ライフサイエンスコーパス: haematoma

1 egion which was compatible with intraorbital haematoma.

2 en for post-operative complications, such as haematoma.

3 ny, are associated with presence of subdural haematoma.

4 tex by immunoblotting at 30 min and 4 h post-haematoma.

5 atoma and this persisted at 2 h and 4 h post-haematoma.

6 h following production of an acute subdural haematoma.

7 n the areas identified during standard US as haematomas.

8 sults indicate that following acute subdural haematoma, a rapid cellular redistribution of apoE occur

9 On post operative day 6, an infected haematoma and an area of proximal skin necrosis were sur

10 Mortality at 90 days and risk of parenchymal haematoma and symptomatic intracranial haemorrhage did n

11 examination, the average difference between haematoma and the renal cortex was 5 dB.

12 hroughout the cortical layers underlying the haematoma and this persisted at 2 h and 4 h post-haemato

13 rious adverse events related to kyphoplasty (haematoma and urinary tract infection); other serious ad

14 ssibility of a persistent idiopathic scrotal haematoma and/or haematoma secondary to a trauma of the

15 taxis); one receiving placebo (infusion site haematoma), and four during dabigatran etexilate pretrea

16 o study the natural history of such subdural haematomas; and to ascertain which obstetric factors, if

17 Subdural haematomas are thought to be uncommon in babies born at

18 Iron deposition appeared on day 3 around the haematoma border but did not change synchronously with A

19 n the cortical levels of apoE at 30 min post-haematoma but, at 4 h post-haematoma, there was a signif

20 Intraorbital haematoma can be managed by conservative approach withou

21 Chronic subdural haematoma causes serious morbidity and mortality.

22 the levels of apoE in cortex underlying the haematoma compared to control levels.

23 difference in the echogenicity of perirenal haematomas compared to the routine examination in B pres

24 Aortic dissection and intramural haematoma comprise an aortopathy involving separation of

25 n reveals intracranial abnormalities such as haematomas, contusions and cerebral oedema.

26 We experienced a case of intraorbital haematoma during a commercial flight.

27 Early surgery combined haematoma evacuation (within 24 h of randomisation) with

28 in 27 countries, we compared early surgical haematoma evacuation within 12 h of randomisation plus m

29 than 2 years (normal mental status, no scalp haematoma except frontal, no loss of consciousness or lo

30 eta-analysis to compare baseline ICH volume, haematoma expansion and clinical outcomes between NOAC-I

31 fect of intensive BP lowering on significant haematoma expansion at 24 hours warrants further investi

32 Haematoma expansion is a major cause of mortality in int

33 and at 3 months, as well as ICH volumes and haematoma expansion rates in the two groups.

34 The rates of haematoma expansion were comparable in NOAC-ICH versus V

35 five (19%) of 27 in the PCC group (none from haematoma expansion), the first of which occurred on day

36 ight (35%) of 23 in the FFP group (five from haematoma expansion, all occurring within 48 h after sym

37 Our results confirm that ICH volume, haematoma expansion, mortality and functional outcome ap

38 activated factor VII (eptacog alfa) reduced haematoma expansion, mortality, and disability when give

39 were judged to be FFP related (four cases of haematoma expansion, one anaphylactic reaction, and one

40 Complications of ICH include haematoma expansion, perihaematomal oedema with increase

41 isation seemed to be associated with smaller haematoma expansion.

42 d 18 years and older with a chronic subdural haematoma for burr-hole drainage were assessed for eligi

43 hrombotic therapy is associated with greater haematoma growth, which may be reduced by early intensiv

44 All were rescanned at 4 weeks and haematomas had completely resolved.

45 troke that is defined by the occurrence of a haematoma in the wall of an intracranial artery.

46 s the echogenicity and the size of perirenal haematomas in patients after kidney transplant during ro

47 ed a more detailed assessment of the size of haematomas in the perirenal space that appeared during e

48 Following the haematoma induction, apoE immunoreactivity was dramatica

49 At 30 min post-haematoma, intense apoE staining was observed in cluster

50 ng logistic regression, and (2) of increased haematoma+intraventricular haemorrhage volume (IVH) with

51 Intraorbital haematoma is a rare clinical entity which can be caused

52 after burr-hole drainage of chronic subdural haematoma is safe and associated with reduced recurrence

53 he interpretation of the images of perirenal haematoma is their ability to change in time.

54 Intensive BP lowering reduced haematoma+/-IVH growth by 4.7/7.1 mL in patients on anti

55 ollow-up CT after 24 h, absolute increase in haematoma+/-IVH volume was larger (5.2/5.0 mL) in those

56 Additionally, at 4 h post-haematoma marked apoE staining of discrete foci within t

57 e neocortex (13 cases, 37%) and intracranial haematoma (nine cases, 26%).

58 Subdural haematomas occurred in two patients.

59 There were also fewer haematomas of any severity in the stenting group than in

60 1.03; p=0.04), the presence of intracerebral haematoma on initial CT scan (OR 2.0, 95% CI 1.2 to 3.5;

61 significant difference in the development of haematomas or seromas after surgery but the non-warmed g

62 l hypotension, ischaemic stroke, retroclival haematoma, pituitary apoplexy, third ventricle colloid c

63 bital exploration and surgical evacuation of haematoma remains a second line intervention.

64 At the first month follow-up, intraorbital haematoma resolved without significant sequelae.

65 rsistent idiopathic scrotal haematoma and/or haematoma secondary to a trauma of the inguino-scrotal r

66 alysis, each copy of APOE epsilon2 increased haematoma size by a mean of 5.3 mL (95% CI 4.7-5.9; p=0.

67 tional outcome following ICH is dependent on haematoma size, with only patients with smaller haemorrh

68 oE at 30 min post-haematoma but, at 4 h post-haematoma, there was a significant elevation (27%, P < 0

69 parenchyma, who were suspected of perirenal haematoma, underwent a CE-US examination after intraveno

70 Admission Glasgow Coma Scale, increasing haematoma volume and cortical involvement were associate

71 Baseline haematoma volume and gender appear to influence PHE grow

72 ssion to establish the effect of genotype on haematoma volume and logistic regression to assess the e

73 associate with severity of ICH, in terms of haematoma volume at presentation and subsequent outcome.

74 e relationship with PHE growth, but baseline haematoma volume had a direct correlation.

75 At baseline, median haematoma volume was 15.0 mL (IQR 7.9-29.2) with median

76 In six patients, the size of haematomas was comparable using both techniques, whereas

77 e were observed in the cortex underlying the haematoma with minimal damage observed in shams.