ライフサイエンスコーパス: gut-associated lymphoid tissue

1 dendritic cells, B cells and T cells in the gut-associated lymphoid tissue.

2 a typhimurium to deliver DNA directly to the gut-associated lymphoid tissue.

3 regions of colonization and necrosis of the gut-associated lymphoid tissue.

4 c immune responses, in a location other than gut-associated lymphoid tissue.

5 fever in mice have been shown to target the gut-associated lymphoid tissue.

6 f cytokine production in discrete regions of gut-associated lymphoid tissue.

7 us distribution of components of the diffuse gut-associated lymphoid tissue.

8 and that this diversification occurs in the gut-associated lymphoid tissue.

9 us distribution of components of the diffuse gut-associated lymphoid tissue.

10 oid tissues from normal mice, chiefly in the gut-associated lymphoid tissue.

11 elium of high endothelial venules in gut and gut-associated lymphoid tissue.

12 he numbers of CD4(+) and IL-17(+) T cells in gut-associated lymphoid tissue.

13 ells (LSEC) supported migration into gut and gut-associated lymphoid tissue.

14 vation or HIV-specific responses in PBMCs or gut-associated lymphoid tissue.

15 correlated with active viral replication in gut-associated lymphoid tissue.

16 -1) results in the dissemination of virus to gut-associated lymphoid tissue.

17 of Peyer's patches, a major component of the gut-associated lymphoid tissue.

18 ally populates epithelia and lung as well as gut-associated lymphoid tissues.

19 tinal epithelium and delivered to underlying gut-associated lymphoid tissues.

20 eted delivery of recombinant antigens to the gut-associated lymphoid tissues.

21 otavirus (RV)-specific Ab-secreting cells in gut-associated lymphoid tissues.

22 nd MHC class II molecule presentation in the gut-associated lymphoid tissues.

23 l immunity through antigen-processing by the gut-associated lymphoid tissues.

24 epithelial cells (IECs) and the interior of gut-associated lymphoid tissues.

25 etention to promote B cell egress from these gut-associated lymphoid tissues.

26 e of IgA production by B cells, derived from gut-associated lymphoid tissues.

27 prominent role in iT(reg) cell generation in gut-associated lymphoid tissues.

28 increase in the frequency of T(H)17 cells in gut-associated lymphoid tissues.

29 be induced in the absence of the spleen and gut-associated lymphoid tissues.

30 o the RA-dependent homing receptor switch in gut-associated lymphoid tissues.

31 lation is present at a high frequency in the gut-associated lymphoid tissue and appears to mediate a

32 somatic hypermutation occur in young rabbit gut-associated lymphoid tissue and are thought to divers

33 nic Salmonella infection is localized to the gut-associated lymphoid tissue and does not extend effic

34 ches aimed at immune reconstitution of human gut-associated lymphoid tissue and for the development,

35 ave compared the responses of T cells in the gut-associated lymphoid tissue and in the periphery to i

36 ets of CD4(+) T cells from peripheral blood, gut-associated lymphoid tissue and lymph node tissue spe

37 nfection with depletion of CD4(+) T cells in gut-associated lymphoid tissue and other pathologic sequ

38 ntly required on potential reservoirs in the gut-associated lymphoid tissue and the central nervous s

39 me paracellular pathway, in concert with the gut-associated lymphoid tissue and the neuroendocrine ne

40 direct impact on the host defense system of gut-associated lymphoid tissue and therefore has potenti

41 vector-modified CD4 T cells was measured in gut-associated lymphoid tissue and was correlated with e

42 nodes (MLN) are structural components of the gut-associated lymphoid tissues and contribute to the in

43 monstrate a B cell subset that is induced in gut-associated lymphoid tissues and is characterized by

44 dCAM-1 RNA is restricted to mucosal tissues, gut-associated lymphoid tissues and spleen.

45 eost SALT structurally resembles that of the gut-associated lymphoid tissue, and it possesses a diver

46 e, a carrier molecule to deliver antigens to gut-associated lymphoid tissues, and possibly an adjuvan

47 ) infections are acquired mucosally, and the gut-associated lymphoid tissues are important sites for

48 in organized lymphoid nodules of the colonic gut-associated lymphoid tissue at 14 days; double-labeli

49 rphine inhibits Ag-specific IgA responses in gut-associated lymphoid tissue at least partially throug

50 lthough both decreased IgA production due to gut-associated lymphoid tissue atrophy and impaired muco

51 soluble oral Ags do not require an organized gut-associated lymphoid tissue but are most likely induc

52 The best studied is gut-associated lymphoid tissue, but distinct epithelium-

53 indicate that T helper cells were induced in gut-associated lymphoid tissues by i.g. immunization wit

54 opies in bulk and resting CD4 T cells and in gut-associated lymphoid tissue, CD4 T-cell-associated HI

55 (CD4(+) CD69(+)) T lymphocytes was found in gut-associated lymphoid tissues collected from animals w

56 mechanism via which functionally specialized gut-associated lymphoid tissue DCs can extend the repert

57 bone marrow, blood, spleen, lymph nodes, and gut-associated lymphoid tissues depleted by day 7, inclu

58 All the mutants colonized the gut-associated lymphoid tissue efficiently, but capaciti

59 T560, a mouse B lymphoma that originated in gut-associated lymphoid tissue, expresses receptors that

60 In mammals that use gut-associated lymphoid tissues for expansion and somati

61 he physiologic conduit for antigens to reach gut associated-lymphoid tissues, for penetration of the

62 eneration of virus-specific Ab production by gut-associated lymphoid tissue (GALT) after i.m. immuniz

63 ) T cells to T reg cells occurs primarily in gut-associated lymphoid tissue (GALT) after oral exposur

64 ing CD4(+) T cells preferentially traffic to gut-associated lymphoid tissue (GALT) and have a key rol

65 duced by B-cell receptor engagement in human gut-associated lymphoid tissue (GALT) and involvement of

66 High levels of viral replication occur in gut-associated lymphoid tissue (GALT) and other lymphoid

67 EL) are a critical effector component of the gut-associated lymphoid tissue (GALT) and play an import

68 s, the transitional 2 (T2) B cells, homes to gut-associated lymphoid tissue (GALT) and that most T2 B

69 y of parenteral nutrition-induced changes in gut-associated lymphoid tissue (GALT) and upper respirat

70 sly or intragastrically had small intestinal gut-associated lymphoid tissue (GALT) atrophy along with

71 We, therefore, examined lymph node (LN) and gut-associated lymphoid tissue (GALT) biopsies from full

72 We hypothesized that T cells primed in the gut-associated lymphoid tissue (GALT) by a specific anti

73 d genes is important for colonization of the gut-associated lymphoid tissue (GALT) by S. typhimurium.

74 The predictive value of acute gut-associated lymphoid tissue (GALT) CD4+ T cell deplet

75 this study, we show that p38alpha regulates gut-associated lymphoid tissue (GALT) formation in a non

76 and memory T cells from peripheral blood and gut-associated lymphoid tissue (GALT) from eight patient

77 Gut-associated lymphoid tissue (GALT) harbors the majori

78 Effects of therapy interruption on gut-associated lymphoid tissue (GALT) have not been well

79 n, and gastrointestinal tract (including the gut-associated lymphoid tissue (GALT) in rhesus monkeys

80 The role of lymphocytes in the gut-associated lymphoid tissue (GALT) in the production

81 Gut-associated lymphoid tissue (GALT) is a major site of

82 Gut-associated lymphoid tissue (GALT) is a sensor region

83 Gut-associated lymphoid tissue (GALT) is a significant b

84 Gut-associated lymphoid tissue (GALT) is an early target

85 Gut-associated lymphoid tissue (GALT) is an early target

86 Although the gut-associated lymphoid tissue (GALT) is an important ea

87 The gut-associated lymphoid tissue (GALT) is constantly expo

88 and profound depletion of CD4(+) T cells in gut-associated lymphoid tissue (GALT) of animals infecte

89 acytomas that develop "spontaneously" in the gut-associated lymphoid tissue (GALT) of interleukin-6 t

90 d B cells are a predominant component of the gut-associated lymphoid tissue (GALT) they may play a ro

91 Diet influences the ability of gut-associated lymphoid tissue (GALT) to maintain mucosa

92 The cytokine profile in the gut-associated lymphoid tissue (GALT) was measured.

93 Somatic diversification occurs in gut-associated lymphoid tissue (GALT), and by about 1-2

94 IELs), previously stimulated to cycle in the gut-associated lymphoid tissue (GALT), proliferated in t

95 V-1-induced depletion of CD4+ T cells in the gut-associated lymphoid tissue (GALT), we first determin

96 ification occurs in the appendix, which is a gut-associated lymphoid tissue (GALT).

97 ntegrin alpha4beta7 and can be identified in gut-associated lymphoid tissue (GALT).

98 ng infection of lymphocytes that home to the gut-associated lymphoid tissue (GALT).

99 on between commensal intestinal bacteria and gut-associated lymphoid tissue (GALT).

100 ad to rapid depletion of CD4(+) T cells from gut-associated lymphoid tissue (GALT).

101 e accumulation of some prion diseases in the gut-associated lymphoid tissues (GALT) is important for

102 upon follicular dendritic cells (FDC) within gut-associated lymphoid tissues (GALT) is important for

103 ction on the number of lymphoid cells in the gut-associated lymphoid tissues (GALT) were determined.

104 Here we show that the absence of gut-associated lymphoid tissues (GALT), such as Peyer's

105 chain (gammac) (Rag-gammac(-/-)), which lack gut-associated lymphoid tissues (GALT), such as Peyer's

106 nal bacteria are required for development of gut-associated lymphoid tissues (GALT), which mediate a

107 mune responses were assessed in the neonatal gut-associated lymphoid tissues (GALT).

108 s in part because of residual replication in gut-associated lymphoid tissues (GALT).

109 g cells, which are generated from B cells in gut-associated lymphoid tissues (GALT).

110 an enhanced homing capacity to draining and gut-associated lymphoid tissues (GALTs) after systemic a

111 re associated with immune cell activation in gut-associated lymphoid tissues (GALTs) and significant

112 Gut-associated lymphoid tissues (GALTs) interact with in

113 nmental localization in intestine and in the gut-associated lymphoid tissues (GALTs).

114 T cell priming by dendritic cells (DC) from gut-associated lymphoid tissues gives rise to effector c

115 cent studies have shown that human and mouse gut-associated lymphoid tissues harbor a unique NK cell

116 hypothesized that, because teleost SALT and gut-associated lymphoid tissue have probably been subjec

117 is likely to affect local gut integrity and gut-associated lymphoid tissue homeostasis, which in tur

118 olute numbers of Tregs declined in blood and gut-associated lymphoid tissue in patients with chronic

119 We examined the cytokine microenvironment in gut-associated lymphoid tissue in response to orally adm

120 l exposure to SEB, suggesting a role for the gut-associated lymphoid tissue in the gastrointestinal m

121 prevalent, although virus genomes persist in gut-associated lymphoid tissue in the majority of indivi

122 mic autoimmune responses to the formation of gut-associated lymphoid tissue in the neonatal period of

123 that elevated interferon-gamma, produced by gut-associated lymphoid tissue in the small intestine, i

124 In the local microenvironment of gut-associated lymphoid tissues, inflammatory cytokines

125 delivery of plant-synthesized insulin to the gut-associated lymphoid tissues, insulin was linked to t

126 Since gut-associated lymphoid tissue is likely to play an impo

127 The migration of lymphocytes to gut-associated lymphoid tissue is mediated by integrin a

128 Gut-associated lymphoid tissue is the dominant site for

129 Gut-associated lymphoid tissue is the major reservoir of

130 Lymphocyte trafficking into gut-associated lymphoid tissues is mediated by interacti

131 its role on CD4 T cells, specifically in the gut-associated lymphoid tissue, is less well understood.

132 ronic HIV infection results in impairment of gut-associated lymphoid tissue leading to systemic immun

133 on was monitored by enumerating listeriae in gut-associated lymphoid tissues, livers, and spleens.

134 iated antiviral immunity in association with gut-associated lymphoid tissue mass atrophy.

135 velopment of antigen-specific T cells in the gut-associated lymphoid tissues, mice were immunized i.g

136 Under physiological conditions the gut-associated lymphoid tissues not only prevent the ind

137 lt rabbits comparable with those produced in gut associated lymphoid tissues of young rabbits.

138 which can influence immune responses in the gut-associated lymphoid tissue of a neonate.

139 e, spontaneous germinal centers developed in gut-associated lymphoid tissue of LMP2A mice, indicating

140 between 4 and 9 d, but were not detected in gut-associated lymphoid tissue or lymph nodes.

141 ral replication, similar to lymphoid tissue, gut-associated lymphoid tissue or semen.

142 tor from activated cells derived from bovine gut-associated lymphoid tissues (Peyer's patch and mesen

143 ut) inflamed sites such as the lung into the gut-associated lymphoid tissues, Peyer's patches, and th

144 nhanced restoration of CD4(+) T cells within gut-associated lymphoid tissue, respectively.

145 CTL were also detected in rectal washes and gut-associated lymphoid tissues, respectively.

146 ymphocytes to peripheral lymph nodes and the gut-associated lymphoid tissues, respectively.

147 ontributing to the HIV-induced impairment of gut-associated lymphoid tissue structure and function, e

148 esenteric lymph nodes, splenic follicles and gut-associated lymphoid tissue that demonstrate high lev

149 tent progenitor (MPP) cell population in the gut-associated lymphoid tissue that promotes T(H)2 cytok

150 ages in systemic lymphoid tissues, including gut-associated lymphoid tissue, the major site of HIV-1

151 es and is responsible for T-cell homing into gut-associated lymphoid tissues through its binding to m

152 Therefore, modulating gut-associated lymphoid tissue to boost Tr1 cells may be

153 int) mice, dendritic cell recruitment in the gut-associated lymphoid tissues was normalized, and tumo

154 eption can be associated with enlargement of gut-associated lymphoid tissue, we studied the capacity

155 3 showed that most of the donor cells in the gut-associated lymphoid tissues were rapidly replaced, b

156 in tissue resident T cells of intestines and gut associated lymphoid tissues, which demonstrated pred

157 nd on the gastrointestinal immune system and gut-associated lymphoid tissue, which may be active site