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1 ssion profile very similar to that of normal granulocyte macrophage progenitors.
2 myeloid progenitors but relative sparing of granulocyte-macrophage progenitors.
3 f multilymphoid progenitors and depletion of granulocyte-macrophage progenitors.
4 normal and CML hematopoietic stem cells and granulocyte-macrophage progenitors.
5 typic characteristics of colony-forming unit-granulocyte-macrophage progenitors.
6 r lymphoid-primed multipotent progenitors or granulocyte/macrophage progenitors.
7 rentiate from common myeloid progenitors and granulocyte/macrophage progenitors.
8 usly proposed, common myeloid progenitors or granulocyte/macrophage progenitors.
9 macrophage differentiation of murine 32Dcl3 granulocyte/macrophage progenitors.
10 rise to either megakaryocyte/erythrocyte or granulocyte/macrophage progenitors.
11 1 gene causes a severe reduction in myeloid (granulocyte/macrophage) progenitors.
12 CSF, along with a hematopoietic shift toward granulocyte macrophage progenitor and myeloid cells.
13 ol I transcription reduces both the leukemic granulocyte-macrophage progenitor and leukemia-initiatin
14 mmon myeloid progenitors committed to become granulocyte-macrophage progenitors and as megakaryocyte-
15 Gfi-1 in the common myeloid progenitors and granulocyte-macrophage progenitors and down-regulation o
16 LSCs compared with their normal counterpart granulocyte-macrophage progenitors and myeloblast precur
17 rogenitors to differentiate into bipotential granulocyte/macrophage progenitors and their progeny.
18 1RI in supporting increased proliferation by granulocyte/macrophage progenitors and, surprisingly, mu
19 fferentiating common myeloid progenitors and granulocyte-macrophage progenitors, and 4-1BB was induci
20 etic stem cells, common myeloid progenitors, granulocyte-macrophage progenitors, and megakaryocyte-er
22 hil/macrophage lineage outputs from a common granulocyte-macrophage progenitor are still not complete
25 lowing infection with cytomegalovirus, human granulocyte-macrophage progenitors carry the viral genom
26 hat its down-regulation in MDS is related to granulocyte-macrophage progenitor cell sensitivity to TR
27 be expressed in bone marrow-derived bipotent granulocyte macrophage progenitor cells (GM-colony formi
29 cells, common myeloid progenitors (CMPs) and granulocyte-macrophage progenitor cells (GMPs) different
30 terized by a dramatic increase in numbers of granulocyte-macrophage progenitor cells in the marrow an
34 s repressed by C/EBPalpha-p42, and in normal granulocyte/macrophage progenitor cells, we detect C/EBP
37 ng-term haematopoietic stem cells (HSCs) and granulocyte-macrophage progenitors compared with wild-ty
39 dd45a-/- and gadd45b-/- colony forming units granulocyte/macrophage progenitors displayed prolonged p
40 rmal granulocyte-macrophage progenitors, CML granulocyte-macrophage progenitors formed self-renewing,
41 I571 on primary leukemic colony-forming unit granulocyte/macrophage progenitors from patients with CM
42 eage, bipotent megakaryocyte-erythrocyte and granulocyte-macrophage progenitors give rise to unipoten
43 expansion of the lin-/Sca-1/c-kit (LSK) and granulocyte macrophage progenitor (GMP) compartments at
44 chronic myelogenous leukemia (CML), abnormal granulocyte macrophage progenitors (GMP) with nuclear be
45 egakaryocyte-erythroid progenitor (MEP), and granulocyte-macrophage progenitor (GMP) cells, accompani
46 on and increased numbers of DCs, even in the granulocyte-macrophage progenitor (GMP), which does not
47 anscription factor critical for formation of granulocyte-macrophage progenitors (GMP) and leukemic GM
52 mbers of common myeloid progenitor (CMP) and granulocyte/macrophage progenitor (GMP) populations, and
53 CBP, the fusion protein selectively expanded granulocyte/macrophage progenitors (GMP) and enhanced th
54 expansion of splenic cells that derive from granulocyte/macrophage progenitors (GMP) compared with w
55 ecifically expands the numbers of LT-HSC and granulocyte/macrophage progenitors (GMP) resulting in ch
56 els, we show that myeloid differentiation to granulocyte macrophage progenitors (GMPs) is critical fo
57 ivate Evi1 expression in MLL-AF9-transformed granulocyte macrophage progenitors (GMPs) that were init
58 erized by elevated beta-catenin signaling in granulocyte macrophage progenitors (GMPs), which enables
59 -primed multi-potential progenitors (LMPPs), granulocyte-macrophage progenitors (GMPs) and multi-lymp
60 lacked common myeloid progenitors (CMPs) and granulocyte-macrophage progenitors (GMPs) but retained m
61 ogenitors [CLPs]) and CMPs and their progeny granulocyte-macrophage progenitors (GMPs) can give rise
62 uncover a unique spatiotemporal mechanism of granulocyte-macrophage progenitors (GMPs) employed in em
64 -deficient Rag1(-/-) mice, lineage-committed granulocyte-macrophage progenitors (GMPs) or bone marrow
65 d that common myeloid progenitors (CMPs) and granulocyte-macrophage progenitors (GMPs) preferentially
66 profiles from wild-type and Dicer1-deficient granulocyte-macrophage progenitors (GMPs) revealed that
67 Here, we have identified a population of granulocyte-macrophage progenitors (GMPs) that were high
68 hils and the generation of granulocytes from granulocyte-macrophage progenitors (GMPs) were markedly
69 e granulocyte differentiation in bipotential granulocyte-macrophage progenitors (GMPs), its role in r
70 ulations: common myeloid progenitors (CMPs), granulocyte-macrophage progenitors (GMPs), megakaryocyte
71 (CMPs), common lymphoid progenitors (CLPs), granulocyte-macrophage progenitors (GMPs), or early thym
72 n aberrant progenitor differentiation toward granulocyte-macrophage progenitors (GMPs), resulting in
74 s (CLPs), common myeloid progenitors (CMPs), granulocyte/macrophage progenitors (GMPs), and thymocyte
75 t-term (ST)-HSCs/multipotent progenitors and granulocyte/macrophage progenitors have self-renewal cap
76 rmally high numbers of colonies derived from granulocyte-macrophage progenitors in cultures supplemen
77 mpounds exhibited some degree of toxicity to granulocyte/macrophage progenitors in the bone marrow of
80 or myeloid progenitor lineage skewing toward granulocyte-macrophage progenitors, increased colony-for
83 ypes through the same myeloid-restricted pre-granulocyte-macrophage progenitor (pre-GM) (Lin(-)Sca-1(
84 lum-induced HSC, multipotent progenitor, and granulocyte/macrophage progenitor proliferation and reac
86 he Id1 gene begins to be up-regulated at the granulocyte-macrophage progenitor stage and continues th
87 ) and acute leukemia evolving from committed granulocyte-macrophage progenitors that have acquired th
88 ng experimental latent infection of cultured granulocyte-macrophage progenitors, the viral genome was
89 d LSC from leukaemias initiated in committed granulocyte macrophage progenitors through introduction
90 s increased, but differentiation from CMP to granulocyte/macrophage progenitor was decreased, and the
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