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1 ished against capsaicin-induced cough in the guinea pig.
2 yocytes and in isolated perfused hearts from guinea pig.
3 he induction of NAbs and T cell responses in guinea pigs.
4 ently HSV-1-infected mice and HSV-2-infected guinea pigs.
5 2)--to subdivide IC GABAergic cells in adult guinea pigs.
6 d Ag preparations were analyzed in immunized guinea pigs.
7 displayed marked attenuation in the lungs of guinea pigs.
8  of rodent models such as mice, rabbits, and guinea pigs.
9 d latent infection of dorsal root ganglia in guinea pigs.
10 ariant (SUDV-GA) that is uniformly lethal to guinea pigs.
11 ditory neurons of the inferior colliculus in guinea pigs.
12 viously seen in MARV-infected mice or inbred guinea pigs.
13  and attenuated phenotypes, respectively, in guinea pigs.
14 mapped to the outer domain of gp120 for some guinea pigs.
15 lication and transmission of bovine FLUDV in guinea pigs.
16 ary auditory cortex of urethane-anesthetized guinea pigs.
17 iber system model of tuberculosis, mice, and guinea pigs.
18 spiratory tract for intratracheally infected guinea pigs.
19 on cardiovascular parameters in anesthetized guinea pigs.
20 sGP does not affect pathogenicity of EBOV in guinea pigs.
21 enuated for replication in immunocompromised guinea pigs.
22  lungs but failed to persist in the lungs of guinea pigs.
23  the most potent NAb responses in vaccinated guinea pigs.
24 the viruses are lethal in mice, hamsters and guinea pigs.
25 d resulted in reduced viral loads in newborn guinea pigs.
26  infection of the lower respiratory tract of guinea pigs.
27 atients with COPD, healthy donors, mice, and guinea pigs.
28  grafts were performed on the calvaria of 36 guinea pigs.
29 induced sudden sensorineural hearing loss in guinea pigs.
30 rvallo termed Car(91) that was attenuated in guinea pigs.
31 n normal hearing, tinnitus, and non-tinnitus guinea pigs.
32 ous than the parental EA viruses in mice and guinea pigs.
33 e faithfully maintained upon transmission to guinea pigs.
34                                           In guinea pigs, a DNA prime-protein boost regimen with thes
35 BOV using a guinea pig model and recombinant guinea pig-adapted EBOV containing mutations at the edit
36 livers and spleens of guinea pigs in vivo, a guinea pig-adapted SUDV variant (SUDV-GA) uniformly leth
37 y-six outbred guinea pigs were infected with guinea pig-adapted ZEBOV and examined sequentially over
38 ell lines and pancreatic tissues of mice and guinea pigs, administration of ethanol reduced expressio
39 enic, we determined if Car(91) could protect guinea pigs against Guanarito virus (GTOV), a distantly
40              This cocktail protected 100% of guinea pigs against JUNV and GTOV lethal disease.
41  (EBOVDeltaVP30) protects immunized mice and guinea pigs against lethal challenge with rodent-adapted
42     Here we show TRPV4-induced activation of guinea pig airway-specific primary nodose ganglion cells
43 sA strain) increased sPLA2-IIA expression in guinea pig airways and was cleared more efficiently, com
44 paring pharmacokinetics from carbon-14 dosed guinea pigs analyzed by both CRDS and accelerator mass s
45 r, ML277, can enhance IKs amplitude in adult guinea pig and canine ventricular myocytes.
46 hibiting capsaicin-induced depolarization of guinea pig and human isolated vagus nerve.
47 o urban air pollution leads to activation of guinea pig and human sensory nerves, which are responsib
48 e cleaned particles evoked depolarization of guinea pig and human vagus, and this was inhibited by a
49 characterized a new set of affinity-purified guinea pig and rabbit antibodies against RNA-binding pro
50 reatic cell lines as well as in tissues from guinea pigs and CFTR knockout mice after administration
51 this, we established a transmission model in guinea pigs and determined the mechanisms behind virus s
52 gimen with trimeric envelope gp140(CA018) in guinea pigs and elicited high anti-gp41 IgG titers.
53 dy responses, we sequentially infected mice, guinea pigs and ferrets with divergent H1N1 or H3N2 subt
54 ers (sgp140) from a CD4-independent HIV-1 in guinea pigs and found that the sgp140 elicited ADCC-medi
55 ng immunity against Lassa virus challenge in guinea pigs and marmosets and virus-specific cell-mediat
56                                           In guinea pigs and mice, InlP increased the placental bacte
57 rod increased motility in inflamed colons of guinea pigs and mice, whereas administration of GR113808
58 o nasal epithelium for intranasally infected guinea pigs and more widespread in the respiratory tract
59  and displays good in vivo safety margins in guinea pigs and rats.
60 estigate viral pathogenesis (mouse, hamster, guinea-pig and ferret) are naturally resistant to MERS-C
61 ted using membrane preparations from animal (guinea pig) and human origin.
62 However, rodent models, including the mouse, guinea pig, and hamster, also exist for Ebola, Marburg,
63 issive small-animal species, namely, ferret, guinea pig, and hamster.
64 c solutions caused depolarization of murine, guinea pig, and human vagus and firing of Adelta-fibers
65 ku strain) in C57BL/6J mice, C3HeB/FeJ mice, guinea pigs, and a mouse hollow-fiber model of infection
66 against all pathogenic ebolaviruses in mice, guinea pigs, and ferrets.
67                Immunization studies in mice, Guinea pigs, and in Rhesus macaques, revealed that LNPs
68 tains motility in healthy colons of mice and guinea pigs, and reduces inflammation in colons of mice
69 higher than class 1 to mice, rats, hamsters, guinea pigs, and/or rabbits in the examinees was 14.1% (
70                                  Here, using guinea pigs as animal models, we report that adaptation
71 t reduction of brain Abeta42 was observed in guinea pig at 30 mg/kg dosed orally.
72 plicated in the nasal turbinate and lungs of guinea pigs at high titers and was also able to transmit
73 itive to either ITDs or ILDs in anesthetized guinea pig, before, during, and following recovery from
74            sigma1 affinities determined with guinea pig brain and human RPMI 8226 tumor cell lines di
75 l entorhinal cortex of the in vitro isolated guinea pig brain during focal interictal and ictal disch
76 e and female mice and in the isolated female guinea pig brain preparation during perfusion with 4-AP.
77                        Studies using ex vivo guinea pig brain slices have revealed that activity-gene
78 oth tangential brain slices and the isolated guinea pig brain with the potassium channel blocker 4-am
79  in the hippocampus of the in vitro isolated guinea pig brain.
80 rhinal cortex of the in vitro isolated whole guinea pig brain.
81 y untreated exhaust ward air, and another 90 guinea pigs breathed only air from the same six-bed tube
82      Over an exposure period of 7 months, 90 guinea pigs breathed only untreated exhaust ward air, an
83 tif, to achieve 24 h duration of action in a guinea pig bronchoprotection model.
84 uated for dissemination in immunocompromised guinea pigs but elicited ELISA and neutralizing response
85 brane fusion activity and viral virulence in guinea pigs, but it did not significantly affect cell en
86  region induce QTc prolongation in immunized guinea-pigs by targeting the HERG channel independently
87 ial Ca(2+) flux and buffering in Pi-depleted guinea pig cardiac mitochondria.
88  workload transitions in isolated murine and guinea pig cardiac myocytes and mitochondria.
89  data suggest that CO induces arrhythmias in guinea pig cardiac myocytes via the ONOO(-)-mediated inh
90 GC synaptic connections in the retina of the guinea pig (Cavia porcellus) by recording inhibitory cur
91 mic contractile indices in detergent-skinned guinea pig (Cavia porcellus) cardiac muscle fibers in th
92 ibed circumstantial evidence suggesting that guinea pigs (Cavia porcellus) lack CD59, at least on ery
93                              Importantly, in guinea pigs CD-160130 produced neither alteration of the
94 blishment of a latent viral reservoir in the guinea pig challenge model of HSV-2 infection.
95 activity in vitro and on the cough reflex in guinea pig challenge models.
96                                  In pregnant guinea pigs, challenge with doses of r129 virus of >/=5
97 f eliciting cross-neutralizing antibodies in guinea pigs, chimpanzees, and healthy human volunteers.
98    AAV.BDNF or AAV.Ntf3 was delivered to the guinea pig cochlea one week following deafening and ears
99 asured the travelling wave in regions of the guinea pig cochlea that respond to low frequencies (<2 k
100 -linear processing of sound performed by the guinea pig cochlea varies substantially between the coch
101  vivo to study the low-frequency part of the guinea pig cochlea, and found that sound stimulation cau
102 distributed over the apical two turns of the guinea pig cochlea.
103 utively expressing GFP (H9 Cre-LoxP) in deaf guinea pig cochleae that were pre-conditioned to reduce
104         In the present study, normal-hearing guinea pig cochleae underwent cochlear implantation thro
105 e bioassay, in vivo electrophysiology, and a guinea pig conscious cough model to investigate a role f
106  expression in sensory neurons supplying the guinea pig corneal epithelium, which have well-defined m
107                   Our study established that guinea pigs could be used as an animal model for studyin
108  a novel vaccine strategy against congenital guinea pig cytomegalovirus (GPCMV) infection was develop
109  an intervention strategy against congenital guinea pig cytomegalovirus (GPCMV) infection.
110  and vaccine efficacy were evaluated using a guinea pig cytomegalovirus (GPCMV) model.
111                                              Guinea pig cytomegalovirus (GPCMV) provides a valuable m
112                                              Guinea pigs developed high titers of broadly cross-react
113 spite exhibiting no clinical signs, infected guinea pigs developed seroconversion and the viral antig
114 id (OA-NO2) on TRP channels were examined in guinea-pig dissociated dorsal root ganglia (DRG) neurons
115 ologs, including mouse, ferret, hamster, and guinea pig DPP4, do not.
116 ant of permissivity for ferret, hamster, and guinea pig DPP4.
117 phin gene inserts, AAV.BDNF and AAV.Ntf3, on guinea pig ears deafened systemically (kanamycin and fur
118 in the lack of L-glutaminase activity in the guinea pig enzyme.
119  inhibited native IKr and cross-reacted with guinea pig ERG channel.
120 %) of PV-ir neurons in macaques, humans, and guinea pigs express m1AChRs.
121 uronic content of the pericellular brush for guinea pig fibroblast cells.
122                   These antibodies protected guinea pigs from lethal disease when given post-virus ch
123 coprotein rabbit antiserum protected Hartley guinea pigs from lethal intraperitoneal infection with J
124 cross-reactive mAb that completely protected guinea pigs from the lethal challenge with heterologous
125 g (GP83), determined via a newly established guinea pig gamma interferon enzyme-linked immunosorbent
126 ith respect to their abilities to ascend the guinea pig genital tract.
127                                              Guinea pigs given 20 mg of antisera or F(ab')2 at or sta
128 with statistical significance, groups of six guinea pigs (gps) were challenged intranasally (i.n.) or
129 zotocin (STZ)-induced type 1 diabetic (T1DM) guinea pigs (GPs).
130 ssible reassortant genotypes demonstrated in guinea pigs (Gt2, Gt3, Gt7, Gt10 and Gt13) were also the
131 rosomes (HLMs) or liver microsomes from male guinea pig, hamster, monkey, mouse, and rabbit or female
132 crease intercellular cleft width in isolated guinea pig heart experiments.
133  excised from aortic-banded or sham-operated guinea-pig hearts.
134 ) flux of recombinant P2X7 receptors of dog, guinea pig, human, monkey, mouse, rat, and zebrafish.
135 lectrically induced contractions in isolated guinea pig ileum via CB1 receptors (pEC50, 6.0 +/- 0.4).
136 r 5-HT3 receptor-induced contractions in the guinea pig ileum, which are down-regulated after chronic
137 ecovery from receptor desensitization in the guinea pig ileum.
138                                           In guinea pigs, immunization with the PGT145 Fab-BG505 SOSI
139  herpes simplex virus 1 (HSV-1)-seropositive guinea pigs immunized and subsequently challenged intrav
140                                              Guinea pigs immunized with a single dose of this vector
141 10(7) PFU of Delta145 or parental virus into guinea pigs immunosuppressed with cyclophosphamide.
142 ial in both murine cochlear explants and the guinea pig in vivo.
143                         The hazard ratio for guinea pigs in the control chamber converting their skin
144 saging SUDV within the livers and spleens of guinea pigs in vivo, a guinea pig-adapted SUDV variant (
145 n viral replication in cell culture and in a guinea pig infection model.
146 more, recordings of neural activity from the guinea pig inferior colliculus have shown that individua
147 lving mammalian intracochlear anatomy, fixed guinea pig inner ears were imaged as whole temporal bone
148                                           In guinea pigs inoculated with a mixture of viruses, parent
149                              In anesthetized guinea pigs intratracheal administration of DEPs activat
150                                          The guinea pig is the only small animal model for congenital
151 hea sections, show that mucosal infection of guinea pigs is an efficient model for Ebola transmission
152 inoid type 2 (CB2) receptors, as well as the guinea pig isolated ileum, using the full agonist CP5594
153  informed by experimental data recorded from guinea pig isolated single ventricular myocytes.
154           A TMA-like state also developed in guinea pigs IV administered PEO+.
155                         We are exploring the guinea pig L-asparaginase (gpASNase1) as a potential rep
156            We recently identified the low KM guinea pig L-asparaginase (gpASNase1).
157                             We conclude that guinea pigs lack an intact gene encoding CD59; to our kn
158                                     Isolated guinea pig left ventricular cardiomyocytes were incubate
159   We applied this to experimentally infected guinea pig lung sections and were able to distinguish bo
160 mes in the persistence of M. tuberculosis in guinea pig lungs and underscore the applicability of thi
161 en localized to the inner ear of the rat and guinea pig mammalian models.
162 igh-frequency, subthreshold resonance in the guinea pig medial superior olive (MSO) was recently link
163                                      Using a guinea pig model and human lung cells, we show that oxid
164 editing in the pathogenicity of EBOV using a guinea pig model and recombinant guinea pig-adapted EBOV
165 tection against lethal EBOV challenge in the guinea pig model at this timepoint.
166 obust growth and contact transmission in the guinea pig model atypical of avian strains and indicativ
167 mouse model and in abortion using a pregnant guinea pig model following oral challenge.
168                                          The guinea pig model for Shigella flexneri invasion of the c
169  of conditioned neutrophils in a neutropenic guinea pig model increased bacterial clearance of Shigel
170                        In a well-established guinea pig model of aerosol infection with Mycobacterium
171                      We document that in the guinea pig model of AHF, mutation F427I in GP2 is also h
172 man chimeric antibodies and evaluated in the guinea pig model of AHF.
173 enetic platform for future studies using the guinea pig model of congenital CMV infection.
174 cts, the DeltamdbA mutant is attenuated in a guinea pig model of diphtheritic toxemia.
175                 When tested in the stringent guinea pig model of EBOV disease, EBOTAb has been shown
176               After promising results in the guinea pig model of EBOV infection, EBOTAb was tested in
177                   Blood samples taken from a guinea pig model of IA were used for WB and serum PCR.
178                                         In a guinea pig model of infection, the severity of disease a
179                             Finally, using a guinea pig model of infection, we assessed the in vivo r
180      Analysis of virulence of the rJUNV in a guinea pig model of lethal infection that closely reprod
181                                      Using a guinea pig model of lipopolysaccharide (LPS)-induced neu
182  fiber system (HFS) model, murine model, and guinea pig model of tuberculosis as well as clinical stu
183 ded inhibitor of protein kinase R, using the guinea pig model of vertical CMV transmission.
184      These findings suggest that the outbred guinea pig model recapitulates ZEBOV infection of primat
185       In the current study, we established a guinea pig model to test the actuator for its ability to
186                              An anesthetized guinea pig model was used to elicit a SOF+AMIO-dependent
187 Based on the promising data generated in the guinea pig model, a formulation containing truncated gD-
188                  We report here that, in the guinea pig model, a human isolate of novel H7N9 influenz
189            Theophylline was antitussive in a guinea pig model, inhibited activation of single C-fiber
190 /34 (H1N1) (PR8), is nontransmissible in the guinea pig model, making it a useful background in which
191                                       In the guinea pig model, rSPN04-P was transmitted to fewer cont
192               Two scenarios were tested in a guinea pig model, using influenza A/Panama/2007/99 (H3N2
193 cs significantly better than does a standard guinea pig model.
194 s sGP and assessed its virulence in the EBOV guinea pig model.
195 ellent protection provided by gC2/gD2 in the guinea pig model.
196 s and impacts growth and transmission in the guinea pig model.
197 ion and transmissibility of the virus in the guinea pig model.
198 s not required for abortion induction in the guinea pig model.
199 responses in a cigarette smoke (CS) exposure guinea pig model.
200 he transition to heart failure, we studied a guinea-pig model of angiotensin II infusion (400 ng kg(-
201 c function in the transition to failure in a guinea-pig model of disease.
202                     Uniformly lethal outbred guinea pig models of MARV-Angola (MARV-Ang) and MARV-Rav
203 d virulence and transmissibility in mice and guinea pig models.
204 e-defective mutants in both cell culture and guinea pig models.
205   Strain-associated virulence was studied in guinea pig, monocyte-derived macrophage, and lysozyme re
206 age-clamp recordings were made from isolated guinea pig myocytes as well as from human embryonic kidn
207 mine the basis for CO-induced arrhythmias in guinea pig myocytes in which action potentials (APs) mor
208 ssion of these TS channels in cultured adult guinea pig myocytes, combined with a quantitative ventri
209 Ps and induced early afterdepolarizations in guinea pig myocytes.
210 cts of CO on Kv11.1 tail currents and APs in guinea pig myocytes.
211                                    In single guinea-pig myocytes, endogenous IKr was replaced by mode
212 ents done on the altricial rat and precocial guinea pig neonate demonstrated that the effect of vasop
213 ation in auditory neurons of chinchillas and guinea pigs of both sexes, and show how heterogeneous tu
214    Motile responses were induced in isolated guinea pig OHCs by stimulation with an 8 V/cm external a
215                                           In guinea pigs, our best mutant, DS-SOSIP.4mut, elicited a
216 ation, we tracked reassortment in coinfected guinea pigs over time and given matched or discordant do
217 ra (Depo) treatment and from oophorectomized guinea pigs (OVX-GPs) treated with vehicle, estradiol (E
218        Based on studies of human, mouse, and guinea pig pancreata, alcohol disrupts expression and lo
219           ABSTRACT: A computational model of guinea-pig pancreatic duct epithelium was developed to d
220  fitting the model to experimental data from guinea-pig pancreatic ducts.
221 ted against EqCXCL16 or by pretreatment with guinea pig polyclonal antibody against EqCXCL16 protein
222                                      We used guinea pig primary cells, tissue bioassay, in vivo elect
223 we generated transgenic (Tg) mice expressing guinea pig prion protein (GPPrP).
224 ere, we show that transgenic mice expressing guinea pig prion protein are fully susceptible to vCJD a
225                 Our results suggest that the guinea pig prion protein is a better, more rapid substra
226  experimental challenge of immunocompromised guinea pigs, r129 induced significant weight loss, longe
227 the cervix of four placental mammals, mouse, guinea pig, rabbit and armadillo, and one marsupial, opo
228 d displaced RGC (dRGC) somata in mouse, rat, guinea pig, rabbit, and monkey retina.
229 KA phosphorylation sites conserved in human, guinea pig, rabbit, rat, and mouse alpha1C subunits.
230  different inducers; untreated male monkeys, guinea pigs, rabbits, and hamsters; and female dogs.
231                                     Deafened guinea pigs received a cochlear implant, and their cochl
232 us was induced by low-dose streptozotocin in guinea pigs rendered glucose intolerant by first feeding
233 d secondary transmissions of BSE and vCJD in guinea pigs result in long incubation periods of approxi
234        M. tuberculosis infection of diabetic guinea pigs resulted in severe and rapidly progressive t
235 from OFF delta retinal ganglion cells in the guinea pig retina and monitored synaptic currents that w
236 ion cells in a dense patch of salamander and guinea pig retinas while displaying a bar moving diffusi
237 ant strain was also impaired for survival in guinea pig sera and sheep blood.
238                   The assay was performed in guinea pig serum.
239                         In summary, isogenic guinea pigs show promise as a model to investigate the r
240              The 52-kDa Ro antigen-immunized guinea pigs showed QTc prolongation on ECG after develop
241 armacokinetic and pharmacodynamic studies in guinea pigs showed that a single 5-min application of a
242 th the upper and lower respiratory tracts of guinea pigs, similarly to virus infection in its native
243                        In a second series of guinea pig studies, the administration of EBOTAb dosing
244 ed from the antisera and used for an in vivo guinea pig study.
245 sed a greater number of coughs in CS-exposed guinea pigs than in control animals; similar increased r
246    We performed a genomic screen in pregnant guinea pigs that led to the identification of 201 lister
247 t animals than rSPN04-A and failed to infect guinea pigs that received a low-dose inoculum.
248 t of virus infections of cell culture and of guinea pigs that the SSP plays an essential role in medi
249                                           In guinea pigs, the Fab-complexed trimer induced 100-fold l
250                                  In mice and guinea pigs, the rLmIII/a30 and rLmI/h30 vaccines were g
251                            Upon infection of guinea pigs, the RNase mutant viruses stimulate strong I
252 tinin (HA) and the binding of virus to fixed guinea pig tissues were also examined.
253 ocked cough and single C-fiber firing in the guinea pig to the transient receptor potential (TRP) V1
254 coinfection was achieved by exposing a naive guinea pig to two cagemates, one infected with wt and th
255 e ability of cells in the auditory system of guinea pigs to compare interaural level differences (ILD
256 delling with experiments in heart cells from guinea pigs to determine how cellular electrical activit
257 p140 protein in a prime-boost combination in guinea pigs to enhance envelope (Env)-specific humoral a
258 virus (ZEBOV) primarily used inbred mice and guinea pigs to model disease.
259 olerance and type 2 diabetes were induced in guinea pigs to model the emerging comorbidity of Mycobac
260 FLUDV was able to transmit from the infected guinea pigs to sentinel animals by means of contact and
261 ectivity profile, long duration of action on guinea pig trachea, and longer than salmeterol duration
262 cacy was evaluated by immunizing GPCMV-naive guinea pigs twice with either 10(5) or 10(6) PFU of Delt
263 ated the effect of theophylline on human and guinea pig vagal sensory nerve activity in vitro and on
264 ents in vivo and depolarization of human and guinea pig vagus in vitro, and inhibited calcium influx
265 aber-Rudy computationally based model of the guinea pig ventricular action potential, we simulated ef
266 ential, we simulated effects of GS-458967 on guinea pig ventricular APD.
267 e effects of GS-458967 on endogenous INaL in guinea pig ventricular myocytes demonstrate a robust con
268 ich it is used to develop models of isolated guinea pig ventricular myocytes that simulate the electr
269 tricular myocytes and acutely isolated adult guinea pig ventricular myocytes.
270 regulation of action potential morphology in guinea pig ventricular myocytes.
271                                       In all guinea pigs, virus titers peaked in nasal secretions at
272 FLUDV replicated in the respiratory tract of guinea pigs was similar to observations described previo
273  of LASV that is uniformly lethal in Hartley guinea pigs, we demonstrate that favipiravir, a broad-sp
274                     In this initial study in guinea pigs, we investigated mSync using combined activa
275             In the present study in deafened guinea pigs, we investigated the long-term effects of te
276                                              Guinea pigs were either (i) mock immunized; (ii) immuniz
277                In the second scenario, naive guinea pigs were exposed to a cagemate that had been coi
278                                              Guinea pigs were immunized with the resulting HIV VLPs t
279                           Thirty-six outbred guinea pigs were infected with guinea pig-adapted ZEBOV
280                            Strain 2 isogenic guinea pigs were inoculated with H1N1pdm09 influenza vir
281 eatures of disease in ZEBOV-infected outbred guinea pigs were largely consistent with disease in huma
282 o GTOV infection, 88% of the Car(91)-exposed guinea pigs were protected.
283                                              Guinea pigs were used as an animal model to understand t
284                            Twelve adult male guinea pigs were used in this experiment.
285                                              Guinea-pigs were immunized with a 31-amino-acid peptide
286 and SEBOV-induced disease in outbred Hartley guinea pigs where we observed protection from effects of
287 of allergen-specific blocking IgG in outbred guinea pigs which had been immunized with recombinant bi
288  We assessed hearing recovery in LPS-applied guinea pigs, which were either left untreated or were sy
289      Here, we describe a study with isogenic guinea pigs, which would uniquely combine the advantages
290  bacteremia in mice and abortion in pregnant guinea pigs, while complementation of capsule expression
291                    By deactivating one IC in guinea pig with cooling or microdialysis of procaine, an
292                                 Infection of guinea pigs with an earlier passage of Carvallo, termed
293                                 Infection of guinea pigs with Pichinde virus (PICV), a prototype aren
294  establishing pregnancy, and challenging the guinea pigs with salivary gland-adapted GPCMV.
295                                We vaccinated guinea pigs with single trimers as well as mixtures of t
296                              Immunization of guinea-pigs with a peptide (E-pore peptide) correspondin
297 e tested the hypothesis that immunization of guinea-pigs with a peptide corresponding to the E-pore r
298                                          The guinea pig, with a placenta structure similar to that in
299 ection in its native host, demonstrates that guinea pigs would be a suitable model host to study the
300 s and asymptomatic HSV-2 genital shedding in guinea pigs, yet the protection was not complete.

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