ライフサイエンスコーパス: growth stimulation

1 n essential component for DNA synthesis upon growth stimulation.

2 SAPK pathway that correlated with a 150-190% growth stimulation.

3 ompounds counteracted exogenous ET-1-induced growth stimulation.

4 e normal control of E2F2 expression during a growth stimulation.

5 S may be an effector of Ras in cAMP-mediated growth stimulation.

6 was expressed at a constant level following growth stimulation.

7 crimination of aberrant and normal levels of growth stimulation.

8 tion, elevated BfeA receptor production, and growth stimulation.

9 d, plant-to-plant signalling for defence and growth stimulation.

10 content or stability of UAP56 mRNA following growth stimulation.

11 of rp mRNA do not change significantly after growth stimulation.

12 le the signal transduction pathways for cell growth stimulation.

13 y the response of these livers to additional growth stimulation.

14 ls and partially mediated the PGE(2)-induced growth stimulation.

15 ivation of the E2F3a promoter in response to growth stimulation.

16 wild-type Smad4 abolished the BMP-2-mediated growth stimulation.

17 can be a determinant of tumor E2 content and growth stimulation.

18 region, designated yfe, essential for SAB11 growth stimulation.

19 define the molecular mechanism of carcinoma growth stimulation, a three-dimensional co-culture model

20 a cells, which are responsive to TGFbeta1 by growth stimulation, a truncating mutation at Gln-311 was

21 On the other hand, growth stimulations also lead to PKM2 phosphorylation, w

22 RNA and protein are increased in response to growth stimulation, although the precise regulatory mech

23 umber of cellular phenotypes as divergent as growth stimulation and growth inhibition.

24 to monitor heparin's potentiation of LEDGF's growth stimulation and heparin's role in the translocati

25 structural factor controlling both microbial growth stimulation and iron uptake.

26 tes to RET kinase activation, signaling, and growth stimulation and may therefore be an attractive th

27 inked to cell division, as it was induced by growth stimulation and repressed by growth inhibition.

28 Remarkably, cell growth stimulation and suppression of apoptosis by endog

29 C1 were hypersensitive to estrogen-dependent growth stimulation and that DLC1 had an accelerating eff

30 te the signal transduction pathways for cell growth stimulation and therapeutic resistance.

31 'Nipponbare') seedlings had a slow onset of growth stimulation, and barley (Hordeum vulgare) had a t

32 in embryogenesis, tissue regeneration, cell growth stimulation, and cell proliferation.

33 ndance of eEF-2 kinase have been observed on growth stimulation, and increased enzyme activity is cha

34 ation rates and mitotic index 24 hours after growth stimulation, and the ability of RPE cells to repo

35 Growth stimulation appears to be due to a reduction in t

36 tivate mitogenesis, while in the presence of growth stimulation, as is frequently seen with vasculopa

37 ular myocytes, including evidence indicating growth stimulation at concentrations in the same range a

38 vitro or entry into the cell cycle following growth stimulation, but it markedly reduced the growth o

39 ay of several factors, including the loss of growth stimulation by 5-LO products, the induction of PP

40 not bovine serum albumin (0.15%) suppressed growth stimulation by ATRA.

41 small interfering RNA against wnt-1 blunted growth stimulation by core gene, whereas transfection of

42 Growth stimulation by IFN-alpha/beta is independent of o

43 metastatic melanoma cells did not respond to growth stimulation by IGF-1 because of a constitutive ac

44 lationship between eicosanoid metabolism and growth stimulation by IL-6 and HGF, two important biliar

45 bdate, and tungstate competitively inhibited growth stimulation by molybdate.

46 term quiescence and down-regulated following growth stimulation by serum.

47 ultured mouse PA-SMCs, TERT was expressed on growth stimulation by serum.

48 the AKAP12alpha CArG boxes are shielded from growth stimulation by the absence of a binding site for

49 Moreover, growth stimulation depended on overall GSK3 activity, co

50 esis, apoptosis, and either growth arrest or growth stimulation depending on the cellular context.

51 having distinct biological effects, such as growth stimulation, differentiation, invasiveness, and m

52 ide with distinct biological effects such as growth stimulation, differentiation, invasiveness, and m

53 hat van der Sleen's conclusions of a lack of growth stimulation do not hold.

54 iferation, and LIF is able to counteract the growth stimulation elicited by EGF.

55 The CO(2) growth stimulation exceeds that of most other woody spec

56 re dependent on exogenous, receptor-mediated growth stimulation for cell cycle entry and progression,

57 mes are emerging as significant mediators of growth stimulation for epithelial cells.

58 tion is involved with growth factor-mediated growth stimulation for lung cancer cell lines.

59 logical effects on cultured cells, including growth stimulation, growth inhibition, and induction of

60 an cells to this family of ligands including growth stimulation, growth inhibition, apoptosis and ind

61 t the physiological basis of light-activated growth stimulation has not yet been determined.

62 xide enrichment, although causing short-term growth stimulation in a range of European tree species,

63 tibody significantly inhibited the T47D cell growth stimulation in coculture with primary fibroblasts

64 ntration of dihydrotestosterone required for growth stimulation in CWR-R1 and LNCaP-C4-2 cells was fo

65 ormones is therefore necessary for autocrine growth stimulation in lung tumors and possibly in the ea

66 receptor as a mediator of MMP-2 release and growth stimulation in response to type I collagen.

67 Growth stimulation in rice correlated with a decrease in

68 VEGF treatment led to extensive growth stimulation in six of seven pancreatic cancer cel

69 ative response of neoblasts to amputation or growth stimulation in Smed-CHD4(RNAi) animals was dimini

70 alter enterobactin transport, but eliminated growth stimulation in the absence of a siderophore.

71 o confer a defect in norepinephrine-mediated growth stimulation in the presence of transferrin.

72 kinase pathway, thereby leading to excessive growth stimulation in this malignancy.

73 Fibroblast-dependent carcinoma cell growth stimulation in three-dimensional coculture was ab

74 After growth stimulation in vivo by 70% partial hepatectomy (P

75 , and E2F3a genes are similarly regulated by growth stimulation, involving a combination of E2F-depen

76 rogen-responsive element (ARE) indicate that growth stimulation is accompanied by androgen receptor (

77 001 which is of particular interest for bone growth stimulation is achievable by this assembly.

78 reases in intrinsic water use efficiency, no growth stimulation is observed.

79 tin 1b for M. hydrocarbonoclasticus-specific growth stimulation may be a poor strategy for oil-spill

80 ution of Ca2+ appears to be a consequence of growth stimulation, not a critical step in the early pha

81 cells, synchronized in late G1 and following growth stimulation, now progressed into S-phase identica

82 s, suggesting a potential autocrine-mediated growth stimulation of a subset of cancers by VEGF.

83 tivities similar to those of IL-2, including growth stimulation of activated T cells, induction of cy

84 D1 may be a target gene for leptin mediated growth stimulation of breast cancer cells and molecular

85 increased expression of ER target genes and growth stimulation of breast cancer cells.

86 se results demonstrate that retinoid-induced growth stimulation of Calu-1 cells is associated with en

87 the fibroblast surface, leading to paracrine growth stimulation of carcinoma cells by Sdc1 ectodomain

88 We now demonstrate that TGFbeta1 growth stimulation of colon carcinoma cells is Ras-depen

89 plasia (OSSN) may result from the continuous growth stimulation of corneal epithelial progenitor cell

90 ient and begins to be turned off 3-6 h after growth stimulation of cultured cells.

91 Thus, additional growth stimulation of diseased uPA-expressing liver indu

92 VciB, but not VciA, was required for growth stimulation of E. coli and Shigella flexneri stra

93 or growth inhibition of epithelial cells and growth stimulation of fibroblasts.

94 he predominant IGF involved in the autocrine growth stimulation of human lung and breast epithelial t

95 d SAPK/JNK signaling pathway, which leads to growth stimulation of MCF-7 breast tumor cells.

96 tivity blocks the induction of S phase after growth stimulation of normal mouse embryo fibroblasts, i

97 tent ERK1/2 activation and serum-independent growth stimulation of only uPAR-rich cells.

98 Growth stimulation of prostate cancer cells with 5alpha-

99 ponse to high glucose concentration required growth stimulation of RMCs by serum and activation of pr

100 Growth stimulation of some colon cancer cells as well as

101 ther, our results suggest that auxin-induced growth stimulation of tobacco leaf strips results primar

102 ion to functioning as a positive effector of growth, stimulation of the MEK/MAPK pathway can result i

103 Meanwhile, loss of scrA led to growth stimulation on fructooligosaccharides, due in lar

104 lls, but is stabilized immediately following growth stimulation or inhibition of protein synthesis.

105 rences in functional v-src expression, or by growth stimulation or suppression via paracrine mechanis

106 oduced by Fusarium moniliforme, resulting in growth stimulation or toxicity.

107 ytes were examined, there was no evidence of growth stimulation over a wide range of PADMA 28 concent

108 fically blocked Sdc1-mediated carcinoma cell growth stimulation, pointing toward MMPs as critical enz

109 In the absence of UCP2, endothelial growth stimulation provoked mitochondrial network fragme

110 Activation of HsOrc1 transcription following growth stimulation requires G1 cyclin-dependent kinase a

111 e to elevated TACE activity, complements the growth stimulation resulting from increased release of T

112 mopexin, Hepa cells become refractory to the growth stimulation seen with 0.1-0.75 microM heme-hemope

113 ee-ring data contain evidence for historical growth stimulation that was concealed due to failing reg

114 of Na/K-ATPase and converts ouabain-induced growth stimulation to growth inhibition in LLC-PK1 cells

115 fects in hematopoietic cells that range from growth stimulation to induction and prevention from apop

116 bolic dose-response growth curve, indicating growth stimulation until the chimera begins to compete w

117 carcinogenesis, including auto- or paracrine growth stimulation, upregulation of angiogenesis, and st

118 eased activation of TGF-beta1) and augmented growth stimulation (via decreased degradation of the IGF

119 enterobactin at approximately 10 microM, but growth stimulation was abolished when an omega (Omega) c

120 We observed that growth stimulation was associated with stimulation of ER

121 In culture, the growth stimulation was evident when senescent cells comp

122 e CoMFA models for receptor binding and cell growth stimulation were optimized through the use of var

123 of 5-HETE to breast cancer cells resulted in growth stimulation, whereas selective biochemical inhibi

124 Moreover, growth stimulation with exogenous, purified apo-Ybt prov

125 Nicotiana sylvestris resulted in substantial growth stimulation, with a 3-fold increase in height in