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1 ew advances that will deepen knowledge of S. haematobium.
2 nown to exhibit activity against Schistosoma haematobium.
3 um that are coinfected with S. mansoni or S. haematobium.
4 red with Schistosoma mansoni and Schistosoma haematobium.
5 parasitic trematode blood fluke Schistosoma haematobium.
6 ure of acetylcholinesterase from Schistosoma haematobium.
7 h endemic Schistosoma mansoni or Schistosoma haematobium.
8 h either Wuchereria bancrofti or Schistosoma haematobium.
9 educes morbidity is proposed for Schistosoma haematobium, a parasite of major public health importanc
12 tterns of recognition of defined Schistosoma haematobium adult worm antigens by serum antibodies from
13 aused by the parasitic trematode Schistosoma haematobium, affects over 112 million people worldwide.
14 indicate a diversity of lesions caused by S. haematobium and a dynamic evolution of the genital lesio
15 ted with Schistosoma mansoni and Schistosoma haematobium and considers the practical implications of
18 ecimens to determine the seroprevalence of S haematobium and S mansoni by ELISA and immunoblot analys
19 The literature search identified Schistosoma haematobium and Schistosoma mansoni surveys done in, res
20 vides a global insight into the kinome of S. haematobium and should assist the repurposing or discove
21 the AChE from the human parasite Schistosoma haematobium and succeeded in expressing functional recom
23 ution of Schistosoma mansoni and Schistosoma haematobium and the incidence of schistosomiasis in area
24 water exposure was limited to Lake Malawi; S haematobium antibodies were found in 135 of 141 (96%) se
25 he most comprehensive characterization of S. haematobium antigens to date and describes novel antigen
26 verrini, Clonorchis sinensis and Schistosoma haematobium are classified as Group 1 biological carcino
27 errini, Clonorchis sinensis, and Schistosoma haematobium are classified as group 1 biological carcino
29 ts, Schistosoma japonicum, S. mansoni and S. haematobium, are blood flukes that have complex life cyc
32 rvention for reducing the transmission of S. haematobium [corrected] is an economically attractive st
33 show that genital infection with Schistosoma haematobium [corrected] may increase the risk for HIV in
34 y between 10% and 70% of WSH for reducing S. haematobium [corrected] transmission, our model predicte
39 eripheral blood of children residing in a S. haematobium-endemic area and in the peripheral blood, sp
42 zed by pooled serum samples from Schistosoma haematobium-exposed Zimbabweans were determined by 2-dim
43 tosomiasis due to infection with Schistosoma haematobium following recreational water exposure at Cap
49 cted Bulinus globosus, the snail vector of S haematobium in Malawi, were found at Cape Maclear and ot
53 resulted in less IL-10(+) T cells in the S. haematobium-infected group, while levels of FoxP3(+) reg
54 re was a lack of treatment effects in the S. haematobium-infected group; however, the small sample si
55 lished study, which included 163 Schistosoma haematobium-infected individuals and 183 matched healthy
57 anemic children with documented Schistosoma haematobium infection (n = 224 for AGP, CRP, SF, sTfR, a
58 ctions in the prevalence and intensity of S. haematobium infection 1 year after treatment were, howev
59 with observed field patterns of Schistosoma haematobium infection and antibody responses, including
60 y that evaluates the relationship between S. haematobium infection and associated morbidity in childr
61 otherapy can have a substantial impact on S. haematobium infection and its associated morbidity in ch
62 osed to increased TNF-alpha production to S. haematobium infection are more likely to develop an exag
65 ether bladder pathology in human Schistosoma haematobium infection is related to the balance of Th17
66 ws promising activity against concomitant S. haematobium infection leading to an important reduction
75 Schistosoma mansoni (and rarely Schistosoma haematobium) intestinal infection is also not very commo
80 schistosomiasis, infection with Schistosoma haematobium, is linked to increased risk for the develop
81 nital schistosomiasis, caused by Schistosoma haematobium, is the most prevalent form of schistosomias
82 nth species and others that are unique to S. haematobium may provide novel diagnostic and drug target
83 ng anti-IL-10 added to PBMC from Schistosoma haematobium patients' enhanced adult worm (SWAP)- or egg
84 esearch into human coinfections (Schistosoma haematobium-Plasmodium falciparum versus Schistosoma man
85 were recruited from areas where Schistosoma haematobium (S.h) infections were high or low endemic.
87 2 patients showed infection with Schistosoma haematobium, S haematobium-Schistosoma bovis hybrids, an
88 velers returned from Mali with a Schistosoma haematobium-Schistosoma bovis hybrid infection, confirme
91 derivatives that kill both S. mansoni and S. haematobium, the two species responsible for >99% of sch
93 are orthologs of M-IPSE, from egg cDNA of S. haematobium Using PCR and immunodetection, we confirmed
94 million people are infected with Schistosoma haematobium, with the most intense infections in childre
95 ciated with increasing levels of Schistosoma haematobium worm IgG1, with adolescents with optical den
97 eproducibly recovered significantly fewer S. haematobium worms and eggs from Mta1-/- mice than wild-t
99 ract coinfection by bacteria and Schistosoma haematobium worms, the etiologic agent of urogenital sch
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