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1 e survival of neutrophils, downstream of the granulocyte colony stimulating factor receptor.
2 econdary, and tertiary granule proteins, and granulocyte colony-stimulating factor receptor.
3 eceptor signal transducing chain, gp130, and granulocyte colony-stimulating factor receptor.
4 of murine myeloid cells transfected with the granulocyte colony-stimulating factor receptor.
5 of CCAAT enhancer binding protein alpha, the granulocyte colony-stimulating factor receptor.
6 ith upregulation of the granulocyte-specific granulocyte colony-stimulating factor receptor and secon
7 cytokine receptors (erythropoietin receptor, granulocyte colony-stimulating factor receptor, and MPL)
8 ause the erythropoietin, thrombopoietin, and granulocyte colony-stimulating factor receptors are all
9 lorimetry experiments using a soluble murine granulocyte colony-stimulating factor receptor construct
11 ed high-frequency oncogenic mutations in the granulocyte-colony stimulating factor receptor (CSF3R) i
12 ction by binding to a specific receptor, the granulocyte colony-stimulating factor receptor, expresse
13 leads to transcriptional suppression of the granulocyte colony-stimulating factor receptor G-CSF-R (
14 AML-1, PU.1, GATA-2 and Lmo2, as well as the granulocyte colony-stimulating factor receptor (G-CSF-R)
15 companied by mutations in CSF3R encoding the granulocyte colony-stimulating factor receptor (G-CSFR)
17 ace C/EBPalpha in vivo, by the expression of granulocyte colony-stimulating factor receptor (G-CSFR)
22 , we have identified distinct regions in the granulocyte colony-stimulating factor receptor (G-CSFR)
23 e carrying a homozygous null mutation in the granulocyte colony-stimulating factor receptor (G-CSFR)
24 to the cytokine receptor group that includes granulocyte colony-stimulating factor receptor (G-CSFR)
25 hosphatase-1 (SHP-1)-dependent inhibition of granulocyte colony-stimulating factor receptor (G-CSFR)
28 actor 8 (IRF-8) transcripts; lacked CD16 and granulocyte colony-stimulating factor receptor (G-CSFR);
29 ruct was associated with upregulation of the granulocyte-colony stimulating factor receptor (G-CSFR)
30 led signaling with ectopic expression of the granulocyte colony-stimulating factor receptor (GCSFR)-g
31 me high frequency recurrent mutations in the granulocyte colony-stimulating factor receptor gene CSF3
32 as downregulation of the granulocytic marker granulocyte colony-stimulating factor receptor, indicati
33 (EpoR), the thrombopoietin receptor, or the granulocyte colony-stimulating-factor receptor, is neces
34 in the secretory pathway and is required for granulocyte colony-stimulating factor receptor-mediated
36 protein is an activator of transcription of granulocyte colony-stimulating factor receptor promoter,
37 nal activation function of C/EBPalpha on the granulocyte colony-stimulating factor receptor promoter.
38 containing 603 amino acid residues of human granulocyte-colony stimulating factor receptor was expre
41 of type I and II cytokine receptors, except granulocyte colony-stimulating factor receptor, which su
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