ライフサイエンスコーパス: grey

1 neurons in the ventrolateral periaqueductal grey.

2 ing the cortical grey matter or the cortical grey and adjacent white matter was observed in 8 patient

3 projections to the amygdala, periaqueductal grey and striatum, and an underactive median raphe nucle

4 ovide the first evidence that they relate to grey and white matter abnormalities seen using MRI.

5 Longitudinal grey and white matter changes were found in the left lan

6 and employed these in volumetric analyses of grey and white matter in a subset of 70 patients (progre

7 ndings on cortical grey matter, suggest that grey and white matter integrity at the start of treatmen

8 2%) presented with a combination of cortical grey and white matter involvement.

9 l diffusivity values were calculated in both grey and white matter of spinal cord.

10 Previous reports of altered grey and white matter structure in Major Depressive Diso

11 Brain grey and white matter volume and diffusion tensor imagin

12 enchyma and fluids, and at junctions between grey and white matter.

13 nce, Global Health, Google Scholar, and Open Grey) and hand-searched the reference lists of relevant

14 ietary allowances of lithium for black, Earl Grey, and green tea samples, respectively.

15 bioaccessibility of lithium from black, Earl Grey, and green teas were evaluated by inductively coupl

16 ese two different age groups and diseases, a grey area exists with regard to hepatocellular carcinoma

17 or cingulate cortex (ACC) and periaqueductal grey, areas involved in pain processing, were decreased

18 s for features of scalp hair (shape, colour, greying, balding) and facial hair (beard thickness, mono

19 nstricts content complexity to simple moving grey blobs, allowing objective mechanistic investigation

20 We studied stopover behaviour of Grey-cheeked Thrush (Catharus minimus) at a site in nort

21 ng methods to define midbrain periaqueductal grey circuits for specific defensive behaviours.

22 Note the heavy coat of epibiotic bacteria (grey colouring) on the individual in the hottest section

23 isinhibition of ventrolateral periaqueductal grey excitatory outputs to pre-motor targets in the magn

24 on was excavated at the presumed site of the Grey Friars friary in Leicester, the last-known resting

25 influence the distributions of non-breeding grey-headed albatrosses Thalassarche chrysostoma tracked

26 regular polarization microscope are seen as grey image, which contrast disappears at certain orienta

27 microclimatological effects of buildings and grey infrastructure through the urban heat island (UHI)

28 ular mechanisms of the responses of maize to grey leaf spot (GLS) disease caused by Cercospora zeina,

29 The grey levels (GLs) from 8 circularly arranged segments po

30 EDLINE, Embase, and PsycINFO), internet, and grey literature databases, and disseminated data request

31 We searched Medline, PsycINFO, Embase, and grey literature from 1999 to 2014 to identify 19 reviews

32 tic review we searched for peer-reviewed and grey literature publications reporting associations betw

33 ) by searching MEDLINE, Embase, CENTRAL, and grey literature published between Jan 1, 2005, and Sept

34 explosions at munitions sites, although the grey literature suggests that AXO is a substantial probl

35 cles, scientific conference proceedings, and grey literature up to Aug 31, 2013, to identify randomis

36 cles, scientific conference proceedings, and grey literature up to Aug 31, 2013, to identify randomis

37 hed PubMed, CINAHL, and Embase databases and grey literature using a predetermined search strategy to

38 ches, reference checking and searches of the grey literature were also undertaken.

39 Literature reviews, conference abstracts and grey literature were excluded from this metasynthesis.

40 Healthcare databases and sources of grey literature were searched in July 2013.

41 eys, Multiple Indicator Cluster Surveys, and grey literature with no date restriction until May 2014.

42 re mixed-methods studies, and four [7%] were grey literature).

43 es were identified via electronic databases, grey literature, and conference proceedings through May

44 arched peer-reviewed databases, searched the grey literature, and disseminated data requests to inter

45 nic databases, including Embase and MEDLINE, grey literature, and reference lists for primary researc

46 e collections of source documents, including grey literature, and the food composition data reported

47 wer of floristic studies, often available in grey literature, for understanding long-term biotic chan

48 Further data sources were: websites, grey literature, research in progress databases, hand-se

49 es, including PubMed and Web of Science, and grey literature.

50 ffects, so we extended the search to include grey literature.

51 reviewed research, organisation reports, and grey literature.

52 heart disease were associated with decreased grey matter (GM) and cortical volumes (p < 0.05), while

53 White matter (WM), grey matter (GM) and thalamic fractions were derived at

54 Grey matter (GM) density and hippocampal volume from the

55 gesting increased transmission time, whereas grey matter (GM) in auditory cortex partially mediates a

56 Cortical grey matter (GM) lesions are common in multiple sclerosi

57 The extent and clinical relevance of grey matter (GM) pathology in multiple sclerosis (MS) ar

58 Our aim was to investigate the impact of grey matter (GM) volume alterations in lobules VI to VII

59 ALS-FTD) showed reduced motor and extramotor grey matter (GM) volume when compared to neurological co

60 h SCI exhibited decreased cord area, reduced grey matter (GM) volumes in anterior cingulate cortex (A

61 Cross-sectional areas of grey matter (GM), white matter (WM), and posterior colum

62 ination and neuronal loss in the spinal cord grey matter (GM).

63 ogen peroxide-reducing enzyme catalase in MS grey matter (GM).

64 ased compared with normal-appearing cortical grey matter (P < 10(-10) and P < 10(-7)), and mean corti

65 onal tissue volumes for the subcortical deep grey matter (SDGM) structures were also obtained.

66 Localized grey matter abnormalities were detected in a topographic

67 in the frontal lobes, affecting the cortical grey matter and adjacent juxtacortical white matter.

68 re, while combining brain-predicted age with grey matter and cerebrospinal fluid volumes (themselves

69 nt-responsive patients include reductions in grey matter and perfusion of frontotemporal regions, and

70 1 years, in four we found moderate to severe grey matter and vascular amyloid-beta (Abeta) pathology.

71 entration (both P < 0.001) were higher while grey matter and white matter intracellular sodium volume

72 Global grey matter and white matter total sodium concentration

73 pite growing evidence of the role that local grey matter architecture plays in a variety of brain dis

74 Imaging measures of the grey matter are necessary, but not sufficient to fully c

75 es overlap with cytoarchitecturally distinct grey matter areas and may serve as the structural basis

76 using a simple ratio method with cerebellar grey matter as reference tissue, taking into account reg

77 The segmentation performance for grey matter at C2/C3 level was close to inter-rater vari

78 poE levels were associated with greater deep grey matter atrophy (partial correlation rp=-0.28, p<0.0

79 resent study was therefore to assess whether grey matter atrophy and amyloid pathology contribute to

80 Conversely, increased rates of global grey matter atrophy are significantly associated with fa

81 of white matter hyperintensity expansion and grey matter atrophy are strongly correlated (Pearson's R

82 hometry was used to characterize patterns of grey matter atrophy associated with task performance.

83 ion tomography and the longitudinal rates of grey matter atrophy in a cohort of clinically diagnosed

84 whereas ApoE levels are associated with deep grey matter atrophy in high risk CIS patients treated wi

85 alysis aims to assess patterns of cerebellar grey matter atrophy in seven neurodegenerative condition

86 Grey matter atrophy in the hallucinators occurred predom

87 al grey matter atrophy, and demonstrate that grey matter atrophy is the major contributor to whole br

88 ls was associated with a 1% increase in deep grey matter atrophy over 2 years.

89 ion is associated with increases in cortical grey matter atrophy rates, in the medial-frontal, orbito

90 as-based parcellation, and rates of regional grey matter atrophy were assessed using tensor-based mor

91 experienced visual hallucinations, exhibited grey matter atrophy with significant voxel-wise differen

92 ntensities with increasing rates of regional grey matter atrophy, and demonstrate that grey matter at

93 tter hyperintensity progression and cortical grey matter atrophy?

94 ellar parallel fibres, an example of typical grey matter axons, to investigate the effects of K(+) ch

95 White matter connections between 165 grey matter brain regions were defined using tractograph

96 gions of interest were limited to 0.5 cm3 of grey matter centred around sites that had been identifie

97 oups, and availability of coordinate data of grey matter cerebellar atrophy in patients were included

98 efault network, correlated with longitudinal grey matter changes in the non-fluent/agrammatic variant

99 Here, we present a new model of intrinsic grey matter connectivity of the human connectome.

100 might help explain histological patterns of grey matter connectivity, highlighting that observed con

101 d morphometry in the patient cohort revealed grey matter correlates of auditory motion detection and

102 Grey matter cortical thickness and shape analysis reveal

103 This relation between grey matter damage and cognitive impairment has been len

104 In patients with multiple sclerosis (MS), grey matter damage is widespread and might underlie many

105 Specifically, we derived grey matter density and standardized uptake value ratios

106 obiological underpinning and correlated with grey matter density in prefrontal and parietal cortex, a

107 region of interest, correcting for regional grey matter density, age, education and disease status,

108 t cortical volume and thickness reduction or grey matter diffusion tensor imaging values alterations

109 menter (attention-getting sounds) differs in grey matter distribution compared to chimpanzees that do

110 les of supratentorial and cerebellar damage (grey matter fraction, T2 lesion volume, metrics of cereb

111 esonance imaging (MRI) measures of white and grey matter in a large population-derived cohort to inve

112 s published up to January 2015 that compared grey matter in MDD (50 data sets including 4101 individu

113 ciated abnormalities of structure (decreased grey matter in right dorsolateral prefrontal cortex and

114 and right inferior temporal gyrus; increased grey matter in right insula, right putamen, left tempora

115 osocial behaviour, associated with decreased grey matter in the anterior insula, lateral orbitofronta

116 tting sounds were characterized by increased grey matter in the ventrolateral prefrontal and dorsal p

117 asking to multiple brain measures, including grey matter in various prefrontal regions and white matt

118 Loss of cortical grey matter is a diagnostic marker of many neurodegenera

119 We then prepared grey matter lesion maps, based on meta-analyses of publi

120 y of magnetisation transfer ratio values and grey matter lesions withint he same regions, and whole-b

121 requent bilateral, large, brainstem and deep grey matter lesions.

122 , in order to discriminate between white and grey matter location of contacts.

123 plain the absence of findings for consistent grey matter loss across studies.

124 Voxel-based morphometry confirmed grey matter loss across the motor and cognitive cerebell

125 ery close association between the pattern of grey matter loss and the regions of interest each scale

126 R = -0.69, P < 1 x 10(-7)), and significant grey matter loss and whole brain atrophy occurs annually

127 nd temperature symptoms were associated with grey matter loss in a right-lateralized network includin

128 That grey matter loss in parietal regions is a part of health

129 ound B standard unit value ratio and greater grey matter loss over time in the posterior cingulate gy

130 Regional grey matter loss was determined on three-dimensional T1-

131 and perform morphometric analyses to assess grey matter loss.

132 echanisms, including, but not restricted to, grey matter loss.

133 by-voxel morphometry revealed no significant grey matter loss.

134 Using grey matter morphometry and probabilistic tractography c

135 tion, on-going inflammation, axonal loss and grey matter neuronal injury are likely pathological proc

136 nal damage occurs preferentially in cortical grey matter next to the outer surface of the brain.

137 tigate the normal-appearing white matter and grey matter of subjects with clinically isolated syndrom

138 is with spinal-cord lesions involving mainly grey matter on imaging, or acute cranial nerve dysfuncti

139 s in the frontal lobe affecting the cortical grey matter or the cortical grey and adjacent white matt

140 ientational complexity (DOC), as an index of grey matter pathology in regions associated with decisio

141 Grey matter pathology was identified through analysis of

142 16% of white and 14% of mixed cortical and grey matter patient regions showed FA decreases greater

143 sed an expected increase in frontal cortical grey matter perfusion but unexpected perfusion decreases

144 In addition, when confined to the grey matter perfusion deficit, intracellular pH (P < 0.0

145 Central grey matter PK11195 BPND was increased in subjects with

146 rols but no difference was found in cortical grey matter PK11195 BPND.

147 activity, and (iii) a module to compute the Grey Matter Proximity Index, i.e. the distance of each c

148 78, p=0.0087; whole-brain r=0.602, p<0.0001; grey matter r=0.518, p<0.0001; white matter r=0.588, p<0

149 arriers had significantly decreased cortical grey matter rCBF in the occipital lobe (mean difference

150 test two hypotheses: (i) glutamate levels in grey matter regions are abnormal in MS, and (ii) patient

151 ch catalyses hydroxymethylation in white and grey matter regions of this animal model.

152 In the brain, grey matter regions were parcellated with Freesurfer and

153 terms of links (correlations) between nodes (grey matter regions) and to extract information out of t

154 and varying degrees of degeneration of other grey matter regions.

155 s (STEPS) algorithm for segmenting white and grey matter simultaneously.

156 These findings suggest that the grey matter structure of right hemisphere posterior dors

157 gh the targets of deep brain stimulation are grey matter structures, axonal modulation is known to pl

158 red with patients' normal-appearing cortical grey matter T2* (paired t-test) and with mean cortical T

159 iated with decision-making and also measured grey matter tissue volumes and white matter lesion volum

160 In responders across 9 datasets grey matter volume (GMV) was significantly higher in the

161 dy, we tested for differences in subcortical grey matter volume (n = 1157) and white matter integrity

162 ce of a mutation was associated with a lower grey matter volume (P = 0.002), even in presymptomatic s

163 ope of the correlation between education and grey matter volume (P = 0.007).

164 We found that impaired cortical grey matter volume and gyrification index in newborns wi

165 a significant negative relationship between grey matter volume and intrinsic cerebellar connectivity

166 s and show that age-related declines in rPPC grey matter volume better account for age-related change

167 First, we obtained a composite measure of grey matter volume by graph-Laplacian principal componen

168 TMEM106B genotype did not influence grey matter volume directly on its own but in mutation c

169 T2 lesion volume, and brain white matter and grey matter volume fractions.

170 .e. TMEM106B polymorphism, rs1990622 T/C) on grey matter volume in a large cohort of presymptomatic s

171 Grey matter volume in a region of right posterior pariet

172 Education directly affected grey matter volume in all the samples (P = 0.02) with lo

173 have, however, reported reduced hippocampal grey matter volume in MDD and reduced white matter integ

174 bility in patients was related to changes in grey matter volume in pre-supplementary motor area, and

175 nishment; this was associated with decreased grey matter volume in the anterior cingulate, orbitofron

176 We found that local grey matter volume in the left anterior superior tempora

177 We identified increased grey matter volume in the right anterior hippocampus/amy

178 muli was significantly associated with lower grey matter volume in the right collateral sulcus, in a

179 hometry, we aimed to determine whether local grey matter volume in the right hemisphere independently

180 for risky rewards in young adults, with less grey matter volume indicating decreased tolerance for ri

181 presumed to be healthy in our sample and its grey matter volume is positively correlated with one's l

182 er's disease exhibited different patterns of grey matter volume loss, with more extensive temporopari

183 between each rating scale and the pattern of grey matter volume loss.

184 healthy aging suggests that diminished rPPC grey matter volume may have a role in modulating risk pr

185 hanged-although less spatially extended-when grey matter volume or 11C-PiB uptake maps were added as

186 There were no significant differences in grey matter volume or structural connectivity between th

187 Significant grey matter volume reductions appeared in OSA throughout

188 ubjects show extensive regionally-demarcated grey matter volume reductions in areas that control cogn

189 Results showed that total cerebellar grey matter volume was robustly reduced in SZ relative t

190 ses in basal forebrain and entorhinal cortex grey matter volume were interdependent and sequential.

191 Increased amygdalo-hippocampal grey matter volume with right-sided changes is consisten

192 Compared to controls, posterior thalamic grey matter volume, an area mediating oxygen regulation,

193 dividuals, did not show typical increases in grey matter volume, and this relative anatomical immatur

194 ith Mini-Mental State Examination scores and grey matter volume, as well as with Pittsburgh compound

195 nce of neuronal injury, measured as regional grey matter volume, in 16 OSA children (8 male, 8.1 +/-

196 equency was associated with lower cerebellar grey matter volume, while patients with posterior cortic

197 l attainment; and (iii) TMEM106B genotype on grey matter volume.

198 ons withint he same regions, and whole-brain grey matter volume.

199 nt associations with cerebral blood flow and grey matter volume.

200 en cognition and both 18F-AV-1451 uptake and grey matter volume.

201 Here, we examined relative grey matter volumes (rGMVs) between three cortical netwo

202 ep habits are associated with regional brain grey matter volumes and school grade average in early ad

203 lly, the ageing brain atrophies as white and grey matter volumes decrease.

204 ue ratio and longitudinal change in regional grey matter volumes from an in-house modified atlas.

205 sed analysis of T1 volume scans, we compared grey matter volumes in 12 cases of sudden unexpected dea

206 sleeping hours correlate with smaller brain grey matter volumes in frontal, anterior cingulate, and

207 iates with later weekend bedtime and smaller grey matter volumes in medial brain regions.

208 areas, lesion size, and demographic factors, grey matter volumes in parts of the right temporoparieta

209 lated to language outcomes, we then compared grey matter volumes in patients and healthy controls to

210 Grey matter volumes in right temporoparietal clusters we

211 ive brain system, manifested through reduced grey matter volumes in the amygdala bilaterally (but not

212 try was then used to determine whether local grey matter volumes in the right hemisphere explained ad

213 Further, grey matter volumes in these areas were greater in strok

214 hips, partial correlations demonstrated that grey matter volumes in these clusters related to verbal

215 In brain areas in which grey matter volumes related to language outcomes, we the

216 For each subject, cortical and subcortical grey matter volumes were generated using a parcellation

217 Mapping toolbox to further control for local grey matter volumes, 11C-PiB uptake, or both.

218 ehavior and suggests that alterations in the grey matter volumes, i.e., brain morphology, of specific

219 inhibition, which manifests through reduced grey matter volumes, this region is presumed to be healt

220 be associated with cingulate and prefrontal grey matter volumes.

221 amyloid burden, but are in part mediated by grey matter volumes.

222 The cortical grey matter was affected in 15 patients (83.3%) and 13 p

223 sclerosis, T2* in normal-appearing cortical grey matter was significantly increased relative to cont

224 The Abeta deposition in the grey matter was typical of that seen in Alzheimer's dise

225 tients and lesions, early neuronal damage in grey matter, and early astrocytic proliferation and acti

226 tion (14/14), present in meninges, white and grey matter, associated with variable tissue destruction

227 sponders show different alterations in brain grey matter, but the findings are inconsistent.

228 ed progressive and widespread changes in the grey matter, notably including the basal ganglia.

229 minantly affects brain white matter and deep grey matter, resulting in ischaemic damage that ranges f

230 , together with earlier findings on cortical grey matter, suggest that grey and white matter integrit

231 Based on the relative sparing of frontal grey matter, we propose to redefine these clinical syndr

232 maps were calculated for each tissue class (grey matter, white matter, white matter hyperintensities

233 extensive spinal-cord lesions of the central grey matter, with predominant anterior horn-cell involve

234 Mediation analyses revealed both direct and grey matter-mediated effects of 18F-AV-1451 uptake on co

235 he contours of strain and strain rate at the grey matter-white matter boundary were mapped.

236 rain injury patients were also mapped at the grey matter-white matter boundary.

237 subpial region-and in spinal cord white and grey matter.

238 ommon type of axons in the mammalian brain's grey matter.We used rat cerebellar parallel fibres, an e

239 sed for PML lesion distribution, appearance, grey matter/white matter involvement and possible signs

240 and eight normal-appearing white-matter and grey-matter regions) and from three controls with non-ne

241 ised by both common and distinct patterns of grey-matter volume changes.

242 Both disorders were associated with lower grey-matter volume relative to healthy individuals in a

243 mparisons indicated that findings of smaller grey-matter volumes relative to controls in the right do

244 alding, and the first reported loci for hair greying, monobrow, eyebrow and beard thickness.

245 egetables and flowers significantly inhibits grey mould disease.

246 Disruption of SEP4 in the plant grey mould fungus Botrytis cinerea completely blocked IF

247 ns showed gram-negative bacilli and regular, grey non-hemolytic colonies appearing the next day on bl

248 de contacts in terms of both relationship to grey or white matter and location in specific brain regi

249 , medial frontal pole (mFP) and periaquiduct grey (PAG) are significantly greater in the verum acupun

250 in individual columns of the periaqueductal grey (PAG) during breathlessness and its conditioned ant

251 The midbrain periaqueductal grey (PAG) lies at the heart of the defence-arousal syst

252 halamic nucleus, STN) and the periaqueductal grey (PAG), which have now been recorded from in humans

253 s 128 nm, at a length scale that exceeds the grey phonon mean-free path in this material by almost an

254 nsky-Pudlak syndrome, May-Hegglin anomaly or grey platelet syndrome.

255 MLAMP, was also developed using ML-SMOTE and grey pseudo amino acid composition.

256 e and most abundant predator at Palmyra, the grey reef shark (Carcharhinus amblyrhynchos).

257 imated a density of 21.3 (95% CI 17.8, 24.7) grey reef sharks/km(2), which is an order of magnitude l

258 The dorsal raphe nucleus/ periaqueductal grey region of the midbrain and hippocampus were found t

259 Transabdominal grey scale and real time 3D ultrasound (US) was done wit

260 gorithm in terms of visual inspection of the grey scale plots , defect area and defect severity.

261 Grey-scale face classification is experimentally demonst

262 tom-up synthetic biology approach can detect grey-scale images and patterns of light moving across th

263 fter a V1 lesion while the monkeys fixated a grey screen.

264 S equipped with thermal sensors, imaging two grey seal (Halichoerus grypus) breeding colonies in east

265 Bovismorbificans may circulate between grey seal and cattle populations and that both S. Typhim

266 om rectal swabs of free-ranging and stranded grey seal pups (21.1%; 37/175) and compared with strains

267 location and dive data from recently-weaned grey seal pups from two regions of the United Kingdom (t

268 Grey seals (Halichoerus grypus) are typical capital bree

269 Milk was collected from Atlantic grey seals (Halichoerus grypus) periodically from birth

270 ng top-down control by their main predators (grey seals and saithe).

271 othermal species: causing severe declines in grey seals, cod, herring and haddock, while eurythermal

272 ene expression in blubber explants from wild grey seals.

273 ow 400 nm the two-beam interference produces grey shades only.

274 indicate the appearance of bright, dark and grey solitons dwelling in the vicinity of the boundary o

275 the Epi-group (event-free survival curves by Grey-test, P=0.03).

276 nts in the Endo and Epi-Group, respectively (Grey-test, P=0.2).

277 amygdala to the ventrolateral periaqueductal grey that produces freezing by disinhibition of ventrola

278 e amygdala, hypothalamus, and periaqueductal grey to angry facial expressions.

279 ins) with similar colourations, ranging from grey to green brownish.

280 nd/or duration, with the color changing from grey to red with time of exposure at high temperature (4

281 howed color changes from light yellow, light grey, to dark grey with the increasing amount of NaBH4.

282 The ventrolateral periaqueductal grey (vlPAG) has a well-established role in fear-evoked

283 hin-group analyses, increased periaqueductal grey volume was associated with role limitations due to

284 n the abundance of a native insectivore, the grey warbler (Gerygone igata).

285 animal waste (e.g., urine, fecal sludge, or grey water) is a critical step in reducing the spread of

286 e-Life Depression: Getting to Remission [IRL-GRey]), we evaluated the effects of the following potent

287 l plate, penetrating cortical blood vessels, grey-white matter junctions, and structures lining the v

288 Our results indicate that the evolution of grey/white matter injury and blood-brain barrier disrupt

289 uptake value ratio normalized to cerebellum grey/white matter.

290 anges from light yellow, light grey, to dark grey with the increasing amount of NaBH4.

291 the macular region of both eyes, parafoveal greying with crystalline deposits and changes in retinal

292 study involving woodland caribou subject to grey wolf predation, DeCesare et al. (2014) show that wh

293 s breeder turnover in cooperatively breeding grey wolves (Canis lupus Linnaeus 1758).

294 bility, recruitment and population growth of grey wolves (Canis lupus) in Denali National Park and Pr

295 We explored multiple linkages among grey wolves (Canis lupus), elk (Cervus elaphus), berry-p

296 of sarcoptic mange (Sarcoptes scabiei) among grey wolves (Canis lupus).

297 For example, in North America, grey wolves Canis lupus are known to kill coyotes Canis

298 rrays in genotyping noninvasive samples from grey wolves, European wildcats and brown bears, and we c

299 une-tolerant (IT), immune-inactive (IC), and grey zone (GZ), respectively, showed active antiviral cy

300 This diagnostic grey zone should be considered in clinical management de