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1 risk in daughters, granddaughters and great-granddaughters.
2 ve and increased intra-abdominal fat mass in granddaughters, accompanied by accelerated accumulation
4 dence interval [CI], 1.6-6.7), but not among granddaughters and nieces of the probands (RR, 1.2; 95%
5 ermal cell files, suggested to be daughters, granddaughters, and great-granddaughters of the direct p
7 d with the slope of the BMI trajectory among granddaughters (beta = 0.10, 95% confidence interval: 0.
8 n grandparents' chronic poverty exposure and granddaughters' BMI growth slope remained even after con
10 curred in daughters, granddaughters or great-granddaughters - but in 5% of the pedigrees cells contin
11 lls start to elongate, suggesting that great-granddaughter cells switch synchronously from the mitoti
14 evaluated empirically by analyzing simulated granddaughter designs consisting of 2000 sons, 20 relate
16 in mammary cancer risk is transmitted to HF granddaughters equally through the female or male germ l
17 ndmaternal diet on reproductive potential of granddaughters in the absence of any further dietary man
18 length declines more rapidly in the exposed granddaughters, indicating accelerated ageing in the rep
22 ry tumourigenesis is higher in daughters and granddaughters of HF rat dams and in daughters and great
23 d to be daughters, granddaughters, and great-granddaughters of the direct progeny of each stem cell.
24 es to the ablation of neighboring cells, the granddaughters of the primary op blast cell are categori
25 t phenotype generally occurred in daughters, granddaughters or great-granddaughters - but in 5% of th
27 ters to instruct the formation of seven-tile granddaughter sequences that are identical to the initia
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