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1 , were taller, more muscular, and had higher grip strength.
2 ysial collagen III deposition, and hind limb grip strength.
3 ociation was attenuated after adjustment for grip strength.
4 uch as elevated pain sensitivity and reduced grip strength.
5 dismutase in spinal motor neurons preserved grip strength.
6 These findings were not explained by grip strength.
7 was generally associated only with increased grip strength.
8 ysmetria and dysdiadochokinesia but not with grip strength.
9 ation, speech and swallowing assessments and grip strength.
10 that was 30 percent of the maximal voluntary grip strength.
11 dent OA at different hand joints and maximal grip strength.
12 trically measured flexed arm muscle mass and grip strength.
13 tion of fibrosis and restoration of forelimb grip strength.
14 were more robust for walking speed than for grip strength.
15 uding robust hind limb elements modified for grip strength.
16 , mechanical hypersensitivity, and decreased grip strength.
17 paired motor control, exercise capacity, and grip strength.
18 tands, 1.9 for standing balance, and 1.7 for grip strength.
19 -m walk, chair stands, standing balance) and grip strength.
20 in, hair, and eye color, blood pressure, and grip strength.
21 .52, p = .001) and left (r = 0.50, p = .002) grip strength.
22 er 3 months on the waiting list: -0.38 kg in grip strength, -0.05 meters/second in gait, 0.03 seconds
23 weight-supported hanging (43% increase) and grip strength (25% increase), were obtained after hypoth
24 he WMFT (-1.39 kg, -2.74 to -0.04), for WMFT grip strength (-4.39 kg, -6.91 to -1.86), for amount of
25 0.26 kg, P < 0.001; 11 studies, n = 308) and grip strength (5.3%, P < 0.050; 4 studies, n = 156), whi
26 ed swollen and tender joint count and score, grip strength, 50-foot walking time, duration of morning
27 e we examine the morphological correlates of grip strength, a defensive combat trait involved in mate
28 ivation of the Col12a1 gene showed decreased grip strength, a delay in fiber-type transition and a de
30 5% confidence interval: 1.04, 6.84), and low grip strength (adjusted odds ratio = 3.29, 95% confidenc
32 ge, sex, race, cognition, comorbidities, and grip strength, AMD subjects showed an increased likeliho
33 ons of radiographic variables with pinch and grip strength among individuals with radiographic hand O
34 sed risk (95% CI, 1%-23%) of developing weak grip strength and a 14% decreased risk (95% CI, 8%-20%)
35 ulted in significant improvement in hindlimb grip strength and a 30% decrease in inflammation in the
36 idisciplinary expert team measured patients' grip strength and assessed their predicted mobilization
38 ligible patients had low performance on hand grip strength and chair rise tests, tested with the proc
39 decline in normalized forelimb and hindlimb grip strength and declines in in vitro EDL force after r
40 observed for cardiovascular mortality.Lower grip strength and excess adiposity are both independent
42 th performance; those in the lowest fifth of grip strength and highest fifth of BMI having particular
43 No association was found between maximal grip strength and incident OA in the DIP joints of men o
45 ured using dual energy x-ray absorptiometry; grip strength and information on lifestyle indicators, i
51 from 5.0 to 2.9 mm; P = .02), but changes in grip strength and pain were not significant for control
53 havioral (Digiscan) and functional outcomes (grip strength and Rotarod) were assessed prior to sacrif
54 function, assessed by skilled paw reaching, grip strength and sensory testing varied with accuracy o
55 to reduce the likelihood of developing weak grip strength and slow walking speed because purpose has
57 ted with a decreased risk of developing weak grip strength and slow walking speed, although the findi
58 nsight into the mechanistic underpinnings of grip strength and the causal role of muscular strength i
60 with a control group differed on functional (grip strength and walk time) and disease activity (total
62 score, a 1.12-kg (95% CI: 0.83, 1.40) lower grip strength, and a 4.7-nm (95% CI: 3.5, 5.9) lower kne
63 ts in dystonic movements, motor performance, grip strength, and body weight that progressively worsen
64 nce score (sum of quartiles of walking pace, grip strength, and chair-stand speed; range, 0-9) were a
66 ositive effects of exercise on walk time and grip strength, and demonstrated that fatigue and perceiv
68 hy Impairment Score of the Lower Limbs, hand grip strength, and evaluation of vegetative dysfunction,
69 strength, measured by hindlimb and forelimb grip strength, and heat nociception, measured by tail-fl
71 -min walking distance, fast gait speed, hand grip strength, and isometric leg extension strength).
73 ipants were stratified by country, age, hand grip strength, and performance on the chair rise test, a
76 ere the number of tender and swollen joints, grip strength, and the erythrocyte sedimentation rate (E
82 otor functions, including breathing pattern, grip strength, balance beam and rotarod performance.
83 treated mice exhibited improved body weight, grip strength, bone integrity, and percent survival at 2
84 icantly increased body weight, lean mass and grip strength by 60-80% over vehicle-treated mdx mice.
87 r adults in the home, body mass index (BMI), grip strength, cognitive ability, mood, or comorbid illn
88 demonstrated improved downward climbing and grip strength compared with those given vehicle, though
89 statistically significant (14 days) delay in grip strength decline but not in the onset of paralysis,
92 9; 95% CI, 0.83-0.95), but associations with grip strength did not reach conventional levels of stati
95 stimulation, and behavioural tests including grip strength, double simultaneous stimulation and joint
96 ound; African-American HRT users had greater grip strength during the study, whereas Caucasian HRT us
97 walk, 5 chair stands, standing balance, and grip strength, each scored from 0 to 4 (0, unable to per
98 but other measurements of strength (forelimb grip strength, ex vivo measurements of contractile funct
99 ds ratio = 2.43; 95% CI, 1.17-5.03) and poor grip strength, exhaustion, and slowed walking speed (haz
104 ailty was measured on a scale from 0 to 5 by grip strength, gait speed, exhaustion, shrinkage, and ph
107 (HDL) cholesterol, forced expiratory volume, grip strength, HbA1c, longevity, obesity, self-rated hea
108 he last year were inversely related to adult grip strength, height, and walking speed in men and wome
109 mmonia, increase in lean body mass, improved grip strength, higher skeletal muscle mass and diameter,
110 .21; 95% CI: 1.32, 3.71) and/or reduced hand grip strength (HR: 1.53; 95% CI: 10.07, 2.17) than in th
111 complex to calcium improves muscle force and grip strength immediately after administration of single
112 d the relation between birth weight and hand grip strength in a prospective national birth cohort of
119 baseline to endpoint were also recorded for grip strength in the dominant hand (treatment difference
120 e yoga groups had significant improvement in grip strength (increased from 162 to 187 mm Hg; P = .009
122 e rates are higher when the HAQ, rather than grip strength, is used to measure physical function.
123 nce intervals -16.786 to -4.482) decrease in grip strength (kg force) (P < 0.001) and -8.74 (95% conf
124 Truncal flexion and extension strength, hand grip strength, leg extension power, and quality of life
126 >/=30) in the lowest tertile of sex-specific grip strength (<35.3 kg for men and <19.6 kg for women).
128 articipants viewing TV >/= 6 hrs/d had lower grip strength (Men, B = -1.20 kg, 95% CI, -2.26, -0.14;
129 ast, internet use was associated with higher grip strength (Men, B = 2.43 kg, 95% CI, 1.74, 3.12; Wom
130 ded hindlimb and forelimb muscle strength by Grip Strength Meter and quantitative muscle fibrosis par
131 onal deficits were tested on the rotarod and grip-strength meter at 24, 48 and 72 h after pMCAO.
134 ain on an accelerating rotarod and increased grip strength observed in the pMCAO rats treated with PR
135 t 10 IU/day (but not at 1 IU/day), increased grip strength of the contralesional paretic forelimb and
143 ght changes, lifespan, RotaRod performances, grip strength, overall activity and no significant effec
145 Participants with CMT2A had the weakest grip strength (P < .05), while those with CMT2A and CMT4
146 The only ray significantly associated with grip strength (P < 0.05) was ray 1, and no individual ra
147 (P = .03) and B (P = .05), right-sided Jamar grip strength (P = .02), Rapid Pace Walk (P = .03), Brak
149 e postmenopausal showed a 1.04-kg decline in grip strength (p = 0.10) and a 0.57-kg decline in pinch
151 of the erythrocyte sedimentation rate (ESR), grip strength, pain scores, tender joint counts, and anx
153 -appearing brain was associated with: weaker grip strength, poorer lung function, slower walking spee
154 ll Pain Questionnaire Short Form, walk time, grip strength, predicted maximum oxygen uptake, and join
155 risk = 0.82, 95% CI: 0.73, 0.92; for highest grip strength quartile vs. lowest: relative risk = 0.76,
156 isease duration (r = 0.51 for each measure), grip strength (r = -0.49 for NDJ, and r = -0.51 for Shar
158 tive relation between birth weight and adult grip strength remained after adjustment first for adult
160 cts with the disease had 10% reduced maximal grip strength, reported more difficulty writing, handlin
161 In multivariate models, the mean ESR, mean grip strength, rheumatoid factor positivity, and tender
162 monstrated significant motor deficits (e.g., grip strength, righting reflex and touch escape) in bf m
164 ent (beam walking, pole climbing, wire hang, grip strength), sensorimotor skills (rotarod), mechanica
165 3 weeks after stroke on locomotor activity, grip strength, sensory neglect, gait impairment, motor c
166 ced vital capacity, resting heart rate, hand grip strength, sit and reach distance, and time standing
167 or more of the following 5 components: weak grip strength, slowed walking speed, poor appetite, phys
168 ss index (Spearman r=0.28, P<0.0001), weaker grip strength (Spearman r=-0.34, P<0.01), and slower wal
169 Ab levels were significantly correlated with grip strength (Spearman r=-0.57, P<0.005), walking speed
170 ures of physical capability at age 53 years: grip strength, standing balance, and chair-rise time.
174 in mdx mice as demonstrated through in vivo grip strength tests and in vitro contraction measurement
175 ysfunction as determined by both rotorod and grip strength tests, as well as enhanced loss of motor n
176 .03), appendicular skeletal muscle mass, and grip strength than did controls, but these differences w
179 for the functional measures of walk time and grip strength: the treatment groups improved more than t
180 ms and four measures of physical capability: grip strength, timed walk or get up and go, chair rises
181 5 and 0.025, respectively) but not with hand-grip strength, triceps skin-fold thickness (TSFT), or mi
184 nd changes in the rate of decline of leg and grip strength, vital capacity, ALS Functional Rating Sca
186 n; joint tenderness, swelling, or deformity; grip strength; walking velocity; and timed button test.
187 urrent work status; vital status at 6 years; grip strength; walking velocity; the timed-button test;
191 A significant HRT-by-race interaction for grip strength was found; African-American HRT users had
192 Among healthy 45- to 68-year-old men, hand grip strength was highly predictive of functional limita
198 ly joint group significantly associated with grip strength was the CMCs, and only OA in the MCP joint
200 ate the genetic determinants of variation in grip strength, we perform a large-scale genetic discover
203 cle alone but to be insufficient to maintain grip strength, whereas delivery to both motor neurons an
204 erformance was assessed by walking speed and grip strength, while global functional limitation, acros
205 , 2.17) than in those with stable weight and grip strength, with the highest risk in those with both
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