ライフサイエンスコーパス: graft tolerance

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1 , which may contribute to the maintenance of graft tolerance.

2 blood of patients that spontaneously develop graft tolerance.

3 plus anti-CD40L antibodies does not disrupt graft tolerance.

4 ty could be used to promote the induction of graft tolerance.

5 peutically to synergize in the generation of graft tolerance.

6 instill long-lived allogeneic and xenogeneic graft tolerance.

7 sorders, including cancer, autoimmunity, and graft tolerance.

8 be in induction of a chimeric state to allow graft tolerance.

9 nological responses and aid the induction of graft tolerance.

10 of each treatment in achieving maximal skin graft tolerance.

11 us BMTx improves graft survival and promotes graft tolerance.

12 ale-reactive T-cell population and permanent graft tolerance.

13 latory functions that contribute to specific graft tolerance.

14 D8(+) T cells critical to the development of graft tolerance.

15 TGF)-beta within islets to achieve long-term graft tolerance.

16 ritical for the induction and maintenance of graft tolerance.

17 mopoietic cells to promote induction of skin graft tolerance across full MHC barriers.

18 or cell infusions can be used to induce skin graft tolerance across MHC barriers, accompanied by spec

19 venous injection of soluble beta-gal-induced graft tolerance and a lack of detectable beta-gal-specif

20 l of CD4 reconstitution correlated with skin graft tolerance and an absence of induced anti-donor xen

21 associated with optimal donor-specific skin graft tolerance and avoidance of the anti-donor xenoanti

22 Highly disparate xenogeneic pig skin graft tolerance and efficient repopulation of mouse CD4+

23 been proposed that this chimerism may imply graft tolerance and permit withdrawal of immunosuppressi

24 gs with anti-CD25 antibodies prevented islet graft tolerance and resulted in rejection.

25 lineage mixed chimerism; donor-specific skin-graft tolerance; and in vitro tolerance were observed in

26 nse was associated with, but did not insure, graft tolerance, as the inopportune timing of B7 blockad

27 Highly disparate xenogeneic pig skin graft tolerance can be achieved by grafting FP THY alone

28 nd allowed 60% of treated animals to develop graft tolerance (>120 days), when donor sMHC were combin

29 treatment in this model to achieve pig skin graft tolerance have not previously been defined.

30 n of mixed chimerism and donor-specific skin graft tolerance in 3 Gy-irradiated mice receiving fully

31 ion facilitates macrochimerism induction and graft tolerance in a mouse skin transplantation model.

32 e early alloimmune response) leads to robust graft tolerance in a purely alloimmune setting and prolo

33 3 Gy TBI is not essential for donor pig skin graft tolerance induction.

34 apoptosis is required for the development of graft tolerance, induction strategies that use IL-2-inde

35 rt in the presence of competent T(reg), then graft tolerance is lost.

36 In a model of T(reg)-dependent graft tolerance, it is shown that GZB- deficient mice ar

37 ecific microchimerism in graft acceptance or graft tolerance remains to be elucidated.

38 Donor-specific skin graft tolerance was evaluated, and CD4 reconstitution an

39 However, spontaneous graft tolerance was restored by parking the irradiated L

40 Using a CD4(+) TCR transgenic model for graft tolerance, we have unveiled the independent contri

41 Mixed chimerism and skin graft tolerance were achieved in NOD mice receiving anti

42 erism induces life-long donor-specific organ graft tolerance while obviating the need for chronic imm

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