ライフサイエンスコーパス: hCG

1 hCG also blocked NF-kappaB activation induced by ceramid

2 hCG also prevents Neisseria gonorrhoeae from developing

3 hCG also retained dendritic cells in a tolerogenic state

4 hCG assembly appears to be limited by the need to disrup

5 hCG cleavage by gonococcal IgA1 proteases in vivo may in

6 hCG cleaved a few additional proteins, although this occ

7 hCG impact on Treg suppressive capacity was studied in v

8 hCG is heterodimeric and functionally defined by its bet

9 hCG is stabilized by a strand of its beta-subunit that h

10 hCG signaling activates silent mating type information r

11 hCG treatment also decreased cell invasion, which was mo

12 hCG treatment prevented the tumor necrosis factor-depend

13 hCG was cleaved in the beta subunit by the type 1 but no

14 hCG was measured using a solid-phase competitive chemilu

15 hCG-generated Tregs were fully functional and could conf

16 hCG-NPM/RAR alpha leukemic cells resembled monoblasts.

17 hCG-PR x PU.1+/- mice developed a typical APL syndrome a

18 capillary glucose [FCG] level, 2-hour CG [2-hCG] level, and glycated hemoglobin A1c [HbA1c] level) a

19 9; FCG level > 7 mmol/L, 4.5% of patients, 2-hCG level > 11 mmol/L, 6.8%; and HbA1c level > 6.5%, 9.3

20 [95% CI, .7-8.7] at follow-up; aOR for the 2-hCG level: 6.6 [95% CI, 4.0-11.1] vs 1.6 [95% CI, .8-2.9

21 In females having spontaneous abortions, hCG provoked not only an augmentation of Treg numbers, b

22 Administered hCG reduces risk of carcinogen-induced breast cancer in

23 Samples of r-alpha hCG obtained from a CHO cell line were also analyzed and

24 tryptic glycopeptides and glycans of r-alpha hCG to enable the assignment of glycan structures to ind

25 inant human chorionic gonadotrophin (r-alpha hCG), a protein that is glycosylated at two sites and is

26 Analysis of r-alpha hCG, using capillary electrophoresis (CE) with a separat

27 adotrophin (hCG), and antibodies, anti-alpha-hCG and anti-beta-hCG, using a sandwich assay by surface

28 logical assay that showed that the hCG-alpha-hCG-beta heterodimer facilitated human CG receptor-media

29 urine of pregnant women consistently altered hCG and PPARgamma expression in primary placental cells.

30 ta strongly suggest that PLA(2)G4A amplifies hCG induction of PTGS2 and colocalizes with the induced

31 ntibodies, we use a short oligopeptide as an hCG receptor to bind hCG.

32 ighest possible screening sensitivity for an hCG-based pregnancy test therefore is estimated to be 90

33 ors labeled with gold-deglycosylated hCG, an hCG antagonist, also exhibit restricted lateral diffusio

34 ip between Thr(250)-Gln(268), exoloop 2, and hCG.

35 They also recommended measuring AFP and hCG shortly before retroperitoneal lymph node dissection

36 these TM with those in hCG-PML/RAR alpha and hCG-PLZF/RAR alpha TM.

37 Both TNF-alpha antibody and hCG reduced TNF-alpha levels in sera by approximately 75

38 of interaction between anti-hCG antibody and hCG that exhibited 6.5 times higher sensitivity for the

39 Ten patients received chemotherapy, and hCG concentrations returned to normal in eight (80%) of

40 Cleavage of the alarmins by HC and hCG suggests a function in regulating excessive inflamma

41 e tested for hCG, hCG free beta subunit, and hCG beta-core fragment.

42 %) patients continued under surveillance and hCG values spontaneously returned to normal without chem

43 n FSH and AR-induced maturation in vitro and hCG-induced maturation in vivo.

44 ivered the scale of interaction between anti-hCG antibody and hCG that exhibited 6.5 times higher sen

45 For anti-hCG (human chorionic gonadotropin) mAb 8F11, studied at

46 nic gonadotropin (hCG)/mouse monoclonal anti-hCG and goat IgG (anti-intact hCG)/ mouse monoclonal ant

47 is was inhibited by treatment with antisense hCG/LH receptor phosphorothioate oligodeoxynucleotide.

48 Similar to LH receptors labeled with Au-hCG, LH receptors labeled with gold-deglycosylated hCG,

49 g in 6,976 B-human chorionic gonadotropin (B-hCG)-positive specimens, as determined by the Auszyme Mo

50 Because hCG also blocked TNF receptor-associated factor-2 and NF

51 The 5HRE beta-hCG reporter system described here enables serial, nonin

52 3-pentafluoropropyl)acetamide (EF5) and beta-hCG and pimonidazole, two extrinsic markers for tumor hy

53 t syntheses of homogeneous beta-hLH and beta-hCG bearing model glycans at all native glycosylation si

54 and antibodies, anti-alpha-hCG and anti-beta-hCG, using a sandwich assay by surface plasmon field-enh

55 tometry indicate colocalization between beta-hCG and 2-(2-nitro-1H-imidazol-1-yl)-N-(2,2,3,3,3-pentaf

56 nes and primary breast tumors expressed beta-hCG, c-Met, beta 1-->4-N-acetylgalactosaminyl-transferas

57 ers: beta-human chorionic gonadotropin (beta-hCG), oncogene receptor (c-Met), beta 1-->4-N-acetylgala

58 ost complex human glycoprotein hormone (beta-hCG) as well as its closely related homologue (beta-hLH)

59 The combination of beta-hCG and MAGE-A3 mRNA markers correlated significantly wi

60 en route to the N-terminal fragment of beta-hCG and the sequential installation of four O-linked gly

61 Secretion of beta-hCG from xenografts that contain these stable constructs

62 ted with this plasmid construct secrete beta-hCG in response to hypoxia or hypoxia-inducible factor 1

63 Structurally, beta-hCG shares a high degree of sequence similarity with bet

64 beta-human chorionic gonadotropin test (beta-hCG < 6 IU/L) 17 days after egg retrieval.

65 e element (HRE) ligated upstream of the beta-hCG gene.

66 We adapted this approach to use urinary beta-hCG as a secreted reporter protein for tumor hypoxia.

67 Similarly, urinary beta-hCG levels decline after treatment with flavopiridol, an

68 We observed no association between hCG and breast cancer risk, overall [Quartile 4 vs. 1, O

69 and that a conformational difference between hCG-beta and mCG-beta was recapitulated in the context o

70 hort oligopeptide as an hCG receptor to bind hCG.

71 d microbeads, simultaneous detection of both hCG and PSA on each gel pad array is achieved with singl

72 d pathology in transgenic mice and that both hCG and antibody to TNF-alpha prevent the development of

73 on of PU.1 for leukemia progression, we bred hCG-PR mice with PU.1+/- mice and assessed their phenoty

74 RTH 43-kDa protein in Leydig cells caused by hCG treatment was prevented by the androgen receptor ant

75 mans, interrupting the inflammatory cycle by hCG opens new perspectives for therapeutic intervention

76 d by TNF and ceramide was also suppressed by hCG.

77 The captured hCG can disrupt a thin layer (~6 mum) of LC covered on t

78 important tumor markers: AFP, ferritin, CEA, hCG-beta, CA 15-3, CA 125, and CA 19-9.

79 quine chorionic gonadotropin (eCG)/human CG (hCG) treatment and mating, ovulation and fertilization r

80 ique structures of human choriogonadotropin (hCG) and related glycoprotein hormones make them well su

81 , teFSH analogs of human choriogonadotropin (hCG) are assembled by a pathway in which the subunits do

82 ty residues of the human choriogonadotropin (hCG) beta-subunit that are wrapped around alpha-subunit

83 he manner in which human choriogonadotropin (hCG) contacts lutropin receptors (LHR) have been stymied

84 and interact with human choriogonadotropin (hCG).

85 cer in animal models, and higher circulating hCG concentrations were associated with significantly lo

86 ired for the formation of assembly-competent hCG subunits and that the invariant Gly residue is requi

87 of the slope ratio of the low concentration hCG protein assay in linear regression analysis was GO-p

88 Like its human counterpart hCG-beta with which it shares 81% identity, macaque (m)C

89 ys, followed by 3 IV doses EPO over 3 days), hCG+Saline using the same schedule, Saline+EPO using the

90 H receptors labeled with gold-deglycosylated hCG, an hCG antagonist, also exhibit restricted lateral

91 of other antigens, and by failure to detect hCG with in-house assays.

92 -free and label-free mechanism for detecting hCG.

93 and wide variations in results for different hCG assays.

94 o four treatment groups: hCG+EPO (3 IM doses hCG over 5 days, followed by 3 IV doses EPO over 3 days)

95 rnal-embryonic crosstalk, in which embryonic hCG via endometrial PROK1 may play a pivotal role in enh

96 The incorporation of an engineered hCG-SNAP fusion reporter protein (human chorionic gonado

97 Interestingly, in coculture experiments, hCG-treated endometrial cells induce an increase in T ce

98 aB-inducing kinase reporter gene expression, hCG must act at a step that causes phosphorylation of Ik

99 ed surface by adsorbing monobiotinylated Fab-hCG in place of the whole antibody.

100 tudy, using an extremely sensitive assay for hCG, 10% of clinical pregnancies were undetectable on th

101 the hormone are thought to be essential for hCG activity.

102 tial to become a portable diagnostic kit for hCG.

103 Compared to other detection methods for hCG, this detection method does not require the use of a

104 other patients who were then registered for hCG follow-up.

105 be modified to include a compulsory test for hCG in urine.

106 Samples were tested for hCG, hCG free beta subunit, and hCG beta-core fragment.

107 This differs from hCG assembly, which occurs by threading the glycosylated

108 Staining of oocytes from hCG-stimulated mice with the anti-PT172 antibody also sh

109 ed under the control of a human Cathepsin G (hCG) minigene.

110 nder the direction of the human cathepsin G (hCG) promoter.

111 ymase (HC) and human neutrophil cathepsin G (hCG) show relatively similar cleavage specificities: the

112 rossed PML(-/-) mice with human cathepsin G (hCG)-PMLRARalpha or mammary tumor virus (MMTV)/neu trans

113 treatment with human chorionic gonadatropin (hCG) prevented death and the expression of HIV-1 mRNA an

114 ve or until a human chorionic gonadotrophin (hCG) test confirmed pregnancy.

115 n an antigen, human chorionic gonadotrophin (hCG), and antibodies, anti-alpha-hCG and anti-beta-hCG,

116 FP) 2.0 ng/mL, human chorionic gonadotropin (hCG) 151,111 IU/L, and lactate dehydrogenase 588 U/L.

117 In contrast, human chorionic gonadotropin (hCG) and other glycoprotein hormones are secured by a st

118 ancer markers, human chorionic gonadotropin (hCG) and prostate specific antigen (PSA), in serum sampl

119 mone (hLH) and human chorionic gonadotropin (hCG) are human glycoprotein hormones each consisting of

120 Using human chorionic gonadotropin (hCG) as a model analyte to describe the properties of ph

121 Serum human chorionic gonadotropin (hCG) concentration was 200 mIU/ml; she was hemoconcentra

122 include raised human chorionic gonadotropin (hCG) concentrations 6 months after uterine evacuation of

123 gnancy hormone human chorionic gonadotropin (hCG) efficiently attracts human Tregs to trophoblasts, f

124 with PMs need human chorionic gonadotropin (hCG) follow-up.

125 Human chorionic gonadotropin (hCG) induces de novo synthesis of STAR, a process shown

126 that human embryonic chorionic gonadotropin (hCG) induces sequential mRNA expression of PROK1 and LIF

127 e that secrete human chorionic gonadotropin (hCG) into the host mouse and include small areas of trop

128 Human chorionic gonadotropin (hCG) is a glycoprotein hormone essential to pregnancy.

129 Human chorionic gonadotropin (hCG) is a glycoprotein secreted during pregnancy that ha

130 Human chorionic gonadotropin (hCG) is a heterodimeric member of a family of cystine kn

131 Human chorionic gonadotropin (hCG) is an important biomarker for the diagnosis of preg

132 Human chorionic gonadotropin (hCG) is necessary for the maintenance of early pregnancy

133 a single serum human chorionic gonadotropin (hCG) level that is diagnostic of ectopic pregnancy.

134 The impact of human chorionic gonadotropin (hCG) on prostate carcinoma viability was investigated.

135 Human chorionic gonadotropin (hCG) promotes proliferation of endogenous neural stem ce

136 pecificity for human chorionic gonadotropin (hCG) protein.

137 The human chorionic gonadotropin (hCG) proteins constitute a diverse group of molecules th

138 t of mice with human chorionic gonadotropin (hCG) resulted in increased circulating testosterone leve

139 The hormone human chorionic gonadotropin (hCG) serves to maintain the fetus during early pregnancy

140 Human chorionic gonadotropin (hCG) suppresses cell-mediated allogeneic reactions, vira

141 We detect human chorionic gonadotropin (hCG) using an antibody-based sandwich assay and quantita

142 stimulated by human chorionic gonadotropin (hCG) via cyclic AMP-induced androgen formation in testic

143 e present work human chorionic gonadotropin (hCG) was used as a model protein in a proof-of-concept s

144 indicate that human chorionic gonadotropin (hCG), a hormone that is present in very high levels duri

145 s suggest that human chorionic gonadotropin (hCG), a major hormone of pregnancy, could play a role in

146 tein (CRP) and human chorionic gonadotropin (hCG), by using fluorescent-labeled polyclonal antibodies

147 n pregnancy hormone, chorionic gonadotropin (hCG), in the liver.

148 iator molecule human chorionic gonadotropin (hCG), we interface the intracellular information to enzy

149 V derived from human chorionic gonadotropin (hCG)-beta.

150 mplantation is human chorionic gonadotropin (hCG).

151 ent binding of Human Chorionic Gonadotropin (hCG).

152 er exposure to human chorionic gonadotropin (hCG).

153 s were investigated: chorionic gonadotropin (hCG)/mouse monoclonal anti-hCG and goat IgG (anti-intact

154 eta-subunit of human chorionic gonadotropin (hCG-beta).

155 protein [AFP], human chorionic gonadotropin [hCG], and lactate dehydrogenase [LDH]) before and after

156 were randomized into four treatment groups: hCG+EPO (3 IM doses hCG over 5 days, followed by 3 IV do

157 Samples were tested for hCG, hCG free beta subunit, and hCG beta-core fragment.

158 b resistance were investigated using a Hep3B-hCG orthotopic human xenograft model of locally advanced

159 pital, London, UK, who had persistently high hCG concentrations 6 months after evacuation of hydatidi

160 ith hydatidiform moles had persistently high hCG concentrations of more than 5 IU/L 6 months after ev

161 nce policy would be clinically acceptable if hCG values returned to normal in 75% of patients or more

162 ance of disease was 84%, compared with 7% in hCG-PR x PU.1+/+ mice (P < 0.0001).

163 The residual PU.1 allele in hCG-PR x PU.1+/- APL cells was expressed, and complete e

164 CG values by the lack of parallel changes in hCG results when serum was diluted, by false detection o

165 tributions that noncysteine residues make in hCG folding, secretion, and assembly.

166 leukemia observed in these TM with those in hCG-PML/RAR alpha and hCG-PLZF/RAR alpha TM.

167 breast cancer risk decreased with increasing hCG (upper tertile OR, 0.67; CI, 0.46-0.99), especially

168 onoclonal anti-hCG and goat IgG (anti-intact hCG)/ mouse monoclonal anti-goat IgG.

169 owever, it is unclear whether LRR4 interacts hCG and the endodomain and how it might be involved in s

170 face was determined by covalent 125I-labeled hCG crosslinking to intact, stably transfected mammalian

171 sence of functional luteinizing hormone (LH)/hCG receptors in human breast cells, prompted us to inve

172 ing hormone/human chorionic gonadotropin (LH/hCG) receptor (LHR) in cultured hypothalamic cells and i

173 vide novel insights into the mechanism of LH/hCG receptor activation.

174 gically active heterodimer, mCG, which, like hCG, is required for pregnancy maintenance.

175 However, they recommended measuring hCG and AFP to monitor for relapse in patients treated f

176 chemotherapy groups, apart from lower median hCG concentrations 6 months after evacuation in those un

177 terodimer assembly and is thought to mediate hCG-LHR interactions.

178 With the assistance of microfluidics, hCG sample was delivered via single-injection to 3-Amino

179 dent protein kinase, dibutyryl cAMP mimicked hCG in preventing NF-kappaB activation, and dideoxyadeno

180 emale mice were treated either with 10 IU/ml hCG or PBS at days 0, 2, 4, and 6 of pregnancy.

181 Moreover, hCG stimulates FasL mRNA and protein expression without

182 This explains why most hCG is assembled by threading the glycosylated end of al

183 7 were of macaque origin (PGVD), the mutated hCG-beta subunit displayed macaque-like conformational r

184 e reviewed centrally and, if confirmed, need hCG follow-up.

185 e compartments despite treatment with 100 nm hCG as do LH receptors on cells treated with cytochalasi

186 s, LH receptors on cells treated with 100 nm hCG exhibit restricted lateral diffusion and are confine

187 ompartments on KGN cells treated with 100 nm hCG.

188 Young, nonleukemic hCG-PR x PU.1+/- mice developed splenomegaly because of

189 and neither central histological review nor hCG follow-up were obtained.

190 inding and signal transduction activities of hCG analogs in which the alpha-subunit C terminus (alpha

191 carboxyl terminus had 10-20% the activity of hCG.

192 Application of hCG increased Treg frequency in vivo and their suppressi

193 her, our results demonstrate that binding of hCG induces aggregation of LH receptors within nanoscale

194 sequently in the SAM, to maximize binding of hCG of the streptavidin-bound antibody.

195 ce and treated in vitro with combinations of hCG, DHT, and BMP4 inhibitors.

196 Depending on the initial concentration of hCG, LC gives distinct optical signals visible to the na

197 nts with low and declining concentrations of hCG 6 months after evacuation of hydatidiform mole.

198 Long-term stability (45 days) of hCG in dried spots at reduced temperatures (</=8 degrees

199 ion of hCG in serum and lack of detection of hCG and its degradation products in urine; and wide vari

200 ere identified by two criteria: detection of hCG in serum and lack of detection of hCG and its degrad

201 he results indicated complete dissolution of hCG from the spots, acceptable limit of detection (LOD)

202 The effect of hCG on NF-kappaB was mediated through inhibition of phos

203 hibition attenuated the induction effects of hCG and DHT on estrogen and progesterone secretion in CM

204 i-proliferative and anti-invasive effects of hCG in human breast cancer MCF-7 cells by down-regulatin

205 The cytotoxic effects of hCG were enhanced by expression of dominant-negative Ika

206 lation and enhanced the cytotoxic effects of hCG.

207 ptavidin-antibody layer showed that a LOD of hCG of 2 mIU mL(-1) (4 x 10(-12) mol L(-1)) could be rea

208 aline and urine samples, and only 0.6 muL of hCG solution is required.

209 This means that as little as 45.5 pg of hCG can be detected by this method.

210 ive state of the receptor in the presence of hCG, the possibility that S431 promotes receptor activat

211 Considering the low toxicity profile of hCG in humans, interrupting the inflammatory cycle by hC

212 The promotion of hCG lethality by lovastatin was abolished by overexpress

213 uppressed the radiosensitizing properties of hCG.

214 Rates of hCG normalisation, relapse, and death were assessed in p

215 When the X(1), X(2), and X(3) residues of hCG-alpha and -beta were substituted by swapping their r

216 In addition, the role of hCG as a tumor marker in a variety of cancers has also a

217 This study examined the sensitivity of hCG to gonococcal IgA1 proteases, by means of autoradiog

218 lation differences, we generated a series of hCG-beta-mCG-beta chimeras and identified domains that c

219 During studies of hCG assembly in mammalian cells, we found that addition

220 loop 2 "like a seatbelt." During studies of hCG synthesis in COS-7 cells, we found that, when the se

221 Part of the beta subunit of hCG has an amino acid sequence similar to that of the hi

222 had similar lutropin activities to those of hCG; that in which it was latched to residue 92 at the c

223 nd qualitative differentiation between other hCG variants in a selective, sensitive, and reproducible

224 lar basis, with occasional detection of 1 pg hCG.

225 tographic assays, a detection limit of 10 pg hCG in a 100-microl sample has been achieved on a regula

226 Our results position hCG in a novel, so far unknown role as modulator of immu

227 False-positive hCG concentrations were identified by two criteria: dete

228 We corroborated false-positive hCG values by the lack of parallel changes in hCG result

229 Panel recommended measuring postorchiectomy hCG and LDH for patients with seminoma and preorchiectom

230 ggest that the association between pregnancy hCG and subsequent maternal risk of breast cancer is mod

231 this study, we investigated early-pregnancy hCG concentrations and subsequent breast cancer risk.

232 reatment of MCF-7 cells with highly purified hCG resulted in a modest dose-dependent and hormone-spec

233 vation in the presence of activated LH/CG R, hCG-stimulated ARF6 activation is reduced to basal level

234 mplex in vivo and plays a role in regulating hCG expression.

235 nd protein levels for four forms of secreted hCG were measured in the conditioned media.

236 s not detected by X-ray analysis of secreted hCG-beta.

237 e relationship between early pregnancy serum hCG concentrations and breast cancer risk.

238 transvaginal sonogram and quantitative serum hCG testing.

239 Repeat serum hCG levels after chemotherapy were 12.3 IU/L at 2 weeks

240 The serum hCG level was 8.1 IU/L, and there were residual lesions

241 Similarly, hCG-PLZF/RAR alpha TM displayed a different phenotype wh

242 In round spermatids, hCG caused a significant decrease of 61 kDa species and

243 e human chorionic gonadotropin beta-subunit (hCG-beta) reveals the presence of a disulfide between Cy

244 ed by the high compatibility with the target hCG protein, the major advantage of GO-peptide-based SPR

245 le, cell surface membrane compartments, that hCG binding also affects the lateral motions of antagoni

246 In summary, our results demonstrate that hCG has anti-proliferative and anti-invasive effects in

247 tructs targeted to PROK1 to demonstrate that hCG-mediated LIF expression in the endometrium is depend

248 The findings that hCG treatment increased cAMP synthesis and activated cAM

249 We hypothesized that hCG may be a placental link for the development of local

250 In this study, we hypothesized that hCG not only acts as a chemoattractant of Tregs but also

251 These findings support the premise that hCG could be responsible for the pregnancy-induced prote

252 Here, we report that hCG regulates the SIRT1/FOXO3a axis in hepatocytes, resu

253 We report that hCG treatment decreases cell proliferation and increases

254 These results suggest that hCG may be a link in the development of peritrophoblasti

255 yl cAMP mimicked the effect, suggesting that hCG suppresses the transcription factors through cAMP-in

256 The hCG effect on Treg frequency and cytokine secretion was

257 TAT-CRAC did not affect the hCG-induced cAMP synthesis and the 22R-hydroxycholestero

258 Each mouse carried the hCG-PML/RARA transgene, a well-characterized initiator o

259 In the hCG-dependent steroidogenic MA-10 mouse Leydig cell line

260 type contrasts with what was observed in the hCG-PML/RAR alpha TM model in which the leukemic phase w

261 o bind [(3)H]promegestone and to inhibit the hCG-stimulated steroidogenesis.

262 ed into MA-10 Leydig cells and inhibited the hCG- and cAMP-stimulated steroid production in a dose-de

263 Some months later, the hCG was rising and repeat uterine evacuation revealed ch

264 2) terminus, cystine knot, and loop 2 of the hCG beta-subunit.

265 e contributions that cystine residues of the hCG subunit cystine knots make in folding, assembly, and

266 rom one man having received injection of the hCG-containing pharmaceutical Pregnyl were analyzed.

267 ng inconsistent with our earlier view of the hCG-LHR complex.

268 n adenylate cyclase inhibitor, prevented the hCG effect on NF-kappaB suggested that the hCG actions a

269 order of magnitude more effectively than the hCG-alpha-mCG-beta chimera.

270 e hCG effect on NF-kappaB suggested that the hCG actions are mediated via the cAMP-dependent protein

271 Pairwise testing of groups found that the hCG+EPO group had significantly better behavior at 6/10

272 sing a biological assay that showed that the hCG-alpha-hCG-beta heterodimer facilitated human CG rece

273 lity, in terms of its ability to bind to the hCG antigen, was studied.

274 a threading mechanism, as observed when the hCG seatbelt was replaced with its salmon FSH counterpar

275 Our aim was to assess whether the hCG concentrations were false-positive test results.

276 clude that all three "X" residues within the hCG cystine knots are collectively, but not individually

277 These hCG actions were abrogated when receptor synthesis was i

278 Thus, hCG, in combination with radiation and lovastatin, may r

279 secondary fluorescently labeled antibody to hCG immobilized on the optimized SAM-streptavidin-antibo

280 Exposure of LNCaP cells to hCG promoted activation of epidermal growth factor recep

281 hese cells also express Cyp17 and respond to hCG stimulation but do not express the Leydig specific m

282 ing, and its levels increased in response to hCG treatment.

283 or to mediate cAMP production in response to hCG, suggesting that S431 stabilizes the active state of

284 main, the site phosphorylated in response to hCG.

285 xpressing mutant receptor were responsive to hCG with respect to production of inositol phosphates.

286 l protein with immunological similarities to hCG.

287 guanine-DNA alkyltransferase) led to LAMP-to-hCG signal transduction on low-cost, commercially availa

288 Total hCG was determined on Immulite 2000 analyzer.

289 This would trap hCG in the 1/3 donut structure of the LRRs and enhance t

290 the placental human chorionic gonado-tropin (hCG) alpha- and beta-subunit genes, however, previous wo

291 In vivo, we investigated whether hCG enhances Treg suppressive capacity indirectly by mod

292 Treatment of cells with hCG followed by exposure to ionizing radiation enhanced

293 Treatment of LNCaP and PC-3 cells with hCG modestly reduced cell viability within 96 h.

294 re preferentially secreted and combined with hCG-beta.

295 eted, and its ability to heterodimerize with hCG-beta.

296 h repeats (LRRs) specifically interacts with hCG, preferentially hCGalpha.

297 t of the CaCOV3 human ovarian cell line with hCG blocked TNF-induced activation of NF-kappaB, IkappaB

298 Treatment with hCG and lovastatin decreased expression of BCL-(XL) and

299 togram analysis indicate that treatment with hCG induces aggregation of YFP-coupled LH receptors stab

300 Treatment with hCG+EPO significantly reduced total lesion volume by 82-