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1 m, other infectious bacteria, host and human gut flora.
2 calf IAP (cIAP) rapidly restores the normal gut flora.
3 out drug targets that may also affect human gut flora.
4 at their expansion is regulated by commensal gut flora.
5 acid degradation pathway is present in human gut flora.
6 extracts but at the same affected the normal gut flora.
7 ly caused by food sensitivity or a change in gut flora.
8 er than indiscriminately eliminating natural gut flora.
9 ed in isolators and colonized with a defined gut flora.
10 appeared to be absent from the normal human gut flora.
11 be endogenously produced by normal microbial-gut flora.
12 ncephalopathy is lactulose and alteration of gut flora.
13 ssex chickens possessing an acquired natural gut flora.
14 Hundreds of bacterial species make up human gut flora.
15 rRNAs distinct from those of common skin and gut flora.
16 ed that in competition with the normal mouse gut flora, an EHEC O157:H7 strain with a nonpolar deleti
18 to inflammatory signals induced by both the gut flora and arthritis, Breg cells increase in number a
20 Studies have shown that probiotics alter gut flora and lead to elaboration of flora metabolites t
23 human health, and alterations of the normal gut flora are associated with a variety of distinct dise
24 nged in the absence of TLR4, suggesting that gut flora associated with the graft may augment alloimmu
25 tedly enhances butyrate production by normal gut flora can influence the outcome of an E. coli O157 i
29 is well recognized, components of the human gut flora critical for colonization resistance are not k
30 s Ib protein, MR1, which is required for the gut flora-dependent development of mucosa-associated inv
33 with control BM monocytes, BM monocytes from gut flora-depleted mice had decreased migratory capacity
36 understand why, among the vast diversity of gut flora, enterococci are so well adapted to the modern
38 le MHC-related protein I (MR1), B cells, and gut flora for development and/or expansion, and they pre
40 ce pointing to the role of altered commensal gut flora in such common conditions as irritable bowel s
42 oligonucleotide (CpG-B), can substitute for gut flora in the induction of neonatal immunoresponsiven
43 toimmunity, the particular role of commensal gut flora in the initiation of colitis, and the role of
44 rmed a critical role for dietary choline and gut flora in TMAO production, augmented macrophage chole
45 n is influenced by the presence of commensal gut flora, in particular increased colonization with seg
47 rgrowth (SIBO) or other abnormalities in the gut flora is believed to contribute to the development o
50 The induction of such cells in response to gut flora may be a mechanism protecting normal individua
53 nsion and selection, known to require normal gut flora, may be driven through indirect effects of mic
58 c compounds, and fermentability by the human gut flora, SCFAs production, nitric oxide and cytokine e
60 ur findings demonstrate in neonatal mice how gut flora synergizes with poly(I:C) to elicit protective
61 intervention to affect this process - on the gut flora, the 'leaky' mucosal membrane and endotoxin co
63 lites derived from dietary tryptophan by the gut flora to activate AHR signaling in astrocytes and su
64 pithelial tight junctions, allowing resident gut flora to promote chronic increases in antimicrobial
66 t change in the composition and diversity of gut flora when the pH of drinking water was altered.
68 s exert highly selective effects on resident gut flora, which, in turn, lead to very specific alterat
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