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1 e of cleanliness of the mouse colony and the gut microflora.
2 is believed to be under the influence of the gut microflora.
3 ue between the immune system of the host and gut microflora.
4 th a broad spectrum of antibiotics to reduce gut microflora.
5 rvival, a function dependent on licensing by gut microflora.
6 nal rabbits and thereby acquired a different gut microflora.
7 enged by higher eukaryotes from the diet and gut microflora.
8 ic disease in individuals with a compromised gut microflora.
9 lasma isoflavone concentrations and modified gut microflora activities [beta-glucoside hydrolysis and
10 o be completely dependent on the presence of gut microflora and could be established by colonization
11 ed mice showed no significant decline in the gut microflora and developed EAE similar to untreated mi
12                                    Commensal gut microflora and dietary fiber protect against colonic
13 llus species are normal members of the human gut microflora and most are regarded as safe when admini
14  suggests that changes in the composition of gut microflora and/or deranged epithelial barrier functi
15  apoptosis of intestinal epithelium, changed gut microflora, and elevated ATP.
16  Aberrant immune response and changes in the gut microflora are the main causes of inflammatory bowel
17 t likely due to effects mediated through the gut microflora, bowel transit, and enhancement of gastro
18 We recently described that alteration of the gut microflora can affect a population of Foxp3(+)T(reg)
19 y of environmental samples such as the human gut microflora, combined with the sustained exponential
20 ronment and with the presence of a different gut microflora compared to mice maintained in SPF facili
21 ns and participate in the development of the gut microflora for infants.
22 microbiome was observed, suggesting that the gut microflora has a direct impact on the drug metabolis
23                              Introduction of gut microflora in GF mice rapidly initiated maturation o
24 an significantly affect the growth of select gut microflora in humans, which suggests the potential p
25 on is currently available on the role of the gut microflora in modulating isoflavone bioavailability
26  Additional investigation revealed commensal gut microflora in the mesenteric lymph nodes and elevate
27  to be modulated according to composition of gut microflora, including ingested probiotics.
28                                    The human gut microflora is important in regulating host inflammat
29 of drinking water affects the composition of gut microflora, leading to an altered autoimmune respons
30 d with amino acid, energy, purine, lipid and gut microflora metabolisms.
31      Concentrations of isoflavones and their gut microflora metabolites in the plasma, urine, and fec
32                        Relationships between gut microflora, morphological and metabolic traits were
33 ved molecular techniques for analysis of the gut microflora, new manufacturing biotechnologies, and i
34 d higher abundance of dominant genera in the gut microflora of group JD.
35 ollectively, these findings suggest that the gut microflora of the honey bee harbours bacterial membe
36 community-based mechanism for protecting the gut microflora, preserving its functional robustness dur
37 hetaiotaomicron, a predominant member of the gut microflora, revealing a mechanism whereby intestinal
38 ditions, such as circulating glucose levels, gut microflora, time of year, and even diurnal rhythm, w

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