ライフサイエンスコーパス: growth potential

1 tep in HMEC acquisition of uniform unlimited growth potential.

2 tic twin pairs naturally matched for genetic growth potential.

3 03X) caused dramatic protection from loss of growth potential.

4 are compromised in their differentiative and growth potential.

5 pulation doublings without any diminution in growth potential.

6 static cancer cells without decreasing their growth potential.

7 tic potential without interfering with their growth potential.

8 in concert to modify an individual's genetic growth potential.

9 regulate genes important in regulating cell growth potential.

10 he fetus to reach its genetically determined growth potential.

11 ufficient to enhance tumor cell survival and growth potential.

12 ich balances leaf construction costs against growth potential.

13 because all existing graft materials have no growth potential.

14 whether it can be altered to enhance axonal growth potential.

15 activity as a critical regulator of neuronal growth potential.

16 ve carbonate budget states and reducing reef growth potential.

17 concerns over reef carbonate production and growth potential.

18 ereby the fetus fails to achieve its genetic growth potential.

19 molog (PTEN) is deleted to enhance intrinsic growth potential.

20 declines are now suppressing Caribbean reef growth potential.

21 cy must be distinguished from modest genetic growth potential.

22 e to growth inhibitors and lack of intrinsic growth potential.

23 butes to the age-related decline of the axon growth potential.

24 rogenitor cell population to preserve future growth potential.

25 ith good clinical status fail to reach their growth potential.

26 tributable to a difference in intrinsic axon growth potential.

27 nd Shh(-/-);Gli3(-/-) lungs exhibit enhanced growth potential.

28 lls were compared in terms of their in vitro growth potential.

29 TX/96), to measure in vitro reassortment and growth potentials.

30 in genomic sequence, iPSCs acquire unlimited growth potential, a characteristic shared with cancer ce

31 nd/or Slug has been correlated with invasive growth potential, a property primarily attributed to the

32 ter inhabited by a population, the lower its growth potential, a relationship presumably molded by na

33 On the basis of growth potential, ability to re-initiate ES cultures and

34 type growth kinetics with subsequent loss of growth potential after which survivors are generated via

35 nic cells displayed unlimited stem-cell-like growth potential and a stable phenotype in culture.

36 al traits beyond the acquisition of enhanced growth potential and decreased cell death.

37 steal cell populations were tested for their growth potential and for expression of conventional mark

38 d cancer stem cell (CSC) phenotypes based on growth potential and gene expression signatures that rep

39 ablish a correlation between diminished axon growth potential and histone 4 (H4) hypoacetylation.

40 Whether disturbed reefs can recover their growth potential and how rapidly, are thus critical rese

41 er, the signalling pathways that enhance the growth potential and induce spontaneous axon regeneratio

42 sed in keratinocytes with long-term in vitro growth potential and is coexpressed with high levels of

43 ssages when the majority of cells lost their growth potential and neared senescence but p21 levels de

44 d cold treatment alone had minimal affect on growth potential and resulted in ~1% flowering.

45 pha(+)Sca-1(+) (PalphaS) MSCs have augmented growth potential and robust tri-lineage differentiation

46 nse to climate change is dependent on innate growth potential and the discrepancy between the two opt

47 nate budgets are declining, threatening reef growth potential and thus capacity to track rising sea-l

48 and the suppression of anchorage-independent growth potential and tumor formation in nude mice.

49 La cells and have lost anchorage-independent growth potential and tumorigenicity.

50 kade with an anti-EGFL7 antibody reduced the growth potential and viability of AML cells.

51 Engineered HSPCs displayed altered in vitro growth potentials and induced acute leukemias following

52 ncluding 6-fold less scarring, 40% increased growth potential, and 4-fold more hypertrophic chondrocy

53 The transformed phenotype, growth potential, and actin cytoskeleton of 749r-1 cells

54 vironmental exposures that can limit genetic growth potential appear to have lessened, and variation

55 red axons must overcome their poor intrinsic growth potential as well as the inhibitory environment o

56 5 cells, resulted in a profound reduction in growth potential, as determined by the colony formation

57 These cells show increased growth potential at 33 degrees C, but on shift to the no

58 rapid growth might lead to an exhaustion of growth potential before progression to clonal immortalit

59 inventory costs and significantly increases growth potential but necessitates active phloem loading.

60 y intraclonal heterogeneity and hierarchy of growth potential, but also plasticity of cellular differ

61 imulates germination not by enhancing embryo growth potential, but by weakening the micropylar region

62 ly acceptable level without sacrificing bone-growth potential, but COX-associated inflammation appear

63 Inosine augments neurons' intrinsic growth potential by activating Mst3b, a component of the

64 mechanosensing of the increase in embryonic growth potential by GA action.

65 oducibly isolated and had markedly increased growth potential compared to uninfected cells; HTLV-2 tr

66 rongly enhanced and Wnt-dependent clonogenic growth potential compared to virtually identical populat

67 steps resulting in acquisition of unlimited growth potential, evasion of apoptosis and non-responsiv

68 phenotypically distinct cells with enhanced growth potential exist within the normal arterial media,

69 to come, this work illustrates the power and growth potential for association studies of human ageing

70 xamined for their levels of gene expression, growth potential in cell culture, and virulence in mice.

71 ed that equine MaSCs (eMaSCs) maintain their growth potential in culture for an indefinite period, wh

72 gotes exhibited a slight decrease in overall growth potential in culture.

73 ilability of t(6)A-modified tRNA, determines growth potential in eukaryotic cells.

74 g carcinoma cells (3LL) have more aggressive growth potential in IL-10 transgenic mice compared with

75 od, whereas canine MaSCs (cMaSCs) lose their growth potential in long term cultures.

76 sed to CC had no alteration of cell shape or growth potential in monolayer culture, however, a statis

77 on and resulted in a concomitant increase in growth potential in response to M-CSF.

78 e side effects of selection, both broadening growth potential in some conditions and narrowing it in

79 oning lesion to stimulate intrinsic neuronal growth potential in the absence of substrate modificatio

80 mediates PI3K-dependent augmentation of the growth potential in the PNS.

81 or negatively enrich for cells with enhanced growth potential in these assays.

82 mine whether clonogenic cells with long-term growth potential in vitro persist in vivo and give rise

83 ia and neuroblastoma, can be induced to lose growth potential irreversibly and terminally differentia

84 Tumor regrowth indicates that clonogenic growth potential is continually maintained, but the dete

85 Cell growth potential is determined by the rate of ribosome b

86 Axon growth potential is highest in young neurons but diminis

87 lecular mechanisms underlying this unlimited growth potential is of broad interest for tooth regenera

88 path to carcinogenesis, the key to unlimited growth potential lies in overcoming the steady loss of t

89 ic cell types also prove to be restricted in growth potential, not identical to the corresponding pos

90 r culture on stromal layers, we assessed the growth potential of 70 cases of newly diagnosed B-lineag

91 striction (IUGR) is a failure to achieve the growth potential of a fetus that is promised by the gene

92 nt of CDC2 but not CDK2 protein; a decreased growth potential of Adp21WAF1/CIP1-infected cells demons

93 A broadly known method to stimulate the growth potential of axons is to elevate intracellular le

94 cultured glioma cells secrete glutamate, the growth potential of brain tumors has not yet been linked

95 We also demonstrated the growth potential of C. formicarius on these two host pla

96 sformation and in elevating the survival and growth potential of cancer cells.

97 uences both the contractile activity and the growth potential of cardiac myocytes.

98 ns has been attributed to a loss of inherent growth potential of cells and to inhibitory signals asso

99 es deleterious effects of Lig4 deficiency on growth potential of embryonic fibroblasts (MEFs) and gen

100 Growth potential of embryos isolated from seeds pretreat

101 In a cross-sectional study, the number and growth potential of eosinophil-lineage-committed progeni

102 r alterations responsible for the aggressive growth potential of epidermal growth factor receptor (EG

103 ng an experimental condition for testing the growth potential of functioning heart in the absence of

104 es suggested that E. faecalis suppressed the growth potential of GBS in SBM.

105 Cell proliferation assay revealed a higher growth potential of GM3 KO MEFs.

106 he above findings suggest that the increased growth potential of human lens epithelial cells by Ad12-

107 cifically attenuates proliferation and tumor growth potential of human melanoma cells expressing BRAF

108 e investigated separately effects of SHAM on growth potential of isolated embryos as well as on endos

109 romosome number of 51 to 65, we assessed the growth potential of leukemic cells from 129 children wit

110 ent in establishing infection, impairing the growth potential of lung epithelial cells and thereby sl

111 s a negative regulator of the metastatic and growth potential of malignant cells and strongly suggest

112 tion inhibited tumorigenesis by reducing the growth potential of melanoma cells.

113 rtially accounts for the different postnatal growth potential of molars and incisors.

114 rming assays demonstrated an increase in the growth potential of monocyte progenitors and a significa

115 alysis was able to qualitatively predict the growth potential of mutant strains in 86% of the cases e

116 lymphoma (BL) cells and to contribute to the growth potential of other B-cell lymphoma-, gastric carc

117 during the diauxic switch and the long-term growth potential of the cell.

118 llular metabolism and cell division with the growth potential of the cell.

119 nly increased after a significant decline in growth potential of the culture.

120 Thus, as the growth potential of the environment decreases, cells app

121 eurotrophin 3 during exercise, the increased growth potential of the exercise-conditioned animals req

122 in the heterozygote mice matches the limited growth potential of the great majority of TSC hamartomas

123 he number of susceptible cells influence the growth potential of the virus?

124 splantation experiments designed to test the growth potential of these lesions.

125 these interactions can explain the unlimited growth potential of these tumors.

126 pecific niche signals can restore metastatic growth potential of tumor cells lacking one of the oncog

127 vels of ROS associated with the uncontrolled growth potential of tumor cells.

128 ted and soil was rehydrated to determine the growth potential of underground adventitious buds.

129 Leaf surface temperatures and growth potentials of plants growing under well-watered c

130 nucleation mechanisms are discussed and the growth potentials of the nuclei are also analyzed and di

131 rum IGF-1 levels, but this neither predicted growth potential or skeletal integrity nor defined growt

132 f certain neuronal populations that retain a growth potential over time, and lead to functional impro

133 of Pak1 regulation of anchorage-independent growth, potential Pak1 regulation of invasiveness, and a

134 ytokines and demonstrate that this increased growth potential precedes polyploidization of the cultur

135 ory factors in the lesion scar and poor axon growth potential prevent axon regeneration.

136 gnificantly reduced capacity to enhance cell growth potential relative to BL cells expressing wild-ty

137 ying this EBER-dependent enhancement of cell growth potential remain to be elucidated.

138 t a fraction of adult mouse hepatocytes have growth potential similar to that of hematopoietic stem c

139 xons are double conditioned to enhance their growth potential, some traverse the lesion core and expr

140 ein (MEZ) results in an irreversible loss in growth potential, suppression of tumorigenic properties

141 os from dormant seeds, however, had a lesser growth potential than those from nondormant seeds.

142 poietic cells leads to functional defects in growth potential that may be of consequence to leukemic

143 merase-deficient cells began to show loss of growth potential, the cells arrested in G2/M and showed

144 ut is most often competitively excluded; and growth potential, the innate capacity for growth at the

145 ic effect most closely related to population growth potential; the colder the winter inhabited by a p

146 roximal-distal growth axes with two types of growth potential: they can be indeterminate, in which ca

147 be crucial for acquisition of the unlimited growth potential thought to be critical for malignant pr

148 control system, suggest that the cell trades growth potential to avert the potential toxicity associa

149 cl-2 is an oncogene that confers deregulated growth potential to B lymphocytes through its ability to

150 Enhanced expression of MYCN confers growth potential to neuroblastoma cells, and a direct li

151 er a complete SCI in rats improves intrinsic growth potential to result in axon regeneration out of a

152 o confer anchorage independence and invasive growth potential to transformed cells.

153 ively active Rheb to enhance their intrinsic growth potential, transplanted a growth supporting perip

154 at exerts its detrimental, highly aggressive growth potential upon escape from cell-cycle blockade, a

155 This extension of the in vitro growth potential was accomplished without any of the obv

156 Embryo growth potential was quantified by incubating decoated e

157 te index of each site, which is a measure of growth potential, was varied to represent different leve

158 PCR in aggressive MPM cells attenuated their growth potential, whereas EPCR silencing in nonaggressiv

159 changes have driven major reductions in reef growth potential, which have declined from an average 4.

160 tween target cell availability and the virus growth potential with a combination of experimental and