ライフサイエンスコーパス: gonorrhoeae

1 /mL for C. trachomatis and 1500CFU/mL for N. gonorrhoeae.

2 as a multifunctional virulence factor for N. gonorrhoeae.

3 tion that is insufficient to clear Neisseria gonorrhoeae.

4 olithromycin against clinical isolates of N. gonorrhoeae.

5 stantial in vitro activity against Neisseria gonorrhoeae.

6 as examined the effects of CrgA in Neisseria gonorrhoeae.

7 and establishment of infection by Neisseria gonorrhoeae.

8 ) from the obligate human pathogen Neisseria gonorrhoeae.

9 suspected cephalosporin-resistant Neisseria gonorrhoeae.

10 mbrane topology, and variation of TraG in N. gonorrhoeae.

11 were >98% for C. trachomatis and 100% for N. gonorrhoeae.

12 iscrimination between N. meningitidis and N. gonorrhoeae.

13 r model of the type IVa pilin from Neisseria gonorrhoeae.

14 merging threat of antimicrobial-resistant N. gonorrhoeae.

15 ted by antimicrobial resistance in Neisseria gonorrhoeae.

16 Aptima assays for the detection of Neisseria gonorrhoeae.

17 of Gram-negative bacteria such as Neisseria gonorrhoeae.

18 m (T4SS) that is found in most strains of N. gonorrhoeae.

19 to protect against serogroup B or against N. gonorrhoeae.

20 ening against highly resistant strains of N. gonorrhoeae.

21 exposure and decreased susceptibility of N. gonorrhoeae.

22 exposure and decreased susceptibility of N. gonorrhoeae.

23 mmon sexually transmitted pathogen Neisseria gonorrhoeae.

24 ections: Chlamydia trachomatis and Neisseria gonorrhoeae.

25 o modulate biofilm accumulation by Neisseria gonorrhoeae.

26 bal spread of antibiotic-resistant Neisseria gonorrhoeae.

27 %; P = 0.005), C. trachomatis (6.2%), and N. gonorrhoeae (1.4%).

28 Biochemical assays using N. gonorrhoeae 1291 wild type and isogenic mutant strains s

29 s isolate containing one copy of a Neisseria gonorrhoeae 16S rRNA gene is described herein.

30 d here expresses both N. meningitidis and N. gonorrhoeae 16S rRNA genes, as shown by positive FISH si

31 ated with the analysis of clinical Neisseria gonorrhoeae 16S rRNA to show its potential value in real

32 lows: syphilis, 3.5% (1.8%-5.2%; n = 851), N gonorrhoeae, 2.7% (1.7%-3.7%; n = 73), C trachomatis, 6.

33 trachomatis, 21 (4.2%) were positive for N. gonorrhoeae, 26 (5.2%) were positive for T. vaginalis, a

34 r than those for Chlamydia trachomatis or N. gonorrhoeae (27.6 and 25.9 years, respectively; P < 0.00

35 (3.9%-5.1%; n = 8346 positive diagnoses), N gonorrhoeae, 3.7% (2.8%-4.6%; n = 626), C trachomatis, 6

36 We previously demonstrated that Neisseria gonorrhoeae, a Gram-negative pathogen responsible for th

37 sma genitalium was associated with Neisseria gonorrhoeae (adjusted OR, 1.84; 95% CI, 1.13-2.98) and w

38 spatus, Gardnerella vaginalis, and Neisseria gonorrhoeae All vaginal microbiota and N. gonorrhoeae ef

39 lis detection rate compared with 2.1% for N. gonorrhoeae and 1.6% for C. trachomatis.

40 technology to examine the epidemiology of N. gonorrhoeae and associated AMR in the Australian populat

41 c acid amplification testing would detect N. gonorrhoeae and C. trachomatis (or T. vaginalis if utili

42 Neisseria gonorrhoeae and Chlamydia trachomatis are well-documente

43 inal swabs; NAATs for detection of Neisseria gonorrhoeae and Chlamydia trachomatis from pharyngeal an

44 cation (TMA) enhances detection of Neisseria gonorrhoeae and Chlamydia trachomatis from rectal and ph

45 lection of urethral discharge to diagnose N. gonorrhoeae and Chlamydia trachomatis infection in certa

46 hral/first-void urine samples) for Neisseria gonorrhoeae and Chlamydia trachomatis using nucleic acid

47 -based tool for genomic contact tracing of N gonorrhoeae and demonstrate local, national, and interna

48 lex, from two species of bacteria: Neisseria gonorrhoeae and Haemophilus ducreyi.

49 st and unveils the molecular link between N. gonorrhoeae and HIV-1.

50 N. gonorrhoeae and human papillomavirus 18 (HPV18) infectio

51 Neisseria gonorrhoeae and Neisseria meningitidis are closely relat

52 Neisseria includes two pathogens, Neisseria gonorrhoeae and Neisseria meningitidis, and at least 13

53 also a capsular polysaccharide for Neisseria gonorrhoeae and nontypable Hemophilus influenzae, and pr

54 However, only infection with pathogenic N. gonorrhoeae and not infection with the other bacteria te

55 Quinolone-resistant N. gonorrhoeae and reduced cefixime susceptibility appear a

56 n males was significantly higher than the N. gonorrhoeae and T. vaginalis infection rates.

57 n intense inflammatory response by Neisseria gonorrhoeae and the persistence of this pathogen in the

58 (ii) swabs seeded with C. trachomatis and N. gonorrhoeae and then placed in transport medium were tes

59 me (OR, 0.24; 95% 0.09, 0.66), and Neisseria gonorrhoeae and/or Chlamydia trachomatis had 92% lower o

60 from the bifunctional enzyme PglB (Neisseria gonorrhoeae) and the full-length acetyltransferase WeeI

61 ples spiked with either C. trachomatis or N. gonorrhoeae, and also containing both bacteria.

62 such as Salmonella spp, Campylobacter spp, N gonorrhoeae, and H pylori.

63 ecific human papillomavirus (HPV), Neisseria gonorrhoeae, and HIV antibody.

64 tube epithelial cells were infected with N. gonorrhoeae, and MMP patterns were examined.

65 on incident Chlamydia trachomatis, Neisseria gonorrhoeae, and Mycoplasma genitalium infection.

66 coccus aureus, Bacillus anthracis, Neisseria gonorrhoeae, and Neisseria meningitidis.

67 nd to have antimicrobial activity against N. gonorrhoeae, and Nuc expression enhanced N. gonorrhoeae

68 rom symptomatic patients were cultured for N gonorrhoeae, and resulting isolates were whole-genome se

69 olone-resistant Campylobacter spp, Neisseria gonorrhoeae, and Salmonella typhi were included in the h

70 on of 2,408 specimens for C. trachomatis, N. gonorrhoeae, and T. vaginalis TMA screening.

71 values for M. genitalium, C. trachomatis, N. gonorrhoeae, and T. vaginalis were 100, 70, 67, and 20%,

72 genitalium, Chlamydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis in liquid-based c

73 ium and for Chlamydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis Sequencing was us

74 ecimens for Chlamydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis via commercial tr

75 ocused experimental data from E. coli and N. gonorrhoeae, and we validate our system's ability to ide

76 Anti-E. coli or anti-N. gonorrhoeae antibodies were conjugated to submicron part

77 In this study, we predicted Neisseria gonorrhoeae Ape1a to be an SGNH hydrolase with an adopte

78 e hypothesis that multiple pili of Neisseria gonorrhoeae are coordinated through a tug-of-war.

79 Inflammatory responses to N. gonorrhoeae are generated by host innate immune recognit

80 Infections by Neisseria gonorrhoeae are increasingly common, are often caused by

81 ncy, and Chlamydia trachomatis and Neisseria gonorrhoeae are recognized microbial causes.

82 lycan O-acetyltransferase using PatB from N. gonorrhoeae as the model system.

83 bicans, Gardnerella vaginalis, and Neisseria gonorrhoeae, as well as to toxic shock syndrome toxin-1.

84 r the qualitative RealTime C. trachomatis/N. gonorrhoeae assay, the overall agreements between the tw

85 %) participants had positive cultures for N. gonorrhoeae at the time of enrollment: 24 of the 28 pers

86 blish quality control (QC) ranges for the N. gonorrhoeae ATCC 49226 control strain for MIC agar dilut

87 utrophils released NETs after exposure to N. gonorrhoeae, but NET integrity declined over time with N

88 o detect Chlamydia trachomatis and Neisseria gonorrhoeae, but no commercial tests are cleared by the

89 o detect Chlamydia trachomatis and Neisseria gonorrhoeae, but no commercial tests are cleared by the

90 ck complement-dependent killing of Neisseria gonorrhoeae by otherwise bactericidal Abs.

91 ated by host innate immune recognition of N. gonorrhoeae by several innate immune signaling pathways,

92 cal urine and swab specimens positive for N. gonorrhoeae by the Cobas assay, 71% could be genotyped.

93 mblies, we analyzed 25 isolates of Neisseria gonorrhoeae by using a high-resolution single nucleotide

94 Neisseria gonorrhoeae causes gonorrhea, a sexually transmitted inf

95 A antibodies that bound to the surface of N. gonorrhoeae cells, as shown by indirect fluorescent anal

96 eic acid amplification testing for Neisseria gonorrhoeae, Chlamydia trachomatis, and Trichomonas vagi

97 All participants were tested for Neisseria gonorrhoeae, Chlamydia trachomatis, Treponema pallidum (

98 e sequenced the genomes of 236 isolates of N gonorrhoeae collected by the Centers for Disease Control

99 The cohort comprised N. gonorrhoeae culture-positive individuals identified betw

100 There were 291 N. gonorrhoeae culture-positive individuals identified.

101 N. gonorrhoeae cultures were genotyped using multiple-locus

102 nd conditioned medium from Nuc-containing N. gonorrhoeae degraded human neutrophil DNA and NETs.

103 This N. gonorrhoeae-derived HMP activates CD4(+) T cells to invo

104 etry analysis of extensively fractionated N. gonorrhoeae-derived supernatants revealed that the LTR-i

105 In considering whether N. gonorrhoeae directly influences B cells, we observed tha

106 study examined the persistence of Neisseria gonorrhoeae DNA following treatment for pharyngeal and r

107 For pharyngeal gonorrhea, positivity of N. gonorrhoeae DNA on both PCR assays was present at days 7

108 ia gonorrhoeae All vaginal microbiota and N. gonorrhoeae efficiently colonized the 3-D surface, local

109 The human-restricted pathogen Neisseria gonorrhoeae encodes a single N-acetylmuramyl-l-alanine a

110 mice to study mechanisms by which Neisseria gonorrhoeae evades host-derived antimicrobial factors an

111 We demonstrate for the first time that N. gonorrhoeae exploits this host strategy in a novel defen

112 homologous NgoAVII RM system from Neisseria gonorrhoeae FA1090 are composed of three genes: a DNA me

113 Three strains, including N. gonorrhoeae FA1090, an nrrF deletion mutant, and a compl

114 These data demonstrate that N. gonorrhoeae filamentous phage can induce antibodies with

115 A for the detection of C. trachomatis and N. gonorrhoeae from rectal swab samples.

116 The sensitivity of Xpert for N. gonorrhoeae from rectal swabs was 100% (95% CI, 88 to 10

117 evaluating UTI (E. coli) and STD (Neisseria gonorrhoeae) from human urine samples.

118 redicted, whereas the hairpin from Neisseria gonorrhoeae functions as an intrinsic transcription term

119 -Acetylpeptidoglycan esterase from Neisseria gonorrhoeae functions to release O-acetyl groups from th

120 ave receptive anal intercourse for Neisseria gonorrhoeae (GC) and Chlamydia trachomatis (CT).

121 dia trachomatis (CT) and/or rectal Neisseria gonorrhoeae (GC).

122 of the input plasmid pools and the output N. gonorrhoeae genomic DNA pools identified mutations prese

123 usters of patients infected with specific N. gonorrhoeae genotypes were related to various epidemiolo

124 amydia trachomatis (chlamydia) and Neisseria gonorrhoeae (gonorrhea) infections in women.

125 ilized the enzyme-DNA cleaved complex for N. gonorrhoeae gyrase and topoisomerase IV.

126 d the stabilization of cleaved complex by N. gonorrhoeae gyrase increased in a fluoroquinolone-resist

127 The lipooligosaccharide of N. gonorrhoeae has a hexa-acylated lipid A.

128 However, induction of cell death by N. gonorrhoeae has also been reported in other cell types.

129 Quinolone-resistant N. gonorrhoeae has arisen multiple times, with extensive cl

130 Neisseria gonorrhoeae has developed resistance to each of the anti

131 sRNA in Neisseria meningitidis and Neisseria gonorrhoeae, has been shown in the meningococcus to cont

132 yses of Kingella denitrificans and Neisseria gonorrhoeae HpuA mutants, although validating the intera

133 , as well as in the human pathogen Neisseria gonorrhoeae identified HemN as a copper toxicity target

134 autotransporter beta-domain of the Neisseria gonorrhoeae IgA protease precursor (IgAbeta), which succ

135 s critical for the growth and survival of N. gonorrhoeae in human cells.

136 and cefixime-resistant strains of Neisseria gonorrhoeae in MSM in England, which was applied to data

137 -of-care test for the detection of Neisseria gonorrhoeae in patients attending a public health clinic

138 ssay (Xpert) to detect C. trachomatis and N. gonorrhoeae in rectal and pharyngeal samples from 224 me

139 ction of Chlamydia trachomatis and Neisseria gonorrhoeae in rectal swabs with regulatory approval.

140 This explains why the probes for N. gonorrhoeae in the Gen-Probe Aptima assays cross-react w

141 tion and alters the infection dynamics of N. gonorrhoeae in vitro Furthermore, miR-718 regulates the

142 o pathogenic species (N. meningitidis and N. gonorrhoeae) in addition to a number of commensal specie

143 has potent bactericidal activity against N. gonorrhoeae, including multidrug-resistant strains and k

144 ndertaken to reveal which component(s) of N. gonorrhoeae induce HIV-1 expression in CD4(+) T lymphocy

145 n populations at high risk of contracting N. gonorrhoeae induces an increase in MIC and may result in

146 n populations at high risk of contracting N. gonorrhoeae induces an increase in MIC, and may result i

147 We now show that msbB-deficient N. gonorrhoeae induces less inflammatory signaling in human

148 In this study, we confirmed that N. gonorrhoeae induces production of cIAP2 in human cervica

149 iated with bacterial burden during Neisseria gonorrhoeae infection and alters the infection dynamics

150 are Gram stain testing to diagnose Neisseria gonorrhoeae infection and nongonococcal urethritis (NGU)

151 nsequences of sexually transmitted Neisseria gonorrhoeae infection and probably involve an imbalance

152 ar, phenotypic, and epidemiologic data on N. gonorrhoeae infection could help develop a more complete

153 Treatment of Neisseria gonorrhoeae infection is empirical and based on populati

154 considered for point-of-care diagnosis of N. gonorrhoeae infection or NGU in men, meatal swabs should

155 is infection, 0.56 [95% CI, .19-1.67] for N. gonorrhoeae infection, and 0.66 [95% CI, .38-1.15] for M

156 iological investigation demonstrated that N. gonorrhoeae infections are dominated by relatively few s

157 spective cohort study of culture-positive N. gonorrhoeae infections at a single sexual health clinic

158 fication tests (NAATs) to diagnose Neisseria gonorrhoeae infections complicates the performance of a

159 , the sensitivity of PS testing to detect N. gonorrhoeae infections increased to 94%.

160 f clinical failure following treatment of N. gonorrhoeae infections with cefixime was relatively high

161 re the cornerstone of treatment of Neisseria gonorrhoeae infections, cefixime is the only oral antimi

162 ly more prevalent than C. trachomatis and N. gonorrhoeae infections, while the M. genitalium infectio

163 ous exposures for the treatment of Neisseria gonorrhoeae infections.

164 N. gonorrhoeae is a human-restricted pathogen that primaril

165 Multidrug-resistant Neisseria gonorrhoeae is a top threat to public health.

166 jor outer membrane porin (PorB) of Neisseria gonorrhoeae is an essential protein that mediates ion ex

167 erestingly, infection with msbB-deficient N. gonorrhoeae is associated with less localized inflammati

168 Antimicrobial resistance (AMR) by Neisseria gonorrhoeae is considered a serious global threat.

169 frequent infectious diseases, and Neisseria gonorrhoeae is emerging as resistant to most available a

170 ead of antimicrobial resistance in Neisseria gonorrhoeae is globally recognised.

171 G monomers by N. meningitidis relative to N. gonorrhoeae is partly due to ampG, since replacement of

172 re component of most lipopolysaccharides, N. gonorrhoeae is peculiar in that it effectively liberates

173 ally transmitted disease caused by Neisseria gonorrhoeae, is an important cause of morbidity worldwid

174 The MtrCDE multidrug pump, from Neisseria gonorrhoeae, is assembled from the inner and outer membr

175 All available N. gonorrhoeae isolates (n = 2452) received from Australian

176 losporin resistance-comprised 8.9% of all N. gonorrhoeae isolates and were primarily observed in male

177 In the United States, 19.2% of Neisseria gonorrhoeae isolates are resistant to ciprofloxacin.

178 n 1102 resistant and susceptible clinical N. gonorrhoeae isolates collected from 2000 to 2013 via the

179 Eight N. gonorrhoeae isolates collected from 7 patients on Oahu,

180 -passage-number clinical-specimen-derived N. gonorrhoeae isolates for Opa expression and assess their

181 We included N. gonorrhoeae isolates of patients visiting the Amsterdam

182 We included N. gonorrhoeae isolates of patients who visited the Amsterd

183 We identified a cluster of Neisseria gonorrhoeae isolates with high-level azithromycin resist

184 study transmission and track resistance in N gonorrhoeae isolates.

185 In N. gonorrhoeae, lack of TsaP results in the formation of me

186 N. gonorrhoeae liberates a soluble factor that potently ind

187 Neisseria gonorrhoeae lipooligosaccharides (LOSs) induce immunoglo

188 suggest that TLR4-mediated recognition of N. gonorrhoeae LOS plays an important role in the pathogene

189 receptor 4 signaling but does not affect N. gonorrhoeae-mediated activation of the inflammasome.

190 The mechanisms by which N. gonorrhoeae modulates cell death are not clear, although

191 We demonstrate that N. gonorrhoeae msbB is dispensable for initiating and maint

192 One man initially infected with N. gonorrhoeae multiantigen sequence type 2400 had type 424

193 dentify multilocus sequence types (MLST), N. gonorrhoeae multiantigen sequence types (NG-MAST), and m

194 We used N. gonorrhoeae multiantigen sequence typing to describe cha

195 es of resistance-associated mutations and N. gonorrhoeae multiantigen sequence typing, and challenges

196 currence of Chlamydia trachomatis, Neisseria gonorrhoeae, Mycoplasma genitalium, and Trichomonas vagi

197 Immune Abs against N. gonorrhoeae need to overcome several subversive mechanis

198 ion of Chlamydia trachomatis (CT), Neisseria gonorrhoeae (NG), and an internal control in the same re

199 k-based mathematical model of HIV, Neisseria gonorrhoeae (NG), and Chlamydia trachomatis (CT) transmi

200 The N. gonorrhoeae NG0969 open reading frame contains a gene (n

201 d extrusion transporter, NorM from Neisseria gonorrhoeae (NorM_NG).

202 In Neisseria gonorrhoeae, one of the first bacteria for which pan-res

203 boratory isolation or detection of Neisseria gonorrhoeae only from a clinical specimen, and controls

204 ual contact with either C. trachomatis or N. gonorrhoeae, or had symptoms of an STI.

205 fected with Chlamydia trachomatis, Neisseria gonorrhoeae, or herpes simplex virus type 2.

206 nce of any bacterial STI (C. trachomatis, N. gonorrhoeae, or M. genitalium infection) was lower in th

207 icantly for C. trachomatis (P = 0.774) or N. gonorrhoeae (P = 0.163).

208 pound library for potential inhibitors of N. gonorrhoeae PBP 2, and 32 compounds were identified that

209 membrane porin (PorB) expressed by Neisseria gonorrhoeae plays multiple roles during infection, in ad

210 hest C. trachomatis prevalence (9.2%) and N. gonorrhoeae prevalence (2.2%) were in women <30 years ol

211 en of >40 years, while C. trachomatis and N. gonorrhoeae prevalence is lowest in that age group.

212 Our recent studies have demonstrated that N. gonorrhoeae proactively suppresses host T-helper (Th) 1/

213 c Chlamydia trachomatis Q(x) (CTQ)/Neisseria gonorrhoeae Q(x) (GCQ), Hologic Aptima Combo 2 (AC2) and

214 Quinolone-resistant Neisseria gonorrhoeae (QRNG) arise from mutations in gyrA (interme

215 were immunized i.m. and s.c. with Neisseria gonorrhoeae recombinant porin B (Ng-rPorB) or i.n. with

216 We demonstrate that the N. gonorrhoeae RecQ helicase can bind and unwind the pilE G

217 D C-terminal" (HRDC) domain, whereas the N. gonorrhoeae RecQ helicase gene encodes three HRDC domain

218 Neisseria gonorrhoeae releases peptidoglycan (PG) fragments during

219 The closely related pathogen Neisseria gonorrhoeae releases PG fragments during normal growth.

220 er, a significant subset of patients with N. gonorrhoeae remain asymptomatic, without evidence of loc

221 we report that Nuc degrades NETs to help N. gonorrhoeae resist killing by neutrophils.

222 Deletion of amiC from N. gonorrhoeae results in severely impaired cell separation

223 s conducted with Escherichia coli, Neisseria gonorrhoeae, Salmonella enterica, Streptococcus pyogenes

224 o 89 years old) undergoing C. trachomatis/N. gonorrhoeae screening using the Aptima Combo 2 assay (Ge

225 During infection, Neisseria gonorrhoeae senses and responds to stress; such response

226 The N. gonorrhoeae Sequence Typing for Antimicrobial Resistance

227 meningitidis sequences, and 29 (24%) were N. gonorrhoeae sequences.

228 fragment release from N. meningitidis and N. gonorrhoeae showed that meningococci release less of the

229 ous studies have demonstrated that Neisseria gonorrhoeae sialylates the terminal N-acetyllactosamine

230 idis isolate described must have obtained N. gonorrhoeae-specific DNA through interspecies recombinat

231 We analyzed 265 urethral Neisseria gonorrhoeae specimens collected from symptomatic males a

232 Inhibition of cIAP2 in N. gonorrhoeae-stimulated epithelial cells resulted in incr

233 Collectively these results indicate that N. gonorrhoeae stimulation of human endocervical epithelial

234 acellular cIAP2 were detected early after N. gonorrhoeae stimulation, which was followed by a marked

235 mydia trachomatis AC2 also detects Neisseria gonorrhoeae Storage and temperature conditions may impac

236 We have generated a panel of mutants of N. gonorrhoeae strain FA1090 expressing a variety of mutant

237 , and recently we reported that wild-type N. gonorrhoeae strain FA1090 has a survival advantage relat

238 The increasing prevalence of N. gonorrhoeae strains exhibiting decreased susceptibility

239 The prevalence of mosaic PBP2 harboring N. gonorrhoeae strains highlight the ability for new N. gon

240 fically, cN supports nitrite reduction in N. gonorrhoeae strains lacking the cytochromes c5 and CcoP

241 N. gonorrhoeae strains that carry an inactivated msbB (also

242 eae strains highlight the ability for new N. gonorrhoeae strains to spread and become established acr

243 All Neisseria gonorrhoeae strains whose DNA sequences have been determ

244 dissemination of antimicrobial-resistant N. gonorrhoeae strains.

245 Neisseria gonorrhoeae successfully overcomes host strategies to li

246 xone, cefixime, and cefpodoxime in Neisseria gonorrhoeae surveillance.

247 gonorrhoeae, and Nuc expression enhanced N. gonorrhoeae survival in the presence of neutrophils that

248 genotypic assay for prediction of Neisseria gonorrhoeae susceptibility to ciprofloxacin.

249 The sensitivity and specificity of the N. gonorrhoeae test were 100% and 100% for AC2 and 76.2% an

250 samples; for Chlamydia trachomatis/Neisseria gonorrhoeae tests, the ability to run batches of 24 samp

251 ltidrug-resistant bacteria such as Neisseria gonorrhoeae The first structure of BamA, the central com

252 The Neisseria gonorrhoeae (the gonococcus [Gc]) opacity-associated (Op

253 ely by the human-specific pathogen Neisseria gonorrhoeae (the gonococcus), is characterized by the in

254 Neisseria gonorrhoeae (the gonococcus, Gc) triggers a potent infla

255 coccal type IV pilus (GC-T4P) from Neisseria gonorrhoeae, the causative agent of gonorrhea.

256 Neisseria gonorrhoeae, the causative agent of the sexually transmi

257 Neisseria gonorrhoeae, the causative agent of the sexually transmi

258 Neisseria gonorrhoeae, the cause of the sexually transmitted infec

259 he syndrome caused by its sister species, N. gonorrhoeae, the etiologic agent of gonorrhea.

260 A, the central component of BAM, was from N. gonorrhoeae, the etiological agent of the sexually trans

261 those of Chlamydia trachomatis and Neisseria gonorrhoeae, the mechanisms by which this pathogen elici

262 In the beta-proteobacterium Neisseria gonorrhoeae, the native PilQ secretin ring embedded in O

263 For N. gonorrhoeae, the sensitivity of PS testing (90%) was sig

264 ithromycin usage and resistance in Neisseria gonorrhoeae threatens current dual treatment.

265 to slow the spread of antibiotic-resistant N gonorrhoeae through augmentation of gonococcal outbreak

266 esting to determine the susceptibility of N. gonorrhoeae to ceftriaxone, cefixime, and cefpodoxime, a

267 or misS severely reduced the capacity of N. gonorrhoeae to colonize mice or maintain infection over

268 We propose that Nuc enables N. gonorrhoeae to escape trapping and killing by NETs durin

269 ding proteins TbpA and TbpB enable Neisseria gonorrhoeae to obtain iron from human transferrin.

270 d mutant gene pools were transformed into N. gonorrhoeae to select for alleles that maintained bacter

271 h control; however, the ability of Neisseria gonorrhoeae to successively develop resistance to differ

272 tudy, we used the type IV pilus of Neisseria gonorrhoeae to test whether variation of surface structu

273 cular typing was used to elucidate Neisseria gonorrhoeae transmission networks among men who have sex

274 Distinct N. gonorrhoeae transmission networks were present in a mixe

275 organisms (Chlamydia trachomatis, Neisseria gonorrhoeae, Trichomonas vaginalis) and the E6/E7 mRNA o

276 We characterized the inhibition of Neisseria gonorrhoeae type II topoisomerases gyrase and topoisomer

277 Neisseria gonorrhoeae uses a type IV secretion system (T4SS) to se

278 Neisseria gonorrhoeae uses a type IV secretion system (T4SS) to se

279 odel for the study of the pathogenesis of N. gonorrhoeae using a well-characterized DeltapilT mutant.

280 ated for Chlamydia trachomatis and Neisseria gonorrhoeae using NAATs and bacterial vaginosis using Gr

281 The strict human pathogen Neisseria gonorrhoeae utilizes homologous recombination to antigen

282 f the glutamine synthetase gene of Neisseria gonorrhoeae was able to tolerate urea concentrations of

283 N. gonorrhoeae was detected from 30 rectal and 40 pharyngea

284 achomatis was detected in 8.7% and Neisseria gonorrhoeae was detected in 6.6%.

285 crobial resistance determinants in Neisseria gonorrhoeae was developed and is publicly accessible.

286 assay for detection of C. trachomatis or N. gonorrhoeae was observed, although some mailed swabs too

287 Repeat testing for N. gonorrhoeae was undertaken using real-time polymerase ch

288 ependent velocity of DNA uptake by Neisseria gonorrhoeae We found that the DNA uptake velocity depend

289 and the mechanism of type IV secretion in N. gonorrhoeae, we examined the expression levels and local

290 t for the detection of C. trachomatis and N. gonorrhoeae were 86%, 99.2%, 92.5%, and 98.4% and 91.1%,

291 All patients with positive cultures for N. gonorrhoeae were cured at all sites of infection.

292 amples (undiluted) spiked with E. coli or N. gonorrhoeae were incubated for 5 min with 1% Tween 80.

293 fteen clinical and laboratory isolates of N. gonorrhoeae were tested following the Clinical Laborator

294 ests for Chlamydia trachomatis and Neisseria gonorrhoeae, which can provide results rapidly to guide

295 Neisseria gonorrhoeae, which causes gonorrhea, is particularly ade

296 the cervicovaginal microbiome can modify N. gonorrhoeae, which will enhance successful transmission

297 limit was 10 CFU/mL for both E. coli and N. gonorrhoeae, while commercially available gonorrhea rapi

298 s human immunodeficiency virus and Neisseria gonorrhoeae with concurrent T. vaginalis infection.

299 The emergence of Neisseria gonorrhoeae with decreased susceptibility to extended sp

300 For N. gonorrhoeae, Xpert had higher sensitivity than Aptima, b