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1 e or guanylate, inosine 5'-monophosphate and guanosine-5'-monophosphate).
2 is independent of global elevation of cyclic guanosine 5'-monophosphate.
3 Evidence is presented for complexation of guanosine 5'-monophosphate 2-methylimidazolide (2-MeImpG
4 dazolide derivatives of nucleotides, such as guanosine 5'-monophosphate 2-methylimidazolide (2-MeImpG
5 olycytidylic acid-directed polymerization of guanosine 5'-monophosphate 2-methylimidazolide (2-MeImpG
6 the concentration of the activated monomer, guanosine 5'-monophosphate 2-methylimidazolide, 2-MeImpG
8 s potentiation by 5'-ribonucleotides such as guanosine-5'-monophosphate and inosine 5'-monophosphate,
9 where TMA is [(CH(3))(4)N](+) and 5'-GMP is guanosine 5'-monophosphate, and K(2)(5'-GMP), containing
10 '-monophosphate, thymidine-5'-monophosphate, guanosine-5'-monophosphate, and cytidine-5'-monophosphat
11 ells to H(2)S increases intracellular cyclic guanosine 5'-monophosphate (cGMP) in a NO-dependent mann
13 to synthesize a N-acetyl-(2-aminofluorenyl)-guanosine 5'-monophosphate (dGpAAF) adduct, which was su
14 nd is responsible for the phosphorylation of guanosine 5'-monophosphate (GMP) and dGMP to guanosine 5
16 arkbound in a fashion similar to that of the guanosine 5'-monophosphate (GMP) and inosine 5'-monophos
17 osphate (ADP) and guanosine conformations of guanosine 5'-monophosphate (GMP) bound to wild type and
18 the hypothesis that nitric oxide (NO) cyclic guanosine 5'-monophosphate (GMP) signaling is deficient
19 Specifically, a known competitive inhibitor, guanosine 5'-monophosphate (GMP), and a synthetic, trans
20 hway is redundant and contains two routes to guanosine-5'-monophosphate (GMP) formation: conversion f
21 inone (H2Q), N-acetyl-tyrosine (N-Ac-Tyr) or guanosine-5'-monophosphate (GMP) was investigated at var
23 onophosphate 2-methylimidazolide (2-MeImpG), guanosine 5'-monophosphate imidazolide (ImpG), and adeno
24 ives are guanosine 5'-monophosphate (5'GMP), guanosine 5'-monophosphate imidazolide (ImpG), and guano
25 In mice, exposure to CS reduced sGC, cyclic guanosine 5'-monophosphate levels, and protein kinase G
29 lla pneumoniae BlrP1, a light-activated c-di-guanosine 5'-monophosphate phosphodiesterase which consi
32 f either MLL exon 7 or exon 8 with the GMPS (GUANOSINE 5'-MONOPHOSPHATE SYNTHETASE) gene from chromos
33 its platelet activation by generating cyclic guanosine 5'-monophosphate, which is metabolized by phos
34 itro in reactions supplemented with biotinyl-guanosine 5'-monophosphate, which led to the specific ad
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