ライフサイエンスコーパス: haemocyte

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1 apoptosis in both S2 cells and H. virescens haemocytes.

2 i-based immunity mediated by macrophage-like haemocytes.

3 t signalling is not autonomously required in haemocytes.

4 s proliferate and mature into differentiated haemocytes.

5 that is present in its inactive form inside haemocytes.

6 , midgut, integument, testis, silk gland and haemocytes.

7 eatment can increase cell number in cultured haemocytes and the Drosophila wing, respectively.

8 hat a reciprocal relationship exists between haemocytes and the VNC and that defects in nerve cord de

9 is along the ventral nerve cord (VNC), where haemocytes are required for the correct development of t

10 , also provides the scaffold on which intact haemocytes assemble during encapsulation; a response tha

11 identify direct Notch targets in Drosophila haemocytes (blood cells), where Notch promotes crystal c

12 wly cycling progenitor cells, whereas mature haemocytes comprising plasmatocytes, crystal cells and l

13 Although PO activity and haemocyte count were weakly correlated across the whole

14 c effects on phenoloxidase (PO) activity and haemocyte count, both indicators of immune system activi

15 ity were substantially higher than those for haemocyte count, indicating that the different component

16 ivity in every case and substantially higher haemocyte counts in all treatments except unheated/low d

17 A higher temperature led to higher haemocyte counts when density was high and food quality

18 and heritability for components of immunity (haemocyte densities, proPhenoloxidase activity, resistan

19 two immune parameters related to resistance (haemocyte density and pre-immune challenge activity of p

20 genetic correlation between growth rate and haemocyte density was estimated).

21 g antibacterial histones together with other haemocyte-derived defence factors, but crucially, also p

22 cell size and cell-cycle progression in two haemocyte-derived Drosophila cell lines.

23 These include proteins that participate in haemocyte development, vesicle transport, actin cytoskel

24 mosquitoes constitutively release a soluble haemocyte differentiation factor into their haemolymph t

25 Our data reveal that the final pattern of haemocyte distribution, and the details and timing of it

26 We show that the failure of haemocyte migration along the VNC in slit mutants is not

27 at defects in nerve cord development prevent haemocyte migration along this structure.

28 VNC, further highlighting the importance of haemocyte migration for correct neural development.

29 This block of haemocyte migration in turn disrupts the formation of th

30 and its receptor Robo are both required for haemocyte migration, but signalling is not autonomously

31 g epithelium, creating a physical barrier to haemocyte migration.

32 ontact inhibition plays in the patterning of haemocyte movements, we have mathematically analysed and

33 Furthermore, we observed the same effects in haemocytes of H. virescens larvae, after TnBVank1 in viv

34 Vank1 in vivo transient transfection, and in haemocytes of parasitised larvae.

35 cently revealed that Drosophila macrophages (haemocytes) require contact inhibition for their uniform

36 Snail haemocyte RNA, extracted from parasite-challenged resist

37 Haemocytes take up dsRNA from infected cells and, throug

38 in the environment, this interaction induces haemocyte trafficking into these tissues.

39 cells associated with host cilia and induced haemocyte trafficking.

40 bryonic development, Drosophila macrophages (haemocytes) undergo a series of stereotypical migrations

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