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1 n-like Ca(2+)-binding proteins termed GCAPs (guanylate cyclase-activating proteins).
3 three Ca(2+)-binding proteins, recoverin and guanylate cyclase activating proteins 1 (GCAP1) and GCAP
6 sites (EF-hands) of the GUCA1A gene encoding guanylate cyclase-activating protein 1 (GCAP1) cause slo
10 outer segments of GC double knock-out mice, guanylate cyclase-activating proteins 1 and 2, and cycli
14 stem is composed of two parts: Ca(2+) sensor guanylate cyclase activating protein (GCAP) and ROS-GC.
15 and transgenic retinal rods with and without guanylate cyclase activating protein (GCAP) to disrupt C
16 addition, two of the proteins, recoverin and guanylate cyclase activating protein (GCAP), appear to b
17 ons were made to rods expressing mutant Y99C guanylate cyclase activating protein (GCAP)-1, to unders
19 ack loop was disrupted in mouse rods lacking guanylate cyclase activating proteins GCAP1 and GCAP2 (G
20 cyclase activator 1A (GUCA1A), the gene for guanylate cyclase activating protein (GCAP1), in a famil
23 hrough the calcium binding proteins: through guanylate cyclase activating proteins (GCAPs) in the cas
25 racteristic of light-adapted photoreceptors, guanylate cyclase-activating protein (GCAPs) activate Re
31 oteins found in retinal photoreceptor cells (guanylate cyclase activating protein, protein kinase A,
32 is coexpressed with its specific modulator, guanylate cyclase activating protein type 1 (GCAP1), in
33 1 coexisted with its other Ca(2+) modulator, guanylate cyclase activating protein type 1 (GCAP1).
34 ies with the Ca(2+) binding proteins, GCAPs (guanylate cyclase activating proteins), which stimulate
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