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1 ine and the adrenergic neuron-blocking agent guanethidine.
2 reatment with the adrenergic neuron blocker, guanethidine (20 mg/kg), also abolished the PVN-mediated
3 sions of the mesentery and by application of guanethidine (3 microM) to the stimulation and recording
6 oxamine (6-20 microM) and in the presence of guanethidine (5 microM), EFS of perivascular sensorimoto
7 rivative, displays the same pH dependence as guanethidine, a heptamethyleniminoethyl- guanidine deriv
10 HDA] treatment) and long-term sympathectomy (guanethidine and 8 days after 6-OHDA) on the distributio
17 he release of sympathetic neurotransmitters (guanethidine or reserpine) inhibited capsaicin-evoked iC
21 of sympathetic fiber function with systemic guanethidine significantly decreased the pain-related be
22 ale Sprague-Dawley rats were pretreated with guanethidine sulfate (40 mg/kg daily for 5 weeks) or veh
23 cted 50% with noradrenaline and treated with guanethidine (to inhibit sympathetic neurotransmission),
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