ライフサイエンスコーパス: green tea

1 urine after single bolus intake of black or green tea.

2 the nutraceutical potential of a beverage as green tea.

3 e and antisecretory benefits of red wine and green tea.

4 cups/d, and we observed no association with green tea.

5 ate (EGCG) is the major active polyphenol in green tea.

6 , cyclooxygenase-2 inhibitors, lycopene, and green tea.

7 ea polyphenols were consumed as black tea or green tea.

8 coctions of Limonium algarvense flowers, and green tea.

9 -3-gallate (EGCG) is the major polyphenol in green tea.

10 tion to two food products, i.e. red wine and green tea.

11 s the most biologically potent polyphenol of green tea.

12 cals in nutraceutical products obtained from green tea.

13 ed on polypropylene added with catechins and green tea.

14 tioxidants to study in coffee, black tea and green tea.

15 re the main contributors to the taste of RTD green tea.

16 nsities of bitterness and astringency in RTD green teas.

17 tioxidant capacity of twenty-four commercial green teas.

18 with seaweed, but inconclusive for black and green teas.

19 pigallocatechin gallate (EGCG) were found in green teas.

20 of the black teas were as efficacious as the green teas.

21 found that treatment of A/J mice with either green tea (0.6% in water) or a defined green tea catechi

22 The method was applied to green tea (10) and royal jelly (8) samples.

23 Catechins from green tea (125-1,000 ppm), including (+)-catechin, (-)-e

24 Green tea (29.8% [25 of 84]), echinacea (13.1% [11 of 84

25 es of three beverages, coffee, black tea and green tea, along with their major components, were inves

26 The anticancer potency of green tea and its individual components is being intense

27 Nine (eight of green tea and one of royal jelly) samples were found to

28 Green tea and placebo gels were placed at test and contr

29 r scaffold (gallotannins), as are present in green tea and red wine.

30 e obtained using C18 and zirconium oxide for green tea and royal jelly, respectively.

31 s were detected after 3 months of storage in green tea and soy products, while 6 months were necessar

32 Green tea and the green tea catechin have been shown to

33 chin gallate (EGCG), the major polyphenol in green tea and the main bioactive compound responsible fo

34 t are reachable by ingestion of pure EGCG or green tea, and are not toxic to hepatocytes.

35 e analysis of liquid samples of grape juice, green tea, and coffee.

36 roxyl radical-scavenging activity, higher on green tea, and iron chelating potential, higher on L. al

37 tea varieties, respectively, known for good green tea, and they were used as reference standards.

38 xidant and anti-inflammatory properties than green tea, and thus, may be useful for alleviating sympt

39 he well-known pleiotropic health benefits of green tea are mainly attributed to epigallocatechin-3-ga

40 ed classes (Argentinean green tea; Brazilian green tea; Argentinean black tea; Brazilian black tea; a

41 Evidence is accumulating that catechins in green tea as well as theaflavins and thearubigins from b

42 andomly assigned to 3 different sequences of green tea, black tea, or a green tea extract supplement

43 asma 8 h after a bolus consumption of either green tea, black tea, or a green tea extract supplement.

44 ion of the five studied classes (Argentinean green tea; Brazilian green tea; Argentinean black tea; B

45 ilability of flavan-3-ols after ingestion of green tea by humans is reviewed.

46 Green tea (Camellia sinensis) is one of the most widely

47 ides in nutraceutical products obtained from green tea (Camellia sinensis).

48 o antioxidant and antifibrotic properties of green tea (Camellia sinensis, Theaceae) were investigate

49 The encapsulation of green tea catechin and epigallocatechin gallate (EGCG) i

50 We selected a green tea catechin derivative, oligomerized (-)-epigallo

51 The green tea catechin epigallocatechin-3-gallate (EGCG) is

52 ither green tea (0.6% in water) or a defined green tea catechin extract (polyphenon E; 2.0 g/kg in di

53 Thermo-reversible sustained-release green tea catechin gel was prepared and tested for its i

54 Green tea and the green tea catechin have been shown to inhibit tumorigene

55 ular targets in cardiac muscle for the major green tea catechin, (-)-epigallocatechin-3-gallate (EGCG

56 The most abundant and biologically active green tea catechin, (-)-epigallocatechin-3-gallate or (-

57 alysis of the interactions between the major green tea catechin, epigallo-catechin 3-gallate (EGCG),

58 y be good carriers of physiologically active green tea catechin.

59 scribed as the most potently chemopreventive green tea catechin; however, the low bioavailability of

60 is observed over time under the influence of green tea catechins (GTC), which are suggested to offer

61 Recent data suggest that green tea catechins (GTCs) reduce acute UVR effects, but

62 Green tea catechins (GTCs) with or without caffeine have

63 phobic and/or cation-pi associations between green tea catechins and cheese fat components.

64 Molecular integrations between green tea catechins and milk fat globules in a cheese ma

65 Green tea catechins are known to have anticarcinogenic e

66 The anti- or prooxidant effects of green tea catechins have been implicated in the modulati

67 ns between the milk fat globule membrane and green tea catechins provided useful information about th

68 Green tea catechins such as (-)epigallocatechin-3-gallat

69 ocatechin gallate (EGCG) is one of the major green tea catechins that is suggested to have a role as

70 We assessed the ability of alternative green tea catechins to inhibit HGF-induced signaling and

71 droxyl groups are important in mediating the green tea catechins' inhibitory effect towards HGF/Met s

72 e, in general, neither aglycones (except for green tea catechins) nor the same as the dietary source.

73 Polyphenon E, a mixture of green tea catechins, had a similar effect but (-)-epicat

74 and laccase, both from Agaricus bisporus, on green tea catechins, the oxidation process was directed

75 The implication of the green tea chemical quality parameters is also discussed.

76 The green tea component epigallocatechin-3-gallate (EGCG) ma

77 tically applied to chemical structures of 75 green tea components, resulting in a virtual library of

78 P, and that acute inhibition of Bcl-2 by the green tea compound epigallocatechin gallate results in a

79 Unexpectedly, we discovered that various green tea constituents, in particular (-)-epigallocatech

80 tion of metabolites over a 24-h period after green tea consumption corresponded to 28.5% of the inges

81 cts are required before a recommendation for green tea consumption for cancer prevention should be ma

82 Green tea consumption is associated with reduced cardiov

83 provided evidence for a protective effect of green tea consumption on breast cancer development.

84 Epidemiological studies have associated green tea consumption with reduced recurrence of invasiv

85 ial vascular and metabolic health effects of green tea consumption.

86 ved metabolites in human urine samples after green tea consumption.

87 late (EGCG), a flavanoid polyphenol found in green tea; curcumin, found in tumeric; and resveratrol (

88 Green tea decoction had the highest content of phenolic

89 (EGCG), the main polyphenolic constituent of green tea, decreased beta-amyloid (Abeta) levels and pla

90 Recent reports suggest green tea-derived (-)-epigallocatechin-3-gallate (EGCG)

91 tudy that epigallocatechin gallate (EGCG), a green tea-derived catechin, acts as a potent suppressor

92 the putative identification of 26 additional green tea-derived metabolites.

93 Green tea-derived polyphenol (-)-epigallocatechin-3-gall

94 e compounds present in new broccoli-enriched green tea drinks and their potential antitumoral activit

95 echin 3-gallate), the major phytochemical in green tea, emerged as an intriguing candidate.

96 eine in real samples (urine, plasma, tablet, green tea, energy and soda drink).

97 ously shown that two of the polyphenols from green tea (epigallocatechin gallate (EGCG) and epicatech

98 on was to determine whether an ingredient of green tea, epigallocatechin gallate (EGCG) could attenua

99 gate the effects of two main constituents of green tea, (-)-epigallocatechin-3-gallate (EGCG) and caf

100 As a major effective component in green tea, (-)-epigallocatechin-3-gallate (EGCG)'s poten

101 The major constituent of green tea, (-)-epigallocatechin-3-O-gallate (EGCG), has

102 allate (EGCG), the major polyphenol found in green tea, exerts antiproliferative and proapoptotic eff

103 These results indicate that green tea exhibits potent protective effects against CCl

104 d provide a molecular signature specific for green tea exposure.

105 e randomly allocated to 1350 mg encapsulated green tea extract (540 mg GTC) with 50 mg vitamin C or p

106 extracts of ulam raja (UREX) and commercial green tea extract (GTE) added individually at 200 and 50

107 type I collagen synthesis and the effects of green tea extract (GTE) and its major catechin, (-)-epig

108 of lettuce extract (LE) with quercetin (QC), green tea extract (GTE) or grape seed extract (GSE) was

109 dy was to examine the efficacy and impact of green tea extract (GTE) supplementation high in epigallo

110 extract (PRE), cherry stem extract (CSE) and green tea extract (GTE) were added to sour cherry juice

111 anufactured containing free catechin or free green tea extract (GTE), and liposomal encapsulated cate

112 f alpha-tocopherol (TOH) in combination with green tea extract (GTE), the green tea polyphenol (-)-ep

113 h the most notable improvement observed with green tea extract addition.

114 Moreover, green tea extract administration significantly increased

115 al trials have shown an inhibitory effect of green tea extract against the progression of prostate pr

116 was to test the chemopreventive potential of green tea extract and EGCG after tannase-mediated hydrol

117 almost 50% of the total catechin content in green tea extract and has very potent antioxidant effect

118 Green tea extract and many other products, in contrast,

119 incubated with epigallocatechin gallate and green tea extract at 62 or 37 degrees C for 180 min.

120 Oral administration of green tea extract at doses of 125, 625 and 1250 mg/kg fo

121 EGCG is a flavonoid present in green tea extract belonging to the subclass of catechins

122 cence quenching measurements showed that the green tea extract contained components that interacted w

123 Green tea extract is a naturally occurring antimicrobial

124 n inhibitory role of oral supplementation of green tea extract on a precancerous lesion of oral cavit

125 Our study found that oral administration of green tea extract prevented CCl(4)-induced hepatic fibro

126 rent sequences of green tea, black tea, or a green tea extract supplement in a 3 x 3 crossover design

127 umption of either green tea, black tea, or a green tea extract supplement.

128 Our observations suggest that green tea extract supplements retain the beneficial effe

129 illus subtilis were found to be sensitive to green tea extract via disc diffusion assay (zone of inhi

130 -catechin, (-)-epigallocatechin gallate, and green tea extract were added to washed MFGs to examine p

131 om rapeseed oil>mix of synthetic tocopherols>green tea extract>sinapic acid>BHT.

132 red the interactions and reactivities of the green tea extract, (-)-epigallocatechin-3-gallate [(2R,3

133 implicated agents include anabolic steroids, green tea extract, and multi-ingredient nutritional supp

134 tor inhibitors, cyclooxygenase-2 inhibitors, green tea extract, and peroxisome proliferator activated

135 phenols (vanillin, epigallocatechin gallate, green tea extract, and protocatechualdehyde) at inhibiti

136 hin-3-gallate (EGCG), a major constituent of green tea extract, exhibits effects of anti-inflammation

137 untargeted LC-MS(n) (n </= 3) data set of a green tea extract, generated on an LC-LTQ/Orbitrap hybri

138 ioxidants were added to the prepared matrix: green tea extract, rosemary extract, a mix of tocopherol

139 In a green tea extract, tyrosinase increased the proportion o

140 model better than the fruit or standardized green tea extract.

141 or fading increased from 2.9 to 6.7days with green tea extract.

142 ration Solutions and 3255 Camellia sinensis (Green Tea) Extract with certified concentrations of cate

143 hin gallate (EGCG) is the major component of green tea extracts and possesses antibacterial, antivira

144 onsistent results of an inhibitory effect of green tea extracts and tea polyphenols on the developmen

145 Administration of green tea extracts containing epigallocatechin 3-gallate

146 The yellow and green tea extracts were characterized by the highest con

147 tures of the natural oil body emulsions with green tea extracts, aiming to serve as a base for functi

148 A major component of green tea extracts, catechin (-)-Epigallocatechin gallat

149 GCG), the major and most active component in green tea extracts, inhibited HSC activation.

150 r the reduction of MbFe(IV)O by catechin and green tea extracts, though possible confounding reaction

151 the mechanism of thearubigin formation from green tea flavan-3-ols, a model system, based on electro

152 Recent studies suggest that green tea flavonoids may be used for the prevention and

153 Treatment with Dyrk1A inhibitor, green tea flavonol epigallocatechin-gallate (EGCG), from

154 keloids and imply a therapeutic potential of green tea for the intervention and prevention of keloids

155 gallate (EGCG), the main catechin present in green tea, forms complexes with the casein micelles and

156 The consumption of black tea, green tea, fruit juices, or soft drinks was not associat

157 evelop a thermo-reversible sustained-release green tea gel and to study its clinical effects on patie

158 ve local drug therapy with thermo-reversible green tea gel has shown to reduce pockets and inflammati

159 the range of 0.375-12mg in totally 1.75g of green tea, green coffee and mixed herbal tea by using FT

160 the first time, detection of sibutramine in green tea, green coffee and mixed herbal tea using ATR-F

161 Previous investigations have implicated green tea (GT) as affording protection against PAH-induc

162 current study investigated the potential of green tea (GT) to improve uncoupling of endothelial nitr

163 properties of pure plant extracts (PPEs) of green tea (GT), black tea (BT) and soybean individually

164 her (p<0.05) in FST than FS, in the order of green tea>oolong tea>black tea>soymilk.

165 Because polyphenols from green tea (GTP) prevent other cutaneous adverse effects

166 L. algarvense and green tea had similar antioxidant properties, except for

167 Green teas had the highest levels of total polyphenols,

168 Green tea has been shown to be a potent chemopreventive

169 Green tea has been suggested to improve cardiovascular d

170 Black tea, although not as well studied as green tea, has also shown cancer-preventive effects in l

171 techin-3-gallate (EGCG), a catechin found in green tea, has been recognized as a potential therapeuti

172 (EGCG), the principal polyphenol present in green tea, has been shown to be effective at preventing

173 -gallate (EGCG), the principal polyphenol in green tea, has been shown to inhibit the growth of many

174 in-3-O-gallate (EGCG), a major ingredient of green tea, has been shown, for example, to possess antic

175 atechin-3-gallate (EGCG), a main catechin of green tea, has been suggested to inhibit hepatic glucone

176 late (EGCG), a major polyphenol component of green tea, has recently been identified as an inhibitor

177 Although, anti-microbial properties of green tea have been studied, its role against bacterial

178 The cancer chemopreventive qualities of green tea have been well documented.

179 hat can predict the exposure and efficacy of green tea in mice.

180 chin gallate (EGCG), the major polyphenol in green tea, in mice treated with carbon tetrachloride.

181 Green tea infused at 95 degrees C for 10min showed highe

182 Green tea infusion is a beverage that also contains anti

183 During staying of green tea infusion, the degradation of some catechins pr

184 radation of catechins and other phenolics in green tea infusions were monitored using fast HPLC/MS se

185 Tannic acid, red wine, and green tea inhibited arterial smooth muscle contraction a

186 ilution of red wine and 100-fold dilution of green tea inhibited CaCCs by >50%.

187 , a major biologically active constituent of green tea, inhibits activation of the epidermal growth f

188 GCG), an anti-inflammatory compound found in green tea, inhibits IL-1beta-induced IL-6 production and

189 Caffeinated soda intake and green tea intake >/=1 cup/d (1 cup = 240 mL) were positi

190 Evidence in support of green tea intake against the development of liver cancer

191 An inverse association between green tea intake and lung cancer risk has been observed

192 evidence in support of a protective role of green tea intake in the development of oral-digestive tr

193 efute a definitive cancer-preventive role of green tea intake.

194 ng white women, whereas caffeinated soda and green tea intakes were associated with increased estradi

195 Green tea is a major source of catechins and may be asso

196 Green tea is consumed globally and is reported to have a

197 in-3-gallate (EGCG), the major polyphenol in green tea, is a direct antagonist of androgen action.

198 -gallate (EGCG), a polyphenol extracted from green tea, is an antioxidant with chemopreventive and ch

199 of coffee and potentially black tea, but not green tea, is associated with lower risk of type 2 diabe

200 atechin-3-O-gallate, the main flavan-3-ol in green tea, is unclear because it appears unmetabolized i

201 activity and the basic mechanism of aqueous green tea leaf extract on selected bacterial strains.

202 In the research ethanolic extracts of green tea leaves (China Lung Ching), yellow tea leaves (

203 Transformation from green tea leaves into black tea involves oxidation of ca

204 Finally, green tea leaves were infused separately with tap water,

205 il samples increased in the fallowing order: green tea<yellow tea<blackberry<BHT<cranberry<lemon<oil

206 f theanine; hence its highly recommended for green tea manufacture.

207 in gallate (EGCG), a bioactive polyphenol in green tea, mimics metabolic actions of insulin to inhibi

208 te (EGCG), the main antioxidant derived from green tea, on evolving and established atherosclerotic l

209 tumors and green tea-treated tumors (either green tea or polyphenon E) were compared to determine th

210 ic for exposure to effective doses of either green tea or polyphenon E.

211 and lung adenomas when mice were exposed to green tea or polyphenon E.

212 ed for antioxidant activity determination of Green tea, orange juice and asparagus extracts.

213 laboratories around the world suggests that green tea, particularly its major polyphenolic constitue

214 to study the antioxidant activity of banana, green tea, pink guava, and honeydew and the results were

215 ombination with green tea extract (GTE), the green tea polyphenol (-)-epicatechin (EC) or the isomeri

216 The green tea polyphenol (-)-epigallocatechin gallate and th

217 Recently, we have shown that green tea polyphenol (-)-epigallocatechin-3-gallate (EGC

218 hat (-)-epigallocatechin-3-gallate (EGCG), a green tea polyphenol antioxidant, stimulates expression

219 Previously, we showed that the green tea polyphenol epigallocatechin 3-gallate (EGCG) i

220 Previously, we showed that the green tea polyphenol epigallocatechin-3 gallate (EGCG) i

221 less invasive phenotype, the effects of the green tea polyphenol epigallocatechin-3 gallate (EGCG) o

222 athway, i.e., wortmannin, celecoxib, and the green tea polyphenol epigallocatechin-3 gallate, induced

223 We encapsulated green tea polyphenol epigallocatechin-3-gallate (EGCG) i

224 Previously, we showed that the bioactive green tea polyphenol epigallocatechin-3-gallate (EGCG) i

225 Continuous green tea polyphenol infusion for 24 weeks to these mice

226 Furthermore, green tea polyphenol infusion resulted in marked inhibit

227 tory effects of Polyphenon E [a standardized green tea polyphenol preparation containing 65% (-)-epig

228 Activation of hINV promoter activity by the green tea polyphenol, (-)-epigellocathecin-3-gallate, is

229 ary chemopreventive agents, sulforaphane and green tea polyphenol, and that this reduction involves u

230 Green tea polyphenol, epigallocatechin-3-gallate (EGCG)

231 igallocatechin-3-gallate (EGCG), a bioactive green tea polyphenol, on EGFR signaling in cervical cell

232 e that PKC-delta is required for calcium and green tea polyphenol-dependent regulation of end respons

233 P-3 signaling pathway is a prime pathway for green tea polyphenol-mediated inhibition of prostate can

234 Green tea polyphenolic catechins exhibit biological acti

235 Green tea polyphenols (GTP) and Tai Chi (TC) have been s

236 Green tea polyphenols (GTP) are highly effective in inhi

237 We have previously demonstrated that green tea polyphenols (GTP) induce apoptosis in prostate

238 cells, we show that 2-day pretreatment with green tea polyphenols (GTPP) and their active ingredient

239 cluding epigallocatechin-3-gallate (EGCG) in green tea polyphenols (GTPs) and sulforaphane (SFN) in b

240 Consumption of green tea polyphenols (GTPs) in drinking water prevents

241 Recently we demonstrated that a subset of green tea polyphenols are potent inhibitors of glutamate

242 itro studies reveal the possible benefits of green tea polyphenols as cancer therapeutic agents to in

243 Green tea polyphenols can be a useful supplement in the

244 A short-term clinical trial of green tea polyphenols has translated the findings from o

245 Green tea polyphenols have been shown to have efficacy i

246 Green tea polyphenols have both antioxidant and antiinfl

247 We examined the effects of green tea polyphenols in carbon tetrachloride-treated mi

248 during chemoprevention of prostate cancer by green tea polyphenols in TRAMP mice.

249 e earlier demonstrated that oral infusion of green tea polyphenols inhibits development and progressi

250 Taken together, our results indicate that green tea polyphenols may have the potential to negate t

251 Here we report the effects of green tea polyphenols on GDH and insulin secretion.

252 mized intervention trials on the efficacy of green tea polyphenols or extracts are required before a

253 Green tea polyphenols reduce the severity of liver injur

254 The green tea polyphenols seem to interact with the oil bodi

255 rammonemia forms of GDH are inhibited by the green tea polyphenols, epigallocatechin gallate and epic

256 late (EGCG), the major polyphenol present in green tea, possesses potent anti-inflammatory and antipr

257 n-3-gallate (EGCG), the major constituent of green tea, possesses significant anti-inflammatory and c

258 Here, we show that green tea prevents or reverses loss of the epithelial ma

259 in-3-gallate (EGCG), the major polyphenol of green tea, prevents photocarcinogenesis in mice.

260 icacy of BZM and suggest that consumption of green tea products may be contraindicated during cancer

261 looxygenase-2 inhibitors, lycopene, soy, and green tea promising agents.

262 GCG), a catechin-base flavonoid derived from green tea protects retina neurones in situ from ischemia

263 echin-3-gallate (EGCG), a major component of green tea, protects against certain types of cancers, al

264 Clones K-purple and TRFK 91/1 showed high green tea quality indices with the latter doubling with

265 These are the major green tea quality parameters.

266 Berry juice, green tea, red wine, and caffeic acid reduced oxygen upt

267 Green tea reduced LDL (-0.23 mmol/L; -0.34, -0.12; 4 stu

268 ination of soy phytochemical concentrate and green tea reduced serum insulin-like growth factor-I con

269 (EGCG), the main polyphenolic constituent of green tea, reduces Abeta generation in both murine neuro

270 Broccoli added to green tea resulted in a combination of phytochemicals wi

271 3-gallate (EGCG), a polyphenolic compound of green tea, results in activation of p53 and induction of

272 f gene expressions that both offer clues for green tea's potential mechanisms of action and provide a

273 gallate (EGCG) and caffeine in 29 commercial green tea samples available in a Saudi Arabian local mar

274 Analysis (PCA), which grouped the black and green tea samples into 3 different clusters, respectivel

275 tile compounds in seven ready-to-drink (RTD) green tea samples were analysed and quantified using liq

276 owances of lithium for black, Earl Grey, and green tea samples, respectively.

277 B1 (5.4 mug/kg) was also found in one of the green tea samples.

278 Green tea seems to have a positive impact on health due

279 EGCG), the major polyphenolic constituent of green tea, sensitizes TRAIL-resistant LNCaP cells to TRA

280 r amount of polyphenols compared to standard green tea shoot (GL) while epigallocatechin gallate (EGC

281 No melatonin could be detected in black and green tea, sour cherry, sour cherry concentrate, kefir (

282 ceutical products based on natural extracts (green tea, soy, royal jelly and grapes) observing the ap

283 basis of all flavonoids present in black and green tea, soybean, red fruits and so on.

284 when tea polyphenols were administered as a green tea supplement in capsule form and led to a small

285 e therapeutic and toxicological potential of green tea supplementation is evaluated and may provide a

286 s incubated with the mixture of broccoli and green tea than on cells exposed to control infusions.

287 chin gallate (EGCG) is a major polyphenol in green tea that has beneficial effects in the prevention

288 The antioxidant activity was high in green teas though some of the black teas were as efficac

289 gallocatechin gallate (EGCG), a component of green tea, to be considered in the context for neuroprot

290 Gene expression in control tumors and green tea-treated tumors (either green tea or polyphenon

291 e those TUM genes whose expression levels in green tea-treated tumors returned to levels seen in norm

292 tea bag storage on antioxidant properties of green tea was evaluated.

293 Green tea was the most antioxidant herb, although oregan

294 L. algarvense was not toxic, whereas green tea was toxic for S17 cells.

295 never drank tea, regular tea intake (mostly green tea) was associated with reduced risk of all diges

296 20 micromol/L (-)-epigallocatechin gallate (green tea) was restored back to wild-type levels in ATPa

297 GCG), the principal polyphenol isolated from green tea, was recently shown to inhibit Hsp90; however,

298 WEWT, like water extracts of green tea (WEGT) and water extracts of Pu-erh tea (WEPT)

299 bility of lithium from black, Earl Grey, and green teas were evaluated by inductively coupled plasma

300 ns between intakes of coffee, black tea, and green tea with the risk of type 2 diabetes in Singaporea