ライフサイエンスコーパス: granulocytosis

コーパス検索結果 (１語後でソート)

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1 lymphocyte reductions to support a reactive granulocytosis.

2 iated by the concomitant induction of marked granulocytosis.

3 ce characterized by massive splenomegaly and granulocytosis.

4 d myelopoiesis leading to thrombocytosis and granulocytosis.

5 tor (G-CSF), a principal cytokine-regulating granulocytosis.

6 2 deficient mice succumbed from overwhelming granulocytosis.

7 [15%]), thrombocytopenia (47/104 [45%]), and granulocytosis (44/104 [42%]).

8 of Sema3E in mice results in increased lung granulocytosis, airway hyperresponsiveness, mucus overpr

9 SCID and NOD-SCID mice exhibited also granulocytosis and increased numbers of hemopoietic prog

10 of ILCs prevented IL-17- and G-CSF-dependent granulocytosis and resistance to sepsis.

11 many proinflammatory cytokines, resulting in granulocytosis and severe sterile inflammation.

12 Granulocytosis and splenomegaly developed in mice that r

13 d by myeloid hyperplasia, including profound granulocytosis and splenomegaly.

14 splenomegaly, extramedullary erythropoiesis, granulocytosis and thrombocytopaenia secondary to a bloc

15 However, these animals did develop mild granulocytosis compared with mice reconstituted with WT

16 f human PV, characterized by erythrocytosis, granulocytosis, extramedullary hematopoiesis, and bone m

17 tion, presumably accounting for the reactive granulocytosis in diseased mice.

18 mulating factor (G-CSF) as a means to induce granulocytosis in donors has rekindled interest in this

19 These data suggest that erythrocytosis and granulocytosis in JAK2(V617F) mice are the net result of

20 CD97 null mice also displayed a mild granulocytosis in the nonchallenged state.

21 myeloid colony-forming activity, and marked granulocytosis in the peripheral blood.

22 a strongly argue against the clinical use of granulocytosis-inducing hematopoietic stem cell mobiliza

23 r the intestinal microbiota in regulation of granulocytosis, neutrophil homeostasis and host resistan

24 eated with G-CSF showed less than 50% of the granulocytosis observed in identically treated WT mice.

25 These observations suggest that the granulocytosis observed in the absence of CD18 occurs th

26 CD97 deficiency did not appear to stimulate granulocytosis secondary to peripheral inflammation and

27 Splenomegaly was prevented and granulocytosis was inhibited by more than 95% in mice th

28 mia, and Icsbp(-/-) mice exhibit progressive granulocytosis with evolution to blast crisis, similar t

29 es death of all animals from an overwhelming granulocytosis within 3 to 4 weeks.

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