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1 However, their correlation has not been clarified.
2 ole of these proteins in IFN-gamma signaling has not been clarified.
3 ossible role of IL-12 in periodontal disease has not been clarified.
4 the free fatty acid receptor family members, has not been clarified.
5 of the sensor signal in experimental studies has not been clarified.
6 s STAT3-inhibiting effect by galiellalactone has not been clarified.
7 may contribute to those therapeutic failures has not been clarified.
8 omaltulose limits postprandial hyperglycemia has not been clarified.
9 tract infections (RTIs) in European children has not been clarified.
10 ication in chronic kidney disease (CKD) risk has not been clarified.
11 their synthesis, in lung health and disease has not been clarified.
12 pe of transplantation for oversight purposes has not been clarified.
13 ifferent molecular subtypes of breast cancer has not been clarified.
14 l peptides; however, its molecular mechanism has not been clarified.
15 tional importance of de novo DNA methylation has not been clarified.
16 0 A, and the molecular mechanism of coupling has not been clarified.
17 ogical significance of HCV-induced autophagy has not been clarified.
18 induced arrest of human T-cell proliferation has not been clarified.
19 The function of OLE RNA has not been clarified.
20 us or too much interference from distraction has not been clarified.
21 although the mechanism by which this occurs has not been clarified.
22 owever, its biological function in cartilage has not been clarified.
23 for the predominant neonatal susceptibility has not been clarified.
24 ors or Galpha(q) involvement in this process has not been clarified.
25 homeostasis or contribute to skin pathology has not been clarified.
26 t of SOD1, ROS, and apoptosis in their death has not been clarified.
27 usion in the mitochondrial signaling pathway has not been clarified.
28 n vivo role of these proteins in causing GCA has not been clarified.
29 although the role of SH2-B in these signals has not been clarified.
30 role of IRF-3 in the induction of IFNA genes has not been clarified.
31 eintervention remodeling on clinical outcome has not been clarified.
32 cing eye movements, but its precise function has not been clarified.
33 hanism underlying the cardiovascular changes has not been clarified.
34 ugh the molecular basis for this degradation has not been clarified.
35 ever, the molecular mechanism of this effect has not been clarified.
36 urons in amyotrophic lateral sclerosis (ALS) has not been clarified.
37 However, the effect on SCD as a mechanism has not been clarified.
38 AIDs in the natural history of biliary colic has not been clarified.
39 iation, and their historical diversification has not been clarified.
40 yavirus GPCs and the host proteases involved have not been clarified.
41 le dissociation with CD8(+) T-cell responses have not been clarified.
42 ween the OPRM1 A118G SNP and these behaviors have not been clarified.
43 e mechanisms associated with human cataplexy have not been clarified.
44 reexisting CLL, the mechanisms leading to RS have not been clarified.
45 alpha and beta isoforms to mechanisms of AD have not been clarified.
46 e secretory cell types, and nature of action have not been clarified.
47 d and the molecules involved in this process have not been clarified.
48 nisms leading to this low treatment efficacy have not been clarified.
49 ever, the characteristics of late ISA in SES have not been clarified.
50 and subcellular localization of DJ-1 protein have not been clarified.
51 ic mutant htt to the pathogenesis of disease have not been clarified.
52 eptor (EGFR) activation and force generation have not been clarified.
53 c functions, their molecular sites of action have not been clarified.
54 of survival in node-negative gastric cancer have not been clarified.
55 ne in the liver, but the mechanisms involved have not been clarified.
56 ympathetic nerve activity and blood pressure have not been clarified.
57 response in the absence of systemic illness have not been clarified.
58 The risks, benefits, and long-term results have not been clarified.
59 many food constituents, such as polyphenols, have not been clarified.
60 rognosis of older patients, its neural basis has not yet been clarified.
61 ryonic mouse neural progenitor cells (eNPCs) has not yet been clarified.
62 nowledge, the cause of this elusive behavior has not yet been clarified.
63 derlying mechanism of the expression of CEAs has not yet been clarified.
64 m small GTPases, but its biological function has not yet been clarified.
65 s by which CdtA interacts with CdtB and CdtC has not yet been clarified.
66 e response in and T cell homing into the CNS has not yet been clarified.
67 of inhibiting constitutive activation of JNK has not yet been clarified.
68 PKC or its isoforms and mitoK(ATP) channels has not yet been clarified.
69 volvement of this process in stress response has not yet been clarified.
70 ed in the dephosphorylation of eNOS(pThr497) have not yet been clarified.
71 alization of the various neurexins, however, have not yet been clarified.
72 ver, the specific cell types targeted by CPF have not been clarified, an issue of vital importance in
73 e contribution of individual pfcrt mutations has not been clarified and other genes have been postula
74 The mechanism underlying this phenomenon has not been clarified and there is no treatment current
76 s underlying the initiation of these lesions have not been clarified but might involve early and fund
77 itively selected thymocytes into the medulla have not been clarified, but several studies have implic
78 amine receptors in agonist-induced signaling has not yet been clarified despite recent advances in 7
81 (CMR) and late gadolinium enhancement (LGE) has not been clarified in acute myocarditis (AM) with pr
83 nd functional relationships among components have not been clarified, nor is it known where Warts act
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