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1 at mediates intestine-specific expression of guanylyl cyclase C.
3 co2 human colon carcinoma cells that express guanylyl cyclase C (GC-C) and SW480 human colon carcinom
11 at-stable enterotoxin binds to and activates guanylyl cyclase C (GC-C), regulating fluid and electrol
14 mutation in the catalytic domain (D853A) of guanylyl cyclase-C (GC-C), the heat-stable enterotoxin (
16 rgeting the intestinal cancer mucosa antigen guanylyl cyclase C (GCC) and its effect on inflammatory
19 to 2ClAdo, demonstrating that inhibition of guanylyl cyclase C (GCC) was mediated by a noncompetitiv
20 tems, and the bacterial enterotoxin receptor guanylyl cyclase C (GCC), the principle source of cGMP i
23 ic tumors all express a unique surface-bound guanylyl cyclase C (GCC), which binds the diarrheagenic
27 nuation of the colonic cell surface receptor guanylyl cyclase C (GUCY2C) that occurs due to loss of i
29 enovirus (Ad5) combination regimen targeting guanylyl cyclase C (GUCY2C), a receptor expressed by int
30 endogenous ligands for the tumor suppressor guanylyl cyclase C (GUCY2C), disrupting a network of hom
32 vealed that intestine-specific expression of guanylyl cyclase C is directed by the proximal promoter.
36 atio for death associated with expression of guanylyl cyclase C mRNA in regional lymph nodes was 15.0
38 prior to defining the baseline expression of guanylyl cyclase C mRNA, a marker for colorectal cancer,
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