ライフサイエンスコーパス: haemostasis

1 ecting the critical function of platelets in haemostasis.

2 the International Society on Thrombosis and Haemostasis.

3 ine protease thrombin is a critical event in haemostasis.

4 grins have a critical role in thrombosis and haemostasis.

5 , II, and III, which is essential for normal haemostasis.

6 s essential for transport, inflammation, and haemostasis.

7 is critical for understanding thrombosis and haemostasis.

8 2) modulates platelet activation to regulate haemostasis.

9 form a plug which is responsible for primary haemostasis.

10 ma fibrinogen concentrations are critical to haemostasis.

11 cular injury without affecting parameters of haemostasis.

12 tment approaches include early diagnosis and haemostasis, aggressive management of blood pressure, op

13 e genetic regulation of proteins involved in haemostasis and atherothrombotic disorders, including my

14 flow conditions and achieve wound-triggered haemostasis and decreased bleeding times in vivo in a tr

15 the distinct roles of individual agonists in haemostasis and have important consequences in the desig

16 Platelets are critical in haemostasis and in arterial thrombosis, which causes hea

17 he third in a series on interactions between haemostasis and inflammation.

18 tivation by thrombin is necessary for normal haemostasis and may be an important target in the treatm

19 va that degrade nucleotides, thus inhibiting haemostasis and minimizing the ensuing pain and inflamma

20 Understanding normal haemostasis and the pathophysiology of its disorders is

21 as apolipoprotein H, has been implicated in haemostasis and the production of anti-phospholipid anti

22 the International Society of Thrombosis and Haemostasis and their associations with the occurrence o

23 derlie a potential role of microparticles in haemostasis and thrombosis as modulators of fibrin forma

24 rtance of platelet activation by thrombin in haemostasis and thrombosis is unknown.

25 Roles for PARs are beginning to emerge in haemostasis and thrombosis, inflammation, and perhaps ev

26 importance of circulating microparticles in haemostasis and thrombosis, there is limited evidence fo

27 ower on the modulation of platelet function, haemostasis and thrombosis.

28 g platelet function, thereby contributing to haemostasis and thrombosis.

29 ates platelets is critical for understanding haemostasis and thrombosis.

30 Platelets undoubtedly play a pivotal role in haemostasis and trauma-induced coagulopathy.

31 te cytoplasm, have a critical role in normal haemostasis, and in thrombotic disorders.

32 a crucial role in the maintenance of normal haemostasis, and perturbations of this system can lead t

33 t activation during inflammation compared to haemostasis, and that hitherto undiscovered platelet act

34 The primary endpoint was effective haemostasis, and the co-primary endpoint was rapid INR r

35 Genes influencing activation of haemostasis are likely to be an important component of t

36 is system may be compromised by disorders of haemostasis associated with a prothrombotic state, we po

37 result in a further deterioration of primary haemostasis at the level of varix.

38 els, International Society of Thrombosis and Haemostasis Bleeding Assessment Tool (ISTH BAT) confirme

39 the International Society on Thrombosis and Haemostasis bleeding assessment tool.

40 STH (International Society on Thrombosis and Haemostasis) bleeding classifications was assessed in 2,

41 of the equilibrium between all components of haemostasis (coagulation, anticoagulation, fibrinolysis,

42 Impaired haemostasis could exacerbate the primary insult with ris

43 ing (International Society of Thrombosis and Haemostasis criteria), cause-specific hospitalization, a

44 2005 International Society on Thrombosis and Haemostasis criteria.

45 g to International Society on Thrombosis and Haemostasis definition).

46 Normal haemostasis depends on an intricate balance between mech

47 s encoding seven clusters or isoforms of the haemostasis-disruptive C-type lectin (CTL) proteins from

48 improved survival with better management of haemostasis early after injury.

49 de a locally inducible treatment to maintain haemostasis for haemophilia A.

50 elet-like structures for the augmentation of haemostasis have focused solely on recapitulating aspect

51 lar efficacy to chrysin in the modulation of haemostasis in mice.

52 plasma for rapid INR reversal and effective haemostasis in patients needing VKA reversal for urgent

53 Haemostasis in the arteriolar circulation mediated by vo

54 nd Factor (vWF), a glycoprotein essential to haemostasis in Weibel-Palade bodies (WPBs), cigar-shaped

55 Accurate assessment of blood haemostasis is essential for the management of patients

56 /SE, International Society on Thrombosis and Haemostasis major bleeding, and the net clinical outcome

57 Haemostasis occurs at sites of vascular injury, where fl

58 ion to being the chief cellular effectors of haemostasis, platelets are innate inflammatory cells tha

59 ur current knowledge of platelet function in haemostasis, possible mechanisms for aberrant activity i

60 Monitoring of the haemostasis status is significant for proper therapeutic

61 ATC is an endogenous impairment of haemostasis that begins at the moment of injury.

62 Platelets are critical for haemostasis, thrombosis, and inflammatory responses, but

63 Effective haemostasis was achieved in 78 (90%) patients in the 4F-