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1 were unrelated to risk after adjustment for gravidity.
2 and the excess risk of infection varies with gravidity.
3 at menarche, age at first birth, parity, and gravidity.
4 esign, we compared antibody levels in women (gravidity 1 to 11) with and without placental malaria.
5 ned statistically significant independent of gravidity: a history of ever (vs. never) having had an i
6 at anti-PfGPI antibody levels increased with gravidity and age and that malaria infection boosted ant
10 t modified by insecticide-treated net use or gravidity, and remained significant in areas with high S
11 n (during pregnancy, not at delivery) and by gravidity, and we used meta-regression to assess factors
12 d with lower mean hemoglobin irrespective of gravidity, and with increased anemia risk in primigravid
13 d among women, it was associated with higher gravidity (aOR for 5 vs 0 pregnancies, 3.2; 95% CI, 1.9-
14 nalyzed plasma samples from women of various gravidities at different gestational stages for antiadhe
15 s in primigravidae appear to account for the gravidity-dependent differential susceptibilities of pre
20 sociated with seropositivity, including age, gravidity, human immunodeficiency virus (HIV) infection
21 uded in the model: study site, maternal age, gravidity, marital status, education, race/ethnicity, sm
23 height and body mass index at school entry, gravidity, maternal age at birth, pregnancy-induced hype
24 ge, low maternal education, race, residence, gravidity, maternal spontaneous abortion history, perina
28 cational level and socioeconomic status, and gravidity, transient hypoglycemia was associated with de
30 sed ITN usage data that can be stratified by gravidity), we estimate that, due primarily to low ITN u
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