ライフサイエンスコーパス: gut lumen

1 companied by increased formate levels in the gut lumen.

2 substantially elevated lactate levels in the gut lumen.

3 reduced the growth of S. Typhimurium in the gut lumen.

4 estinal tract and aid in absorption from the gut lumen.

5 resulting in high fucose availability in the gut lumen.

6 h M-cell-specific transcellular pores to the gut lumen.

7 ion of ovalbumin (OVA) to the blood from the gut lumen.

8 a and other pathogens that also exist in the gut lumen.

9 nsitizing antigens when they reappear in the gut lumen.

10 go with nuclei of neighboring cells, and the gut lumen.

11 etic microspheres were administered into the gut lumen.

12 rchenteron cavity actually gives rise to the gut lumen.

13 from the proteinase-rich environment of the gut lumen.

14 nteron lining does not become the definitive gut lumen.

15 migration into the intestinal mucosa and the gut lumen.

16 "blebbing" of the midgut epithelium into the gut lumen.

17 lization dependent on their proximity to the gut lumen.

18 that the worms harbour several taxa in their gut lumen absent from their diets and substrates.

19 be transported to internal tissues from the gut lumen across gut cells that lack SID-1.

20 revealed Helicobacter-like organisms in the gut lumen and attached to intestinal epithelial cells.

21 decreased clearance of C. difficile from the gut lumen and diminished inflammation.

22 by mouse Paneth cells, is secreted into the gut lumen and has bactericidal activity against intestin

23 ial cells interact with both microbes in the gut lumen and host immune cells.

24 unlike these it is secreted into the larval gut lumen and is an active beta1,3-glucanase.

25 sociated with increased endotoxin within the gut lumen and its associated portal circulation.

26 ransform into the procyclic stage within the gut lumen and later migrate to the ectoperitrophic space

27 s Listeria monocytogenes colonization of the gut lumen and prevents systemic dissemination.

28 ormed calcium phosphate nanoparticles in the gut lumen and show how this helps to shape intestinal im

29 (despite the immunological inertness of the gut lumen), and (iii) that there are very high benefits

30 VCBPs are secreted into the gut lumen, and direct binding to bacterial surfaces can

31 demonstrates that symbiotic bacteria in the gut lumen appear to function as partners both for symbio

32 Upon feeding, chitinase is secreted into the gut lumen as an inactive pro-enzyme that is later activa

33 on by 1 week, beginning patch extrusion into gut lumen at 3 weeks, and total separation of patch with

34 uced in the intestine was transported to the gut lumen at a rate that exceeded transport to plasma by

35 al stimuli induces active DC sampling of the gut lumen at sites distant from organized lymphoid tissu

36 that sorafenib-glucuronide excreted into the gut lumen can be cleaved by microbial enzymes to sorafen

37 lts in defective IgA transportation into the gut lumen, causing a dramatic increase in serum polymeri

38 unoglobulin A (IgA) across the mucosa to the gut lumen depends on the epithelial transport protein, p

39 The gut lumen-dwelling tapeworm Hymenolepis diminuta, unlike

40 e peritrophic matrix (PM) that separates the gut lumen from the epithelium.

41 ntestinal epithelial cells (IECs) lining the gut lumen functions as the site of nutrient absorption a

42 LPS administered enterally was found in the gut lumen in close proximity to the mucosa but was not d

43 omplex communities of bacteria reside in the gut lumen in direct contact with the ingested materials

44 he intestine in vivo Uric acid levels in the gut lumen increased in response to exogenous DNA, and th

45 ansporter that would transport heme from the gut lumen into intestinal epithelial cells.

46 n the initial propagation of prions from the gut lumen into Peyer's patches.

47 translocation of microbial products from the gut lumen into the bloodstream.

48 ses move through the insect vector, from the gut lumen into the hemolymph or other tissues and finall

49 Acid-base status in the hemolymph and gut lumen of insects is generally well regulated but var

50 irochete, Borrelia burgdorferi, inhabits the gut lumen of the tick vector.

51 in their hosts (i.e. as promastigotes in the gut lumen of their sandfly vectors and as amastigotes in

52 ally in acidic pH compartments either in the gut lumen or in vacuoles of the intestinal lining cells.

53 e changes in the bacterial population in the gut lumen or lamina propria that cause inflammation at t

54 pteran and dipteran insects that have acidic gut lumen, recombinant AtVSP2 significantly delayed deve

55 emic IgG antibodies neutralize toxins in the gut lumen remains unresolved, although it has been sugge

56 This is accompanied by an increase in the gut lumen size and a compromise in the intestine's abili

57 dding of LPS-containing enterocytes into the gut lumen, that has not been previously described.

58 yphimurium by promoting its outgrowth in the gut lumen through unknown mechanisms.

59 f how prions are initially conveyed from the gut lumen to establish infection on FDC.

60 processes between epithelial cells into the gut lumen to sample pathogens.

61 translocation of the scrapie agent from the gut lumen to the GALT from which neuroinvasion subsequen

62 h TSE agents are initially conveyed from the gut lumen to the GALT is not known.

63 e of toxic and infectious molecules from the gut lumen while allowing selective paracellular absorpti

64 re mainly produced by micro-organisms in the gut lumen, with presumably important alterations in adva

65 motes absorption of monosaccharides from the gut lumen, with resulting induction of de novo hepatic l