome several subversive mechanisms whereby gonococcus evades complement, including binding to C4b-b

13 ABC transport systems are functional in the gonococcus for the acquisition of iron.

14 DE and FarAB-MtrE efflux systems protect the gonococcus from hydrophobic antimicrobial substances tha

15 redundant defense mechanisms may protect the gonococcus from reactive oxygen species during infection

16 The pilus of Neisseria gonorrhoeae (the gonococcus Gc), the causative agent of gonorrhoea, promo

17 chnique to create tandem duplications in the gonococcus (Gc) genome.

18 stigated the effect of Neisseria gonorrheae (gonococcus [GC]) exposure on HIV replication in primary

19 The Neisseria gonorrhoeae (the gonococcus [Gc]) opacity-associated (Opa) proteins media

20 Neisseria gonorrhoeae (gonococcus [GC]), is highly adapted to the human host, t

21 oaches for confirming Neisseria gonorrhoeae (gonococcus [GC])-positive nucleic acid amplification tes

22 block of the pilus of Neisseria gonorrhoeae (gonococcus [GC]).

23 Phagocytosis of Opa+ Neisseria gonorrhoeae (gonococcus, GC) by neutrophils is in part dependent on t

24 enic variation in Neisseria gonorrhoeae (the gonococcus, Gc) is regulated by iron availability.

25 Neisseria gonorrhoeae (the gonococcus, Gc) is the causative agent of gonorrhoea, an

26 Neisseria gonorrhoeae (the gonococcus, Gc) triggers a potent inflammatory response

27 mutate the genome of Neisseria gonorrhoeae (gonococcus; Gc).

28 urvive this interaction, suggesting that the gonococcus has evolved many defenses against PMN killing

29 in the level of inhibitory activity between gonococcus-infected and noninfected patients in either c

30 were not elevated in genital secretions from gonococcus-infected compared with uninfected patients.

31 ristic of IgA1 protease activity was seen in gonococcus-infected or control patients.

32 The level of serum IL-6 was higher in gonococcus-infected than in uninfected patients at recru

33 The subject with sialylated gonococcus infection had an extended incubation period,

34 n response to Neisseria gonorrhoeae (GC, for gonococcus) infection and that conditioned medium from G

35 This raises the question of how the gonococcus infects men if N-acetyllactosamine residues a

36 data indicate that PLD augments CR3-mediated gonococcus invasion of and survival within cervical epit

37 Despite the fact that the gonococcus is not normally exposed to UV irradiation or

38 Neisseria gonorrhoeae (the gonococcus) is an obligate human pathogen and the causat

39 Neisseria gonorrhoeae (the gonococcus) is the causative agent of the sexually trans

40 specific pathogen Neisseria gonorrhoeae (the gonococcus), is characterized by the influx of polymorph

41 chocystis sp. and, like those organisms, the gonococcus lacks a norC homolog.

42 tion to the genitourinary niche, making them gonococcus-like and distinguishing them from other N. me

43 e data suggest that biofilm formation by the gonococcus may represent a response that is linked to th

44 cus and meningococcus but inactivated in the gonococcus on speciation.

45 ed to share genetic characteristics with the gonococcus, raising the question of the extent to which

46 aim is to articulate a shared agenda across gonococcus-related fields from microbiology to epidemiol

47 o slow the increasing incidence of resistant gonococcus require insight into the factors that contrib

48 Lack of PorA expression in the gonococcus resulted from mutations in the promoter regio

49 identified a locus that, when mutated in the gonococcus, results in a significant increase in toxicit

50 tance to reinfection, with the generation of gonococcus-specific circulating immunoglobulin G and vag

51 he human pathogen Neisseria gonorrhoeae (the gonococcus), the pathways of homologous recombination ar

52 in the recombination-based processes of the gonococcus, these genes were inactivated in the N. gonor

53 nd benefit associated with resistance of the gonococcus to a clinically relevant antibiotic.

54 subsequently mutagenized and returned to the gonococcus to generate a Kat- strain that was phenotypic

55 roposed to function in the adaptation of the gonococcus to host environments.

56 res a regulatory response on the part of the gonococcus to respond to this significant change in envi

57 nt of gonorrhoea, promotes attachment of the gonococcus to the host epithelium and is essential for t

58 endent ABC transport system that enables the gonococcus to transport iron into the cell subsequent to

59 unique features of ihf transcription in the gonococcus which questions whether certain aspects if ih

60 ignificant effect in determining whether the gonococcus will be able to successfully establish an inf

61 ous work has identified several genes in the gonococcus with significant identity to the pilin invers