ライフサイエンスコーパス: growing cell

1 ry networks and with cellular processes of a growing cell.

2 the demand for membrane expansion of rapidly growing cells.

3 bosomes are stably maintained even in slowly growing cells.

4 e only partially methylated in exponentially growing cells.

5 entially to the synthesis of TAG in actively growing cells.

6 was not substantially methylated in actively growing cells.

7 sphatase is strongly active in exponentially growing cells.

8 equivalents and synthetic materials in fast-growing cells.

9 rowing bacteria among populations of rapidly growing cells.

10 are viable but have reduced expansion of tip-growing cells.

11 hh1 and Pat1 from ribosomes in exponentially growing cells.

12 I localization to chromatin in exponentially growing cells.

13 asmid ColE1 in a population of exponentially growing cells.

14 om a TA (toxin-antitoxin) operon in normally growing cells.

15 n RpoS-independent promoter in exponentially growing cells.

16 ratio and temporal resolution in individual growing cells.

17 ential for chromosome segregation in rapidly growing cells.

18 e phospholipid metabolism in logarithmically growing cells.

19 rized growth, For2 are apically localized in growing cells.

20 regulon in a DNA damage response in actively growing cells.

21 in the observed variability in shapes of tip-growing cells.

22 ng domain in the chromosome of exponentially growing cells.

23 ase is a component of the division septum in growing cells.

24 de novo synthesis by 75% in logarithmically growing cells.

25 tolytic enzymes must be tightly regulated in growing cells.

26 vation but not when CDC2 is highly active in growing cells.

27 their environment differently from actively growing cells.

28 results from changes in peptidoglycan in non-growing cells.

29 p positively regulates sigD transcription in growing cells.

30 tgrowth from the ER maintains peroxisomes in growing cells.

31 curred that was not evident in exponentially growing cells.

32 and predominantly at the regions of actively growing cells.

33 Escherichia coli chromosome in exponentially growing cells.

34 as only E2F3 is necessary for the S phase in growing cells.

35 salt but at lower levels than in comparable growing cells.

36 nown to be involved in turgor maintenance in growing cells.

37 Here, we investigate bypassing in unstarved, growing cells.

38 depletion is toxic to virtually all actively growing cells.

39 most of the gene expression in exponentially growing cells.

40 expressed higher p66(Shc) protein than slow-growing cells.

41 B(-) strain in sessile, planktonic, and free-growing cells.

42 s higher in confluent cells than in actively growing cells.

43 by mechanisms distinct from those in rapidly growing cells.

44 ation products generated by Hin catalysis in growing cells.

45 se are around 55% of those in asynchronously growing cells.

46 ramatically more Cln protein than did slowly growing cells.

47 eflect non-stress-specific processes in rare growing cells.

48 delivery of a subset of vacuolar proteins in growing cells.

49 following the irradiation of logarithmically growing cells.

50 d reduction of phosphorylated Rb in normally growing cells.

51 ibonucleases are more toxic for more rapidly growing cells.

52 nterorganelle lipid transfer is extensive in growing cells.

53 (cephalexin, aztreonam, and piperacillin) in growing cells.

54 inducible hsp70 promoter, into exponentially growing cells.

55 l tension along cell junctions within faster-growing cells.

56 lk to meet the need for protein synthesis in growing cells.

57 t such a gradient is absent in exponentially growing cells.

58 re of transcription factors expressed in tip-growing cells.

59 onger distances in quiescence as compared to growing cells.

60 d effectively reduces fluorescence signal in growing cells.

61 which are mainly effective against actively growing cells.

62 aining genetically identical to the actively growing cells.

63 e ethanol in mitochondria as opposed to slow growing cells.

64 h pseudopod orientation during chemotaxis of growing cells.

65 were mapped by UV cross-linking in actively growing cells.

66 centrioles and aberrant assembly of cilia in growing cells.

67 differently in those cells than in actively growing cells.

68 origin and terminus domains in exponentially growing cells.

69 the idea of chemotherapy targeting actively growing cells.

70 ate the ClpP protease, resulting in death of growing cells.

71 dmark is a broadly conserved strategy in tip-growing cells.

72 nificantly increases plasmalemma in normally growing cells.

73 articipation of myosin XI and F-actin in tip growing cells.

74 lipid composition is very similar to that of growing cells?

75 mine the particularly simple geometry of tip-growing cells [1, 3, 15, 16], which elongate via the ass

76 rigenic in forming bone metastases than fast-growing cells (55 vs. 15%) and had a unique gene express

77 the half-lives of total mRNA in resting and growing cells (about 9 h).

78 rays are the major sites of transcription in growing cells, accounting for as much as 50% of RNA synt

79 How populations of growing cells achieve cell-size homeostasis remains a ma

80 ls or by protein phosphatase M1A in normally growing cells activates CDK9.

81 elopmental differences between P. patens tip-growing cells and A. thaliana pollen tubes and root hair

82 tion are only slightly increased compared to growing cells and are 10-fold below the levels observed

83 CC2, and XRCC3) are expressed in mitotically growing cells and are thought to play mediating roles in

84 natural populations, selection is imposed on growing cells and can detect the more common mutations t

85 activity is partially suppressed by cdk2 in growing cells and derepressed upon cell confluence.

86 the unique metabolic requirements of rapidly growing cells and geminivirus-infected cells that have b

87 ces transcriptional slippage within actively growing cells and in vitro.

88 ent cells would mechanically isolate rapidly growing cells and limit their impact on organ shape.

89 between polarization mechanisms in moss tip-growing cells and multicellular tissues of seed plants.

90 enotypic switching occurred between normally growing cells and persister cells having reduced growth

91 ls are known to be very low in exponentially growing cells and rise sharply as cells enter stationary

92 Logarithmically growing cells and spores were each harvested from the cu

93 pXP localize in foci often near the poles of growing cells and that ClpP and the ATPase are each capa

94 rall accuracy of DNA replication in normally growing cells and that repair pathways may exist primari

95 chastically in a population of exponentially growing cells and that the high (p)ppGpp level in rare c

96 ls to correct DNA replication errors both in growing cells and under nongrowing conditions.

97 in differentiated stationary-phase (not rare growing) cells and, second, that transient genetic insta

98 or CP110 promotes primary cilia formation in growing cells, and enforced expression of CP110 in quies

99 ng protein synthetic capacity in dividing or growing cells, and requires coordinated production of ri

100 sm regulates cellulase expression in rapidly growing cells, and that the presence of hemicelluloses h

101 evealed nucleolin at the surface of actively growing cells, and these cells bound and internalized fl

102 1 and Pom1 were enriched at opposite ends of growing cells, and they phosphorylated largely non-overl

103 In growing cells, apurinic/apyrimidinic (AP) sites generate

104 , while, conversely, certain types of faster-growing cells are able to eliminate adjacent wild-type c

105 Many tip-growing cells are capable of responding to guidance cues

106 that macropinosomes, but not pseudopods, in growing cells are dependent on PIP(3).

107 Viable but slower growing cells are eliminated during embryonic developmen

108 pression of HIF-1alpha results in death when growing cells are exposed to hypoxia.

109 cally switch into slow growth, that the slow-growing cells are multidrug tolerant, and that they are

110 of Hg transformations suggests that actively growing cells are primarily responsible for methylation,

111 We further found that while growing cells are rapidly killed by iron starvation, sta

112 ecular composition of isogenic, steady-state growing cells arises spontaneously from the inherent sto

113 which old OMPs are displaced to the poles of growing cells as new OMPs take their place.

114 The problem was easily rectified by growing cells at a low glucose concentration.

115 t ABCC4/Abcc4 proteins are mostly rescued by growing cells at subphysiological temperatures.

116 associated with a constant-thickness rim of growing cells at the cluster edge, as well as the emerge

117 mophila, highly phosphorylated histone H1 of growing cells becomes partially dephosphorylated when ce

118 n to that of cells grown in suspension (free-growing cells), bfr mRNA expression by sessile cells on

119 adaption of the ontology to the continuously growing cell biological knowledge.

120 The enzyme is extremely unstable in rapidly growing cells but becomes stabilized under conditions of

121 RNA and protein can be detected in actively growing cells but decline sharply when the replicon cell

122 yogenes SF370 is functional in exponentially growing cells but defective when resources are limiting.

123 Conversely, Fis levels are high in growing cells but fall to nearly zero in stationary-phas

124 nced by the repressive Polycomb complexes in growing cells but is activated in response to oncogenic

125 cell wall-derived muropeptides derived from growing cells but not lysed cells.

126 pathway affected the cell length of actively growing cells but not their growth rate.

127 ave features not required for mRNA export in growing cells but which enhance the ability of mRNAs to

128 tion of protein homeostasis in exponentially growing cells, but ERAD became relevant when the gene do

129 t Mdm2 inhibits the p53 response in normally growing cells by binding to chromatin-associated p53.

130 Conversely, growth suppression of fast-growing cells by cellular form of prostatic acid phospha

131 ed the redox state of the C-terminal HRMs in growing cells by thiol-trapping experiments using the is

132 ) isolation of synchronous subpopulations of growing cells by zonal centrifugation.; (ii) fractionati

133 enome content, responded to Fe starvation in growing cells characterized by low photosynthetic effici

134 overall data revealed that, in exponentially growing cells, CodY and CovRS act in opposite directions

135 ugh HNP-3 were bound to the periphery of the growing cell colonies.

136 We show that moss tip growing cells contain a prominent subapical cortical F-a

137 ospores, it was determined that vegetatively growing cells contain one to two copies of the genome, b

138 Growing cells coordinate protein translation with metabo

139 P signals that locally reduce Anillin at the growing cell cortex.

140 d mRNA expression profiling of synchronously growing cell cultures progressing through the cell cycle

141 In both sucrose-starved and exponentially growing cells, CYCD3;1 protein abundance increases in re

142 wn of p66(Shc) expression by RNAi in rapidly growing cells decreased their proliferation as evidenced

143 Slow-growing cells deep in the biofilms had little expression

144 proteins (Twis) preferentially expressed in growing cells differ in their genetic essentiality and s

145 A wide range of tip-growing cells display highly polarized cell growth, and

146 Tip-growing cells do not display neighbor-induced growth con

147 % of ribosomal proteins expressed in rapidly growing cells does not contribute to translation.

148 have reexamined the cidal activity of bSi on growing cells, dormant and germinated spores of B. subti

149 ed diversity of 23- to 24-nt sRNA classes in growing cells, each with distinct genetic requirements f

150 residues seem to mediate crystal adhesion to growing cells, either directly or via linkage to other c

151 ver, the engineered expression of CuZnSOD in growing cells eliminated superoxide release, confirming

152 g new proteins can be enforced after rapidly growing cells encounter energy limitation.

153 n monopolar distribution of GTP-Cdc42 to the growing cell end.

154 lished, POs and LDs drift slowly towards the growing cell end.

155 owth with abnormal Tea1 aggregate at the non-growing cell end; this abnormal aggregate fails to recru

156 t GTP-bound, active Cdc42 is concentrated to growing cell ends accompanied by developed F-actin struc

157 n further detail and show that exponentially growing cells exhibit Ca(2+) elevation resulting exclusi

158 ass, which are specifically expressed in tip-growing cells, exhibited exocytosis-related functional e

159 tion with cell proliferation, that is, rapid-growing cells expressed higher p66(Shc) protein than slo

160 Free-growing cells expressed more sspA-specific mRNA and sspB

161 Free-growing cells expressed the same levels of sspA and sspB

162 Rapidly growing cells expressed, and required, dramatically more

163 eous transformation are selected in vitro by growing cells for many low density passages at maximal e

164 n, while a small fraction of elongated, slow-growing cells formed colicin-expressing hotspots, placin

165 In growing cells, H2A.Y is incorporated into micronuclei on

166 trigger intracellular responses in actively growing cells have not yet been experimentally character

167 Tip-growing cells have the unique property of invading livin

168 Because the rapidly growing cells have very different geometry from that of

169 stoma protein (Rb), whereas in exponentially growing cells, HIN-1 induces apoptosis without apparent

170 so generate variable resistance levels among growing cells in a population.

171 ct of pH on cell viability was determined by growing cells in broth at various initial glucose concen

172 Bacillus subtilis, we analyzed exponentially growing cells in different media.

173 Enhancing CtBP-mediated repression by growing cells in low oxygen increased the association of

174 The method involves growing cells in multiwell plates, incubating cells with

175 rowth are likely similar to those of non-tip growing cells in seedlings.

176 of FGF-induced MAPK activation than actively growing cells in sparse culture.

177 kton blooms, we mimicked those conditions by growing cells in the presence of increased amounts of di

178 After growing cells in the presence of labeled palmitate compl

179 ion," which manifests in apoptosis of slower-growing cells in the vicinity of faster growing tissue,

180 and limitations on the technologies used for growing cells in vitro, perturbing them, and measuring t

181 In aerobically growing cells, in which reactive oxygen species are prod

182 this phenomenon occurs ubiquitously for fast-growing cells, including bacteria, fungi and mammalian c

183 was observed only in the presence of nearby growing cells, indicating cross-inhibition.

184 ed from polysomal complexes in exponentially growing cells, indicating that they may not be associate

185 RpoS, RpoS must regulate Pol IV activity in growing cells indirectly via one or more intermediate fa

186 In rapidly growing cells, initiation of replication occurs before t

187 The conversion of a growing cell into an endospore in Bacillus subtilis invo

188 (the major vegetative holoenzyme) in rapidly growing cells is engaged in transcribing the rrn operons

189 Metabolite exchange among co-growing cells is frequent by nature, however, is not nec

190 Temporal increase in EB fluorescence of growing cells is indicative for toxic but reversible eff

191 by TORC1, whose inhibition in exponentially growing cells is shown to match an interruption in endom

192 In growing cells, it is found in active and inactive forms.

193 Because aerobic glycolysis is only useful to growing cells, it is tightly regulated in a proliferatio

194 to push old OMP islands towards the poles of growing cells, leading to a binary distribution when cel

195 nSOD by adenovirus transfection in the rapid growing cell line MIA PaCa-2 increased MnSOD immunoreact

196 tic RanGTP gradients were present in rapidly growing cell lines and were required for chromosome cong

197 Aggressively growing cell lines displayed ERK1/2 activation, which st

198 How exponentially growing cells maintain size homeostasis is an important

199 e genes are highly expressed in vegetatively growing cells, many pombe-specific genes are expressed a

200 ate sources, which are incorporated into the growing cell mass; PKA signaling responds to the availab

201 show via computational modeling that rapidly growing cells may distort organ shape.

202 progeny cells, and cell wall homeostasis in growing cells, may also be essential for Mtb's survival

203 B concentration was about 122 microM in 4HPA-growing cells, much higher than the total intracellular

204 Malignant cells, like all actively growing cells, must maintain their telomeres, but geneti

205 -599 site, suggesting that in asynchronously growing cells NF-Y functions only to stimulate expressio

206 Growing cells of Bacillus subtilis are a bistable mixtur

207 hromosome replication origin and terminus in growing cells of Escherichia coli have been visualized s

208 tors in live cells by artificially deforming growing cells of Escherichia coli in curved agar microch

209 t the perturbation of cell wall synthesis in growing cells of S. aureus induces strong repression of

210 re and organization of the S layer on intact growing cells of the Gram-negative bacterium Caulobacter

211 When starved for nitrogen, non-growing cells of the photosynthetic bacterium Rhodopseud

212 In the rapidly growing cells of the root elongation zone, we found MAP6

213 d application of GA to the relatively slowly growing cells of the unexpanded light-grown Arabidopsis

214 When growing cells on these substrates, it is found that cell

215 re stored in LDs, are typically mobilized in growing cells or upon hormonal stimulation by LD-associa

216 In growing cells, P-TEFb exists in active and inactive form

217 cipitation indicates that, in asynchronously growing cells, p53 occupies this element.

218 The CPAM, which is found only around growing cell plate regions, is suggested to be responsib

219 ects vesicles to the CPAM and thereby to the growing cell plate.

220 onsible for the majority of transcription in growing cells, Pol I regulation is poorly understood com

221 oriented Z rings and not by deformation of a growing cell pole or emergence of new tips from the side

222 the Rv1422 protein localizes uniquely to the growing cell pole, the site of peptidoglycan synthesis i

223 (SILAC) can be used to differentially label growing cell populations for quantitation of protein lev

224 Slow-growing cell populations located within solid tumors are

225 n alterations in their relative abundance in growing cell populations that have been transduced with

226 and competition that maximizes the health of growing cell populations.

227 We suggest that steadily growing cells prepare for conditions that demand increas

228 Normally growing cells prevent this response through constitutive

229 ce correlates with growth rate, where faster growing cells produce more flagella.

230 Rapidly growing cells produce thousands of new ribosomes each mi

231 Even when deformations are acquired, growing cells progressively recover their original shape

232 exert more severe cytotoxic effects on fast growing cells, providing an important molecular basis fo

233 Rather, Spo0A is present at high levels in growing cells, rapidly rising to yet higher levels under

234 ind that addition of hydrogen sulfide gas to growing cells recapitulates all aspects of reversible MT

235 Our results show that rapidly growing cells require significant methylation, likely fo

236 In growing cells, Sav1866 expression conferred resistance t

237 We found that non-growing cells shifted their metabolism to use the tricar

238 Wall expansion in tip-growing cells shows variations according to position and

239 Indeed, in exponentially growing cells, sprE deletion mutants exhibit significant

240 In growing cells, Ste11p is present in low levels and is pa

241 cond messenger, plays essential roles in tip-growing cells, such as animal neurons, plant pollen tube

242 which are normally repressed in vegetatively growing cells, such as targets of the Clr6 histone deace

243 are present in approximately 11% of actively growing cells, suggesting that the low frequency of Z ri

244 Rapidly growing cell suspension and adherent cells were effectiv

245 erium that is capable of differentiating non-growing cells that constitute gliding filaments termed h

246 However, in rapidly growing cells the prophage excises and replicates as an

247 In growing cells, the circuit gives rise to an occasional r

248 While BRI1 is ubiquitously expressed in growing cells, the expression of BRL1 and BRL3 is restri

249 In intercalary growing cells, the normal actin organization is disrupte

250 In exponentially growing cells, the nucleoid-binding protein H-NS downreg

251 anical signals are propagated around rapidly growing cells, the resistance to stress in adjacent cell

252 In tip-growing cells, the tip-high Ca(2+) gradient is thought t

253 with the high secretory dynamics of the tip growing cell; they show a pattern to the endoplasmic ret

254 For fast-growing cells, this necessitates rapid recycling of DNA

255 in the GTP pool within 10 min of addition to growing cells; this response does not result from the re

256 adjacent cells mechanically isolates rapidly growing cells, thus contributing to organ shape reproduc

257 collar of endocytic activity encircling the growing cell tip, which elongates through directed membr

258 rexpression leads to reduced diameter of the growing cell tip.

259 nthesized by membrane-bound synthases at the growing cell tip.

260 c42, found in a cap at the inner membrane of growing cell tips, as an important regulator of local ce

261 teins are interdependent for localization to growing cell tips.

262 Mitochondria must grow with the growing cell to ensure proper cellular physiology and in

263 Exposure of exponentially growing cells to iron-limited media, oxidative stress, a

264 single cyanobacteria enable a population of growing cells to maintain synchrony for weeks.

265 uction systems that are adapted to allow non-growing cells to sense and respond to their environment

266 nversely correlated with the capacity of non-growing cells to synthesize protein.

267 a function of acute and chronic exposure of growing cells to the drug.

268 ddition to increasing genetic instability in growing cells, Top1 activity in transcriptionally active

269 In exponentially growing cells translating ribosomes are interspersed amo

270 mply that PolIV can also promote mutation in growing cells under genome stress due to excess single-s

271 Slow-growing cells upregulate transposons and express more ch

272 resting cells and about 8 h in exponentially growing cells, values which are similar to the half-live

273 ates the activity of Pol IV in exponentially growing cells via a second, indirect pathway.

274 te that splicing is prevented in mitotically growing cells via inhibition, in contrast to the positiv

275 cks from Lipid II carrier molecules onto the growing cell wall scaffold during growth and division.

276 of the major load-bearing mechanisms of the growing cell wall.

277 Arabidopsis thaliana as a model of primary, growing cell wall.

278 of cross-links between peptide stems in the growing cell wall.

279 -mediated delivery of new polysaccharides to growing cell walls.

280 program with this array, an RNA extract from growing cells was compared with one prepared from develo

281 ion angle on the affected area of adherently growing cells was investigated both theoretically and ex

282 ddition to a 3.5-fold higher H2 yield by non-growing cells we also observed an accumulation of polyhy

283 For exponentially growing cells we observe high levels of inter-centromeri

284 In rapidly growing cells, we show that the major sites of RNA polym

285 uidic lab-on-a-chip device developed for tip-growing cells, we tested the ability to exert penetrativ

286 Exponentially growing cells were infected with the adenovirus vector,

287 gnal for the release, although only actively growing cells were proficient eDNA donors.

288 sHA, surrounding planktonic cells, and free-growing cells were recovered separately.

289 Similar changes were also observed when growing cells were treated with rapamycin, which is know

290 Most RNase R is sequestered on ribosomes in growing cells where it is stable and participates in tra

291 E conferred acid resistance to exponentially growing cells, whereas GadX overexpression did not.

292 sigma(S) is highly unstable in exponentially growing cells, whereas its stability increases dramatica

293 d cycle to metabolize acetate in contrast to growing cells, which used the glyoxylate cycle exclusive

294 While single cellular organisms and rapidly growing cells with high protein production have short NR

295 ested in both synchronized and exponentially growing cells with only one replicating chromosome.

296 network that controls the development of tip-growing cells with rooting functions among most extant l

297 Treatment of exponentially growing cells with the protein synthesis inhibitor cyclo

298 The distribution shifted to that found in growing cells within 3 h after serum stimulation.

299 We predict that growing cells without MreB exhibit an instability that f

300 In growing cells, xyloglucan is thought to connect cellulos