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1 estrogen-replaced nonhuman primates (African green monkeys).
2 ces were obtained for cynomolgus macaque and green monkey.
3 otype 3 (SFVagm-3), isolated from an African green monkey.
4 -3-fold higher in cynomolgus monkeys than in green monkeys.
5 sterol levels were 50% lower than was fed to green monkeys.
6 CAT2 mRNA nor protein was diet-responsive in green monkeys.
7 ary cholesterol, and less responsive African green monkeys.
8 simian immunodeficiency viruses from African green monkeys.
9 tralization titers in RSV-preexposed African green monkeys.
10 vet, grivet, and tantalus species of African green monkeys.
11 nted in the striatum of MPTP-treated African green monkeys.
12 n the dorsal and ventral striatum of African green monkeys.
13 candidates after a single passage in African green monkeys.
14 immunodeficiency virus infections of African Green Monkeys.
15 both LDL receptor-deficient mice and African green monkeys.
16 g natural SIV variants isolated from African green monkeys.
19 uman A3C (hA3C), human A3DE (hA3DE), African green monkey A3F (agmA3F), and rhesus macaque A3F (rhA3F
20 V) Vif was shown to bind and degrade African green monkey A3G (agmA3G) and, unexpectedly, human A3C.
21 re, we report the DNA sequence of an African green monkey AAV integration site isolated from CV-1 cel
22 t rhesus macaque APOBEC3G (rhA3G) or African green monkey (AGM) APOBEC3G (agmA3G) because of a failur
23 for studying HeV infection, with the African green monkey (AGM) appearing to most faithfully reproduc
27 mined the pathogenesis of HeV in the African green monkey (AGM) following intratracheal inoculation.
29 his issue, we established a neonatal African green monkey (AGM) nonhuman primate model that could be
30 sely, the Vif protein encoded by the African green monkey (agm) simian immunodeficiency virus (SIV) c
31 the A3G-Vif interaction within four African green monkey (AGM) subspecies, which are each naturally
33 n, we isolated cDNA clones of human, African green monkey (AGM), and NIH/Swiss mouse CCR5s, and we qu
34 b from its natural host, the sabaeus African green monkey (AGM), to a new host, the pigtailed macaque
35 investigate replication in primates, African green monkeys (AGM) and rhesus macaques (n = 4) were ino
42 were assessed in naturally infected African green monkeys (AGM) of the vervet subspecies (Chlorocebu
44 genic differences between strains, 4 African green monkeys (AGM) were exposed to NiVM and 4 AGMs were
45 ssive infection (SIVagm infection of African green monkeys (AGM)), and transient, controlled infectio
50 iral pathogens in two populations of African green monkeys (AGMs) (Chlorocebus sabaeus) from Africa a
53 geographically dispersed species of African green monkeys (AGMs) are all infected with highly divers
60 roximately 98-99% identical) CCR5 of African green monkeys (AGMs) avidly binds beta-chemokines and fu
61 est the hypothesis that SIV-infected African green monkeys (AGMs) avoid AIDS due to virus replication
64 fection in sooty mangabeys (SMs) and African green monkeys (AGMs) is associated with low levels of im
65 s of chronically SIV-infected sabeus African green monkeys (AGMs) revealed that gastrointestinal memo
66 d from pigtailed macaques (PTMs) and African green monkeys (AGMs) that experience different SIVsab in
68 p of 10 rhesus macaques (RMs) and 10 African green monkeys (AGMs) was exposed to aerosolized B. pseud
70 ans from chronically SIVagm-infected African green monkeys (AGMs) were frequently CXCR5(+) and entere
77 eficiency virus (SIV) hosts, such as African green monkeys (AGMs), sustain nonpathogenic SIV infectio
86 caques [PTMs]) and nonpathogenic (in African green monkeys [AGMs]) SIVsab infections to assess the si
87 lizing antibody titers obtained from African green monkeys and after human vaccination and natural in
88 strate that nonhuman primates (NHPs; African green monkeys and cynomolgus macaques) harbor serosal B
89 tified multiple SAMHD1 haplotypes in African Green Monkeys and find that the vpr gene from different
90 hosts (for example, sooty mangabeys, African green monkeys and mandrills) share many features of HIV
92 utcomes of SIVsab infection in PTMs, African green monkeys, and rhesus macaques are different, the us
93 ciently in the respiratory tracts of African green monkeys, and the infected animals developed a high
94 TRIM5alpha B30.2 domain v1 region of African green monkeys are also associated with broader antiretro
96 administered separately to groups of African green monkeys by the intranasal/intratracheal route.
99 pr proteins are capable of arresting African green monkey cells but are completely inactive in human
103 IgA did not inhibit HAV infection of African green monkey cells, suggesting that the IgA and the viru
104 n after entry into rhesus macaque or African green monkey cells, where, paradoxically, the interactio
109 s musculus), hamster (Mesocricetus auratus), green monkey (Ceropithecus aethiops) and human (Homo sap
110 Herein we report that two species of African green monkeys (Chlorocebus sabaeus and C. pygerythrus) i
111 cyte-depleting antibodies to sabaeus African green monkeys (Chlorocebus sabaeus) before challenge wit
112 herpesviruses recently identified in African green monkeys, Chlorocebus rhadinovirus types 1 and 2 (C
114 d a range of restriction in mice and African green monkeys comparable with that of two attenuated RSV
117 rom chimpanzees] and SIVagmSab [from African green monkeys]) discordantly in different regions of the
118 nts from humans, rhesus monkeys, and African green monkeys displayed different but overlapping restri
119 e show that many CD4(+) T cells from African green monkeys downregulate CD4 in vivo as they enter the
120 ery atherosclerosis were examined in African green monkeys fed diets containing cholesterol and 35% o
123 y shown that intranasal SV protected African green monkeys from challenge with the related human para
124 ruses (family Arteriviridae) in wild African green monkeys from Zambia (malbroucks [Chlorocebus cynos
130 uated the immunological responses of African green monkeys immunized with multiple F and G protein-ba
133 d/nonprogressive infection) and from African green monkeys infected with SIVsab9315BR (nonpathogenic
134 set of clinical anthrax disease, the African green monkey is a suitable animal model exhibiting a dis
135 enuated viruses adapted to growth in African green monkey kidney (AGMK) and MRC-5 cells, respectively
139 ed from a cDNA expression library of African green monkey kidney (AGMK) cells by using protective mon
140 Hepatitis A virus (HAV) infects African green monkey kidney (AGMK) cells via the HAV cellular re
141 ar receptor 1 (havcr-1) and protects African green monkey kidney (AGMK) clone GL37 cells (GL37 cells)
143 sackievirus B6 replication in living Buffalo green monkey kidney (BGMK) cells via Tat peptide deliver
144 To characterize interactions between African green monkey kidney (BS-C-1) cell proteins and the predi
145 M175/P16, enhance growth in cultured African green monkey kidney (BS-C-1) cells but not in fetal rhes
147 and establishing stably transfected African green monkey kidney (CV1) cell lines expressing reporter
149 , and produced very small plaques on African green monkey kidney (Vero) cells that were similar in si
150 N2, H5N1 and H7N9 vaccine viruses in African green monkey kidney and Madin-Darby canine kidney cells.
152 s and deletions were monitored in an African green monkey kidney cell culture system (COS-7 cells) as
153 reliminary studies indicated that an African green monkey kidney cell line (Vero) is a suitable syste
155 within a few cycles of infection in African green monkey kidney cell lines CV-1, CV-1P, TC-7, MA-134
156 generates H2O2, was introduced into African green monkey kidney cells (CV-1 cells) under the control
157 ell homeostasis were investigated in African green monkey kidney cells (CV-1) by assessing the appear
158 1.3, was heterologously expressed in African Green Monkey kidney cells (CV-1) using a vaccinia virus/
159 n U937 cells (histiocytic lymphoma), African green monkey kidney cells (MARC-145 and Vero), primary m
160 e microsurgically removed from BSC-1 African green monkey kidney cells before the completion of S pha
162 Hepatitis A virus (HAV) infects African green monkey kidney cells via HAV cellular receptor 1 (h
165 lanoma cell line but not to the CV-1 African green monkey kidney cells, which express CD44 at low lev
170 eir respective intracellular niches, African green monkey kidney epithelial (Vero) cells, A/J mouse b
171 tion was detected in only six: three African green monkey kidney epithelial cell lines (Vero, Vero E6
172 0 DNA replication in infected BSC-1 (African green monkey kidney epithelial) cells, albeit at a great
173 ve in 293T (embryonic kidney), Vero (African-green monkey kidney epithelial), 3T12 (mouse fibroblast)
174 interact with CD66a-transfected COS (African green monkey kidney) and CHO (Chinese hamster ovary) cel
179 ferent species of naturally infected African green monkeys living in different regions across Africa.
180 , complete suppression of macaque or African green monkey Lv1 was achieved by the additive effect of
182 natural SIV hosts (sooty mangabeys, African green monkeys, mandrills, sun-tailed monkeys, and chimpa
184 ed the use of RSV (Memphis 37) in an African green monkey model of intranasal infection and identifie
185 IV5/G) protein in the cotton rat and African green monkey models for their replication, immunogenicit
186 pliced exon 10 were identical between human, green monkey, mouse, rat, and pig, while 207 consecutive
188 mbinant viruses were administered to African green monkeys (NDV-BC and NDV-LS) and rhesus monkeys (ND
189 The infecting SFV originated from an African green monkey (one person) and baboons (three people).
198 tion and deletion analysis in BSC-1 (African green monkey, renal epithelial) cells revealed that the
201 ; the human and, to a low level, the African green monkey sequences bound soluble HCV E2 (sE2) and in
202 eavage-efficient mutant, R-R-R-R, in African green monkeys showed that there was no detectable change
204 We now report a novel function of African green monkey simian immunodeficiency virus (SIVagm) Vif
205 only human Apo3G (hApo3G), whereas, African green monkey simian immunodeficiency virus (SIVagm) Vif
206 ian immunodeficiency virus SIV(SM)), African green monkey (SIV(AGM)), and Sykes' monkey (SIV(SYK)) is
207 munodeficiency viruses isolated from African green monkeys (SIVagm) contain a single accessory gene h
208 odeficiency virus (SIV) that infects African green monkeys (SIVagm) contains a vpr homologue, which e
209 Simian immunodeficiency virus from African green monkeys (SIVagm) results in asymptomatic infection
210 onpathogenic infection with SIV from African green monkeys (SIVagm), follicles remain generally virus
211 d simian immunodeficiency virus from African green monkeys (SIVagm), in one round of viral replicatio
215 (simian immunodeficiency virus from African green monkeys [SIVagm] and Rhesus macaques [SIVmac]), th
217 apped mangabey (SIVrcm), the sabaeus African green monkey (SIVagmSAB), and the chimpanzee (SIVcpz) an
218 odeficiency virus (SIV) that infects African green monkeys (SIVagmTAN), unlike human Apobec3DE, which
221 developed to measure SIVagm from two African green monkey subspecies demonstrated high levels of SIV
222 ed with that previously observed for African green monkeys, suggesting that the HAE model has potenti
224 D4(+) T cells of young mandrills and African green monkeys than on those of adults, we propose that l
229 e compared the plasma virome of West African green monkeys to that in their descendants after importa
233 east two different retroviruses, and African green monkey TRIM5alpha was able to inhibit infection by
236 s in one eye of juvenile rhesus macaques and green monkeys, we combined cDNA subtractions, microarray
237 It was found that all vaccinated African green monkeys were completely protected against subseque
239 otective efficacy in cotton rats and African green monkeys, which are among the best available animal
240 HAV) was originally isolated from an African green monkey with hepatitis and appears to represent a t
241 We infected 35 Asian macaques and African green monkeys with viruses that do or do not express Vpx
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