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1 yer of the IFE and in the dermal papilla and hair bulb.
2 bulge, and subsequently spreads towards the hair bulb.
3 haft fragility and untimely apoptosis in the hair bulb.
4 ted with increased cell proliferation in the hair bulb.
5 s, and the proliferative cells of the anagen hair bulb.
6 dermal papilla (DP), that is embedded in the hair bulb.
7 and, for the cyclic formation of new anagen hair bulbs.
9 type has been described as adendritic, since hair bulb and Harderian gland melanocytes appear to be r
10 xpression onset at anagen IIIa above the new hair bulb and subsequent spreading towards the bulge.
11 mal tissues, except for keratinocytes of the hair bulb and syncytiotrophoblasts of the placenta, but
12 tion was detectable in DNA from the father's hair bulbs and buccal cells but not his white blood cell
13 Dermal papilla cells (DPCs) located in the hair bulb are the main site of androgen activity in the
14 (SC) are maintained in the bulge region, and hair bulbs at the base contain rapidly dividing, yet gen
17 significant reduction in apoptotic (TUNEL+) hair bulb cells, thus mitigating the degree of follicle
19 sequent hair cycle, i.e., after a new anagen hair bulb has been constructed, which points to the exis
20 mal hair follicles was virtually absent from hair bulbs, it was expressed by a subset of keratinocyte
22 rmal transfer of pigment granules to ectopic hair bulb locations, extrafollicular melanin incontinenc
23 cted in progenitor cell compartments; in the hair bulb matrix and basal epidermal layer, a small subc
28 genitor population that resides in the lower hair bulb of anagen-phase follicles and expresses Hopx.
29 , and a novel role for Hopx-expressing lower hair bulb progenitor cells in contributing to stem cell
30 cally, after low-dose IR, keratin 5(+) basal hair bulb progenitors, rather than bulge SCs, were quick
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