ライフサイエンスコーパス: group B Streptococcus

1 cific role for IKKbeta during infection with group B streptococcus.

2 stinguishable clinically from that caused by group B streptococcus.

3 bacteria such as Listeria monocytogenes and group B streptococcus.

4 with a putative peptidoglycan hydrolase from group B streptococcus.

5 ccus aureus, Staphylococcus epidermidis, and group B Streptococcus.

6 hore A23187, nigericin, Candida albicans and Group B Streptococcus.

7 emic infection with the pathogenic bacterium group B Streptococcus.

8 etence and survival following infection with group B Streptococcus.

9 sociated with lower incidence of early-onset group B streptococcus (0.23 per 1000 livebirths [95% CI

10 Incidence of early-onset group B streptococcus (0.43 per 1000 livebirths [95% CI

11 neumoniae, 1.1; Neisseria meningitidis, 0.6; group B streptococcus, 0.3; Listeria monocytogenes, 0.2;

12 open reading frame, spb1 (surface protein of group B streptococcus 1).

13 The alpha C protein of group B streptococcus, a virulence determinant for this

14 s of murine and human macrophages induced by group B Streptococcus agalactiae (GBS) is likely an impo

15 Group B Streptococcus agalactiae bacteria (group B strep

16 Many group B Streptococcus agalactiae strains and other patho

17 tent cytokine response of blood monocytes to group B Streptococcus, although monocytes serve as the k

18 n endothelial cells show enhanced binding to group B Streptococcus and are more susceptible to apopto

19 Although decreased killing of group B streptococcus and H. influenzae was observed in

20 that nonopsonic recognition between type III group B streptococcus and human neutrophils would occur

21 4-kb foreign DNA element that is shared with group B Streptococcus and is present in all serotype M28

22 Several polysaccharides from group B Streptococcus and other bacterial species were s

23 Group B streptococcus and respiratory syncytial virus ar

24 maternal immunisation strategies to prevent group B streptococcus and respiratory syncytial virus in

25 hat is known about immune protection against group B streptococcus and respiratory syncytial virus, i

26 cell wall fragments from lysates of type III group B Streptococcus and showed that the complexes cont

27 reduced phagocytosis of bacteria, including group B streptococcus and Staphylococcus aureus.

28 red for capsule polymerization and export in group B Streptococcus and Streptococcus pneumoniae.

29 in 87.0% of the women who were positive for group B streptococcus and who delivered at term, but in

30 ons with strains of the sialylated pathogen, group B Streptococcus, and with sialoglycans presented a

31 Nonpregnant adults with group B streptococcus bacteremia were identified by acti

32 The rate of screening for group B streptococcus before delivery increased from 48.

33 re born to women who had tested negative for group B streptococcus before delivery.

34 acrophage (MPhi) receptor in the response to group B Streptococcus, both in bone marrow-derived MPhis

35 monas aeruginosa, Staphylococcus aureus, and group B streptococcus by increasing membrane permeabilit

36 s derived from a wild-type strain of type Ia group B Streptococcus by selectively inactivating each g

37 The Group B Streptococcus capsular polysaccharide type IX wa

38 Type III group B Streptococcus capsular polysaccharide was linked

39 K1, groups W-135, Y, and C meningococci, and group B Streptococcus capsular polysaccharides modifies

40 capsular serogroup C (MenC) or Gram-positive group B Streptococcus, capsular type III (GBS-III) bacte

41 Group B Streptococcus causes a variety of morbid and som

42 ated polysaccharides supports a model of the group B Streptococcus cell surface in which the group B

43 ch 31, 2015, that reported the prevalence of group B streptococcus colonisation in pregnant women.

44 untry and regional heterogeneity in maternal group B streptococcus colonisation is unlikely to comple

45 We did a systematic review of maternal group B streptococcus colonisation studies by searching

46 he estimated mean prevalence of rectovaginal group B streptococcus colonisation was 17.9% (95% CI 16.

47 h enrichment culture method for detection of group B streptococcus colonization in pregnant women.

48 xtraction is a suitable method for detecting group B streptococcus colonization in pregnant women.

49 Group B Streptococcus colonization is frequent and dynam

50 The estimated average duration of any group B Streptococcus colonization was longer for women

51 t broth by utilizing the Gen-Probe AccuProbe Group B Streptococcus Culture Identification Test (GPGB)

52 culture method with the Gen-Probe AccuProbe Group B Streptococcus Culture Test (APGB) and the BD Gen

53 Group B Streptococcus detection directly from Copan ESwa

54 tion against both early-onset and late-onset group B streptococcus disease.

55 Routine screening for group B streptococcus during pregnancy prevents more cas

56 to accurately estimate the global burden of group B streptococcus, especially in low-income countrie

57 These results demonstrate that Group B Streptococcus expresses a specific ecto-5'-nucle

58 agar plate (SBAP) and AccuProbe detection of group B streptococcus from overnight LIM broth enhanceme

59 Group B Streptococcus (GBS) (150/302 [50%]; incidence, 0

60 e (n=5), or 2) choriodecidual inoculation of Group B Streptococcus (GBS) 1x10(6) colony forming units

61 The sialylated capsular polysaccharide of group B Streptococcus (GBS) also presents terminal Siaal

62 006, cross-reacts with serogrouping kits for group B Streptococcus (GBS) and could be misidentified i

63 e of the BBB to the human meningeal pathogen group B Streptococcus (GBS) and the organism's major vir

64 Infections such as group B Streptococcus (GBS) are an important cause of ma

65 nant monkeys (Macaca nemestrina) with either group B streptococcus (GBS) at 1 x 10(6) CFU (n = 5) or

66 rd most common cause of neonatal death, with Group B Streptococcus (GBS) being the leading bacterial

67 Group B Streptococcus (GBS) beta C protein elicits prote

68 We examined the virulence role of group B Streptococcus (GBS) beta-hemolysin/cytolysin (be

69 tinguish between the nine known serotypes of group B streptococcus (GBS) by classical antibody-antige

70 SP-A -/- and control mice were infected with group B streptococcus (GBS) by intratracheal instillatio

71 G-independent opsonophagocytosis of type III group B Streptococcus (GBS) by peripheral blood leukocyt

72 Colonizing group B Streptococcus (GBS) capsular polysaccharide (CPS

73 Group B streptococcus (GBS) capsular serotypes are major

74 Group B streptococcus (GBS) carriers were identified and

75 Group B Streptococcus (GBS) causes invasive infections i

76 Group B Streptococcus (GBS) causes severe disease in neo

77 Group B Streptococcus (GBS) causes substantial morbidity

78 Group B Streptococcus (GBS) causes urinary tract infecti

79 Group B Streptococcus (GBS) colonizes mucosal surfaces o

80 We compared five approaches for group B streptococcus (GBS) detection: three culture-bas

81 rea to determine risk factors for late-onset group B streptococcus (GBS) disease (onset of disease or

82 During the 1990s the focus of group B streptococcus (GBS) disease research has shifted

83 te substantial progress in the prevention of group B Streptococcus (GBS) disease with the introductio

84 with HIV are at increased risk for invasive group B streptococcus (GBS) disease.

85 ed infants are at increased risk of invasive Group B Streptococcus (GBS) disease; however, the reason

86 rs for Disease Control guidelines to prevent group B Streptococcus (GBS) early-onset sepsis (EOS) has

87 Pathogenic bacteria such as group B Streptococcus (GBS) express and secrete hyaluron

88 Group B Streptococcus (GBS) frequently colonizes the hum

89 Transmission of group B Streptococcus (GBS) from mothers to neonates dur

90 (GM-CSF) gene-targeted mice (GM-/-) cleared group B streptococcus (GBS) from the lungs more slowly t

91 By sequence analysis of available group B streptococcus (GBS) genomes, we discovered a con

92 Immunogenic vaccines against group B Streptococcus (GBS) have been created by couplin

93 nd universal screening of pregnant women for group B streptococcus (GBS) have further changed the epi

94 flight mass spectrometry (MALDI-TOF MS) for group B streptococcus (GBS) identification, we recovered

95 Group B streptococcus (GBS) imposes a major health threa

96 itive predictive values for the detection of group B Streptococcus (GBS) in 206 LIM enrichment broths

97 Group B streptococcus (GBS) induces apoptosis in macroph

98 Here, we show that Group B Streptococcus (GBS) induces IFN-beta production

99 Group B streptococcus (GBS) infection is a leading cause

100 Meningitis was induced by injecting Group B Streptococcus (GBS) into the cisterna magnae of

101 Group B Streptococcus (GBS) is a common cause of neonata

102 Group B streptococcus (GBS) is a common commensal of the

103 Group B Streptococcus (GBS) is a frequent agent of life-

104 Group B Streptococcus (GBS) is a leading cause of bacter

105 Group B Streptococcus (GBS) is a leading cause of invasi

106 The group B Streptococcus (GBS) is a leading cause of neonat

107 Group B Streptococcus (GBS) is a leading cause of neonat

108 Group B Streptococcus (GBS) is a leading cause of neonat

109 Group B streptococcus (GBS) is a leading neonatal pathog

110 Group B Streptococcus (GBS) is a major cause of bacteria

111 Group B Streptococcus (GBS) is a major cause of invasive

112 Group B Streptococcus (GBS) is a major cause of neonatal

113 Group B streptococcus (GBS) is a major cause of neonatal

114 Group B Streptococcus (GBS) is a major cause of neonatal

115 Group B Streptococcus (GBS) is a major cause of neonatal

116 Group B Streptococcus (GBS) is a major cause of newborn

117 Group B Streptococcus (GBS) is a major cause of pneumoni

118 The group B streptococcus (GBS) is a major cause of pneumoni

119 The alpha C protein of group B Streptococcus (GBS) is a major surface-associate

120 Maternal colonization with group B Streptococcus (GBS) is a risk factor for neonata

121 Group B Streptococcus (GBS) is an encapsulated, gram-pos

122 Infection by group B streptococcus (GBS) is an important cause of bac

123 Group B Streptococcus (GBS) is an important cause of inf

124 Group B Streptococcus (GBS) is an important cause of inv

125 Group B Streptococcus (GBS) is an important human bacter

126 The group B streptococcus (GBS) is an important human pathog

127 Group B streptococcus (GBS) is an important human pathog

128 Group B Streptococcus (GBS) is an important pathogen of

129 Group B Streptococcus (GBS) is an important perinatal pa

130 Group B Streptococcus (GBS) is an important perinatal pa

131 Group B Streptococcus (GBS) is an opportunistic organism

132 The facultative anaerobe group B Streptococcus (GBS) is an opportunistic pathogen

133 Group B Streptococcus (GBS) is classified into nine sero

134 The process of human infection by group B Streptococcus (GBS) is complex and multifactoria

135 Group B Streptococcus (GBS) is currently the leading cau

136 Serotype IV group B Streptococcus (GBS) is emerging in Canada and th

137 Group B Streptococcus (GBS) is frequently carried in the

138 Group B Streptococcus (GBS) is major cause of invasive d

139 Group B Streptococcus (GBS) is one of the most common or

140 Genital tract carriage of group B streptococcus (GBS) is prevalent among adult wom

141 accharide (CPS) and some surface proteins by group B Streptococcus (GBS) is regulated by growth rate.

142 Group B Streptococcus (GBS) is the foremost bacterial ca

143 Group B Streptococcus (GBS) is the foremost cause of neo

144 Group B Streptococcus (GBS) is the leading cause of bact

145 Group B Streptococcus (GBS) is the leading cause of bact

146 Group B Streptococcus (GBS) is the leading cause of bact

147 Group B Streptococcus (GBS) is the leading cause of huma

148 Group B Streptococcus (GBS) is the leading cause of meni

149 Group B Streptococcus (GBS) is the leading cause of neon

150 Group B Streptococcus (GBS) is the most common bacterium

151 Maternal rectovaginal colonization with group B Streptococcus (GBS) is the most common pathway f

152 99 nonpregnant adult Maryland residents with group B Streptococcus (GBS) isolated from a normally ste

153 , we studied the population structure of 102 group B Streptococcus (GBS) isolates prospectively sampl

154 Studies have suggested that group B Streptococcus (GBS) maternal rectovaginal coloni

155 Maternal vaccination against group B Streptococcus (GBS) might provide protection aga

156 nces of the AmpliVue, BD Max, and illumigene group B Streptococcus (GBS) nucleic acid amplification t

157 Group B Streptococcus (GBS) or Streptococcus agalactiae

158 Group B streptococcus (GBS) or Streptococcus agalactiae

159 Group B streptococcus (GBS) pili may enhance colonizatio

160 Group B Streptococcus (GBS) remains a leading cause of n

161 Group B streptococcus (GBS) remains a major cause of mor

162 Group B streptococcus (GBS) remains the leading cause of

163 Infection with group B streptococcus (GBS) results in 12,000 to 15,000

164 Neonatal Group B streptococcus (GBS) sepsis and pneumonia result

165 To determine whether 2 monovalent group B streptococcus (GBS) serotype II or III capsular

166 on of the human epithelial cell line A549 by group B streptococcus (GBS) serotype VIII strains were c

167 Maternal group B streptococcus (GBS) serotype-specific capsular a

168 We investigated the association between group B Streptococcus (GBS) serotype-specific capsular p

169 Group B streptococcus (GBS) serotypes causing neonatal d

170 report that neuD, a gene located within the Group B Streptococcus (GBS) Sia biosynthetic gene cluste

171 dies for molecular and serological typing of group B streptococcus (GBS) strains as part of DEVANI (D

172 Detection of group B Streptococcus (GBS) strains at various bacterial

173 Streptococcal pathogens, such as the group B streptococcus (GBS) Streptococcus agalactiae, ar

174 ansplacental antibody transfer specific to 8 group B Streptococcus (GBS) surface proteins among 81 HI

175 Serotypes of group B streptococcus (GBS) that cause urinary tract inf

176 Resistance of group B streptococcus (GBS) to antibiotics, particularly

177 of immunoglobulin G (IgG) antibodies against group B streptococcus (GBS) type III polysaccharide (PS)

178 to study maternal transfer of antibody to a group B Streptococcus (GBS) type III polysaccharide-teta

179 level of maternal immunoglobulin (Ig) G anti-group B streptococcus (GBS) type III required to protect

180 Although rarely encountered, group B Streptococcus (GBS) types IV and VII have been i

181 Group B Streptococcus (GBS) types VI and VIII are preval

182 Serotype V group B Streptococcus (GBS) was first isolated from huma

183 Mutagenesis of group B streptococcus (GBS) with TnphoZ, a transposon de

184 acterial polysaccharide exotoxin produced by group B Streptococcus (GBS), also referred to as GBS tox

185 women who are rectovaginally colonized with group B Streptococcus (GBS), but the risk of EOGBS from

186 rate kinase (PGK), present on the surface of group B streptococcus (GBS), has previously been demonst

187 rst comprehensive estimates of the burden of group B Streptococcus (GBS), including invasive disease

188 Streptococcus agalactiae, or group B Streptococcus (GBS), is an important opportunist

189 pathogen Streptococcus agalactiae, known as group B Streptococcus (GBS), is the leading cause of bac

190 , 7 produced at least 1 stool that contained group B Streptococcus (GBS), Serratia marcescens, or Esc

191 Group B Streptococcus (GBS), the leading cause of neonat

192 nducting efficacy trials of vaccines against group B streptococcus (GBS), the licensure of these vacc

193 nital inhabitant and opportunistic pathogen, group B Streptococcus (GBS), when present at the time of

194 Here, we define Group B Streptococcus (GBS)-mediated activation of the N

195 SA-colonized women were less likely to carry group B streptococcus (GBS).

196 etween these families was first described in Group B Streptococcus (GBS).

197 ence factors in the important human pathogen group B Streptococcus (GBS).

198 nal vaccines against neonatal disease due to group B Streptococcus (GBS).

199 ons caused by the leading neonatal pathogen, group B streptococcus (GBS).

200 cause invasive disease in humans, including group B Streptococcus (GBS).

201 uence typing (MLST) system was developed for group B streptococcus (GBS).

202 he pathogens group A Streptococcus (GAS) and group B Streptococcus (GBS).

203 ctors associated with vaginal acquisition of group B Streptococcus (GBS).

204 the pathogenesis of septic shock induced by group B Streptococcus (GBS).

205 accine target of the major neonatal pathogen group B Streptococcus (GBS).

206 Group B streptococcus (GBS; Streptococcus agalactiae) in

207 es of serotype III Streptococcus agalactiae (group B streptococcus [GBS]) can be divided into three s

208 Streptococcus agalactiae (group B Streptococcus [GBS]) causes serious infections i

209 Streptococcus agalactiae (group B streptococcus [GBS]) colonizes the rectovaginal

210 Streptococcus agalactiae (group B Streptococcus [GBS]) has not been described as a

211 Streptococcus agalactiae (group B streptococcus [GBS]) is a Gram-positive bacteriu

212 Streptococcus agalactiae (group B streptococcus [GBS]) is a leading cause of neona

213 Streptococcus agalactiae (group B Streptococcus [GBS]) is a leading cause of neona

214 tal infection with Streptococcus agalactiae (group B Streptococcus [GBS]) is a leading cause of sepsi

215 colonization with Streptococcus agalactiae (Group B Streptococcus [GBS]) is a precursor to chorioamn

216 Streptococcus agalactiae (group B Streptococcus [GBS]) is an important neonatal pa

217 Streptococcus agalactiae (group B Streptococcus [GBS]) remains a leading cause of

218 coli, E. faecalis, Streptococcus agalactiae (group B streptococcus [GBS]), or Streptococcus pyogenes

219 Streptococcus agalactiae (Group B Streptococcus, GBS) causes life-threatening infe

220 Streptococcus agalactiae (group B streptococcus, GBS) causes neonatal disease and

221 Streptococcus agalactiae (group B streptococcus, GBS) expresses either Srr1 or Srr

222 Streptococcus agalactiae (Group B Streptococcus, GBS) is a commensal of the digest

223 Streptococcus agalactiae (group B Streptococcus, GBS) is a leading cause of invasi

224 Streptococcus agalactiae (group B Streptococcus, GBS) is the predominant cause of

225 Streptococcus agalactiae (group B Streptococcus; GBS) is a significant bacterial p

226 Streptococcus agalactiae (group B Streptococcus; GBS) produces a CPS that represen

227 -positive bacteria Streptococcus agalactiae (Group B Streptococcus; GBS) type III (GBSIII) and Strept

228 These include pneumococcus, group B Streptococcus, Haemophilus influenzae type b, an

229 of the CpsA protein of the zoonotic pathogen group B Streptococcus in capsule production and cell wal

230 Mean incidence of group B streptococcus in infants aged 0-89 days was 0.53

231 pathogens, Streptococcus pneumoniae and the group B streptococcus, induced neuronal injury in primar

232 Group B streptococcus infection can be associated with i

233 ntibodies during the acute phase of invasive group B streptococcus infection in nonpregnant adults ma

234 more protective in a neonatal mouse model of group B Streptococcus infection than a vaccine construct

235 vival and conferred resistance to an in vivo group B streptococcus infection, we show that mice with

236 uggish mice displayed high susceptibility to group B streptococcus infection, with impaired TNF-alpha

237 ted aneurysms of visceral arteries caused by Group B streptococcus infection.

238 to the observed increase in dissemination of group B Streptococcus into the brain of Hs2st-deficient

239 The capsular polysaccharide of group B Streptococcus is a key virulence factor and an i

240 Group B streptococcus is a leading infectious cause of m

241 Group B Streptococcus is the most common cause of bacter

242 Streptococcus agalactiae (Group B Streptococcus) is a commensal of the human intes

243 psonophagocytic killing of the corresponding group B streptococcus isolate in vitro.

244 by analysis of DNA band sizes of our control group B streptococcus isolate.

245 ive disease and the serotype distribution of group B streptococcus isolates.

246 & Microbe, Andrade et al. (2016) report that Group B Streptococcus limits type I IFN by expressing a

247 to understand these regional differences in group B streptococcus maternal colonisation and early-on

248 e concern that increasing efforts to prevent group B streptococcus neonatal disease may lead to an in

249 Streptococcus agalactiae (group B Streptococcus or GBS) is a common cause of invas

250 Streptococcus agalactiae (group B Streptococcus or GBS) is a common colonizer of t

251 -D-/-) and wild-type mice were infected with group B streptococcus or Haemophilus influenzae by intra

252 The repeating units of the group B Streptococcus polysaccharides all contain an aci

253 d use of intrapartum antibiotic prophylaxis, group B streptococcus remains a leading cause of morbidi

254 ss several maternal immunisation initiatives-group B streptococcus, respiratory syncytial virus, pert

255 ment of the [D130A,S512A] mutant of SCP from group B Streptococcus (S. agalactiae, SCPB) revealed SCP

256 tions, poliovirus immunization schedule, and group B streptococcus screening and treatment.

257 eningococcal type C polysaccharide (MCPS) or group B Streptococcus serotype V (GBS-V) were unresponsi

258 Until recently, group B streptococcus, serotype V (GBS-V), was an infreq

259 esponses to intact Pn14, isolated PPS14, and Group B Streptococcus (strain COH1-11) expressing capsul

260 In this report, we used a mutant strain of group B Streptococcus (Streptococcus agalactiae) type II

261 F upon stimulation with Escherichia coli and group B Streptococcus, the leading pathogens of early-on

262 ning of pregnant women for colonization with group B streptococcus to identify candidates for intrapa

263 Group B streptococcus was the predominant pathogen among

264 n antiangiogenic polysaccharide derived from group B streptococcus, was administered by i.v. injectio

265 Women with no documented culture for group B streptococcus were considered to have been cared

266 g cause of community-acquired pneumonia, and group B Streptococcus, which causes neonatal sepsis and

267 c.), a selective and differential medium for group B streptococcus, with culture using neomycin-nalid