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1 the antibodies in the blood pools (heart and great vessels).
2 are required for remodeling of the heart and great vessels.
3 truncus arteriosus and abnormalities of the great vessels.
4 s to direct blood flow through the heart and great vessels.
5 the formation of the mature aortic arch and great vessels.
6 eate casts of blood flow of the chambers and great vessels.
7 nexpected plasticity in the formation of the great vessels.
8 o avoid laparotomy and cross-clamping of the great vessels.
9 en to the innermost endothelial cells of the great vessels.
10 utermost, adventitial cell population in the great vessels.
11 dventitial cell population of the developing great vessels.
12 rolling chronic inflammatory diseases of the great vessels.
13 a function of blood volume in the heart and great vessels.
14 Six needles (5.2%) were placed into the great vessels.
15 he immune system, and chronic disease of the great vessels.
16 measurements of blood flow in the heart and great vessels.
17 icular septal defect, 4 transposition of the great vessels, 3 patient ductus arteriosus, 3 partial an
19 ith a constellation of heart, outflow tract, great vessel and pharyngeal gland defects that phenocopi
20 ing thoracic aorta, such as proximity to the great vessels and arch tortuosity, have been and remain
22 lular matrix protein abundantly expressed in great vessels and cardiac valves during embryogenesis, a
24 r, with higher rates of transposition of the great vessels and common ventricle patients compared wit
27 malformations of the cardiac outflow tract, great vessels and heart due, at least in part, to failur
28 e are extensive defects in remodeling of the great vessels and heart resulting in death at ~E14.5.
29 are essential for normal development of the great vessels and the heart, giving rise to a range of c
30 nary veins, scattered within the wall of the great vessels, and a strictly delimited cluster between
31 allot, Ebstein anomaly, transposition of the great vessels, and common ventricle) and (2) patients <2
32 In addition to information about the heart, great vessels, and coronary arteries, these examinations
35 ac magnetic resonance reference Z scores for great vessel areas in normal children and adolescents in
36 maging (ventricular volumes and function and great vessel blood flow) and hemodynamic assessment (inv
37 ng the function and anatomy of the heart and great vessels, but its emerging role as one of the domin
38 lood vessels, nor by defects in the heart or great vessels, but were due to abnormal development of t
39 uses a variety of pharyngeal arch artery and great vessel defects, as well as malformations in many o
42 enital heart disease, but normative data for great vessel dimensions in pediatric subjects are scarce
43 is in native (2C) or mechanical valves (1B), great vessel disease and injury (2C), penetrating chest
47 onship between the coronary arteries and the great vessels in subjects with normal cardiac morphology
49 c neural crest in formation of the heart and great vessels in the mouse and, furthermore, shows that
50 to the blood pool, visible as the heart and great vessels in the trunk and limbs, plus diffuse signa
51 dibulum (OFT myocardial remnant) beneath the great vessels, indicating failure of OFT shortening.
52 fined as pneumothorax, hemothorax, aortic or great vessel injury, 2 or more rib fractures, ruptured d
53 metric embryonic aortic arches to the mature great vessels is a complex morphogenetic process, requir
55 malities eventually lead to various types of great vessel malformations highly similar to those seen
56 ithin the DGS phenotypic spectrum, including great vessel malformations, hypoplastic pulmonary and ao
57 E axis signaling in the vulnerable heart and great vessels may be essential in controlling and preven
58 ly with respect to assessment of cardiac and great vessel morphology and left ventricular function.
59 ammation (n = 4), blood pool activity in the great vessels (n = 2), bowel uptake (n = 1) and unknown
63 umental in diagnosing cardiac and associated great vessel pathology and in identifying structural abn
64 e studies assign the embryonic origin of the great vessel progenitors to the interface between the ph
66 s, as well as defects in cardiac outflow and great vessel structures, which are derived from cephalic
68 FT cardiomyocytes by PCD is required for the great vessels to make their proper connections with the
69 e instances, the infundibulum connected both great vessels to the right ventricle in a side-by-side a
70 re injuries in children, such as cardiac and great vessel trauma, may remain undiagnosed precisely be
71 ssures and samples from cardiac chambers and great vessels using antegrade, transseptal, and retrogra
72 s, eg, neural tube defects, transposition of great vessels, ventricular septal defect, atrial septal
75 2 patients (50%), and d-transposition of the great vessels was the diagnosis for 4 patients (16%).
77 ded surface display images of the airway and great vessels were generated from volumetric CT data and
80 nary patterns, both of which loop around the great vessels, were associated with significant mortalit
82 %-58%) and proximity to the aortic valve and great vessels (with potential for dissection complicatio
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