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1 neurons in the ventrolateral periaqueductal grey.
2 ing the cortical grey matter or the cortical grey and adjacent white matter was observed in 8 patient
3 projections to the amygdala, periaqueductal grey and striatum, and an underactive median raphe nucle
6 and employed these in volumetric analyses of grey and white matter in a subset of 70 patients (progre
7 ndings on cortical grey matter, suggest that grey and white matter integrity at the start of treatmen
13 nce, Global Health, Google Scholar, and Open Grey) and hand-searched the reference lists of relevant
15 bioaccessibility of lithium from black, Earl Grey, and green teas were evaluated by inductively coupl
16 ese two different age groups and diseases, a grey area exists with regard to hepatocellular carcinoma
17 or cingulate cortex (ACC) and periaqueductal grey, areas involved in pain processing, were decreased
18 s for features of scalp hair (shape, colour, greying, balding) and facial hair (beard thickness, mono
19 nstricts content complexity to simple moving grey blobs, allowing objective mechanistic investigation
22 Note the heavy coat of epibiotic bacteria (grey colouring) on the individual in the hottest section
23 isinhibition of ventrolateral periaqueductal grey excitatory outputs to pre-motor targets in the magn
24 on was excavated at the presumed site of the Grey Friars friary in Leicester, the last-known resting
25 influence the distributions of non-breeding grey-headed albatrosses Thalassarche chrysostoma tracked
26 regular polarization microscope are seen as grey image, which contrast disappears at certain orienta
27 microclimatological effects of buildings and grey infrastructure through the urban heat island (UHI)
28 ular mechanisms of the responses of maize to grey leaf spot (GLS) disease caused by Cercospora zeina,
30 EDLINE, Embase, and PsycINFO), internet, and grey literature databases, and disseminated data request
31 We searched Medline, PsycINFO, Embase, and grey literature from 1999 to 2014 to identify 19 reviews
32 tic review we searched for peer-reviewed and grey literature publications reporting associations betw
33 ) by searching MEDLINE, Embase, CENTRAL, and grey literature published between Jan 1, 2005, and Sept
34 explosions at munitions sites, although the grey literature suggests that AXO is a substantial probl
35 cles, scientific conference proceedings, and grey literature up to Aug 31, 2013, to identify randomis
36 cles, scientific conference proceedings, and grey literature up to Aug 31, 2013, to identify randomis
37 hed PubMed, CINAHL, and Embase databases and grey literature using a predetermined search strategy to
39 Literature reviews, conference abstracts and grey literature were excluded from this metasynthesis.
41 eys, Multiple Indicator Cluster Surveys, and grey literature with no date restriction until May 2014.
43 es were identified via electronic databases, grey literature, and conference proceedings through May
44 arched peer-reviewed databases, searched the grey literature, and disseminated data requests to inter
45 nic databases, including Embase and MEDLINE, grey literature, and reference lists for primary researc
46 e collections of source documents, including grey literature, and the food composition data reported
47 wer of floristic studies, often available in grey literature, for understanding long-term biotic chan
52 heart disease were associated with decreased grey matter (GM) and cortical volumes (p < 0.05), while
55 gesting increased transmission time, whereas grey matter (GM) in auditory cortex partially mediates a
58 Our aim was to investigate the impact of grey matter (GM) volume alterations in lobules VI to VII
59 ALS-FTD) showed reduced motor and extramotor grey matter (GM) volume when compared to neurological co
60 h SCI exhibited decreased cord area, reduced grey matter (GM) volumes in anterior cingulate cortex (A
64 ased compared with normal-appearing cortical grey matter (P < 10(-10) and P < 10(-7)), and mean corti
67 in the frontal lobes, affecting the cortical grey matter and adjacent juxtacortical white matter.
68 re, while combining brain-predicted age with grey matter and cerebrospinal fluid volumes (themselves
69 nt-responsive patients include reductions in grey matter and perfusion of frontotemporal regions, and
70 1 years, in four we found moderate to severe grey matter and vascular amyloid-beta (Abeta) pathology.
71 entration (both P < 0.001) were higher while grey matter and white matter intracellular sodium volume
73 pite growing evidence of the role that local grey matter architecture plays in a variety of brain dis
75 es overlap with cytoarchitecturally distinct grey matter areas and may serve as the structural basis
76 using a simple ratio method with cerebellar grey matter as reference tissue, taking into account reg
78 poE levels were associated with greater deep grey matter atrophy (partial correlation rp=-0.28, p<0.0
79 resent study was therefore to assess whether grey matter atrophy and amyloid pathology contribute to
81 of white matter hyperintensity expansion and grey matter atrophy are strongly correlated (Pearson's R
82 hometry was used to characterize patterns of grey matter atrophy associated with task performance.
83 ion tomography and the longitudinal rates of grey matter atrophy in a cohort of clinically diagnosed
84 whereas ApoE levels are associated with deep grey matter atrophy in high risk CIS patients treated wi
85 alysis aims to assess patterns of cerebellar grey matter atrophy in seven neurodegenerative condition
87 al grey matter atrophy, and demonstrate that grey matter atrophy is the major contributor to whole br
89 ion is associated with increases in cortical grey matter atrophy rates, in the medial-frontal, orbito
90 as-based parcellation, and rates of regional grey matter atrophy were assessed using tensor-based mor
91 experienced visual hallucinations, exhibited grey matter atrophy with significant voxel-wise differen
92 ntensities with increasing rates of regional grey matter atrophy, and demonstrate that grey matter at
94 ellar parallel fibres, an example of typical grey matter axons, to investigate the effects of K(+) ch
96 gions of interest were limited to 0.5 cm3 of grey matter centred around sites that had been identifie
97 oups, and availability of coordinate data of grey matter cerebellar atrophy in patients were included
98 efault network, correlated with longitudinal grey matter changes in the non-fluent/agrammatic variant
100 might help explain histological patterns of grey matter connectivity, highlighting that observed con
101 d morphometry in the patient cohort revealed grey matter correlates of auditory motion detection and
104 In patients with multiple sclerosis (MS), grey matter damage is widespread and might underlie many
106 obiological underpinning and correlated with grey matter density in prefrontal and parietal cortex, a
107 region of interest, correcting for regional grey matter density, age, education and disease status,
108 t cortical volume and thickness reduction or grey matter diffusion tensor imaging values alterations
109 menter (attention-getting sounds) differs in grey matter distribution compared to chimpanzees that do
110 les of supratentorial and cerebellar damage (grey matter fraction, T2 lesion volume, metrics of cereb
111 esonance imaging (MRI) measures of white and grey matter in a large population-derived cohort to inve
112 s published up to January 2015 that compared grey matter in MDD (50 data sets including 4101 individu
113 ciated abnormalities of structure (decreased grey matter in right dorsolateral prefrontal cortex and
114 and right inferior temporal gyrus; increased grey matter in right insula, right putamen, left tempora
115 osocial behaviour, associated with decreased grey matter in the anterior insula, lateral orbitofronta
116 tting sounds were characterized by increased grey matter in the ventrolateral prefrontal and dorsal p
117 asking to multiple brain measures, including grey matter in various prefrontal regions and white matt
120 y of magnetisation transfer ratio values and grey matter lesions withint he same regions, and whole-b
125 ery close association between the pattern of grey matter loss and the regions of interest each scale
126 R = -0.69, P < 1 x 10(-7)), and significant grey matter loss and whole brain atrophy occurs annually
127 nd temperature symptoms were associated with grey matter loss in a right-lateralized network includin
129 ound B standard unit value ratio and greater grey matter loss over time in the posterior cingulate gy
135 tion, on-going inflammation, axonal loss and grey matter neuronal injury are likely pathological proc
136 nal damage occurs preferentially in cortical grey matter next to the outer surface of the brain.
137 tigate the normal-appearing white matter and grey matter of subjects with clinically isolated syndrom
138 is with spinal-cord lesions involving mainly grey matter on imaging, or acute cranial nerve dysfuncti
139 s in the frontal lobe affecting the cortical grey matter or the cortical grey and adjacent white matt
140 ientational complexity (DOC), as an index of grey matter pathology in regions associated with decisio
142 16% of white and 14% of mixed cortical and grey matter patient regions showed FA decreases greater
143 sed an expected increase in frontal cortical grey matter perfusion but unexpected perfusion decreases
147 activity, and (iii) a module to compute the Grey Matter Proximity Index, i.e. the distance of each c
148 78, p=0.0087; whole-brain r=0.602, p<0.0001; grey matter r=0.518, p<0.0001; white matter r=0.588, p<0
149 arriers had significantly decreased cortical grey matter rCBF in the occipital lobe (mean difference
150 test two hypotheses: (i) glutamate levels in grey matter regions are abnormal in MS, and (ii) patient
153 terms of links (correlations) between nodes (grey matter regions) and to extract information out of t
157 gh the targets of deep brain stimulation are grey matter structures, axonal modulation is known to pl
158 red with patients' normal-appearing cortical grey matter T2* (paired t-test) and with mean cortical T
159 iated with decision-making and also measured grey matter tissue volumes and white matter lesion volum
161 dy, we tested for differences in subcortical grey matter volume (n = 1157) and white matter integrity
162 ce of a mutation was associated with a lower grey matter volume (P = 0.002), even in presymptomatic s
165 a significant negative relationship between grey matter volume and intrinsic cerebellar connectivity
166 s and show that age-related declines in rPPC grey matter volume better account for age-related change
167 First, we obtained a composite measure of grey matter volume by graph-Laplacian principal componen
170 .e. TMEM106B polymorphism, rs1990622 T/C) on grey matter volume in a large cohort of presymptomatic s
173 have, however, reported reduced hippocampal grey matter volume in MDD and reduced white matter integ
174 bility in patients was related to changes in grey matter volume in pre-supplementary motor area, and
175 nishment; this was associated with decreased grey matter volume in the anterior cingulate, orbitofron
178 muli was significantly associated with lower grey matter volume in the right collateral sulcus, in a
179 hometry, we aimed to determine whether local grey matter volume in the right hemisphere independently
180 for risky rewards in young adults, with less grey matter volume indicating decreased tolerance for ri
181 presumed to be healthy in our sample and its grey matter volume is positively correlated with one's l
182 er's disease exhibited different patterns of grey matter volume loss, with more extensive temporopari
184 healthy aging suggests that diminished rPPC grey matter volume may have a role in modulating risk pr
185 hanged-although less spatially extended-when grey matter volume or 11C-PiB uptake maps were added as
186 There were no significant differences in grey matter volume or structural connectivity between th
188 ubjects show extensive regionally-demarcated grey matter volume reductions in areas that control cogn
190 ses in basal forebrain and entorhinal cortex grey matter volume were interdependent and sequential.
192 Compared to controls, posterior thalamic grey matter volume, an area mediating oxygen regulation,
193 dividuals, did not show typical increases in grey matter volume, and this relative anatomical immatur
194 ith Mini-Mental State Examination scores and grey matter volume, as well as with Pittsburgh compound
195 nce of neuronal injury, measured as regional grey matter volume, in 16 OSA children (8 male, 8.1 +/-
196 equency was associated with lower cerebellar grey matter volume, while patients with posterior cortic
202 ep habits are associated with regional brain grey matter volumes and school grade average in early ad
204 ue ratio and longitudinal change in regional grey matter volumes from an in-house modified atlas.
205 sed analysis of T1 volume scans, we compared grey matter volumes in 12 cases of sudden unexpected dea
206 sleeping hours correlate with smaller brain grey matter volumes in frontal, anterior cingulate, and
208 areas, lesion size, and demographic factors, grey matter volumes in parts of the right temporoparieta
209 lated to language outcomes, we then compared grey matter volumes in patients and healthy controls to
211 ive brain system, manifested through reduced grey matter volumes in the amygdala bilaterally (but not
212 try was then used to determine whether local grey matter volumes in the right hemisphere explained ad
214 hips, partial correlations demonstrated that grey matter volumes in these clusters related to verbal
216 For each subject, cortical and subcortical grey matter volumes were generated using a parcellation
218 ehavior and suggests that alterations in the grey matter volumes, i.e., brain morphology, of specific
219 inhibition, which manifests through reduced grey matter volumes, this region is presumed to be healt
223 sclerosis, T2* in normal-appearing cortical grey matter was significantly increased relative to cont
225 tients and lesions, early neuronal damage in grey matter, and early astrocytic proliferation and acti
226 tion (14/14), present in meninges, white and grey matter, associated with variable tissue destruction
229 minantly affects brain white matter and deep grey matter, resulting in ischaemic damage that ranges f
230 , together with earlier findings on cortical grey matter, suggest that grey and white matter integrit
231 Based on the relative sparing of frontal grey matter, we propose to redefine these clinical syndr
232 maps were calculated for each tissue class (grey matter, white matter, white matter hyperintensities
233 extensive spinal-cord lesions of the central grey matter, with predominant anterior horn-cell involve
234 Mediation analyses revealed both direct and grey matter-mediated effects of 18F-AV-1451 uptake on co
238 ommon type of axons in the mammalian brain's grey matter.We used rat cerebellar parallel fibres, an e
239 sed for PML lesion distribution, appearance, grey matter/white matter involvement and possible signs
240 and eight normal-appearing white-matter and grey-matter regions) and from three controls with non-ne
242 Both disorders were associated with lower grey-matter volume relative to healthy individuals in a
243 mparisons indicated that findings of smaller grey-matter volumes relative to controls in the right do
247 ns showed gram-negative bacilli and regular, grey non-hemolytic colonies appearing the next day on bl
248 de contacts in terms of both relationship to grey or white matter and location in specific brain regi
249 , medial frontal pole (mFP) and periaquiduct grey (PAG) are significantly greater in the verum acupun
250 in individual columns of the periaqueductal grey (PAG) during breathlessness and its conditioned ant
252 halamic nucleus, STN) and the periaqueductal grey (PAG), which have now been recorded from in humans
253 s 128 nm, at a length scale that exceeds the grey phonon mean-free path in this material by almost an
257 imated a density of 21.3 (95% CI 17.8, 24.7) grey reef sharks/km(2), which is an order of magnitude l
258 The dorsal raphe nucleus/ periaqueductal grey region of the midbrain and hippocampus were found t
260 gorithm in terms of visual inspection of the grey scale plots , defect area and defect severity.
262 tom-up synthetic biology approach can detect grey-scale images and patterns of light moving across th
264 S equipped with thermal sensors, imaging two grey seal (Halichoerus grypus) breeding colonies in east
266 om rectal swabs of free-ranging and stranded grey seal pups (21.1%; 37/175) and compared with strains
267 location and dive data from recently-weaned grey seal pups from two regions of the United Kingdom (t
271 othermal species: causing severe declines in grey seals, cod, herring and haddock, while eurythermal
274 indicate the appearance of bright, dark and grey solitons dwelling in the vicinity of the boundary o
277 amygdala to the ventrolateral periaqueductal grey that produces freezing by disinhibition of ventrola
280 nd/or duration, with the color changing from grey to red with time of exposure at high temperature (4
281 howed color changes from light yellow, light grey, to dark grey with the increasing amount of NaBH4.
283 hin-group analyses, increased periaqueductal grey volume was associated with role limitations due to
285 animal waste (e.g., urine, fecal sludge, or grey water) is a critical step in reducing the spread of
286 e-Life Depression: Getting to Remission [IRL-GRey]), we evaluated the effects of the following potent
287 l plate, penetrating cortical blood vessels, grey-white matter junctions, and structures lining the v
288 Our results indicate that the evolution of grey/white matter injury and blood-brain barrier disrupt
291 the macular region of both eyes, parafoveal greying with crystalline deposits and changes in retinal
292 study involving woodland caribou subject to grey wolf predation, DeCesare et al. (2014) show that wh
294 bility, recruitment and population growth of grey wolves (Canis lupus) in Denali National Park and Pr
298 rrays in genotyping noninvasive samples from grey wolves, European wildcats and brown bears, and we c
299 une-tolerant (IT), immune-inactive (IC), and grey zone (GZ), respectively, showed active antiviral cy
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