ライフサイエンスコーパス: growth state

1 ilm in a viable but antibiotic-tolerant slow-growth state.

2 influences tumor progression to an invasive growth state.

3 red to reflect a switch back to a vegetative growth state.

4 rowth and entry into a stationary phase-like growth state.

5 ulent at the skin, regardless of its initial growth state.

6 a coli cells from an aerobic to an anaerobic growth state.

7 lation of CAD were measured as a function of growth state.

8 is enzyme is required, according to cellular growth state.

9 growth factor and not mediated by changes in growth state.

10 ransition from a stationary to proliferative growth state.

11 nsition from a stationary to a proliferative growth state.

12 ooth muscle cells into a quiescent G(0)-like growth state.

13 olonged period of suppressed budget and reef growth states.

14 h its formulation and use to compute optimal growth states.

15 to assess transcription during each of these growth states.

16 rcoil barriers than wild-type strains in all growth states.

17 stoma and WI38 fibroblast cells in different growth states.

18 ires reactivating injured neurons' intrinsic growth state and enabling growth in an inhibitory enviro

19 nglion cells (RGCs) to switch into an active growth state and extend lengthy axons down the nerve.

20 esults suggest that the response may be cell growth state and protein kinase C-dependent.

21 en nuclei and mitochondria, depending on the growth state and stress environment of the cell.

22 visual input, the eyes revert to a "default" growth state and that the similarities between the effec

23 o transit from the biofilm to the planktonic growth state and to spawn novel communities in new local

24 fully transit from a biofilm to a planktonic growth state and to spawn novel communities in new local

25 ding nutrient availability, and adjust their growth state and virulence functions accordingly.

26 es MnSOD expression in response to different growth states and radiation.

27 hen E. coli cells transition to an anaerobic growth state, and that the expression of 712 (49%) of th

28 expression profile and switch into a strong growth state; and (2) whether inactivating RhoA, a conve

29 When neurons are triggered into a growth state, as in the conditioning lesion paradigm, H4

30 BP mRNA, and its expression was regulated by growth state, being most prominent in quiescent endothel

31 motor neurons to stimulate their endogenous growth state, bone marrow stromal cell grafts in lesion

32 To elucidate the factors responsible for growth state-dependent regulation of pyrimidine biosynth

33 These data show that the growth-state-dependent tyrosine phosphorylation of p27 m

34 y and noninhibitory, due to its differential-growth-state-dependent tyrosine phosphorylation.

35 indicating that the bacteria were in a rapid growth state despite specific nutrient limitations.

36 bet hedging results from multiple epigenetic growth states determined by a combination of stochastic

37 eradicates cells grown in numerous different growth states (e.g. planktonic cultures and highly robus

38 the existence of bacteria in two additional growth states, filamentous and biofilm encased.

39 IGF-1R was observed during the transition of growth states from exponential to quiescent.

40 Maintenance of the stationary phase growth state has been proposed to be critical for the vi

41 t the new pole attractor; and (5) the robust growth state identified by Wang et al. corresponds to ou

42 ricyte shape, contractility, and endothelial growth state in microvascular cell co-cultures.

43 ransition from the biofilm to the planktonic growth state in response to various cues.

44 for maintenance of the anchorage-independent growth state in RK3E cells transformed by beta-catenin,

45 y, but rather is expressed during a range of growth states in a dynamic environment.

46 Adjusting endothelial metabolism to the growth state is central to normal vessel growth and func

47 ics in response to nutrient availability and growth state is poorly understood, yet essential for the

48 e with cells that accumulate during abnormal growth states, like prostate cancer.

49 mounting for the importance of the intrinsic growth state of a neuron as a crucial determinant of its

50 scriptional switch that promotes an enhanced growth state of adult sensory neurons.

51 (FI, B-50 or neuromodulin) in regulating the growth state of axon terminals.

52 cate that this is achieved through a boosted growth state of dorsal column projecting sensory neurons

53 The intrinsic growth state of injured neurons is, therefore, a key det

54 rve regeneration by increasing the intrinsic growth state of injured neurons.

55 tion through the mature optic nerve when the growth state of neurons is activated.

56 ong regeneration even without activating the growth state of neurons.

57 The growth state of P. aeruginosa in CF airways is probably

58 nvestigated whether increasing the intrinsic growth state of primary sensory neurons by a conditionin

59 nts that synergistically alter the intrinsic growth state of RGCs produce unprecedented levels of axo

60 tion of retraction was dependent on both the growth state of the axon and the activation state of the

61 , modified backbone, probe concentration and growth state of the bacteria were investigated.

62 not known whether the metabolic pathways and growth state of the bacterium influence synthesis and se

63 ption, tightly coupled to the cell cycle and growth state of the cell, is a key process for understan

64 operons, regulons, and stimulons to suit the growth state of the cell.

65 ed the effect of critical parameters such as growth state of the cells and availability of intracellu

66 to grow within D. discoideum depended on the growth state of the cells.

67 ct seems to be primarily associated with the growth state of the epithelium.

68 hibition was conditional, dependent upon the growth state of the inhibitory cell and the pili express

69 ontact with agar ingredients rather than the growth state of the microorganisms determined the infect

70 ion of effectors that increase the intrinsic growth state of the neurons.

71 we have found to be expressed inversely with growth state of the SMC.

72 um-derived NO controls the contractility and growth state of the underlying vascular smooth muscle ce

73 We determined the influence of cellular growth state on DEN type 2 (DEN2) replication in mosquit

74 stresses impact a cell's decision to enter a growth state or a quiescent state.

75 e-entry into the cell cycle from sub-optimal growth states rather than promoting or controlling the p

76 s cerevisiae exhibits alternative vegetative growth states referred to as the yeast form and the fila

77 ted mRNA was in the polysome fraction in all growth states regardless of labeling time, indicating th

78 and beta-lactams, giving rise to an abnormal growth state termed persistence.

79 ect and glutamine addiction as features of a growth state that provides resistance to metabolic stres

80 oietic stem cells (HSCs) can convert between growth states that have marked differences in bioenerget

81 al microtubules and was dependent on polymer growth state; the microtubule-related fluorescence dissi

82 ironmental stresses and transitions from one growth state to another, the transcriptional coupling th

83 ady-state cellular pre-16S rRNA pools during growth state transitions in Escherichia coli.

84 during metabolic adjustments associated with growth state transitions.

85 mooth muscle cells (SMCs) are in a quiescent growth state under normal physiological conditions, but

86 This enhanced growth state was accompanied by an increase in the expre

87 both regions, transitions from quiescent to growth states were accompanied by reorganization of MTs

88 ely other factors) defines two distinct axon growth states, which are critical for proper circuit for

89 the hypothesis that manipulation of cellular growth state will facilitate identification of viral and

90 s) are plastic cells that can switch between growth states with different bioenergetic and biosynthet