ライフサイエンスコーパス: growth phase

1 teria consume nutrients and enter stationary growth phase.

2 with the lack of Ace expression in the early growth phase.

3 isms to the drug is independent of bacterial growth phase.

4 ere age, site, Breslow thickness, and radial growth phase.

5 rization phase, an intermediate phase, and a growth phase.

6 into ordered structures that then enter the growth phase.

7 utions to obtain a sequential nucleation and growth phase.

8 criptional program that depend mainly on the growth phase.

9 and reaches its maximum at late exponential growth phase.

10 ty near neutral pH and during the stationary-growth phase.

11 mpact of infection occurring during the host growth phase.

12 ted and responsive to both growth medium and growth phase.

13 ylakoids when cells were in post-exponential growth phase.

14 itial seed concentration to the onset of the growth phase.

15 smegmatis is selectively modulated with the growth phase.

16 cripts were most abundant at the exponential growth phase.

17 hyma, and vascular tissues) at their maximal growth phase.

18 nt and wild-type (WT) strains in exponential growth phase.

19 cause of a shortage of ribosomes during this growth phase.

20 rature raised it; radiation impact varied by growth phase.

21 ulture is detectable only in the exponential growth phase.

22 n the resting pool and more committed in the growth phase.

23 eping close to 100% viability throughout the growth phase.

24 ination efficiency seems to be unaffected by growth phase.

25 ximal expression observed during exponential-growth phase.

26 ch as that seen during the early exponential growth phase.

27 of the tetQ-rteA-rteB operon is affected by growth phase.

28 nvert into protofibrils that elongate in the growth phase.

29 by RteA did not make excision independent of growth phase.

30 conditions made NBU1 excision independent of growth phase.

31 were expressed very early in the exponential growth phase.

32 wth phase and the potentially fatal vascular growth phase.

33 lf at NIH in 1951 during its most productive growth phase.

34 etermined by a global mechanism dependent on growth phase.

35 n as compared to cultures in the exponential growth phase.

36 zed to the inner bulge during the hair cycle growth phase.

37 rains of varying serotypes in the stationary growth phase.

38 es are translationally induced at transition growth phase.

39 y phase of ENSO is more predictable than the growth phase.

40 July 2014, when the outbreak entered a rapid growth phase.

41 enous jasmonates across the plant vegetative growth phase.

42 by increase in beta-sheet content during the growth phase.

43 terial growth, but not during the stationary growth phase.

44 ion-collapsing alkaline pH of the stationary growth phase.

45 ensity data for cycles near the onset of the growth phase.

46 for this process, but can enhance the later growth phase.

47 acterized by a lag phase followed by a rapid growth phase.

48 models, which involve an initial exponential growth phase.

49 pylori strains, genetic loci, and bacterial growth phases.

50 syn amyloid formation affecting both lag and growth phases.

51 depth and its evolution during the different growth phases.

52 ant occurred at middle, late, and stationary growth phases.

53 merases were constitutively expressed in all growth phases.

54 ulator (TcdC) allows toxin production at all growth phases.

55 es in M. pneumoniae during distinct in vitro growth phases.

56 es in cells grown to mid to late exponential growth phases.

57 n response to environmental changes, such as growth phases.

58 ed HL-60, and HL-60 synchronized in specific growth phases.

59 d found that ace mRNA levels were low in all growth phases.

60 chromosome during exponential and stationary growth phases.

61 BB0449 protein levels are low during various growth phases.

62 SELEX targeted bacterial cells at different growth phases.

63 During stationary growth phase, 285-GHz continuous wave EPR measurements s

64 After the first exponential growth phase, a short lag phase of nongrowth is observed

65 xamine differential gene expression in these growth phases, a microarray was constructed based on the

66 ep-wise transition to latent and overt tumor growth phases, a process that is preceded by recruitment

67 g phase at the proximal poly(A) site, but as growth phase advances, it extends to the distal site.

68 ia also illustrate that an extended pubertal growth phase allows very considerable height recovery, e

69 ls early insulin hypersecretion and a robust growth phase along with hyperexpression of related genes

70 Independence of growth phase also occurred when rteA and rteB were place

71 pression is somewhat constant throughout the growth phases, although it gradually decreases as cells

72 cular mechanism responsible for the end of a growth phase, an event called catastrophe, is still not

73 either Drosha or Dicer during an established growth phase (anagen) caused failure of hair follicles t

74 ed for hair follicle development and for the growth phase (anagen) of postnatal follicles.

75 hair follicle repetitively progresses from a growth phase (anagen) to a rapid apoptosis-driven involu

76 viduals, with an increased proportion in the growth phase, anagen.

77 t abundance of B. pertussis as a function of growth phase and availability of glutamate, a key nutrie

78 enes so that regulation is synchronized with growth phase and cellular energy status.

79 Depending on growth phase and culture conditions, cardiolipin (CL) ma

80 a continuous increase in activity during the growth phase and decreased during the stationary phase.

81 s to regulate gene expression in response to growth phase and environmental change.

82 eculation represents the initial ventricular growth phase and is necessary for embryonic survival.

83 The effects of salts on the growth phase and lag phase of IAPP amyloid formation are

84 l lines WM793 and especially WM239 (vertical growth phase and metastatic cells, respectively) overexp

85 Growth phase and nutrient availability may serve as cues

86 e expression of virulence genes according to growth phase and nutritional status.

87 expression of 60 genes independently of the growth phase and of 122 genes in a growth phase-dependen

88 val after ampicillin exposure throughout its growth phase and protected the population against exposu

89 ction between Cdc31 and Rad23/Rad4 varied by growth phase and reflected oscillations in Cdc31 levels.

90 data showed a transition between the initial growth phase and stable-state phase that, in the case of

91 Amount of 7-heptadecene varied with growth phase and temperature and was strictly dependent

92 riation in their binding profiles across the growth phase and the genome-scale nature of their impact

93 h, linking the relatively harmless avascular growth phase and the potentially fatal vascular growth p

94 and protects sigma(S) during the exponential growth phase and thus enables rapid gene activation by s

95 ice exhibit enhanced resting and abbreviated growth phases and are delayed in response to tissue-rege

96 normal cell replacement cycle throughout all growth phases and do not undergo spontaneous involution.

97 In woody species, growth phases and dormancy follow one another consecutiv

98 herichia coli) over time through lag and log growth phases and following antibiotic administration an

99 -3 cross-linkages predominate throughout all growth phases and the ratio of 4-3/3-3 linkages does not

100 show that production of BoNTs depends on the growth-phase and is under the control of positive and ne

101 , Spd-sr37 (80 nt; strongly expressed in all growth phases), and CcnA (93 nt; induced by competence s

102 al levels of 2.5 muM in the late logarithmic growth phase, and both wild-type and pigmentation (pgm)

103 ary-state markers are present throughout the growth phase, and increase in frequency with cell densit

104 t repression of hdeAB by MarA depends on pH, growth phase, and other regulators of hdeAB and is assoc

105 to eradicate persisters, MRSA in stationary growth phase, and showed significant clearance of intrac

106 into a fibrillar form occurred, marking the growth phase, and still more changes in the luminescence

107 lla larvae that was strain, infectious dose, growth phase, and T4SS dependent.

108 genome is modified in response to change in growth phase, and that 5% (68 genes) of the genome is te

109 attains the maximum level at mid-exponential growth phase, and the half-life of the transcript is les

110 es with a few more DP cells can re-enter the growth phase, and those that do exploit an intrinsic mec

111 , Spitzoid or Non-Spitzoid histology, radial growth phase, and vascular invasion.

112 rly flowering time, a prolonged reproductive growth phase, and, thereby, increased seed yield suggest

113 a that are expressed during the necrotrophic growth phase, as well as programmed cell death mediated

114 ate enhanced body and muscle size during the growth phase associated with elevated IGF1 levels.

115 ase at 37 degrees C and the late-exponential growth phase at 28 degrees C.

116 were expressed mostly in the mid-exponential growth phase at 37 degrees C and the late-exponential gr

117 om bacteria in the mid- and late-exponential growth phases at 28 degrees C and 37 degrees C correlate

118 ssful in prototroph yeast during exponential growth phase but not in stationary phase.

119 o be present in cotyledons during their main growth phase, but not later.

120 MCSP expression in a radial growth phase cell line also promotes an epithelial-to-me

121 ss the plasma membrane whereas in stationary growth phase cells Ca(2+) influx from intracellular and

122 d platelets, compared with that of the early growth phase cells in the high shear regime.

123 d accumulation of myosin II in the cortex of growth-phase cells.

124 During the early growth phase, ClfA is identified as the dominant staphyl

125 We show that cells harvested in exponential growth phase consistently display mixtures of 2-fold and

126 early a century ago, the molecular basis for growth phase control of speB gene expression remains unk

127 drate regulation of lctO as a key element of growth phase control.

128 ell into the late logarithmic and stationary growth phases, demonstrating the importance of DksA for

129 rential gene expression, morphology, and the growth-phase dependence of Hg transformations suggests t

130 nical strain of S. pyogenes and relieved its growth phase dependency.

131 of LspA2 but not LspA1 was shown to be both growth phase dependent and affected by the presence of f

132 The transcription of sarX was growth phase dependent and was expressed maximally durin

133 The expression of wosA was growth phase dependent during growth in liquid and on ag

134 his pattern of transcription was mirrored by growth phase dependent expression of the K1 capsule.

135 erogeneity of cap expression or the strictly growth phase dependent expression.

136 The growth phase dependent transcription was regulated by In

137 Expression of sarZ was growth phase dependent with maximal expression in the ea

138 The expression of covR is growth phase dependent, with maximal expression observed

139 xpression is also heterogeneous and strongly growth-phase dependent.

140 mutations on gene expression was analyzed in growth phase-dependent conditions using C medium, report

141 involved in polysaccharide utilization show growth phase-dependent expression in human saliva.

142 that the T2S pathway is characterized by the growth phase-dependent expression of genes encoding carg

143 nteracts with the cofactor LacD.1 to control growth phase-dependent expression of genes, including sp

144 This growth phase-dependent gene expression is controlled tra

145 axis is determinative for regulation of the growth phase-dependent gene expression.

146 Growth phase-dependent gene regulation has recently been

147 Bvg(-) phase-locked mutants, indicating that growth phase-dependent gene regulation in B. bronchisept

148 analysis revealed and qRT-PCR confirmed that growth phase-dependent gene regulation occurred in both

149 ative real-time PCR (qRT-PCR) confirmed that growth phase-dependent gene regulation occurs in B. bron

150 iseptica-like ancestor, we hypothesized that growth phase-dependent gene regulation would also occur

151 olymerase sigma initiation factors regulates growth phase-dependent gene transcription.

152 anning hours to weeks, we establish distinct growth phase-dependent hierarchies of polymerase mutant

153 e III secretion system (TTSS) genes led to a growth phase-dependent increase in a TTSS-dependent func

154 B0693) as a negative regulator that controls growth phase-dependent induction of rpoS and bosR in Bb.

155 Our data identify CPK28 as a growth phase-dependent key negative regulator of distinc

156 sigmaE activity increases in a growth phase-dependent manner as a culture enters statio

157 of several JA metabolites were elevated in a growth phase-dependent manner in cpk28, and accumulation

158 In addition, PepO protein was secreted in a growth phase-dependent manner to the culture supernatant

159 coli that nanoRNAs prime transcription in a growth phase-dependent manner, resulting in alterations

160 ated by the PhoB/R two-component system in a growth phase-dependent manner, which is coordinated with

161 unt of Pah1p; enzyme abundance declined in a growth phase-dependent manner.

162 DnaB is truncated at its C-terminus in a growth phase-dependent manner.

163 ly of the growth phase and of 122 genes in a growth phase-dependent manner.

164 at rpf gene expression may be regulated in a growth phase-dependent manner.

165 in Streptococcus pyogenes is controlled in a growth phase-dependent manner.

166 Production of H(2)O(2) follows a growth phase-dependent pattern that mimics that of many

167 dative phosphorylation is a primary cause of growth phase-dependent persistence to quinolone antibiot

168 es, demonstrating the importance of DksA for growth phase-dependent regulation of fis.

169 , we show that RNase BN itself is subject to growth phase-dependent regulation, because both rbn mRNA

170 In this study, we examined the growth phase-dependent speB mRNA level and decay using q

171 ughout V. cholerae growth, whereas there was growth phase-dependent transcriptional activity of genes

172 lycogen accumulation were carbon source- and growth phase-dependent, and were repressed by glucose.

173 wo closely related genes whose expression is growth phase-dependent.

174 NAs and formation of the L1 and L2 loops are growth phase-dependent.

175 f atcRS during late-stage growth, indicating growth-phase-dependent expression.

176 BCP-deficient B. cenocepacia exhibit a growth-phase-dependent hypersensitivity to oxidative kil

177 er and slowly yields to the EA1 S layer in a growth-phase-dependent manner.

178 amount and type of recombination events in a growth-phase-dependent manner.

179 e with iron transport and the correlation of growth-phase-dependent morphology with MeHg production a

180 group B streptococci) during the exponential growth phase (designated early GPCSs) or at the senescen

181 iably distinguish between the nucleation and growth phases, despite extensive and diverse attempts.

182 r to adulthood with no secondary accelerated growth phase during adolescence, 4) beta-cell mass (and

183 developmental switch out of an initial "pro-growth" phase during which muscle arms grow out and form

184 itory phase between the attack phase and the growth phase, during which the bdelloplast (the invaded

185 Moreover, RteA is not required for the growth phase effect, because a mutant form of RteB that

186 four-electrode configuration along all cell growth phases, enabling determination of relevant cell g

187 found that excision is also associated with growth phase; even after exposure to tetracycline, excis

188 factor with IHF playing a role in regulating growth phase expression.

189 During the late growth phase, fibrinogen in concert with von Willebrand

190 ctivation, quiescent naive T cells undergo a growth phase followed by massive clonal expansion and di

191 Follicles recruited into the growth phase following formation of multiple layers of g

192 at cell cultures have reached an appropriate growth phase for addition of an agent to induce protein

193 on natural killer T cells helped to launch a growth phase for this field of research.

194 Substrates consumed in the second growth phase (glutamate, proline, and associated organic

195 haping the Drosophila wing during its larval growth phase has been limited, impeding our ability to u

196 In the subsequent growth phase, hierarchical agglomeration could be a domi

197 ontrolled virulence genes at the exponential growth phase; however, mutations of RocA but not mutatio

198 he pathogen's early (day 2 to 3) exponential-growth phase in an experiment involving synchronized inf

199 s significantly stabilized at the stationary growth phase in Cue1p-deficient yeast.

200 omic DNA methylation levels as a function of growth phase in Escherichia coli.

201 teobacteria, it is ubiquitous throughout all growth phases in S. aureus The biological significance o

202 mples was in accord with different bacterial growth phases in sequential lavage specimens.

203 us, and its increased production during late growth phases indicates that c-di-AMP controls processes

204 the crossover time (t(c)), the slope of the growth phase (k(g)), and the arrest time (t(a)).

205 Paradoxically, the growth phase-mediated inductions of PAH1 and phosphatida

206 mental and cellular state, including strain, growth phase, medium, oxygen level, antibiotic and carbo

207 in normal immortal melanocytes, early radial growth phase melanoma, and metastatic melanoma cells.

208 t forced expression of OPN in early vertical-growth-phase melanoma cells dramatically increased their

209 Overexpression of OPN in vertical-growth-phase melanoma cells induced down-regulation of C

210 c melanoma cell lines, derived from vertical growth phase (MelJuSo) and metastatic (SKMel28) melanoma

211 mperature (35 degrees C or 40 degrees C) and growth phase (mid-exponential or stationary phase).

212 otein-DNA complexes formed by Fis, the major growth phase nucleoid protein, have a markedly different

213 h a first-order kinetic model for the single growth phase observed.

214 Here we show that the growth phase of Arabidopsis seedlings in diurnal light c

215 ly, consistent with aestivation, whereas the growth phase of both A. gambiae s.s. and A. arabiensis l

216 ile influenced by compound glycosylation and growth phase of cultured cells.

217 veloping on hard substrates and report a new growth phase of MDCK II cells on soft gels.

218 ein expressed at high levels in the vertical growth phase of melanoma, promotes disease progression t

219 trated by the coincidence of the exponential growth phase of Nitella and the point of maximum enhance

220 ining characteristics showed variations with growth phase of the bacteria.

221 es that enabled direct identification of the growth phase of the bacteria.

222 ants/mug DNA) was obtained at the stationary growth phase of the bacterium (OD 6.0) using 25 ng of pl

223 at binucleate cells are delayed in the first growth phase of the cell cycle (G1) and undergo interpha

224 nuclei/10(6) myocyte nuclei, P<0.05) and the growth phase of the cell cycle (Ki67; 410+/-82 to 1261+/

225 logical evidence of myocytes re-entering the growth phase of the cell cycle and increases in the numb

226 r they originate from skin in the resting or growth phase of the hair cycle or skin with beta-catenin

227 t for V. cholerae PBP1a pathway mutants, the growth phase of the inoculum is a key modulator of infec

228 in culture may be strongly influenced by the growth phase of the target cells.

229 tions in the radial (RGP) and vertical (VGP) growth phases of individual melanoma tumors.

230 ression of ldtMt2 is dominant throughout the growth phases of M. tuberculosis.

231 yzing the metabolic changes accompanying the growth phases of medically treated AAA.

232 In contrast, cells in stationary growth phase or cells treated with a protonophore causin

233 ABC mutant lacks detectible CL regardless of growth phase or growth conditions.

234 gulation of speB transcription at stationary growth phase or in subcutaneous infection of mice.

235 rapid and synchronous cell divisions without growth phases or cell cycle checkpoints.

236 expression in radial growth phase, vertical growth phase, or metastatic cell lines causes sustained

237 e found to increase in number throughout the growth phases, particularly in the stationary phase.

238 an uncoupling of lctO transcription from its growth phase pattern.

239 hafniense strain Y51 was affected by neither growth phase, pceA transcription, or translation, nor by

240 ssion changes between biofilm and stationary growth-phase planktonic cultures.

241 ntiation in which exponential and stationary growth phases play key biological roles.

242 S exposure on the surface of late stationary growth phase promastigotes from patients with LCL, compa

243 macrophage interaction with late stationary growth phase promastigotes in which PS was blocked by an

244 expressed on the surface of early stationary growth phase promastigotes.

245 We studied the role of the growth-phase-regulated outer membrane protein OpnS in ho

246 e distinct mechanisms and that disruption of growth phase regulation alters transcriptional patterns

247 ctivity and may therefore play a role in the growth phase regulation of toxin activity.

248 requirements, toxin gene expression is under growth phase regulation.

249 stal promoter indicated that it accounts for growth-phase regulation and DNA damage inducibility.

250 To begin an analysis of growth-phase regulation, we compared the transcriptome 2

251 No comprehensive investigation of growth phase-related gene regulation in Bordetella pertu

252 Cells in exponential growth phase released 220 exosomes/cell in culture medi

253 ession during the logarithmic and stationary growth phases respectively.

254 ring carbon availability or in fueling later growth phases, respectively, has been proposed.

255 low phosphate and this increase prolongs the growth phase, resulting in the development of long root

256 nsition of melanoma from non-invasive radial growth phase (RGP) to invasive and metastatically compet

257 but not vWF, supports the adhesion of early growth phase S. aureus at the high wall shear rates.

258 d to quantify the adhesion of early and late growth phase S. aureus to immobilized platelet layers as

259 ciency for the adhesion of early versus late growth phase S. aureus to immobilized platelets.

260 t adhesion mechanisms between early and late growth phase S. aureus under physiological shear conditi

261 During both growth phases, several genes carried in O-islands were a

262 ystem, and a poorly defined post-exponential growth phase signal independent of Agr.

263 genetic stability approximately tenfold and growth phase-specific productivity approximately fourfol

264 Our findings identify StpA as a novel growth phase-specific regulator that plays an important

265 es C), to probe for temperature-specific and growth phase-specific transcriptomes.

266 f cobalt, nickel, and sodium showed distinct growth-phase-specific patterns.

267 arkedly elevated binding efficiency for late growth phase staphylococci to immobilized platelets, com

268 Regulation by StpA varied with growth phase; StpA controlled sigma(38) levels at mid-ex

269 l conditions such as antibiotic exposure and growth phase, suggesting that observed shifts in the pha

270 gene in a codY mutant during the exponential-growth phase, suggesting that the quorum-sensing system,

271 of uptake was no longer documented after the growth phases, suggesting a pattern of cyclic metabolic

272 roscopy performed at different points in the growth phase support our proposed model in which quorum

273 expression of numerous genes throughout the growth phases tested, namely, late log to early stationa

274 phase (of approximately 150 h) followed by a growth phase that plateaus, whereas 4-HNE, MDA, and GLY

275 cally homogenous, whereas in the exponential growth phase, the average wall stiffness increased, with

276 ssive phenotype and the postexponential (PE) growth phase, the pathogens express virulence factors, b

277 ition if initial infection occurs during the growth phase then an additional "invasion zone" can exis

278 reorganization of the oligomers, whereas the growth phase ultimately results in the formation of fibr

279 essed during late exponential and stationary growth phase under normal growth conditions, whereas the

280 chicken cecum and in two different in vitro growth phases using strand-specific RNAseq.

281 cycle of flowering plants into two distinct growth phases, vegetative and reproductive, is marked by

282 ed levels of Ace surface expression at early growth phase versus those of wild-type OG1RF.

283 results show that MCSP expression in radial growth phase, vertical growth phase, or metastatic cell

284 vasive and metastatically competent vertical growth phase (VGP) is a major step in tumor progression,

285 -factorial experimental design we found that growth phase, voltage, and resistance all significantly

286 of the LCL strain during the late stationary growth phase was highly expressed on the surface of earl

287 changes in response to temperature shift and growth phase was the induction of known B. quintana viru

288 produced during the late stage of stationary growth phase, was discovered and purified from the cultu

289 isms coupling virulence factor expression to growth phase, we investigated the molecular basis for H(

290 ant strain during logarithmic and stationary growth phases, we determined that PknK regulates the exp

291 ogen removal rates during the initial linear growth phase were 17 and 122 mg.L(-1).d(-1), respectivel

292 ss section of the solutes during the fibrils growth phase were thus recovered.

293 speB transcript level during the exponential growth phase, whereas provision of only the amino-termin

294 antanethiolate monolayer nucleates an island growth phase, which is followed by slow ordering of the

295 growth pattern and exhibited only the first growth phase, which is marked by the consumption of aspa

296 normally exhibits a lag phase followed by a growth phase, which leads to amyloid fibrils.

297 and declined sharply during mid-to-late log growth phases, which was in direct contrast to other myc

298 ntracellular bacteria are in the exponential growth phase with a reduced replication rate and biochem

299 is disabled, consistent with the exponential growth phase with an infinite replicative capacity.

300 r dioxide, and nitrogen dioxide in different growth phases with clinically assessed pubertal stage at

301 d melanoma cells originating from the radial growth phase (WM35) and from lung metastasis (A375-P).