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1 turation, leading to the nanomelic long bone growth disorder.
2  mechanism of how JAK2 mutation induces cell growth disorder.
3 gs useful for the treatment of various human growth disorders.
4  underscored by its association with several growth disorders.
5 s are known as the genetic basis of skeletal growth disorders.
6 tations are associated with several dominant growth disorders.
7 ing genes associated with both developmental growth disorders and hematological malignancies.
8  overlap genes that are mutated in monogenic growth disorders and highlight new biological candidates
9 ew knowledge about the physical basis behind growth disorders and highlight the fact that a single RT
10 erlying principles of hair development, hair growth disorders, and skin cancers.
11 vide useful therapeutic tools for human hair growth disorders based on premature or retarded catagen
12 hylation of this gene is associated with the growth disorders Beckwith-Wiedemann and Silver-Russell s
13  IGF2-H19 expression and the two contrasting growth disorders, Beckwith-Wiedemann syndrome (BWS) and
14  in the long-term treatment of children with growth disorders, but there is considerable treatment re
15 yndrome is an autosomal-recessive primordial growth disorder characterized by significant intrauterin
16 sts may be useful for the management of hair growth disorders characterized by premature entry into c
17 dopsis (Arabidopsis thaliana) shows retarded growth, disordered chloroplast development, and delayed
18  an open study of consecutive referrals to a growth disorder clinic from which 16 short children (hei
19                           Common sequelae of growth disorders include neurodevelopmental defects, som
20 ith sporadic Wilms tumor without features of growth disorders, including 12% of bilateral cases (P =
21 ormation on the placental phenotype in fetal growth disorders--information which might contribute to
22                   3-M syndrome, a primordial growth disorder, is associated with mutations in CUL7 an
23 transcription factor in the brain in certain growth disorders may contribute to neurocognitive defici
24 onsidered a candidate gene for the imprinted growth disorder, the Silver-Russell syndrome (SRS), but
25 cling abnormalities underlie many human hair growth disorders, the accurate classification of individ
26 asia type I (TDI) is a lethal human skeletal growth disorder with a prevalence of 1 in 20,000 to 1 in

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