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1 m, other infectious bacteria, host and human gut flora.
2  calf IAP (cIAP) rapidly restores the normal gut flora.
3  out drug targets that may also affect human gut flora.
4 at their expansion is regulated by commensal gut flora.
5 acid degradation pathway is present in human gut flora.
6 extracts but at the same affected the normal gut flora.
7 ly caused by food sensitivity or a change in gut flora.
8 er than indiscriminately eliminating natural gut flora.
9 ed in isolators and colonized with a defined gut flora.
10  appeared to be absent from the normal human gut flora.
11 be endogenously produced by normal microbial-gut flora.
12 ncephalopathy is lactulose and alteration of gut flora.
13 ssex chickens possessing an acquired natural gut flora.
14  Hundreds of bacterial species make up human gut flora.
15 rRNAs distinct from those of common skin and gut flora.
16 ed that in competition with the normal mouse gut flora, an EHEC O157:H7 strain with a nonpolar deleti
17                                              Gut flora analyses have allowed gut microbiota signature
18  to inflammatory signals induced by both the gut flora and arthritis, Breg cells increase in number a
19                                    DCs sense gut flora and damaged epithelium via expression of C-typ
20     Studies have shown that probiotics alter gut flora and lead to elaboration of flora metabolites t
21 enteritis and be associated with an abnormal gut flora and with food intolerance.
22                                              Gut flora appears to act as an important determinant in
23  human health, and alterations of the normal gut flora are associated with a variety of distinct dise
24 nged in the absence of TLR4, suggesting that gut flora associated with the graft may augment alloimmu
25 tedly enhances butyrate production by normal gut flora can influence the outcome of an E. coli O157 i
26 ein-coupled receptors influenced by diet and gut flora composition.
27 and insulin resistance via modulation of the gut flora composition.
28           Although it is known that resident gut flora contribute to immune system function and homeo
29  is well recognized, components of the human gut flora critical for colonization resistance are not k
30 s Ib protein, MR1, which is required for the gut flora-dependent development of mucosa-associated inv
31          Discovery of a relationship between gut-flora-dependent metabolism of dietary phosphatidylch
32                                              Gut flora-depleted mice expressed lower levels of F4/80
33 with control BM monocytes, BM monocytes from gut flora-depleted mice had decreased migratory capacity
34 s of Lactobacillus, important members of the gut flora, differentially activate DC.
35                 In this study, we found that gut flora DNA (gfDNA) plays a major role in intestinal h
36  understand why, among the vast diversity of gut flora, enterococci are so well adapted to the modern
37 obacteria are important members of the human gut flora, especially in infants.
38 le MHC-related protein I (MR1), B cells, and gut flora for development and/or expansion, and they pre
39 nce supporting the role of altered commensal gut flora in human disease.
40 ce pointing to the role of altered commensal gut flora in such common conditions as irritable bowel s
41 ry input from innate immune responses to the gut flora in the establishment of oral tolerance.
42  oligonucleotide (CpG-B), can substitute for gut flora in the induction of neonatal immunoresponsiven
43 toimmunity, the particular role of commensal gut flora in the initiation of colitis, and the role of
44 rmed a critical role for dietary choline and gut flora in TMAO production, augmented macrophage chole
45 n is influenced by the presence of commensal gut flora, in particular increased colonization with seg
46          The loss of this bacterium from the gut flora is associated with an increased risk of hypero
47 rgrowth (SIBO) or other abnormalities in the gut flora is believed to contribute to the development o
48 an aberrant T cell response directed against gut flora is sufficient to trigger IBD.
49             However, C3H mice with a limited gut flora (LF) were efficiently colonized at high levels
50   The induction of such cells in response to gut flora may be a mechanism protecting normal individua
51                               Alterations in gut flora may be important in the pathophysiology of the
52                       Evidence suggests that gut flora may play an important role in the pathophysiol
53 nsion and selection, known to require normal gut flora, may be driven through indirect effects of mic
54 whereby T1D induction can be circumvented by gut flora-mediated Th17 differentiation.
55                                              Gut flora modify primary bile acids to produce secondary
56                        We then characterized gut flora of patients during onset of intestinal inflamm
57 ased cohorts including American Gut, Flemish Gut Flora Project and the extended TwinsUK cohort.
58 c compounds, and fermentability by the human gut flora, SCFAs production, nitric oxide and cytokine e
59  humanity has experienced a depletion of the gut flora since diverging from Pan.
60 ur findings demonstrate in neonatal mice how gut flora synergizes with poly(I:C) to elicit protective
61 intervention to affect this process - on the gut flora, the 'leaky' mucosal membrane and endotoxin co
62  required additional signals provided by the gut flora through TLR5 and MyD88 signaling.
63 lites derived from dietary tryptophan by the gut flora to activate AHR signaling in astrocytes and su
64 pithelial tight junctions, allowing resident gut flora to promote chronic increases in antimicrobial
65                     Among differences in the gut flora were increased abundances of Lactobacillus and
66 t change in the composition and diversity of gut flora when the pH of drinking water was altered.
67 AFLD because of its modulating effect on the gut flora, which can influence the gut-liver axis.
68 s exert highly selective effects on resident gut flora, which, in turn, lead to very specific alterat

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